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1 stored in cells treated with the antioxidant butylated hydroxyanisole.
2  the two antioxidant standards quercetin and butylated hydroxyanisole.
3 xidant chemopreventive agents ethoxyquin and butylated hydroxyanisole.
4 d reactive oxygen species and was blocked by butylated hydroxyanisole.
5 ession of ROS accumulation by ROS scavengers butylated hydroxyanisole, and N-acetyl-L-cysteine preven
6 itable by the antioxidants N-acetylcysteine, butylated hydroxyanisole, and pyrrolidine dithiocarbamat
7 23-0.3g/L) was very high, similar to that of butylated hydroxyanisole (BHA) and higher than that of o
8           Inhibition of ATO-induced ROS with butylated hydroxyanisole (BHA) does not affect Nrf2 acti
9                         Phenolic antioxidant butylated hydroxyanisole (BHA) is a commonly used food p
10                                              Butylated hydroxyanisole (BHA), a commonly used food pre
11 e presence or absence of orally administered butylated hydroxyanisole (BHA), an antioxidant.
12 re fed a diet that contained the antioxidant butylated hydroxyanisole (BHA), and others were given da
13                                              Butylated hydroxyanisole (BHA), butylated hydroxytoluene
14       Three synthetic phenolic antioxidants, butylated hydroxyanisole (BHA), butylated hydroxytoluene
15 idants pyrrolidinedithiocarbamate (PDTC) and butylated hydroxyanisole (BHA), indicating that the gene
16 xidants [tocopherol, catechin, lycopene, and butylated hydroxyanisole (BHA)] for conjugated dienes (d
17 ted factor-2 (Nrf2) activators (oltipraz and butylated hydroxyanisole) can induce hepatic Mrp transpo
18 ld-type and Nrf2-null mice with oltipraz and butylated hydroxyanisole demonstrated that the induction
19 n this type of cell death, the ROS scavenger butylated hydroxyanisole efficiently blocked TNF-induced
20                              The antioxidant butylated hydroxyanisole inhibited CYP2S1-catalyzed epox
21 d by the structurally unrelated antioxidants butylated hydroxyanisole, N-acetyl-L-cysteine, and pyrro
22 ncubation of CHO cells with the antioxidants butylated hydroxyanisole or butylated hydroxytoluene blo
23 AD, the antioxidants N-acetyl-l-cysteine and butylated hydroxyanisole, or an inhibitor of NADPH oxida
24 xidants, including butylated hydroxytoluene, butylated hydroxyanisole, TEMPOL (4-hydroxy-2,2,6, 6-tet
25 tment with a pancaspase inhibitor but not by butylated hydroxyanisole, which inhibits reactive oxygen

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