ライフサイエンスコーパス: cAMP response element

1 ynthetic promoters containing CREB1-specific cAMP response elements.

2 , constant GnRH stimulation gave very little cAMP-response element activation but very strong AP-1 ac

3 ents: an inverted CCAAT box element (ICE), a cAMP response element, and a B-cell lymphoma 6 (Bcl6)-bi

4 hich contains evolutionarily conserved AP-1, cAMP-response element, and C/EBP half-sites, in synergis

5 and the subsequent release of ATF3 from its cAMP response element/ATF target site.

6 Here we show that the cAMP response element binding (CREB) coactivator Crtc3 p

7 mRNA expression and secretion by inhibiting cAMP response element binding (CREB) protein and AKT pho

8 n (KID) of the DNA transcriptional activator cAMP response element binding (CREB) protein and the KIX

9 ough the PKA-mediated phosphorylation of the cAMP response element binding (CREB) protein and via the

10 , which decreased two transcription factors, cAMP response element binding and Nurr1, controlling syn

11 in vivo and leads to the phosphorylation of cAMP response element binding and the induction of the e

12 The transcription factors CREB (cAMP response element binding factor), SRF (serum respon

13 itutively activates the transcription factor cAMP response element binding protein (CREB) and CREB ta

14 CF included increased phosphorylation of the cAMP response element binding protein (CREB) and elevate

15 s function via increasing phosphorylation of CAMP response element binding protein (CREB) and PSD95 a

16 we found that activated PERK phosphorylates CAMP response element binding protein (CREB) and PSD95 d

17 ecessary role for two transcription factors, cAMP response element binding protein (CREB) and serum r

18 t) and downstream transcription factors, the cAMP response element binding protein (CREB) and signal

19 activating transcription factors such as the cAMP response element binding protein (CREB) and the ser

20 neurons transfected with a dominant-negative cAMP response element binding protein (CREB) and was eli

21 nts for IL-1beta and IFN-gamma were bound by cAMP response element binding protein (CREB) and zinc-fi

22 ssociates with the transcriptional regulator cAMP response element binding protein (CREB) in both mou

23 of cellular cAMP and the phosphorylation of cAMP response element binding protein (CREB) in INS-1 ce

24 ing, and its downstream transcription factor cAMP response element binding protein (CREB) in the cont

25 cAMP response element binding protein (CREB) is a key re

26 In particular, the PKC target protein cAMP response element binding protein (CREB) is a major

27 monophosphate (cAMP)-protein kinase A (PKA)-cAMP response element binding protein (CREB) pathway.

28 anization all impair efficient TSH-dependent cAMP response element binding protein (CREB) phosphoryla

29 Furthermore, TDCA significantly increased cAMP response element binding protein (CREB) phosphoryla

30 ate gyrus of the adult hippocampus rely upon cAMP response element binding protein (CREB) signaling f

31 us slices through mechanisms associated with cAMP response element binding protein (CREB) signaling.

32 fying the stimulatory effects of the drug on cAMP response element binding protein (CREB) signalling.

33 ootshock) activates the transcription factor cAMP response element binding protein (CREB) within the

34 te (cGMP) leading to increased levels of the cAMP response element binding protein (CREB), a transcri

35 levels was associated with activation of the cAMP response element binding protein (CREB), an essenti

36 ed binding site for the transcription factor cAMP response element binding protein (CREB), and we dem

37 in striatal MSNs and resultant activation of cAMP response element binding protein (CREB), in rat pri

38 phosphomimetic mutant S386/396E bound to the cAMP response element binding protein (CREB)-binding pro

39 The starvation-inducible coactivator cAMP response element binding protein (CREB)-cAMP-regula

40 /calmodulin kinase-I (CaMKI), which triggers cAMP response element binding protein (CREB)-dependent W

41 This photic resetting involves cAMP response element binding protein (CREB)-mediated up

42 Chronic exposure to addictive drugs enhances cAMP response element binding protein (CREB)-regulated g

43 For example, the cAMP response element binding protein (CREB)-regulated t

44 slocation that generates an unusual chimeric cAMP response element binding protein (CREB)-regulated t

45 eptors, adenyl cyclase, protein kinase A and cAMP response element binding protein (CREB).

46 the GcgR cDNA restored hepatic GcgR, phospho-cAMP response element binding protein (P-CREB), and phos

47 in acetylated histone H3 (AcH3) and phospho-cAMP response element binding protein (pCREB) associatio

48 rmal rats, riluzole increased phosphorylated cAMP response element binding protein (pCREB) expressing

49 Luman/cAMP response element binding protein 3 is an endoplasmi

50 a (~1,200 pg/mL), but hepatic phosphorylated cAMP response element binding protein and phosphoenolpyr

51 signaling, leading to the phosphorylation of cAMP response element binding protein and, consequently,

52 nscription levels of specific members of the cAMP Response Element Binding protein gene family.

53 lin-dependent protein kinase IV (CaMKIV) and cAMP response element binding protein mediate the Ca(2)(

54 providing the first evidence that enhancing cAMP response element binding protein signaling can alle

55 we reveal a previously unappreciated role of cAMP response element binding protein signaling in RTT p

56 eting human Apolipoprotien C3 (Apoc3), CREB (cAMP Response Element Binding Protein) Regulated Transcr

57 morphine suppressed binding of phospho-CREB (cAMP response element binding protein) to Bdnf promoters

58 cAMP-regulated phosphoprotein-32) and CREB (cAMP response element binding protein), and locomotor ac

59 roaches that boost memories by targeting the cAMP response element binding protein-CCAAT enhancer bin

60 se kinase-3, ribosomal S6 kinase, c-Jun, and cAMP response element binding protein.

61 ERK1/2, calmodulin-dependent kinase II, and cAMP response element binding proteins; and such phospho

62 hosphorylated AKT (p-AKT) and phosphorylated cAMP response element binding-factor (p-CREB) than simil

63 ing pathway culminating in activation of the cAMP response element binding.

64 es PKA- and p38-dependent phosphorylation of cAMP response-element binding and activating transcripti

65 he cAMP pathway and accompanied by increased cAMP response-element binding protein (CREB) activity.

66 fferential regulation of CRH relies upon the cAMP response-element binding protein coactivator CRTC2,

67 ption factor cyclic adenosine monophosphate (cAMP) response element binding protein (CREB) and the ne

68 F1 to the GH promoter along with cyclic AMP (cAMP) response element binding protein (CREB) binding pr

69 e demonstrated that silencing of cyclic AMP (cAMP) response element binding protein 3-like 1 (CREB3L1

70 gene expression after learning requires the cAMP-response element binding (CREB) interaction domain

71 cAMP-response element binding protein (CREB) is a nuclea

72 intracellular stores, causing an increase in cAMP-response element binding protein (CREB) phosphoryla

73 itical role in memory formation, such as the cAMP-response element binding protein (CREB), have been

74 tes the formation of Abeta by activating the cAMP-response element binding protein (CREB), which in t

75 ed a novel intracellular signaling molecule, cAMP-response element binding protein (CREB), which serv

76 phokinase A (PKA)-mediated activation of the cAMP-response element binding protein (CREB1) to transcr

77 activity toward protein substrates, such as cAMP-response element binding protein and cardiac tropon

78 itute for WT-PKD1 as an in vivo activator of cAMP-response element binding protein and ERK phosphoryl

79 ound an enduring reduction in phosphorylated cAMP-response element binding protein levels in the NAcS

80 e activity of the transcription factor CREB (cAMP-response element binding protein).

81 ria, cytochrome oxidase subunit 4 isoform 1, cAMP-response element binding, p38 MAPK and adenosine mo

82 the induction of a persistent activation of cAMP-response element binding-protein (CREB) and C/EBPbe

83 ults in decreased cAMP signaling and reduced cAMP-response-element binding protein (CREB) activation,

84 observed a significant increase in levels of cAMP response element- binding (CREB) transcripts in the

85 tigated the role of Transducers of Regulated cAMP response element-binding (CREB) protein activity (T

86 IL-10 production via the phosphorylation of cAMP response element-binding (CREB) protein on the IL-1

87 ower, behavioral-stimulant doses enhance the cAMP response element-binding (CREB) signaling system, w

88 e production, at least in part by increasing cAMP response element-binding (CREB) stability.

89 complex acted as a competitive inhibitor of cAMP response element-binding (CREB) transcription facto

90 n actions with histone acetylation enhancing cAMP response element-binding (CREB)-associated transcri

91 h increased NF-kappaB-mediated and decreased cAMP Response Element-Binding (CREB)-mediated Tax activi

92 ption factor 5 (ATF5) is a member of the ATF/cAMP response element-binding family of transcription fa

93 cells and induced Treg expansion through the cAMP response element-binding pathway.

94 PKA-induced cAMP response element-binding phosphorylation is normal

95 rophage-derived IL-10 resulted in epithelial cAMP response element-binding protein (CREB) activation

96 to increased Erk1/2 MAP-kinase signaling and cAMP response element-binding protein (CREB) activation

97 ha) activation by free fatty acid (FFA), and cAMP response element-binding protein (CREB) activation

98 trigger the cAMP-mediated phosphorylation of cAMP response element-binding protein (Creb) and dephosp

99 kers of antidepressant action: activation of cAMP response element-binding protein (CREB) and inducti

100 cAMP response element-binding protein (CREB) and nuclear

101 TX phosphorylated the cAMP response element-binding protein (CREB) and recruit

102 genic program through the phosphorylation of cAMP response element-binding protein (CREB) and the dep

103 he activated PKA subsequently phosphorylated cAMP response element-binding protein (CREB) at Ser-133

104 For example, deficits in cAMP response element-binding protein (CREB) binding pro

105 R) and the fasting transcriptional activator cAMP response element-binding protein (CREB) coordinatel

106 t CaMKK2/CaMKIV-dependent phosphorylation of cAMP response element-binding protein (CREB) correlates

107 ar mechanisms of transcriptional control via cAMP response element-binding protein (CREB) during indu

108 The transcription factor cAMP response element-binding protein (CREB) has been im

109 dulin-dependent protein kinase (CaMK) II and cAMP response element-binding protein (CREB) in cultured

110 demonstrated that E2 rapidly phosphorylates cAMP response element-binding protein (CREB) in GnRH neu

111 ssion of FTO delays the dephosphorylation of cAMP response element-binding protein (CREB) in human ne

112 reasing activity of the transcription factor cAMP response element-binding protein (CREB) in young ad

113 The cAMP response element-binding protein (CREB) is a highly

114 The cAMP response element-binding protein (CREB) is a nuclea

115 Phosphorylation of cAMP response element-binding protein (CREB) is also sig

116 The activity-regulated transcription factor cAMP response element-binding protein (CREB) is an essen

117 The transcription factor cAMP response element-binding protein (CREB) is required

118 nd that the p90 ribosomal S6 kinase 2 (RSK2)-cAMP response element-binding protein (CREB) pathway is

119 ic administration of rolipram also increased cAMP response element-binding protein (CREB) phosphoryla

120 -WT, exhibited higher basal PKA activity and cAMP response element-binding protein (CREB) phosphoryla

121 hallenge in drug-naive mice increases Ser133 cAMP response element-binding protein (CREB) phosphoryla

122 Low picomolar PregS similarly activates cAMP response element-binding protein (CREB) phosphoryla

123 blasts have been identified and culminate in cAMP response element-binding protein (CREB) regulation

124 n part via the PKA-mediated induction of the cAMP response element-binding protein (CREB) signaling p

125 IP) and whole-genome sequencing) to identify cAMP response element-binding protein (CREB) targets fol

126 t uncovered that HDAC2 is a direct target of cAMP response element-binding protein (CREB) that is act

127 mitogen-activated protein kinase (MAPK), and cAMP response element-binding protein (CREB) transcripti

128 We previously demonstrated that the cAMP response element-binding protein (CREB) transcripti

129 endent protein kinase II (CaMKII) and Ca(2+)/cAMP response element-binding protein (CREB) transcripti

130 ression of targets of protein kinase A (PKA)-cAMP response element-binding protein (CREB), a pathway

131 Here, we use outbred rats to investigate if cAMP response element-binding protein (CREB), a transcri

132 on of Arc and phosphorylation of cofilin and cAMP response element-binding protein (CREB), a transcri

133 n cortisol production and phosphorylation of cAMP response element-binding protein (CREB), a transcri

134 inhibition did not affect phosphorylation of cAMP response element-binding protein (CREB), a well-est

135 ntisense to mRNA for a transcription factor, cAMP response element-binding protein (CREB), and by an

136 G1 also enhanced cAMP response element-binding protein (CREB), as well as

137 ased mRNA of ERK2 and its downstream targets cAMP response element-binding protein (CREB), BDNF, c-Fo

138 ylation and activity of transcription factor cAMP response element-binding protein (CREB), binding si

139 HCV increased activation of cAMP response element-binding protein (CREB), CCAAT/enha

140 enhanced phosphorylation of a CaN substrate, cAMP response element-binding protein (CREB), in the bra

141 Phosphorylated cAMP response element-binding protein (CREB), nuclear NF

142 egulation of prosurvival signaling (i.e. the cAMP response element-binding protein (CREB)-Bdnf cascad

143 Here, we show that high glucose induced cAMP response element-binding protein (CREB)-binding pro

144 ssion restored normal ryanodine receptor and cAMP response element-binding protein (CREB)-dependent g

145 ffects of HT-0712 on memory formation and on cAMP response element-binding protein (CREB)-regulated g

146 duction by inducing the dephosphorylation of cAMP response element-binding protein (CREB)-regulated t

147 ription factor mediator of Ca(2+)-signals is cAMP response element-binding protein (CREB).

148 downstream cascade adenylyl cyclase-cAMP-PKA-cAMP response element-binding protein (CREB).

149 n of ID1 by prostaglandin E2 was mediated by cAMP response element-binding protein (CREB).

150 synthases and reduced phosphorylation of the cAMP response element-binding protein (CREB).

151 SCO rapidly enhanced the phosphorylation of cAMP response element-binding protein (CREB).

152 ippocampi had lower levels of phosphorylated cAMP response element-binding protein (pCREB), an activi

153 phosphorylation of the transcription factor cAMP response element-binding protein (pCREB).

154 hippocampal neurogenesis and phosphorylated cAMP response element-binding protein (pCREB).

155 suggests that the transcriptional activator cAMP response element-binding protein 1 (CREB1) is impor

156 ate (cAMP) to synthetic promoters containing cAMP response element-binding protein 1(CREB1)-specific

157 y decreasing MCU's transcriptional regulator cAMP response element-binding protein 1.

158 of the downstream transcriptional repressor cAMP response element-binding protein 2 in SNs.

159 inhibits proteolytic processing of CREB3L1 (cAMP response element-binding protein 3-like 1), a membr

160 ing pathway and required for PACAP-dependent cAMP response element-binding protein activation and neu

161 ZIP12 knockdown reduces cAMP response element-binding protein activation and pho

162 Constitutive cAMP response element-binding protein activation restore

163 also increased the nuclear translocation of cAMP response element-binding protein and CCAAT/enhancer

164 ells in part by inhibiting the activation of cAMP response element-binding protein and expression of

165 cer-binding protein beta was required, while cAMP response element-binding protein and signal transdu

166 PKG1 and cAMP response element-binding protein are involved in th

167 The protein acetyltransferases p300 and cAMP response element-binding protein binding protein (C

168 e transcriptional coactivators p300 and p300/cAMP response element-binding protein binding protein-as

169 33 (miR-433) inhibition of expression of the cAMP response element-binding protein CREB1 represses he

170 They also showed reduced phosphorylated cAMP response element-binding protein expression within

171 sing a repressor of the transcription factor cAMP response element-binding protein or a calcium/calmo

172 ndent of the canonical cAMP/Protein Kinase A/cAMP response element-binding protein pathway downstream

173 s --> adenylate cyclase --> cAMP --> PKA --> cAMP response element-binding protein pathway mediating

174 pe, we administered rolipram to activate the cAMP response element-binding protein pathway, which led

175 n of the cAMP-dependent protein kinase (PKA)/cAMP response element-binding protein pathway.

176 Consistently, leptin promotes cAMP response element-binding protein phosphorylation in

177 ments of hippocampal synaptic plasticity and cAMP response element-binding protein phosphorylation.

178 and pEC50 of ACEA-induced Galphas-dependent cAMP response element-binding protein phosphorylation.

179 Consequently, the cAMP-->PKA-->cAMP response element-binding protein signaling axis is

180 phosphorylation of the transcription factor "cAMP response element-binding protein" (CREB) are also a

181 c leucine zipper domain (bZip) protein CREB (cAMP response element-binding protein) as a key effector

182 cient neuronal cell lines have reduced CREB (cAMP response element-binding protein) expression and in

183 tream target of ERK1/2, pCREB [phospho-CREB (cAMP response element-binding protein)].

184 oxygen species production, and activation of cAMP response element-binding protein, a critical transc

185 activation was followed by downregulation of cAMP response element-binding protein, and LTP impairmen

186 y between NF-kappaB (cRel: p50), C/EBPdelta, cAMP response element-binding protein, and nuclear facto

187 n factors, including activator protein 1 and cAMP response element-binding protein, both of which wer

188 ciated with a reduction in the activation of cAMP response element-binding protein, but not the activ

189 sponse element-binding protein (ChREBP), and cAMP response element-binding protein, hepatocyte specif

190 rylation status of their respective targets, cAMP response element-binding protein, p38, and extracel

191 ecretase activates the transcription factor, cAMP response element-binding protein, regulating miR-21

192 ads to induction of the transcription factor cAMP response element-binding protein-3-like-2 (CREB3L2)

193 RORalpha recruited cAMP response element-binding protein-binding protein (C

194 ylation is diminished, thereby enabling p300/cAMP response element-binding protein-binding protein to

195 acid receptor alpha, and HATs (p300 and p300/cAMP response element-binding protein-binding protein-as

196 ediated ERalpha protein methylation and p300/cAMP response element-binding protein-binding protein-de

197 llular outputs: p38-dependent growth arrest, cAMP response element-binding protein-dependent cell sur

198 Inhibition of cAMP response element-binding protein-dependent signalin

199 kinase 2 (SIK2) is an important regulator of cAMP response element-binding protein-mediated gene expr

200 active oxygen species, and the activation of cAMP response element-binding protein.

201 shown by reduction in phosphorylation of the cAMP response element-binding protein.

202 on of a key histone acetyltransferase (HAT), cAMP response element-binding protein.

203 phosphorylation of the transcription factor cAMP response element-binding protein.

204 ivation of a pathway involving Akt1/Akt2 and cAMP response element-binding protein.

205 ated by the transcription factors CREB/CREM (cAMP response element-binding protein/modulator) is link

206 that synaptopodin functions independently of cAMP response element-binding.

207 m channels couple membrane depolarization to cAMP response-element-binding protein (CREB)-dependent t

208 factor (ATF)-adenosine 3',5'-monophosphate (cAMP) response element-binding protein (CREB) family tra

209 l-regulating kinase 1 (ASK1) and cyclic AMP (cAMP) response element-binding protein (CREB).

210 we found that PKA-induced phosphorylation of cAMP-response element-binding protein ((P)CREB) and EPAC

211 phosphorylation of the transcription factor cAMP-response element-binding protein (CREB) as well as

212 he murine RGS2 promoter that is critical for cAMP-response element-binding protein (CREB) binding and

213 A cAMP-response element-binding protein (CREB) binding seq

214 AT/enhancer-binding protein (C/EBPbeta), and cAMP-response element-binding protein (CREB) have been i

215 inase A activity, thus reducing both phospho-cAMP-response element-binding protein (CREB) levels and

216 to G transition under basal/nicotine-induced/cAMP-response element-binding protein (CREB) overexpress

217 -3 expression via the protein kinase C (PKC)-cAMP-response element-binding protein (CREB) pathway.

218 tracellular cAMP production was impaired and cAMP-response element-binding protein (CREB) phosphoryla

219 hrough a cAMP-dependent binding of FGFR1 and cAMP-response element-binding protein (CREB) to a conser

220 nstrate that RNS60 induced the activation of cAMP-response element-binding protein (CREB) via the PI

221 The activation of cAMP-response element-binding protein (CREB), but not NF

222 attenuating the expression of phosphorylated cAMP-response element-binding protein (CREB), c-Fos, and

223 e endogenous Galphas promoter is occupied by cAMP-response element-binding protein (CREB), Egr-1, and

224 However, phosphorylation of cAMP-response element-binding protein (CREB), the cAMP-r

225 ost notable throughout was the high level of cAMP-response element-binding protein (CREB)-response el

226 cAMP-response element-binding protein 1 (CREB1) has been

227 nalysis reveals C1q-activated phosphorylated cAMP-response element-binding protein and AP-1, two tran

228 te (R(P)-cAMPS)) decreased butaprost-induced cAMP-response element-binding protein and ERK activation

229 eir key transcription factors phosphorylated cAMP-response element-binding protein and forkhead box O

230 rgets with functions in adipogenesis such as cAMP-response element-binding protein and FOXO1; however

231 regulates hepatic glucose production through cAMP-response element-binding protein co-activators, we

232 protein, which induced the expression of the cAMP-response element-binding protein family repressor c

233 sured by phospho-GSK3beta (p-GSK3beta) and p-cAMP-response element-binding protein formation.

234 n neurons, and in the efficient signaling to cAMP-response element-binding protein in neurons.

235 ed inactivation of the cAMP/protein kinase A/cAMP-response element-binding protein signaling pathway,

236 was regulated by transcription factor CREB (cAMP-response element-binding protein) and silencing of

237 ta-mediated increases in PKA activity, CREB (cAMP-response element-binding protein) phosphorylation,

238 activity and elevated STMN Ser-63 and CREB (cAMP-response element-binding protein) Ser-133 phosphory

239 ceptor-gamma accompanied with ATF2 and CREB (cAMP-response element-binding protein) was enhanced acro

240 nal activators, Tax and phosphorylated CREB (cAMP-response element-binding protein), recruited the p3

241 s required for activation of SREBP and CREB (cAMP-response element-binding protein)/ATF family transc

242 Furthermore, cAMP-response element-binding protein, as a downstream t

243 Silencing of cAMP-response element-binding protein, LRP1B or GPR6 exp

244 phosphorylation of the transcription factor cAMP-response element-binding protein, which induced the

245 monstrate that the lysine acetyltransferases cAMP-response element-binding protein-binding protein (C

246 Overexpression of the p300/cAMP-response element-binding protein-binding protein (C

247 n and mouse islet alpha cells by GS/cAMP/PKA/cAMP-response element-binding protein-dependent activati

248 t not PDE4B ablation significantly prolonged cAMP-response element-binding protein-mediated transcrip

249 y MSK1 and -2 and their downstream effectors cAMP-response element-binding protein/ATF1 as mediators

250 e classical nuclear calcium-CaMKIV-CREB/CBP (cAMP-response element-binding protein/CREB-binding prote

251 orylation at Thr-200 and its activity toward cAMP-response element-binding transcription factor.

252 racellular cAMP levels and the activation of cAMP-response element-binding, a protein kinase A downst

253 ad determined that nuclear sAC activates the cAMP-response-element-binding (CREB) transcription facto

254 lin-dependent protein kinase type (CAMK)-IV, cAMP-response-element-binding protein (CREB) and brain-d

255 ed CREB with consequent augmented binding on cAMP response element consensus sequence on peroxisome p

256 ng PPARgamma target genes and ERK-regulated, cAMP response element-containing target genes.

257 lement-binding protein (CREB) to a conserved cAMP response element (CRE) contiguous with the NFAT bin

258 tergic selection gene tlx3 through a variant cAMP response element (CRE) in its promoter.

259 d transcriptional activation mediated by the cAMP response element (CRE) in reporter gene studies (10

260 differentially expressed genes containing a cAMP response element (CRE) in their proximal promoter,

261 ly through the methylation of a CpG within a cAMP response element (CRE) motif defined by its promote

262 promoter region contains putative conserved cAMP response element (CRE) regions, which we found can

263 t AC inhibitors in HEK293 cells expressing a cAMP response element (CRE) reporter gene, and MDL-12,33

264 ER stress or ATF4 coexpression: the -267 ATF/cAMP response element (CRE) site and a novel -248 ATF/CR

265 A newly identified cAMP response element (CRE) site on the SYK promoter was

266 found that the synapsin promoter contained a cAMP response element (CRE), raising the possibility tha

267 The cAMP response element (CRE)-binding protein (CREB) is a

268 oter of human GLUT3 contains three potential cAMP response element (CRE)-like elements, CRE1, CRE2 an

269 activated T cells (NFAT) and the cyclic AMP (cAMP) response element (CRE) modulate KSHV-mediated tran

270 Moreover, we identified a conserved cAMP-response element (CRE) in the murine RGS2 promoter

271 cantly inhibited when either the AP-1 or the cAMP-response element (CRE)-like sequence (TGCGTCA, -752

272 or the function of a proximal renin promoter cAMP-response element (CRE).

273 r binding sites for either CREB (cyclic AMP [cAMP] response element [CRE]) or NF-kappaB (kappaB) part

274 eceptor binding elements [RARE], cyclic AMP [cAMP] response elements [CRE], NF-kappaB binding sites [

275 ack binding sites for RA-response element or cAMP-response element (CREB) in their promoter regions.

276 that inflammatory mediators use to activate cAMP response elements (CREs) are poorly understood.

277 ffinity on palindromic and variant half-site cAMP response elements (CREs).

278 Two functional cAMP-response elements (CREs) were identified in the nmn

279 transcriptional control by the c-Jun-binding cAMP response element in the GADD34 gene promoter and po

280 (PGC-1alpha) and involved activation of the cAMP response element in the PEPCK promoter.

281 porter gene constructs revealed a functional cAMP response element in the proximal promoter of Lrrtm2

282 19 gene transcription through binding to the cAMP-response element in the p19 promoter.

283 ockdown of CREB, the presence of a consensus cAMP-response element in the promoter of CNTF, and the r

284 binding of phosphorylated CREB1 and ATF2 to cAMP-response element-like sites was significantly incre

285 gical function in reproduction, we used Cre (cAMP response element)/LoxP technology to generate GnRH

286 The ICE and the cAMP response element mediate activation, and the Bcl6 s

287 The transcriptional repressor cAMP response element modulator (CREM) alpha has importa

288 EB knock-outs (KOs)] with and without global cAMP response element modulator (CREM) deletion (global-

289 s a transcriptional repressor isoform of the cAMP response element modulator (CREM).

290 Transcription factor cAMP response element modulator (CREM)alpha contributes

291 cription through decreased activation of the cAMP response element modulator alpha (CREM-alpha) and r

292 Upregulation of the CREB-family protein cAMP response-element modulator (CREM) was observed afte

293 report the critical role of ventral striatal cAMP-response element modulator (CREM) in mediating impu

294 In turn, cAMP-response element modulator was found to associate w

295 nse element-binding protein family repressor cAMP-response element modulator.

296 r (DAF) promoter activity via binding to the cAMP response element, mutation of which attenuated prom

297 transcriptional level, both a cAMP-dependent cAMP-response element reporter and a DAG/calcium-depende

298 ene expression through the binding to tandem cAMP-response element sites in the proximal promoter reg

299 39 promoter although not when the promoter's cAMP-response element sites were mutated.

300 f3 encode transcription factors that contain cAMP response elements, suggesting that the methylation