戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 scription factor and increased expression of c-Fos gene.
2 cers underlies the broad inducibility of the c-fos gene.
3 is a repression of the ability to induce the c-fos gene.
4 s amphetamine-induced desensitization of the c-fos gene.
5 results in transcriptional activation of the c-fos gene.
6 cificity factor (CPSF73), was reduced at the c-FOS gene.
7 ired for Fyn activation and induction of the c-fos gene.
8 transcriptional activation of the endogenous c-fos gene.
9  activates the serum response element of the c-fos gene.
10  phosphoCREB-stimulated transcription of the c-fos gene.
11  transcriptional responses of the endogenous c-fos gene.
12  for STAT activation or for induction of the c-fos gene.
13 obes for a natural DNA sequence in the mouse c-fos gene.
14  the major CRE in the promoter region of the c-fos gene.
15 2+-induced transcription of prodynorphin and c-fos genes.
16  represses transcription of prodynorphin and c-fos genes.
17 response elements (DREs) in prodynorphin and c-fos genes.
18 NA response elements in the prodynorphin and c-fos genes.
19  element (DRE) of either the prodynorphin or c-fos genes.
20 um glucocorticoid kinase, inhibin alpha, and c-fos genes.
21  BMP2 inhibits PDGF-induced transcription of c-fos gene, a natural target of Elk-1 that normally form
22 s had no effect on AII-induced MAP kinase or c-fos gene activation.
23                                          The c-fos gene (also known as Fos) is induced by a broad ran
24 k factor 1 inhibits the transcription of the c-fos gene and antagonizes the activating effects of the
25 ells by functionally affecting expression of c-fos gene and AP-1 luciferase gene reporter activity.
26 hat PKC couples to the activation of the the c-fos gene and MAP kinases.
27                       AII also activated the c-fos gene and mitogen-activated protein (MAP) kinases.
28                   The activation of PKC, the c-fos gene, and MAP kinases was blocked by inhibition of
29 in the adenovirus 2 major late promoter, the c-fos gene, and the c-myc P1 and P2 promoters reveal var
30 rate that multiple enhancers surrounding the c-fos gene are crucial for ensuring robust c-fos respons
31 hermore, transcripts from the ARE-containing c-fos gene are selectively retained in the nucleus, whil
32                                    Using the c-fos gene as a candidate PRG, we addressed here how it
33  is required for regulated expression of the c-fos gene as well as other immediate-early genes and so
34 articipate in the process that represses the c-fos gene at the onset of cellular senescence.
35 induced c-fos expression, but also decreased c-fos gene basal expression.
36 null ES cells permitted transcription of the c-fos gene but was unable to rescue expression of myogen
37 ing to DNA response elements upstream of the c-fos gene (c-sis-inducible enhancer element; Stat 1 and
38   One CNS located in the first intron of the c-fos gene conferred regulation by cAMP-dependent protei
39 nsactivating a luciferase reporter driven by c-fos gene enhancer elements, suggesting that PRL allowe
40 tion of sialylated O-glycan and increases of c-fos gene expression and AP-1 activity, which are chara
41  a transcriptionally-mediated suppression of c-fos gene expression associated with the malignant tran
42                  UVB significantly increased c-fos gene expression at both the transcriptional and pr
43 ctivation of MAP kinase and the induction of c-fos gene expression both correlated with STAT but not
44 nse factor (SRF), and Tax is known to induce c-fos gene expression by activating SRF-responsive trans
45 rdiac tissues and represses prodynorphin and c-fos gene expression by binding to DNA response element
46       This study investigated the pattern of c-fos gene expression corresponding with auditory adapta
47 gnaling components in mediating induction of c-fos gene expression downstream of epidermal growth fac
48 vlPOA) of rats was found to exhibit elevated c-fos gene expression during sleep, indicating that thes
49 he same area where neurons exhibit increased c-fos gene expression during sleep.
50 RelA contribute significantly to EGF-induced c-fos gene expression in a manner independent of the cla
51 ve of the present study was to determine how c-fos gene expression in brainstem structures after a br
52 nt with the hypothesis that the induction of c-fos gene expression in GnRH neurons leads to an increa
53 inistration of cholecystokinin (CCK) induced c-fos gene expression in NTS POMC-EGFP neurons, suggesti
54                      Our results reveal that c-fos gene expression in the adult rat is attenuated in
55 with CRH there was a significant increase in c-fos gene expression in the CeA and in the hippocampal
56  dopamine-regulated CREB phosphorylation and c-fos gene expression in the striatum.
57 a Rho kinase and myocardin and also regulate c-fos gene expression independently via CaMK.
58 hway that activates CREB phosphorylation and c-fos gene expression is likely activated by Ca(2+) entr
59                             We conclude that c-fos gene expression occurs transiently in granule cell
60 rnary complex factors (TCFs), which regulate c-fos gene expression through the serum response element
61 asticity, and (3) stress are proposed to use c-fos gene expression to signal molecular changes in neu
62  have shown that nitric oxide (NO) regulates c-fos gene expression via cGMP-dependent protein kinase
63                  Specifically, activation of c-fos gene expression was found to occur exclusively thr
64                                              c-fos gene expression was increased by PMA but not invol
65 PI 3-kinase signaling pathway in UVB-induced c-fos gene expression was investigated in a human kerati
66                                  Patterns of c-fos gene expression were compared between Saffan-anest
67                                  Patterns of c-fos gene expression were site-specific and correlated
68 cts of p38 MAP kinase and ERK on UVB induced c-fos gene expression were studied in a human keratinocy
69 tress generates only a small contribution to c-fos gene expression while novel stimuli are potent sig
70 alloproteinase-13 (MMP-13), c-jun, junB, and c-fos gene expression, binding of activator protein 1 (A
71 ediated by the induction of c-jun, junB, and c-fos gene expression, by the binding of AP1 to DNA, by
72 n had no effect on AP-1 activation, c-jun or c-fos gene expression, or CREB phosphorylation.
73 while not inhibiting smooth muscle-unrelated c-fos gene expression, suggesting that RGC-32 is an impo
74               While Epo is known to activate c-fos gene expression, the mechanism of AP-1 activation
75 s transcription factor significantly reduces c-fos gene expression.
76  and respond to leptin with a stimulation of c-fos gene expression.
77  pathway that led to CREB phosphorylation or c-fos gene expression.
78 ndicate that novelty of sound stimuli induce c-fos gene expression.
79 s/luc) was generated to continuously monitor c-fos gene expression.
80 ic acid receptors (RARs) and AP-1 (c-Jun and c-Fos) gene expression, chronic ethanol (36% of total ca
81 y to inhibit endogenous transcription of the c-fos gene in NIH3T3 cells at micromolar concentrations.
82 rmation of an active promoter complex at the c-fos gene, including the linkage of specific signal res
83                     We evaluated patterns of c-fos gene induction as a monitor of spinal neurons resp
84 xtracellular signal-related kinase (ERK) and c-fos gene induction.
85 ays is also required for the response of the c-fos gene itself to UV stimulation.
86  were processed immunocytochemically for the c-fos gene product, Fos and related antigens.
87 gative Src (SrcK-) blocked activation of the c-fos gene promoter by CaMK II 290, a constitutively act
88  and the reduction in transcription involved c-fos gene promoter elements from -327 to +40.
89 uced methylation at CpG dinucleotides in the c-Fos gene promoter, effects reversed by MET treatment.
90 ruiting histone deacetylase 1 (HDAC1) to the c-fos gene promoter, which, in turn, deacetylates surrou
91 taining the TATA elements from the hsp70 and c-fos gene promoters but failed to significantly activat
92  the role of MAP kinases in retinoid-induced c-fos gene regulation.
93      The induction of transactivation by the c-fos gene regulator Elk-1 mirrored this requirement for
94  altering the chromosomal environment of the c-fos gene, thereby rendering it more accessible to the
95 t JNK regulates Elk-1 transactivation at the c-fos gene to promote the formation of AP-1 complexes im
96  p38 almost completely abrogated UVB induced c-fos gene transcription and c-Fos protein synthesis.
97                                              c-Fos gene transcription was lower in tumorigenic HBE ce
98  expression, (2) Ras-dependent activation of c-fos gene transcription, inferred from the induction of
99 hibition of Akt also attenuated PDGF-induced c-fos gene transcription, with concomitant inhibition of
100  c-fos mRNA and protein levels by decreasing c-fos gene transcription.
101      Previous studies have reported that the c-fos gene via formation of activator protein-1 (AP-1) t
102                            Activation of the c-fos gene was blocked by coexpression with a Raf-C4 cat
103                                          The c-fos gene was one of the earliest vertebrate genes show
104 taining for the protein product (Fos) of the c-fos gene was used as an index of neuronal activation.
105 ls of housekeeping genes, but not of p21 and c-fos genes, which are involved in the DNA damage respon

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top