ライフサイエンスコーパス: c-Fos protein

1 dissociates c-Fos from KDM2B and stabilizes c-Fos protein.

2 fter mFSS exposure for CRF and AVP mRNAs and c-Fos protein.

3 neocortex (GNC) expresses enhanced levels of c-Fos protein.

4 reased levels of c-fos promoter activity and c-fos protein.

5 neurons were identified through detection of c-Fos protein.

6 me associated with massive CNS expression of c-Fos protein.

7 then processed immunohistochemically for the c-Fos protein.

8 ound as a heterodimer comprised of c-Jun and c-Fos proteins.

9 ation and nuclear translocation of EGR-2 and c-fos proteins.

10 ent in JNK activity, had increased c-jun and c-fos proteins.

11 GHF-1 interacted directly with c-Jun but not c-Fos proteins.

12 As expected, Ex4 induced expression of c-Fos protein, a marker of neuronal activation, in hindb

13 .8% standardized green tea extract inhibited c-fos protein accumulation by 18.5% and 46.2% (p < 0.05)

14 trations in fetal peripheral plasma and Fos (c-fos protein) activation in corticotropin-releasing hor

15 nslational modification of newly synthesized c-Fos protein after H2O2 exposure.

16 activity and accumulation of c-fos mRNA and c-Fos protein after UVB irradiation.

17 stibular neurons were conducted to visualize c-Fos protein and Fluoro-Gold concomitantly.

18 in (i.e. PP2B), resulted in disappearance of c-Fos protein and MG132 was able to prevent this loss.

19 lpha-dihydrotestosterone and R1881) suppress c-Fos protein and mRNA expression induced by 12-O-tetrad

20 ity involving transient de novo synthesis of c-Fos protein and prolonged synthesis of c-Jun, mediated

21 tochemistry for NMDA NR1 glutamate receptor, c-fos protein, and apoptosis [nuclear immunoreactivity f

22 In addition, we used the expression of c-Fos protein as a marker of neuronal activity to assess

23 The present study used the expression of c-Fos protein as a marker to identify brain areas throug

24 re, expression of either wild-type or mutant c-fos proteins augmented or diminished the response of t

25 increased the expression of the AP-1 subunit c-Fos protein, but not in the presence of PD98095 and wo

26 by in situ hybridization) and immunoreactive c-Fos protein (by immunohistochemistry) were elevated by

27 cells in serum-free medium, indicating that c-Fos protein can induce apoptotic cell death in these c

28 quence similarity with AREs, the purine-rich c-fos protein-coding region determinant of instability.

29 pid increase in c-fos mRNA, but the level of c-Fos protein decreases due to degradation by the multic

30 tion by assembling an E3 ligase to targeting c-Fos protein degradation that is antagonized by mitogen

31 hosphorylation contributes to degradation of c-Fos protein during oxidative stress response of cardio

32 er cypermethrin, extended the time course of c-Fos protein elevation after H2O2 exposure.

33 some Inhibitor I extended the time course of c-Fos protein elevation.

34 e perfused either 5 min or 2 h after SCS and c-fos protein examined immunohistochemically.

35 Here we report that cortical c-Fos protein expression declines significantly followin

36 ata show a dynamic and nonuniform pattern of c-Fos protein expression in brain nuclei known to mediat

37 We used the activation of c-fos protein expression in spinally projecting neurons

38 , MK801, partially reduces cocaine-activated c-Fos protein expression in the CPu.

39 iated with lithium chloride-induced malaise, c-Fos protein expression increased dramatically as compa

40 Fos to the p28 promoter but had no effect on c-Fos protein expression or phosphorylation in response

41 nalysis which showed METH-induced changes in c-Fos protein expression that were blocked by pretreatme

42 MMP-9 through increased AP-1/DNA binding and c-Fos protein expression via ERK and PI3K signaling path

43 Using immunohistochemical methods, brain c-Fos protein expression was analyzed in rats that extin

44 on-associated protein (Arc) mRNA and Arc and c-Fos protein expression was detected in RSP from fear-c

45 Immunohistochemical quantification of c-Fos protein expression was used as the surrogate measu

46 ess-induced 5-HT neural activity, indexed by c-Fos protein expression, in the DRN and c-Fos expressio

47 NAc, SCH23390 also inhibits cocaine-induced c-Fos protein expression.

48 -1 DNA binding activity through induction of c-Fos protein expression.

49 DHEA increased c-Jun, but not c-Fos, protein expression after 2 h.

50 ic area (vlPOA) express immunoreactivity for c-Fos protein following sustained sleep, and display ele

51 The c-Fos proteins form heterodimers with Jun family protein

52 failure (HF) by mapping neuronal staining of c-Fos protein (Fos) 6-8 weeks following coronary artery

53 sponsive to novel tastes, immunostaining for c-fos protein (Fos-like immunoreactivity [FLI]) was used

54 The c-fos protein, Fos, was identified immunocytochemically

55 An increase in ubiquitin was detectable in c-Fos protein from H2O2-treated cells.

56 The presence of the c-Fos protein has been evidenced in the piriform cortex,

57 Expression of c-Fos protein immunoreactivity (IR) in GABAergic neurons

58 We examined the pattern of c-Fos protein immunoreactivity throughout the rostral-ca

59 increase in neural activity (as measured by c-fos protein immunoreactivity) when spontaneous recover

60 These results suggest that the decrease in c-Fos protein in cortical neurons during sleep may be at

61 euronal loss, an expression of dynorphin and c-Fos protein in neurons in the deep laminae of the dors

62 fiber primary afferent input, and upregulate c-Fos protein in response to noxious stimuli.

63 s, we examined the patterns of expression of c-Fos protein in the brain and correlated these with phy

64 This study examined the expression of the c-Fos protein in the rabbit's central nervous system to

65 (as indicated by the regional expression of c-Fos protein) in rats during acquisition and throughout

66 Western blot analysis showed that the c-fos protein level increased in the nuclear fraction af

67 c-Fos protein levels are slightly increased in these can

68 kinase, pp90 ribosomal S6 kinase (RSK), and c-Fos protein levels in the caudate/putamen of Fischer r

69 ase in brain-derived neurotrophic factor and c-FOS protein levels, key regulators of synaptic plastic

70 fficient to induce AP-1 and increase nuclear c-Fos protein levels, PI 3-kinase may need to cooperate

71 s c-Fos for polyubiquitylation and regulates c-Fos protein levels.

72 -Fos mRNA but caused no alternation in total c-Fos protein levels.

73 activity and phosphorylation, and increased c-fos protein levels.

74 upershift experiments revealed that JunD and c-Fos proteins mediated anti-CD19 induced AP-1-binding a

75 The TP functionality anchors the c-Fos protein onto the metal substrate and works as an e

76 Chimeric ZEBRA/c-Fos proteins overexpressed in Escherichia coli bound D

77 The data suggest that c-Fos protein plays a causal role in the activation of a

78 ctions, immunohistochemical detection of the c-Fos protein product was used to study the distribution

79 Western blot analyses of c-MYC and c-FOS protein products support the overexpression of the

80 Decreased levels of c-fos protein result in decreased AP-1 activity, as dete

81 c-Fos protein(s) expression was high at 1.5 h and decrea

82 Finally, c-fos protein staining after novelty suppressed feeding

83 t TGFbeta, presumably through its effects on c-Fos protein synthesis and/or stability, abrogates the

84 ted UVB induced c-fos gene transcription and c-Fos protein synthesis.

85 ium release induces a sustained elevation of c-Fos protein that precedes grp78 induction.

86 by increased nuclear abundance of c-jun and c-fos proteins that bound specifically to the proximal c

87 histochemical analysis of rapidly developing c-Fos protein to elucidate neurobiological loci involved

88 the present study we used the expression of c-Fos protein to identify CNS regions activated during i

89 manner, and increased binding of endogenous c-fos protein to the cyclin A CRE precedes the onset of

90 In addition, the amount of c-Fos protein was estimated by chemiluminescent immunobl

91 In reticular formation c-fos protein was induced in circumscribed columns in th

92 Expression of c-Fos protein was not detected in calretinin-positive ne

93 d phosphomimetic mutations at S374 result in c-Fos protein which cannot be induced by EGF or accumula

94 e phosphatase A (PP2A) and immunoblotting of c-Fos protein with antibodies against phosphorylated ser

95 ones are GABAergic by combining staining for c-Fos protein with staining for glutamic acid decarboxyl

96 Treatment of immunoprecipitated c-Fos protein with the type 2 serine/threonine phosphata