ライフサイエンスコーパス: c-jun gene

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1 owth factor (EGF) induction of a transfected c-jun gene.

2 activation is increased transcription of the c-jun gene.

3 in cells that harbor a null mutation in the c-jun gene.

4 down-regulation, respectively, of the GR and c-jun genes.

5 If the products of the c-fos and/or c-jun genes are essential components in the cascade of e

6 by JNK and subsequent auto-activation of the c-JUN gene by recruitment of c-JUN and ATF2 to two AP-1

7 l observation that AP-1 sequences within the c-JUN gene can function as transcriptional amino acid-re

8 c-jun gene deletion reduced c-Src expression, hyperactiv

9 hila homolog of the mammalian proto-oncogene c-Jun gene (Djun).

10 lts indicate that heat shock activates c-Fos/c-Jun gene expression and AP-1 DNA binding and suggests

11 tion is followed by an increase in c-fos and c-jun gene expression and enhanced activating protein 1

12 The induction of c-fos and c-jun gene expression by bFGF or by PMA was blocked by t

13 A dose-dependent upregulation of c-fos and c-jun gene expression by the PKC activator PMA was also

14 un suggested that up-regulation of c-fos and c-jun gene expression does not directly contribute to th

15 dicate that exogenous bFGF induces c-fos and c-jun gene expression in cultured rat Muller cells throu

16 increases in c-fos in the mNTS and cRVLM and c-jun gene expression in the mNTS and rRVLM caused by NP

17 ate that the coordinate regulation of GR and c-jun gene expression is dose-dependent and cell type-sp

18 Induction of c-fos and c-jun gene expression started at a bFGF concentration of

19 The temporal pattern of c-jun gene expression was similar to that of c-fos, wher

20 HDAC inhibitors suppressed the induction of c-jun gene expression, resulting in reduced COX-2 transc

21 itor, did not inhibit bFGF-induced c-fos and c-jun gene expression, whereas forskolin (5 microM), an

22 PMA transiently increases egr-1 and c-jun gene expression.

23 bound to the c-Jun promoter and upregulated c-Jun gene expression.

24 tivity and an efficient inducer of c-fos and c-jun gene expression.

25 ethal; therefore, transgenic mice encoding a c-Jun gene flanked by LoxP sites (c-jun(f/f)) were used.

26 These findings indicate that the c-jun gene has a biphasic response to hypoxia consisting

27 To determine the role of the endogenous c-jun gene in apoptosis, we performed mammary epithelial

28 enuates the expression of the closely linked c-jun gene in neurons implicated in centrally mediated h

29 n is needed for the expression of the GR and c-jun genes in F9 cells.

30 inal kinase/stress-activated protein kinase, c-jun gene induction, activation of caspase-3/CPP32-like

31 deacetylase 1 (HDAC1) and recruits it to the c-Jun gene promoter, resulting in an inhibition of histo

32 nal level by its direct association with the c-jun gene promoter.

33 The expression of the endogenous c-jun gene, regulated through a promoter-proximal AP-1-b

34 The c-jun gene regulates cellular proliferation and apoptosi

35 immunodeficiency virus (HIV), and the human c-Jun gene's enhancer (CJE), an element newly identified

36 tants blocked EGF induction of a transfected c-jun gene suggesting that MSK1 or a similar family memb

37 ympathetic neurons of mice carrying a mutant c-Jun gene that lacks Ser63/Ser73 phosphorylation sites

38 made use of null mutations in the c-fos and c-jun genes that were produced by gene targeting to inve

39 of histone H3 associated with the c-fos and c-jun genes through an ERK-dependent pathway.

40 n of MKK1/2-Erk1/2 signaling and by blocking c-Jun gene transcription via inactivation of MKK4-JNK1/2

41 ria and the endoplasmic reticulum as well as c-jun gene transcription.

42 consequence of MEF2C activation is increased c-jun gene transcription.

43 embryonic lethality, somatic deletion of the c-jun gene was conducted using floxed c-jun (c-jun(f/f))

44 by targeting the excision of the endogenous c-jun gene within the mouse mammary epithelium, we have

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