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1 inhibitor, 3, 4-dihydro-5-[4-(1-piperidinyl)butoxy]-1(2H)-isoquinolinone (DPQ; IC50=1 microM/l) was
2 RP inhibitor 3,4-dihydro-5-[4-(1-piperidinyl)butoxy]-1(2H)-isoquinolinone both showed increased resis
3 er by 10 muM 3,4-dihydro-5-[4-(1-piperidinyl)butoxy]-1(2H)-isoquinolinone, an established PARP inhibi
6 ist (R)-3-{4-[3-(4-chloro-2-phenoxy-phenoxy)-butoxy]-2-ethyl-phenyl}-propionic acid (20) for the trea
7 ble L-glutamic acid derivative, (4S)-5-(tert-butoxy)-4-[(tert-butoxycarbonyl)amino]-5- oxopentanoic a
10 blocked by the pan-caspase inhibitor N-tert-butoxy-carbonyl-Val-Ala-Asp-fluoro methylketone (zVAD-fm
13 ong the compounds studied, n-butyl- and/or n-butoxy-containing guanidines showed superior biological
16 performed at 120-130 degrees C, whereas tert-butoxy derivatives undergo the retro-ene reaction at 90
19 fied a substituted quinazolinone (Quin-C1, 4-butoxy-N-[2-(4-methoxy-phenyl)-4-oxo-1,4-dihydro-2H-quin
22 These mechanistic data imply that the tert-butoxy radical reacts with the C-H bonds of alkanes, and
23 oportionation pathway, catalyzed by the tert-butoxy radical, of mixed peroxides for the formation of
24 cal traps support the intermediacy of a tert-butoxy radical, which forms an alkyl radical intermediat
27 to vapors of trimethylaluminum and tris(tert-butoxy)silanol deposits highly conformal layers of amorp
28 supported by sterically demanding tris(tert-butoxy)siloxide ligands induces a large cooperative effe
29 uced via grafting reactions of the tris(tert-butoxy)siloxy iron(III) complex Fe[OSi(O(t)Bu)(3)](3)(TH
31 ction between the methyl hydrogens on a tert-butoxy substituent and the cyclooctatetraene (COT) ring
33 of sulfonyl halide derivatives of (S)-(tert-butoxy)-[(tert-butoxycarbonyl)amino]oxoalkanoic acids.
36 [(R)-3,3'-dibromo-2,2'-binaphthoxy](di-tert-butoxy)titanium(IV) (R-3) exists as a crystallographic C
37 ) or pentane (17-19); OR(f) = perfluoro-tert-butoxy) via photolysis of the precursor complex [(HEB)Re
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