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1 The mutation causes these effects by suppressing a conformational change that normally occ

2 y effective in targeting KRAS-mutant cancers by suppressing a newly discovered resistance mechanism.

3 nding protein RBM4 supports ULBP1 expression by suppressing a novel alternatively spliced isoform of

4 the plant will in turn bias stomatal opening by suppressing ABA sensitivity, which then aggravates tr

5 ua/CIC maintains peripheral immune tolerance by suppressing aberrant activation of adaptive immunity.

6 their proper development and maturation and by suppressing aberrant proliferation mediated by the T

7 nced the production of full-size transcripts by suppressing abortive loss of short RNAs.

8 vent neuroinflammation and neurodegeneration by suppressing activation of innate and adaptive immune

9 he responsiveness of macrophages to IFN-beta by suppressing activation of Jak-STAT signaling.

10 Id protein Extramacrochaetae enables growth by suppressing activation of the Salvador-Warts-Hippo pa

11 ess that can be weakened or even interrupted by suppressing activity in such nodes.

12 uffering from post-traumatic stress disorder by suppressing activity in the amygdala and thereby inhi

13 es both innate and adaptive immune responses by suppressing alpha-SYN-induced microglia and macrophag

14 ping mechanisms, maintain beta-cell identity by suppressing alternate pathways leading to neuronal, h

15 x vivo, ibuprofen also affected Sertoli cell by suppressing AMH production and mRNA expression of AMH

16 A1AR) protects against cisplatin ototoxicity by suppressing an inflammatory response initiated by ROS

17 e (AMPK), which regulates energy homeostasis by suppressing anabolic and activating catabolic process

18 DFTD spreads between hosts by suppressing and evading the immune system, and our re

19 available drugs aim to decrease inflammation by suppressing and/or modulating immune responses and th

20 promotes effective dorsal migration in part by suppressing anterior/animalward cell polarity and mov

21 thesis of these expensive products, and (ii) by suppressing antibiotic production in competitors, the

22 hesis that people can attenuate future fears by suppressing anticipatory simulations of dreaded event

23 Bacteria subvert the immune system by suppressing antimicrobial responses or directly killi

24 at AM promote cancer metastasis to the lungs by suppressing antitumor T cells in this organ.

25 inhibited the AAA development in AdAPN mice by suppressing aortic inflammatory cell infiltration, me

26 By suppressing APC(Cdc20), antimitotic agents activate t

27 with doxorubicin, paclitaxel, or carboplatin by suppressing apoptosis and upregulating NF-kappaB Over

28 L5/CCR5 may influence proplatelet production by suppressing apoptosis.

29 of mouse PGCs and spermatogonial stem cells by suppressing apoptosis.

30 ng antagonist sgp130Fc ameliorated emphysema by suppressing augmented alveolar type II cell apoptosis

31 re moderately effective at reversing disease by suppressing autoimmune inflammation in the skin and p

32 te of omega-3, decreased disease progression by suppressing autoreactive T cells and inducing an M2 p

33 the maintenance of immunological homeostasis by suppressing autoreactive T cells in liver and lung.

34 tumor development and resistance to therapy by suppressing BAX and its activators.

35 triggers caspase-dependent apoptosis in CLL by suppressing BCR, CD49d, CD40, and Toll-like receptor

36 more, p75ECD can also reduce amyloidogenesis by suppressing beta-secretase expression and activities.

37 selected thymocytes from negative selection by suppressing Bim expression.

38 to inhibit premature chondrocyte hypertrophy by suppressing BMP-2/Smad1 activity.

39 colysis and cancer stem-like characteristics by suppressing both H19 and PDK1.

40 rs, we show that foretinib protected neurons by suppressing both known degenerative pathways and a ne

41 ting oncogenic and tumor suppressive effects by suppressing both tumor suppressive mRNAs and oncogeni

42 -red light (FR; an indicator of competition) by suppressing bud outgrowth from lower rosette position

43 ced HO-1 activity, inhibits MMP-1 expression by suppressing c-Jun/AP-1 activation.

44 may enhance (or intensify) cryptosporidiosis by suppressing C. parvum-induced cell turnover and caspa

45 RAF3 limits RANKL-induced osteoclastogenesis by suppressing canonical and noncanonical NF-kappaB sign

46 and extent of inflammatory cell infiltration by suppressing canonical BMP signaling, thereby providin

47 nadotropin responsiveness in granulosa cells by suppressing canonical WNT signaling.

48 which EZH2 controls GC B cell proliferation by suppressing CDKN1A, enabling cell cycle progression w

49 intestinal cancer formation and progression by suppressing cell cycle inhibitors.

50 t monensin effectively inhibits tumor growth by suppressing cell proliferation through targeting EGFR

51 Loss of Dnmt3a delayed tumorigenesis by suppressing cellular proliferation during disease pro

52 inib may reduce CLL cell entry and retention by suppressing chemokine-induced integrin activation.

53 d its colonization of the inflamed intestine by suppressing commensal Enterobacteriaceae, which are s

54 (QDs) enhances triplet energy transfer (TET) by suppressing competitive charge transfer from QDs to m

55 g that it dampens the innate immune response by suppressing complement.

56 By suppressing conformational dynamics, IIA(Glc) blocks

57 This is accomplished by suppressing contractions of the detrusor muscle that

58 poietic stem cell transplantation (allo-HCT) by suppressing conventional T cells (Tconvs).

59 that miR-23a prevents ROS-elicited necrosis by suppressing cyclophilin D (PPIF), a regulator of ROS

60 S-induced inflammation and promote tolerance by suppressing DC IL-12 production.

61 ve molecule that promotes beta-cell survival by suppressing DDR signaling and attenuating DNA damage-

62 e excitability of SLC5A11-expressing neurons by suppressing dKCNQ channels, thereby conferring the hu

63 body, dE2F/dDP limits cell-cycle progression by suppressing DNA damage responses.

64 luding DNA repair and inflammatory response, by suppressing downstream targets IKBKB, WEE1, FGF2, RAD

65 t-negative effect on BRCA1 expression and HR by suppressing E2F1-mediated transactivation of BRCA1 pr

66 Kr-h1 maintains the immature stage by suppressing E93 (early ecdysone response gene) in N4.

67 de oxytocin contributes to metabolic control by suppressing eating behavior.

68 ) cells orchestrate memory T cell quiescence by suppressing effector and proliferation programs throu

69 y a convergent strategy of TE load reduction by suppressing element origination in their independent

70 sting that it may inhibit bud growth in part by suppressing elements of the cell cycle machinery and

71 c cells and functions to favor NHEJ over HDR by suppressing end resection, which is the rate-limiting

72 to reduce nanoparticle uptake in macrophages by suppressing endocytosis.

73 signaling in cancer cells promotes survival by suppressing endogenous DNA damage, and may control ce

74 rated the enhancement of dendritic formation by suppressing endogenous TRIO, and similarly decreasing

75 hus, TGF-beta induces SG ILC differentiation by suppressing Eomes.

76 ing Hz, contribute to severe malarial anemia by suppressing Epo-induced proliferation of erythroid pr

77 pound 13d alleviates ER stress in beta-cells by suppressing ER stress-mediated activation of all thre

78 ealed that Cdc42 attenuates MMP-1 expression by suppressing ERK activity.

79 d (cmpd) 28, ameliorates glucose homeostasis by suppressing excessive HGP and enhancing insulin signa

80 10 also indirectly supports humoral immunity by suppressing excessive IFN-gamma, which induces T-bet

81 on of the master osteoclast regulator NFATC1 by suppressing expression and recruitment of its newly i

82 ion protects against fatty liver development by suppressing expression of peroxisome proliferator-act

83 role for BLIMP1 in modulating host defenses by suppressing expression of the chemokine CCL8.

84 inhibited cell surface localization of CXCR4 by suppressing expression of the G protein receptor kina

85 amelanotic phenotype of motile cells in vivo by suppressing expression of the P-glycoprotein Oca2.

86 MF expression inhibited EGF-induced invasion by suppressing extracellular signal-regulated kinase sig

87 ration, clonogenic ability, and invasiveness by suppressing extracellular signal-regulated kinases 1/

88 breast cancer cells to PTX/CBP chemotherapy by suppressing FBXO11-dependent inhibition of p53.

89 itation of hippocampal CA1 pyramidal neurons by suppressing feedforward inhibition.

90 found to regulate flagellum-driven motility by suppressing flagellar reversal rates in a manner inde

91 bolished dopamine-induced salivary secretion by suppressing fluid transport in type III acini.

92 MafB promotes hyperlipidemic atherosclerosis by suppressing foam-cell apoptosis.

93 glucose production and promotes lipogenesis by suppressing FOXO1-dependent activation of G6pase and

94 at ethylene plays a role in tomato fruit set by suppressing GA metabolism.

95 s (Hdacs) regulate endochondral ossification by suppressing gene transcription and modulating cellula

96 ance and lineage determination of stem cells by suppressing genes that regulate cellular differentiat

97 ves robust axon growth and branching in part by suppressing GSK3beta.

98 tes hepatic steatosis and insulin resistance by suppressing hepatic TAG and DAG accumulation through

99 us, ERFE contributes to the recovery from AI by suppressing hepcidin and increasing iron availability

100 ayers (HTLs), which increase nucleus spacing by suppressing heterogeneous nucleation and facilitate g

101 cient and flexible coding in hippocampal CA1 by suppressing heterogeneously tuned excitation.

102 s endochondral ossification at the epiphysis by suppressing HIF signaling pathways.

103 pontaneous and drug-induced latency reversal by suppressing HIV-1 transcriptional elongation.

104 ugs might prove to be an effective treatment by suppressing HK2 infectivity in diseases where these v

105 efficiency from 60.3+/-6.1 % to 71.8+/-6.2 % by suppressing hole transfer.

106 n may, therefore, increase TB susceptibility by suppressing IFN-gamma and IL-17A production during th

107 lopment and onset of SS in NOD mice, in part by suppressing IFN-gamma production.

108 protumorigenic microenvironment, potentially by suppressing IFNgamma-producing natural killer cells a

109 ly reduced late, but not early, infarct size by suppressing IGF-1 degradation and, consequently, dimi

110 i-TNF-alpha therapy may act at least in part by suppressing IL-22BP and point toward a more specific

111 h2 cells and tolerization of dendritic cells by suppressing IL-23 production.

112 pDCs alleviates AHR and airway inflammation by suppressing ILC2 function and survival.

113 eutic approach in treating allergic diseases by suppressing ILC2 function.

114 uate sleep has deleterious effects on health by suppressing immunity and promoting inflammation.

115 mprovements of survival and cardiac function by suppressing infiltration of multiple kinds of inflamm

116 e that GCN2 controls intestinal inflammation by suppressing inflammasome activation.

117 predisposes to invasive pneumococcal disease by suppressing inflammatory processes of the upper respi

118 c damage in response to pathological stimuli by suppressing inflammatory reactions through AdipoR1/AM

119 A (JAM-A) renders protection from thrombosis by suppressing integrin outside-in signaling.

120 orate the pathological outcomes of infection by suppressing interleukin 6 production in the brain.

121 revealed to impair YFV and DENV2 propagation by suppressing intracellular viral RNA accumulation but

122 t Exo1 damage association, at least in part, by suppressing its association with PCNA.

123 nase delta (PI3Kdelta) pathway regulates AID by suppressing its expression in B cells.

124 Derepression of wild-type p53 by suppressing its negative inhibitor iASPP (Inhibitor o

125 y regulated the type I IFN signaling pathway by suppressing JAK1 and IRF9.

126 ID4 maintains the mammary stem cell pool by suppressing key factors required for luminal differen

127 suppressed pancreatic tumor growth, in part, by suppressing KRAS signaling.

128 at lower visual contrast and was diminished by suppressing L1 activity.

129 relieved when nuclear stiffness is decreased by suppressing lamin A/C expression.

130 ts demonstrate that mAChRs in CWC spines act by suppressing large-conductance calcium-activated potas

131 es a critical contribution to tumorigenicity by suppressing Let-7.

132 es adrenal glucocorticoid production, likely by suppressing liver X receptor-mediated activation of s

133 astatic process of lung cancer cells in vivo by suppressing local invasion and distant colonization.

134 tly attenuate ischemia-induced VA, primarily by suppressing LSG activity.

135 AtRAP negatively regulates defense responses by suppressing LSU2 through physical interaction.

136 PN impairs innate mucosal immunity by suppressing luminal sPLA2 activity and MUC2 density c

137 and the development of premalignant lesions by suppressing M1 macrophage polarization.

138 ced IL-35 inhibits LPS-induced EC activation by suppressing MAPK-AP1-mediated VCAM-1 expression and a

139 acute lung injury provoked by LPS inhalation by suppressing mast-cell activation and induction of med

140 important role in the tumor microenvironment by suppressing metastasis-promoting characteristics of t

141 n disease, complements the function of Hsp31 by suppressing methylglyoxal and oxidative stress, thus

142 nhibited alpha-SYN-induced neuroinflammation by suppressing microglial activation, macrophage and CD4

143 drive this learning-induced memory mechanism by suppressing microRNA-mediated translational silencing

144 a-secretase system decreases neuronal losses by suppressing miR-212 and increasing its target surviva

145 stically, ATDC exerted its oncogenic effects by suppressing miR-29 and subsequent upregulation of DNM

146 metformin inhibits cancer cell proliferation by suppressing mitochondrial-dependent biosynthetic acti

147 They regulate gene expression by suppressing mRNA translation and reducing mRNA stabil

148 inhibitor, affects the mucus hypersecretion by suppressing MUC5AC via signal transducer and activato

149 n modeled responses to MYCN-directed therapy by suppressing MYCN expression in MYCN-driven retinoblas

150 Intriguingly, Deltex2 inhibits myogenesis by suppressing MyoD transcription, and the Deltex2 knock

151 Traf2 in myocardial survival and homeostasis by suppressing necroptosis.

152 ndependent, prosurvival pathway in the heart by suppressing necroptotic signaling, which may serve as

153 licit a novel pro-survival function of APC/C by suppressing necrosis.

154 SI-09, phenocopied the effects of Epac1 null by suppressing neointima formation and proliferative VSM

155 genetically complement the loss of syndecan by suppressing neuronal guidance and locomotory defects

156 ve regulation of acute inflammatory response by suppressing NF-kappaB activation and proinflammatory

157 bited ox-LDL-induced macrophage inflammation by suppressing NF-kappaB p65 phosphorylation, nuclear tr

158 tor of HIV-1 gene-expression and replication by suppressing NF-kappaB-mediated activation of viral tr

159 niche pinwheel structures, at least in part by suppressing Notch activation in radial glial cells, w

160 EZH2 activated the Notch pathway by suppressing Notch repressors Numb and Fbxw7 via trime

161 suggest that the A1AR provides otoprotection by suppressing NOX3 and inflammation in the cochlea and

162 androgen-independent prostate cancer growth by suppressing nuclear receptor-mediated oxidative stres

163 line graphene domains in approximately 12 h, by suppressing nucleation events on the growth substrate

164 trophils after inclusion in the nanovesicles by suppressing O2(-) generation and elevating cAMP.

165 el components in high dynamic range mixtures by suppressing one-bond (13)C satellite signals in (1)H

166 Teriparatide increases osteoblast numbers by suppressing osteoblast apoptosis and activating bone-

167 markedly inhibited bone loss and resorption by suppressing osteoclast activity.

168 oading regulates subchondral bone remodeling by suppressing osteoclast development, and prevents degr

169 ore, knee loading exerted protective effects by suppressing osteoclastogenesis through Wnt signaling.

170 4E phosphorylation and was not recapitulated by suppressing other cabozantinib targets.

171 ductance enhances rate coding in place cells by suppressing out-of-field excitation and by limiting d

172 atory role in lineage-committed tissue cells by suppressing overactivation of multiple signalling pat

173 le in promoting HCV infection and HCC tumors by suppressing p53.

174 ilon inhibition promoted cellular senescence by suppressing p65-NF-kappaB and inducing p16(Ink4a).

175 er aerosol chemical properties, for example, by suppressing particle volatility.

176 reduces GBM cell migration and invasiveness by suppressing PCP signaling.

177 ductive and effector sites of oral tolerance by suppressing peripheral regulatory T cell (pTreg) conv

178 COX inhibition abrogated immune tolerance by suppressing PGI2 IP signaling, suggesting that PGI2 s

179 Deleting Ask1 abrogated diabetes-induced UPR by suppressing phosphorylation of inositol-requiring enz

180 phogenesis and modulates thermomorphogenesis by suppressing PIF4 activity, through a reduction in PIF

181 microbes can favor or improve insect fitness by suppressing plant defenses and detoxifying defensive

182 wn to mediate cancer stem-like cell function by suppressing pluripotency and cellular dedifferentiati

183 stability among all reported Mg/S batteries by suppressing polysulfide dissolution.

184 may protect the brain against its I/R injury by suppressing post-ischemic inflammation.

185 1 functions in all these regulatory contexts by suppressing predominant rearrangement of D-proximal V

186 peptides into amyloid fibrils, in particular by suppressing primary nucleation.

187 ion by directly activating transcription and by suppressing PRMT5/MEP50-dependent arginine dimethylat

188 diolipin inhibits resolution of inflammation by suppressing production of anti-inflammatory IL-10 by

189 was able to modulate HIF-1alpha degradation by suppressing prolyl hydroxylase 3 expression.

190 t that mTORC1 inhibition reduces proteolysis by suppressing proteasome expression.

191 ential for prometaphase entry or progression by suppressing protein phosphatase 2A (PP2A) activity.

192 rmation of a more reactive phenotype enabled by suppressing proteolytic cleavage of sIL-6R from IL-6R

193 signals enforce positive-feedback regulation by suppressing PTEN function.

194 promote divergence and facilitate speciation by suppressing recombination.

195 st integration in these oscillatory circuits by suppressing regenerative activity in their dendrites.

196 n of corticostriatal glutamatergic synapses, by suppressing regulator of G protein signaling type 4 (

197 tective role, promotes pathogen colonization by suppressing related commensal bacteria.

198 r, vitamin D may also promote renoprotection by suppressing renin transcription through cross-talk be

199 rts in the maintenance of immune homeostasis by suppressing responses to harmless antigens and by enf

200 omotes male differentiation in XY germ cells by suppressing retinoic acid (RA)-dependent meiotic entr

201 increased spontaneous EPSC (sEPSC) frequency by suppressing RIM1alpha-facilitated CaV2.2 expression i

202 li NusG increases transcription processivity by suppressing RNAP pausing, whereas Bacillus subtilis N

203 f bone marrow-derived osteoblast progenitors by suppressing Runx2 gene expression.

204 her salience-driven distraction is prevented by suppressing salient distractors or by preferentially

205 we show that the host responds to infection by suppressing siderophore synthesis while up-regulating

206 marrow-derived cells and limits inflammation by suppressing signaling through stimulatory receptors.

207 Attenuating apprehensiveness by suppressing simulations of feared events may thus be

208 mation encoding for complex auditory stimuli by suppressing spontaneous "noise" activity and that the

209 ulatory protein protects against nephropathy by suppressing STAT-mediated cell responses to diabetic

210 moting environmental condition, acts in part by suppressing stomatal defense and is linked to hormone

211 es the surface and improves carrier mobility by suppressing surface-defect-related scattering.

212 en implicated in immune regulation, not only by suppressing T cell responses but also by regulating a

213 lation crucially induces Th2 differentiation by suppressing T-bet expression, followed by the inducti

214 cancer cell stem-like properties as well as by suppressing T-cell activation.

215 ode co-infection altered malaria propagation by suppressing target cell availability.

216 of TCF4, we created an in vivo model of PTHS by suppressing Tcf4 expression in rat prefrontal neurons

217 ells that specifically controls GC responses by suppressing Tfh and B cells.

218 al growth factor (VEGF) secretion in Mos/Mps by suppressing TGFbeta1 signalling and subsequently inhi

219 We show that LOX regulates EGFR by suppressing TGFbeta1 signalling through the secreted

220 t may support ustekinumab efficacy, possibly by suppressing TH17 responses through the retinoid-relat

221 hat MHC-matching reduces the immune response by suppressing the accumulation of microglia (Iba-1+) an

222 demonstrate that Notch maintains type II NBs by suppressing the activation of earmuff (erm) by Pointe

223 Signals that promote growth do so in part by suppressing the activation of NDR kinases by STE20/Hi

224 ctivation of transcription of the BIC genes, by suppressing the activity of CONSTITUTIVE PHOTOMORPHOG

225 -124-3p on neuronal inflammation was exerted by suppressing the activity of mTOR signaling.

226 he inhibitory effect of the spacing was lost by suppressing the activity of Ras or mitogen-activated

227 ced secretion of TNF-alpha from macrophages, by suppressing the activity of the TNF-alpha-converting

228 pancreatitis in EtOH-sensitized acinar cells by suppressing the adaptive unfolded protein response si

229 affects the RNA remodeling ability of LAF-1 by suppressing the affinity, dynamics, and annealing act

230 latory network controlling tumor progression by suppressing the Akt pathway.

231 C1 as a novel regulator of B cell activation by suppressing the alternative NF-kappaB pathway.

232 ose uptake favors osteoblast differentiation by suppressing the AMPK-dependent proteasomal degradatio

233 viate pain and reduce fever and inflammation by suppressing the biosynthesis of prostacyclin (PGI2) a

234 4a) in obesity inhibits fat browning in part by suppressing the browning activators fibroblast growth

235 othesized that PIN1 relieves CDK2 inhibition by suppressing the CDK inhibitory activity of p27.

236 regulatory cytokine during RSV pathogenesis by suppressing the development of Th17 cells, but it als

237 By suppressing the different components of host epigenet

238 ecular strategy to adapt to hot environments by suppressing the efficiency of TRPV1-mediated heat det

239 primes progenitor cells for differentiation by suppressing the enhancer of zeste2 gene (EZH2), invol

240 otes the development of CS-induced emphysema by suppressing the enzymatic activities of the LTA4H ami

241 not Tyr(701)) and STAT1 luciferase activity by suppressing the ERK1/2, p38, and JNK mitogen-activate

242 pectral resolution, which can be implemented by suppressing the experimental noises.

243 y, CS and SULT2B1b inhibited gluconeogenesis by suppressing the expression of acetyl coenzyme A (acet

244 inhibition in an experience-dependent manner by suppressing the expression of EphB4; PV cells hemizyg

245 miR-126-3p enhanced angiogenesis by suppressing the expression of its known target, SPRED

246 beta inhibits macrophage foam cell formation by suppressing the expression of key genes implicated in

247 or of thymocyte survival and that it does so by suppressing the expression of p53.

248 er engagement of the B cell antigen receptor by suppressing the expression of the autoimmune suppress

249 t Btd prevents the premature differentiation by suppressing the expression of the homeodomain protein

250 node that switches off antimigratory miR-198 by suppressing the expression of the regulatory factor,

251 d that IDR-1002 reduced sterile inflammation by suppressing the expression of transmembrane G protein

252 erial uptake of iron-laden siderophores, and by suppressing the ferritin iron pool.

253 g mechanisms, however, can evade these tests by suppressing the forces in regions of high density, su

254 The compounds likely enhanced cell viability by suppressing the formation of reactive oxygen species

255 IFN-gamma repressed genes by suppressing the function of enhancers enriched for bi

256 by inducing chromosomal instability but also by suppressing the function of the cell cycle inhibitor

257 ysaccharide (LPS)- and H2O2-stimulated cells by suppressing the generation of ROS and pro-inflammator

258 IDH3alpha significantly delayed tumor growth by suppressing the HIF-1-mediated Warburg effect and ang

259 , simvastatin improved restenosis indicators by suppressing the HIF-1alpha/calpains/MMP2/TGF-beta1 pa

260 on independently of extrinsic SHH activation by suppressing the induction of GLI3.

261 thelialization, and collagen deposition, and by suppressing the inflammatory response and apoptosis.

262 atments to symptomatically alleviate disease by suppressing the inflammatory response and restoring b

263 Although BMI1 operates by suppressing the Ink4a/Arf locus in progenitors and ST

264 miR160 appears to promote auxin activity by suppressing the levels of the ARF10/16/17 family of r

265 iminished the medium after hyperpolarization by suppressing the M-current.

266 hieve more control over the mode selectivity by suppressing the mode that has the highest gain (i.e.

267 in SEB-1 sebocytes, and reduced inflammation by suppressing the NF-kappaB pathway in SEB-1 sebocytes

268 nes and the capability to attract leukocytes by suppressing the NF-kappaB pathway.

269 liferation and myofibroblast differentiation by suppressing the phosphorylation of extracellular sign

270 r stress counteracts endothelial dysfunction by suppressing the pro-inflammatory non-canonical TGF-be

271 metformin inhibits cancer cell proliferation by suppressing the production of mitochondrial-dependent

272 hat RNF17 shapes adult meiotic piRNA content by suppressing the production of secondary piRNAs.

273 ay contribute to the pathogenesis of anthrax by suppressing the responses of immune cells and interfe

274 the field has been shown to increase yields by suppressing the Rubisco oxygenase reaction and, in tu

275 utes to female germline stem cell unipotency by suppressing the somatic program and is potentially in

276 l monitoring of genetic and protein circuits by suppressing the spectral cross-talk among multiple fl

277 ffective hydrate-nucleation sites), and also by suppressing the thermal fluctuation of solvated Na(+)

278 Additionally, by suppressing the triggering surface undulations, sinuo

279 y regulates hepatic cancer stem cells (CSCs) by suppressing the tumor suppressor p53.

280 miR-200 family promotes the epithelial state by suppressing the Zeb1/Zeb2 epithelial gene transcripti

281 n by removing unused or unwanted memories or by suppressing their expression.

282 People often respond to intrusions by suppressing their retrieval, excluding them from awar

283 s willingness to cross the dangerous barrier by suppressing this pathway.

284 -10b promotes PCC proliferation and invasion by suppressing TIP30, which enhances EGFR signaling, fac

285 AAL(S) reduced the severity of acute AAD by suppressing tissue eosinophils and inflammation, mucu

286 dendritic cell (DC) responses, specifically by suppressing TLR4 signalling to inhibit Th1/Th17-drive

287 naive CD8(+) T cells required for activation by suppressing TMEM20, a negative regulator of Ca(2+) ex

288 exosomes that inhibit macrophage activation by suppressing Toll-like receptor signalling, thereby de

289 es TNF-induced JNK activation and cell death by suppressing TRAF2 K63-polyubiquitination; upon TNF st

290 tially recapitulates the amphibian condition by suppressing transit amplification.

291 overed that OCTR-1 regulates innate immunity by suppressing translation and the unfolded protein resp

292 nce of the persister phenotype are regulated by suppressing translation.

293 inhibits primary tumor growth and metastasis by suppressing tumor angiogenesis.

294 nt role in modulating disease-risk in humans by suppressing tumour progression, decreasing inflammati

295 he FGF19-SHP-LSD1 axis maintains homeostasis by suppressing unnecessary autophagic breakdown of cellu

296 on, tube formation and thereby, angiogenesis by suppressing VEGFR-2 phosphorylation.

297 h revealed that Daam2 promotes tumorigenesis by suppressing VHL expression.

298 ined that DKK3 inhibits neuroblast formation by suppressing WNT signaling and Dkk3 deletion or pharma

299 umor suppressor, which inhibits tumor growth by suppressing Wnt/beta-catenin signaling in lung adenoc

300 ppears to modify the specificity of the MAPK by suppressing Y kinase and enhancing S/T kinase activit

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