ライフサイエンスコーパス: butanone

1 androsterone to approximately 940-fold for 2-butanone.

2 ts of 4-(methylnitrosoamino)-1-(3-pyridyl)-1-butanone.

3 nogen, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.

4 ene or 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.

5 doacetate, 1,3-dibromoacetone, and 1-bromo-2-butanone.

6 ced by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.

7 )P and 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.

8 molecule(-1) s(-1) for 3-methyl-3-nitrooxy-2-butanone.

9 ly depended on the proportions of hexane and butanone.

10 inary solvent system, composed of hexane and butanone.

11 cleavage in 2-butanol partial oxidation to 2-butanone.

12 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.

13 obtained with dimethyl methylphosphonate and butanone.

14 carcinogen 4-methylnitrosamino-1-3-pyridyl-1-butanone.

15 nogen, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone.

16 , trans-2-hexenal (0.49-17.49%), 3-hydroxy-2-butanone (0.08-7.39%), 1-hexanol (0.56-16.39%), 1-octano

17 ring-containing ketones, such as 4-phenyl-2-butanone (1a) and 1-phenyl-1,3-butadione (2a), were redu

18 acetaldehyde, acetic acid, ethyl acetate, 2-butanone, 2,3,-butanedione, and acetone, or the monoterp

19 bon disulfide, dimethyldisulfide, acetone, 2-butanone, 2,3-butanedione, 6-methyl-5-hepten-2-one, indo

20 GC-MS identified 2-butanol, 2-butanone, 2-pentanone and 1-propanol to be possibly elev

21 2-butanone, 2-pentanone, 2-heptanone and 3-methyl-1-butano

22 2-butanone, 2-pentanone, 2-heptanone and 3-methyl-1-butano

23 The longer-chain ketones butanone, 2-pentanone, 3-pentanone, and 2-hexanone were

24 methylpyrazine, 2-vinylpyrazine, 3-hydroxy-2-butanone, 3-(methylsulfanyl)-propanal, and 5-methyl-2-fu

25 methyl 2-propenal), four ketones (acetone, 2-butanone, 3-buten-2-one, and 6-methyl-5-hepten-2-one), o

26 sugar damage to DNA: glycolate, 3-hydroxy-2-butanone, 3-phenylbutyraldehyde, and alpha-hydroxy-gamma

27 lose 5-phosphate (Ru5P) to l-3,4-dihydroxy-2-butanone 4-phosphate and formate.

28 he Escherichia coli gene for 3,4-dihydroxy-2-butanone 4-phosphate synthase, a key enzyme for riboflav

29 previously shown to code for 3,4-dihydroxy-2-butanone-4-phosphate synthase (DHBP synthase), an enzyme

30 The enzyme 3,4-dihydroxy-2-butanone-4-phosphate synthase (DHBPS) catalyzes one of t

31 taining GTP cyclohydrase and 3,4-dihydroxy-2-butanone-4-phosphate synthase activities.

32 rotein GTP cyclohydrolase II/3,4-dihydroxy-2-butanone-4-phosphate synthase and a negative regulator o

33 -ray crystal structures of L-3,4-dihydroxy-2-butanone-4-phosphate synthase from Magnaporthe grisea ar

34 ioning GTP cyclohydrolase II-3,4-dihydroxy-2-butanone-4-phosphate synthase, an enzyme essential for r

35 dependent OH radical reaction of 4-hydroxy-2-butanone (4H2B) is presented.

36 -methyl-1-butanol, 3-methyl-1-butanol, and 2-butanone after storage.

37 nogen 4-(methylnitrosamino)-1-(3-pyridyl)-1- butanone also had a low incidence (16.7%) of ER methylat

38 Methyl-2-buten-1-ol (prenol) and 3-Hydroxy-2-butanone (AMC) were selected for further behavioral assa

39 droxylation, 4-(methylamino)-1-(3-pyridyl)-1-butanone (aminoketone), can be converted to the potent t

40 eduction of such challenging substrates as 2-butanone and 3-hexanone.

41 2-Butanone and 3-methyl-1-butanol were identified in Lacto

42 2-Butanone and 3-methyl-1-butanol were identified in Lacto

43 orea and Ruminococcus and higher levels of 2-butanone and 4-methyl-2-pentanone compared to CTRLs.

44 s (TC) 4-(methylnitrosamino)-I-(3-pyridyl)-1-butanone and benzo(a)pyrene.

45 otein bound with small molecules 4-hydroxy-2-butanone and FK506.

46 am for 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone and N'-nitrosoanabasine.

47 gens 4-(methylnitrosamino)-1-(3-pyridinyl)-1-butanone and N-nitroso nornicotine were also detected.

48 (TSNA) 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone and N-nitrosonornicotine by repairing O(6)-[4-o

49 3) cm(3) molecule(-1) s(-1) for 3-nitrooxy-2-butanone, and (2.6 +/- 0.9) x 10(-13) cm(3) molecule(-1)

50 same conditions, cycloheptanone, 3-methyl-2-butanone, and 2-butanone were converted to dienes.

51 m enolates derived from acetone, pinacolone, butanone, and 3-methyl-2-butanone to give fair to good y

52 ncies of alpha-nitrooxyacetone, 3-nitrooxy-2-butanone, and 3-methyl-3-nitrooxy-2-butanone have been d

53 ncluding 2- and 3-methylbutanal, 3-hydroxy-2-butanone, and 6-methyl-5-hepten-2-one.

54 ains: ethyl butyrate (EB), propionic acid, 2-butanone, and ethyl acetate.

55 ake of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, and nicotine, respectively, in relation to lun

56 otine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, and volatiles.

57 entanedione) or the absence of a keto group (butanone) are not ODR-10 agonists.

58 migration as evident from the formation of 2-butanone as the major product via the enol 2-hydroxy-2-b

59 e obtained in high yields and selectivity in butanone by changing the ligand to 1,3,5,7-tetramethyl-2

60 four analytes (methanol, ethanol, acetone, 2-butanone) by systematically varying the oscillating freq

61 during prolonged starvation, nor butanol or butanone, compounds closely related to BOH, had an effec

62 ceae, 4-methyl-2-pentanone, 1-butanol, and 2-butanone could discriminate NAFLD patients from CTRLs.

63 ion of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone could occur in humans.

64 . vaginalis T016 with >/=20 mM 1,4-diamino-2-butanone (DAB) to inhibit ODC resulted in growth arrest,

65 transformant was inhibited by 1, 4-diamino-2-butanone (DAB), a known inhibitor of eukaryotic ODC.

66 panone (7a) was reduced with lower ee than 2-butanone derivatives.

67 sm for 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone detoxification.

68 g 4-(N-methyl-N-nitrosamino)-1-(3-pyridyl)-1-butanone-exposed hamsters revealed that MUC1 mRNA increa

69 unds (acetaldehyde, diacetyl, acetoin, and 2-butanone) followed a sigmoidal trend described by the mo

70 tudied by varying the volume ratio of hexane/butanone from 85:15 to 45:55.

71 trooxy-2-butanone, and 3-methyl-3-nitrooxy-2-butanone have been determined at room temperature at 100

72 well as hydroxy acetone (HA) and 1-hydroxy-2-butanone (HB) as donor substrates, in each case using gl

73 ospira and higher levels of 1-pentanol and 2-butanone in NAFL patients compared to CTRLs.

74 ion of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone-induced pulmonary adenoma formation by myo-inos

75 ocking 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone-induced short-term O(6)-methylguanine and long-

76 mine, 4-(methylnitrosoamino)-1-(3-pyridyl)-1-butanone, is activated to lung DNA methylating and pyrid

77 Adaptation to one odorant, butanone, is disrupted by ODR-1 overexpression.

78 amine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, is one of the most potent and abundant procarc

79 tivity even for the hydrazone derived from 2-butanone (methyl vs ethyl, 91% ee).

80 THF was analyzed for a series of ketones (2-butanone, methyl acetoacetate, and N,N-dimethylacetoacet

81 The rate constants for the reduction of 2-butanone, methylacetoacetate, N, N-dimethylacetoacetamid

82 ion of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) (0.05 ng/L) and 4-(methylnitrosamino)-1-(

83 ncluding (methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and 4-(methylnitrosamino)-1-(3-pyridyl)-1

84 ure of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and benzo[a]pyrene (BaP) in A/J mice.

85 amines 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and N'-nitrosonornicotine (NNN) can under

86 amines 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and N-nitrosonicotine (NNN) are potent ca

87 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and vinyl carbamate (VC) during mouse lun

88 nogens 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and vinyl carbamate, and the occupational

89 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) by measuring the frequency of NNK-induced

90 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) by s.c. injection at a dose of 1.5 mg/kg

91 cromol 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) given by gavage once weekly for 8 weeks.

92 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) in a 2-year bioassay.

93 ced by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) in male F344 rats that had been fed eithe

94 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) in people who had stopped smoking: 4-(met

95 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) induces Ca(2+) signalling, a mechanism th

96 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) into DNA-altering compounds that cause lu

97 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is an important component in cigarette sm

98 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is formed by nitrosation of nicotine and

99 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is formed by nitrosation of nicotine and

100 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is of particular interest due to its pote

101 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is the most potent carcinogen contained i

102 ion by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) or vinyl carbamate (VC) resulted in a sig

103 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) reproducibly induces pulmonary adenocarci

104 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) were previously shown to be highly persis

105 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) within DNA duplexes representing p53 muta

106 inogen 4-(methylnitrosamino)-I-(3-pyridyl)-1-butanone (NNK), 4ebp1(-/-)/4ebp2(-/-) mice showed increa

107 4-(Methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), a tobacco-specific lung carcinogen, is b

108 (NNN), 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), and 4-(methylnitrosamino)-1-(3-pyridyl)-

109 nogen, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), and its inhibition by black tea have bee

110 samine-4-(methylnitrosamino)-1-(3 pyridyl)-1-butanone (NNK), BEAS-2B cells exhibited evidence of tran

111 e.g., 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), have not been elucidated.

112 amine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), in the canine trachea.

113 amine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), is a chemical carcinogen thought to be i

114 he NOC 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), O(6)-methyl-dG (O(6)-Me-dG) and O(6)-pyr

115 amine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), selectively induces lung tumors in F344

116 ole in 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced lung cancer by producing carcinog

117 ine on 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced lung tumor progression from adeno

118 ncy on 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced lung tumorigenesis.

119 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK).

120 amine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK).

121 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK).

122 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK).

123 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK).

124 tes of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK).

125 , and (4-methylnitrosoamino)-1-(3-pyridyl)-1-butanone (NNK).

126 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK).

127 inogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK); and r-1-,t-2,3,c-4-tetrahydroxy-1,2,3,4-

128 sed to 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, NNK, a potent carcinogen in tobacco smoke.

129 4-(acetoxymethylnitrosamino)-1-(3-pyridyl)-1-butanone (NNKOAc), enhanced the tumorigenic activity of

130 4-(acetoxymethylnitrosamino)-1-(3-pyridyl)-1-butanone (NNKOAc).

131 ose of 4-(methylnitrosamino)-I-(3-pyridyl)-1-butanone (P = 0.03) per cigarette than noncarriers.

132 .1 g/mol), 4-aminopiperidine, 3,3-dimethyl-2-butanone (pinacolone) and methylisobutyl ketone for whic

133 luding 4-(methylnitrosamino)-I-(3-pyridyl)-1-butanone plus benzo(a)pyrene.

134 found to modify the kinetic parameters for 2-butanone reduction by R-HPCDH in a saturable fashion, wi

135 , and increasing the enantioselectivity of 2-butanone reduction to a theoretical value of 100% (S)-2-

136 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone results in accumulation of Bcl2 in the nucleus

137 samine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone results in accumulation of Bcl2 in the nucleus,

138 ation leading to the eventual formation of 2-butanone than for the other two pathways.

139 lectivity for producing (S)-2-butanol from 2-butanone that was unaffected by modulators that interact

140 sion of 4-hydroxy-4-(6-methoxy-2-naphthyl)-2-butanone to 6-methoxy-2-naphthaldehyde and acetone.

141 cetone, pinacolone, butanone, and 3-methyl-2-butanone to give fair to good yields of 2H-1,2-benzothia

142 An Oscillospira decrease coupled to a 2-butanone up-regulation and increases in Ruminococcus and

143 The effect of proportions of hexane and butanone was first studied by varying the volume ratio o

144 The optimal mixture ratio of hexane to butanone was found to be 65:35.

145 D or L dibenzoyl tartaric acid (DBTH2) in a butanone/water or 2-pentanone/water solution.

146 , cycloheptanone, 3-methyl-2-butanone, and 2-butanone were converted to dienes.

147 rcinogen 4-methylnitrosamino-1-(3-pyridyl)-1-butanone were hypermethylated at the p16 gene promoter;

148 nder Dakin-West conditions, forms 4-phenyl-2-butanone when the reaction is catalyzed by 1-methylimida

149 te the partial oxidation of 2-butanol into 2-butanone with near 100% selectivity at low oxygen covera

150 nogen, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, with a catalytic efficiency much greater than