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2 ion in the early phase of liver regeneration by relieving Akt1/mTOR complex 1 signaling (and thus eIF
3 , Eco1 promotes sister DNA entrapment partly by relieving an antiestablishment activity associated wi
4 tes ERK activity in immature granulosa cells by relieving an inhibition imposed by a 100-kDa phosphot
5 dings suggest that Gib2 promotes cAMP levels by relieving an inhibitory function of Ras1 on Cac1 in t
7 src homology 3 domain-containing Cb variants by relieving autoinhibition and thus define an alternati
12 CTT mitigates infarct risk in pediatric SCA by relieving cerebral metabolic stress at patient- and t
14 he Nox structure promotes oxidase activation by relieving constraint induced by regulatory components
17 DEPTOR overexpression suppresses S6K1 but, by relieving feedback inhibition from mTORC1 to PI3K sig
18 nsulin/IGFs regulate beta cell proliferation by relieving Foxo1 inhibition of Pdx1 expression in a su
19 um, which augmented the heparin acceleration by relieving Gla domain inhibition as previously shown f
20 s6 is essential for XmyoD induction and acts by relieving Groucho-mediated repression of gene express
23 s formin-dependent capping of the barbed end by relieving inhibition of elongation by FRL1-FH1FH2, mD
25 Smac, a pro-apoptotic protein that functions by relieving inhibitor-of-apoptosis protein (IAP)-mediat
26 een shown to regulate its catalytic activity by relieving intramolecular autoinhibitory interactions.
27 tivates the transcriptional function of LAP1 by relieving its autoinhibited state effected by intramo
35 , has the potential to aid smoking cessation by relieving nicotine withdrawal symptoms and reducing t
36 dependent manner it induces the QS hierarchy by relieving PraR-mediated repression of rhiR and raiR.
37 gen (N) should enhance herbivore performance by relieving protein limitation, increasing herbivorous
40 y a critical role in transcriptional control by relieving repressive effects of chromatin, and yet ho
41 to increase the excitability of nociceptors by relieving resting slow inactivation of tetrodotoxin-r
44 sulin-like growth factor I-dependent manner, by relieving S6K1-dependent negative regulation of insul
48 DNA topoisomerase II modulates DNA topology by relieving supercoil stress and by unknotting or decat
49 umor regression directly but also indirectly by relieving suppressive signals that restrict potent an
51 suggest that external Zn(2+) activates ENaC by relieving the channel from Na(+) self-inhibition, and
53 and USF enhanced Ets-1 DNA binding in vitro by relieving the influence of an autoinhibitory domain i
54 naptotagmin-1 activates fast release, likely by relieving the inhibition caused by Complexins and coo
55 golic acid, decreases alpha-synuclein levels by relieving the inhibition exerted on alpha-synuclein p
56 y on the Bcl-2 family members Bax and/or Bak by relieving the inhibition imposed by antiapoptotic mem
57 tein levels by Cdc5 promotes FEAR activation by relieving the inhibition on Clb2-Cdk1, the kinase for
58 te cancer disrupts this regulatory mechanism by relieving the inhibitory effects of C2 and WW on Hect
59 CaBP4 increases Cav1.4 channel availability by relieving the inhibitory effects of the ICDI domain o
60 tion extended the T-cell-receptor repertoire by relieving the requirement for a charged residue in CD
62 nable the binding of 4(R)-hydroxy-2-oxoacids by relieving the steric hindrance between the 5-methyl g
63 tor Twist1 in inducing mesenchymal migration by relieving the suppression of NEDD9 and DOCK3 by the m
64 rmed that adenosine receptor activation acts by relieving the translational repressive effect of the
65 -PD-1 invigorates subdominant TCD8 responses by relieving their lysis-dependent suppression by immuno
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