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1 tylase inhibitors, and overexpression of the c-Myc proto-oncogene.
2 from PAF1 complex-mediated inhibition of the c-myc proto-oncogene.
3 iption complex paused at the promoter of the c-myc proto-oncogene.
4 ed within the chromosomal band 8q24 near the c-myc proto-oncogene.
5 ression limits the tumorigenic effect of the c-myc proto-oncogene.
6 vo and in vitro decay of mRNA encoded by the c-myc proto-oncogene.
7 ing integration of LTR sequences next to the c-myc proto-oncogene.
8 4 also showed a proviral insertion near the c-myc proto-oncogene.
9 s that require the products of the c-ras and c-myc proto-oncogenes.
12 ickens after proviral integration within the c-myc proto-oncogene and induces erythroblastosis after
24 is a sexual dimorphism in expression of the c-myc proto-oncogene in intact and/or damaged rat caroti
25 and also blunted mitogenic induction of the c-myc proto-oncogene in nontransformed cells with high l
31 le of cell adhesion in the regulation of the c-Myc proto-oncogene is not clear, and the adhesion-depe
32 dMyc, the Drosophila homologue of the human c-myc proto-oncogene, is regulated in vitro and in vivo
33 activation of one target in particular, the c-Myc proto-oncogene, is required for colon cancer patho
36 pleted a biophysical characterization of the c-MYC proto-oncogene P1 promoter quadruplex and its inte
37 ll proliferation and increases levels of the c-myc proto-oncogene product, a DNA-binding protein and
39 by chromosomal rearrangements involving the c-myc proto-oncogene that lead to its inappropriate expr
42 a, involving proviral integration within the c-myc proto-oncogene, while certain strains are genetica
43 hromosomal translocations that juxtapose the c-myc proto-oncogene with regulatory elements of the imm
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