ライフサイエンスコーパス: butanedione

1 pha-diketone butter flavoring, diacetyl (2,3-butanedione).

2 hyl acetate, 2,3-butanediol, acetoin and 2,3-butanedione.

3 0.61 ng mL(-1) (RSD mean value 3.1%) and 2,3-butanedione 0.10 ng mL(-1) (RSD mean value 2.9%).

4 After baking, 2,3-butanedione, 1-propanol, 2-methyl-1-propanol, 3/2-methyl

5 current study was conducted to evaluate 2,3-butanedione 2-monoxime (BDM), a compound that interferes

6 ivated fibres (pCa 4.5) exposed to 10 mM 2,3-butanedione 2-monoxime (BDM), stretch was accompanied by

7 e length and were insensitive to 5-10 mM 2,3-butanedione 2-monoxime (BDM), which inhibited active con

8 h that of the myosin-specific inhibitor, 2,3-butanedione 2-monoxime (BDM).

9 fects were reversed by addition of 10 mM 2,3-butanedione 2-monoxime.

10 al (2.02-25.70%), ethanol (9.84-68.21%), 2,3-butanedione (2.31-14.71%), trans-2-hexenal (0.49-17.49%)

11 tor ML-7 and the myosin ATPase inhibitor 2,3-butanedione-2-monoxime perturbed NF translocation within

12 s were decreased by the myosin inhibitor 2,3-butanedione-2-monoxime, suggesting that tension promotes

13 ition of actomyosin by cytochalasin D or 2,3-butanedione-2-monoxime.

14 dimethyldisulfide, acetone, 2-butanone, 2,3-butanedione, 6-methyl-5-hepten-2-one, indole, and 4-meth

15 The alcohols from fermentation, 2,3-butanedione, acetoin, acetic acid, isobutyric acid and e

16 orted 4,5-dimethyl-1,2-benzoquinone from 2,3-butanedione/amino acid model systems has not been invest

17 g low-boiling compounds such as ethanal, 2,3-butanedione and alcohols, while SPME extracted more effi

18 The arginine-specific modifiers 2,3-butanedione and phenylglyoxal were found to be inactivat

19 modification of the wild-type enzyme by 2,3-butanedione and substrate protection by CoA indicated th

20 a mixture of glyoxal, methylglyoxal and 2,3-butanedione and the digestive enzymes (pepsin and pancre

21 acetic acid, ethyl acetate, 2-butanone, 2,3,-butanedione, and acetone, or the monoterpene, 1,8-cineol

22 g reagents, namely diethylpyrocarbonate, 2,3-butanedione, and the reagent pair EDC/GEE, are used toge

23 DCs), such as glyoxal, methylglyoxal and 2,3-butanedione, are highly reactive substances occurring in

24 t the lead hypoxia imaging agents (64)Cu-2,3-butanedione bis(N4-methylthiosemicarbazone) (ATSM) and (

25 ic-to-normoxic contrast (14:1 for (64)Cu-2,3-butanedione bis(thiosemicarbazone) (ATS), 17:1 for (64)C

26 The trapping of 2,3-butanedione by this in situ generated 4,5-dimethyl-1,2-p

27 ure, the generation of methylglyoxal and 2,3-butanedione due to the oxidation of nucleic acids may le

28 lbutyl butanoate, 2-phenylethyl acetate, 2,3-butanedione, hexanedioic acid, guaiacol, 2,3-dihydro-2-m

29 mino acid analyses of enzyme modified by 2,3-butanedione in the presence and absence of S-HPC suggest

30 The Arg-specific modification reagent, 2,3-butanedione, inactivated SULT1A1 in an efficient, time-

31 ot only to catalyse the cyclocondensation of butanedione into 4,5-dimethyl-1,2-benzoquinone but also

32 Adding the myosin ATPase inhibitor, 2,3-butanedione monoxide abolished all surplus myocardial ox

33 we show that inhibitors of myosin function, butanedione monoxide and ML-7, significantly blocked the

34 ng myosin adenosine triphosphatase using 2,3-butanedione monoxime (BDM) and found that phagocytosis w

35 r outer hair cells (OHCs) while applying 2,3-butanedione monoxime (BDM) by pressure.

36 2,3-Butanedione monoxime (BDM) is widely believed to act as

37 d the effect of the chemical phosphatase 2,3-butanedione monoxime (BDM) on glycine current (IGly) of

38 ogenetically activated eggs treated with 2,3-butanedione monoxime (BDM) undergo a dramatic large-scal

39 sate containing 20 nM dopamine, dopamine+2,3-butanedione monoxime (BDM), a MgATPase-inhibitor, or the

40 e the actions of a chemical phosphatase, 2,3-butanedione monoxime (BDM), on endogenous and expressed

41 The myosin ATPase inhibitor, butanedione monoxime (BDM), reversibly inhibited gliding

42 cal-mechanical uncoupling drugs, such as 2,3-butanedione monoxime (BDM).

43 ose of this investigation was to utilize 2,3-butanedione monoxime (BDM; an inhibitor of contractile a

44 ves a carbamidodiacetyl reaction between 2,3-butanedione monoxime (diacetylmonoxime) and the de novo

45 ke tension response was affected by both 2,3-butanedione monoxime and sarcomere length in the same ma

46 d reactivators monoisonitrosoacetone and 2,3-butanedione monoxime and shows potential for further ref

47 The addition of 3 mm 2,3-butanedione monoxime at pCa 9.0 showed that there was ap

48 tors, since treatment with cytochalasin D or butanedione monoxime blocks the streaming motion.

49 The addition of the neutral oxime 2,3-butanedione monoxime increases the rate of reactivation

50 n firm substrates with myosin inhibitors 2,3-butanedione monoxime or KT5926 caused the reduction of b

51 hydro-1,4-diazapine, cytochalasin D, and 2,3-butanedione monoxime significantly inhibited (P < 0.05)

52 in the presence of the tension inhibitor 2,3-butanedione monoxime suggests that regulatory light chai

53 affect strong crossbridge inhibition by 2,3-butanedione monoxime when near-neighbour thin filament R

54 Cross-bridge inhibition (with butanedione monoxime) or augmentation (with 2 deoxy-ATP)

55 ilaments (cytochalasin D) and myosin ATPase (butanedione monoxime), indicating that they rely on an a

56 n more about the cardioplegic agent BDM (2,3-butanedione monoxime), its effects on the force-velocity

57 ensitive to an inhibitor of myosin function (butanedione monoxime).

58 response was reversibly reduced by 5-mM 2,3-butanedione monoxime, a known cross-bridge inhibitor.

59 n specifically inhibited a slow phase, while butanedione monoxime, a myosin ATPase inhibitor, inhibit

60 al stimulation in the presence of 7.5 mM 2,3-butanedione monoxime, an inhibitor of actin-myosin cross

61 The drug butanedione monoxime, an inhibitor of myosins, blocked t

62 ADE, butanedione monoxime, and NP were given for cardioprotec

63 ith altered concentrations of phosphate, 2,3-butanedione monoxime, or adenosine triphosphate at a fix

64 ference was abolished in the presence of 2,3-butanedione monoxime, suggesting that the basal level of

65 he myosin cross-bridge (inorganic phosphate, butanedione monoxime, trifluoperazine, and blebbistatin)

66 tochalasin D, genistein, wortmannin, and 2,3-butanedione monoxime-sensitive pathway and replicated wi

67 ytoskeleton, and by the myosin inhibitor 2,3-butanedione monoxime.

68 but these increases were not blocked by 2,3-butanedione monoxime.

69 ction coupling) were estimated by use of 2,3-butanedione monoxime.

70 nhibited with 0.5 mM AlF4- or with 30 mM 2,3 butanedione-monoxime (BDM) during measurements of the Ca

71 hemotaxis toward one such odorant, diacetyl (butanedione), requires the function of a seven-transmemb

72 presence of 9,10-phenanthrenequinone or 2,3-butanedione results in the formation of pyrazines 6a,b,

73 er product of nucleic acid oxidation was 2,3-butanedione, the 1,2-diaminobenzene derivative of which

74 2,3-Butanedione was found to reduce enzyme activity with R-H

75 this study such model systems containing 2,3-butanedione were investigated under pyrolytic conditions

76 The chemical interaction of 2,3-butanedione with amino acids through Strecker reaction h