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1 ll lymphoma by insertional activation of the c-myb gene.
2 ranscription in the 3' ends of the pim-1 and c-myb genes.
3 emise of mice with targeted deletions of the c-myb gene and incomplete of knowledge about c-myb's exp
4 tes in the 5'-flanking sequence of the human c-myb gene, and we found that the positive regulators di
5 biological phenotypes produced by A-myb and c-myb genes are mediated by their COOH-terminal domains.
6 ice frequently up-regulate expression of the c-myb gene, but it is unclear whether this is a cause or
7 mutant embryos that carry a deletion of the c-myb gene demonstrates that c-myb is not required for p
12 , revealed an increase in endogenous NmU and c-myb gene expression at day 8 and a 16% expansion of ea
15 nal activation domain in the ancestral A-Myb/c-Myb gene generated from the initial duplication of an
18 nal assays demonstrated that deletion of the c-myb gene leads to a slightly reduced proliferative cap
21 ce analysis of the 5' flanking region of the c-myb gene revealed a consensus PU box at position +16 t
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