ライフサイエンスコーパス: cAMP response element-binding protein

1 al, decreased apoptosis, and activated CREB (cAMP response element-binding protein).

2 e activity of the transcription factor CREB (cAMP-response element binding protein).

3 g other factors such as C/EBPalpha and CREB (cAMP-response element-binding protein).

4 tream target of ERK1/2, pCREB [phospho-CREB (cAMP response element-binding protein)].

5 se kinase-3, ribosomal S6 kinase, c-Jun, and cAMP response element binding protein.

6 shown by reduction in phosphorylation of the cAMP response element-binding protein.

7 on of a key histone acetyltransferase (HAT), cAMP response element-binding protein.

8 phosphorylation of the transcription factor cAMP response element-binding protein.

9 induced the activity of transcription factor cAMP response element-binding protein.

10 ivation of a pathway involving Akt1/Akt2 and cAMP response element-binding protein.

11 active oxygen species, and the activation of cAMP response element-binding protein.

12 kinase (ERK)1/2 and the transcription factor cAMP-response element-binding protein.

13 The cAMP response element binding protein 1 (CREB1) and acti

14 suggests that the transcriptional activator cAMP response element-binding protein 1 (CREB1) is impor

15 ate (cAMP) to synthetic promoters containing cAMP response element-binding protein 1(CREB1)-specific

16 y decreasing MCU's transcriptional regulator cAMP response element-binding protein 1.

17 cAMP-response element-binding protein 1 (CREB1) has been

18 ian locomoter output cycles kaput) and CREB (cAMP-response element-binding protein-1) activated gga-m

19 of the downstream transcriptional repressor cAMP response element-binding protein 2 in SNs.

20 Luman/cAMP response element binding protein 3 is an endoplasmi

21 inhibits proteolytic processing of CREB3L1 (cAMP response element-binding protein 3-like 1), a membr

22 e demonstrated that silencing of cyclic AMP (cAMP) response element binding protein 3-like 1 (CREB3L1

23 ads to induction of the transcription factor cAMP response element-binding protein-3-like-2 (CREB3L2)

24 oxygen species production, and activation of cAMP response element-binding protein, a critical transc

25 -binding protein and transducer of regulated cAMP-response-element-binding proteins actions on the PG

26 We studied the effects of cAMP response element binding protein/activating transcr

27 ing pathway and required for PACAP-dependent cAMP response element-binding protein activation and neu

28 ZIP12 knockdown reduces cAMP response element-binding protein activation and pho

29 Constitutive cAMP response element-binding protein activation restore

30 a (~1,200 pg/mL), but hepatic phosphorylated cAMP response element binding protein and phosphoenolpyr

31 signaling, leading to the phosphorylation of cAMP response element binding protein and, consequently,

32 also increased the nuclear translocation of cAMP response element-binding protein and CCAAT/enhancer

33 ells in part by inhibiting the activation of cAMP response element-binding protein and expression of

34 cer-binding protein beta was required, while cAMP response element-binding protein and signal transdu

35 activity toward protein substrates, such as cAMP-response element binding protein and cardiac tropon

36 itute for WT-PKD1 as an in vivo activator of cAMP-response element binding protein and ERK phosphoryl

37 nalysis reveals C1q-activated phosphorylated cAMP-response element-binding protein and AP-1, two tran

38 te (R(P)-cAMPS)) decreased butaprost-induced cAMP-response element-binding protein and ERK activation

39 eir key transcription factors phosphorylated cAMP-response element-binding protein and forkhead box O

40 rgets with functions in adipogenesis such as cAMP-response element-binding protein and FOXO1; however

41 ellular calcium, which causes an increase in cAMP-response-element-binding protein and transducer of

42 was regulated by transcription factor CREB (cAMP-response element-binding protein) and silencing of

43 cAMP-regulated phosphoprotein-32) and CREB (cAMP response element binding protein), and locomotor ac

44 activation was followed by downregulation of cAMP response element-binding protein, and LTP impairmen

45 y between NF-kappaB (cRel: p50), C/EBPdelta, cAMP response element-binding protein, and nuclear facto

46 ERK1/2, calmodulin-dependent kinase II, and cAMP response element binding proteins; and such phospho

47 PKG1 and cAMP response element-binding protein are involved in th

48 c leucine zipper domain (bZip) protein CREB (cAMP response element-binding protein) as a key effector

49 Furthermore, cAMP-response element-binding protein, as a downstream t

50 s required for activation of SREBP and CREB (cAMP-response element-binding protein)/ATF family transc

51 S1P cleaves and activates cAMP response element binding protein/ATF transcription

52 y MSK1 and -2 and their downstream effectors cAMP-response element-binding protein/ATF1 as mediators

53 The protein acetyltransferases p300 and cAMP response element-binding protein binding protein (C

54 e transcriptional coactivators p300 and p300/cAMP response element-binding protein binding protein-as

55 ive histone methylation and in phospho-CREB (cAMP response element-binding protein) binding, in the N

56 RORalpha recruited cAMP response element-binding protein-binding protein (C

57 ylation is diminished, thereby enabling p300/cAMP response element-binding protein-binding protein to

58 airs its interaction with acetyltransferase, cAMP response element-binding protein-binding protein, w

59 acid receptor alpha, and HATs (p300 and p300/cAMP response element-binding protein-binding protein-as

60 ediated ERalpha protein methylation and p300/cAMP response element-binding protein-binding protein-de

61 ioxidant response element and to coactivator cAMP response element-binding protein-binding protein/p3

62 ression of ZO-1 by JunD was mediated through cAMP response element-binding protein-binding site withi

63 monstrate that the lysine acetyltransferases cAMP-response element-binding protein-binding protein (C

64 Overexpression of the p300/cAMP-response element-binding protein-binding protein (C

65 tor coactivator family coactivators and p300/cAMP-response element-binding protein-binding protein.

66 n factors, including activator protein 1 and cAMP response element-binding protein, both of which wer

67 ciated with a reduction in the activation of cAMP response element-binding protein, but not the activ

68 I inhibited cGMP-mediated phosphorylation of cAMP response element-binding protein, cAMP response ele

69 roaches that boost memories by targeting the cAMP response element binding protein-CCAAT enhancer bin

70 regulates hepatic glucose production through cAMP-response element-binding protein co-activators, we

71 fferential regulation of CRH relies upon the cAMP response-element binding protein coactivator CRTC2,

72 itutively activates the transcription factor cAMP response element binding protein (CREB) and CREB ta

73 CF included increased phosphorylation of the cAMP response element binding protein (CREB) and elevate

74 indicate that HBZ protein can interact with cAMP response element binding protein (CREB) and Jun fam

75 s function via increasing phosphorylation of CAMP response element binding protein (CREB) and PSD95 a

76 we found that activated PERK phosphorylates CAMP response element binding protein (CREB) and PSD95 d

77 ecessary role for two transcription factors, cAMP response element binding protein (CREB) and serum r

78 t) and downstream transcription factors, the cAMP response element binding protein (CREB) and signal

79 activating transcription factors such as the cAMP response element binding protein (CREB) and the ser

80 neurons transfected with a dominant-negative cAMP response element binding protein (CREB) and was eli

81 nts for IL-1beta and IFN-gamma were bound by cAMP response element binding protein (CREB) and zinc-fi

82 h utilizes two families of coactivators, the cAMP response element binding protein (CREB) binding pro

83 Activated cAMP response element binding protein (CREB) has been st

84 ssociates with the transcriptional regulator cAMP response element binding protein (CREB) in both mou

85 of cellular cAMP and the phosphorylation of cAMP response element binding protein (CREB) in INS-1 ce

86 ing, and its downstream transcription factor cAMP response element binding protein (CREB) in the cont

87 cAMP response element binding protein (CREB) is a key re

88 In particular, the PKC target protein cAMP response element binding protein (CREB) is a major

89 AngII stimulated phosphorylation of cAMP response element binding protein (CREB) on Ser-133

90 monophosphate (cAMP)-protein kinase A (PKA)-cAMP response element binding protein (CREB) pathway.

91 anization all impair efficient TSH-dependent cAMP response element binding protein (CREB) phosphoryla

92 cAMP response element binding protein (CREB) phosphoryla

93 Furthermore, TDCA significantly increased cAMP response element binding protein (CREB) phosphoryla

94 ate gyrus of the adult hippocampus rely upon cAMP response element binding protein (CREB) signaling f

95 us slices through mechanisms associated with cAMP response element binding protein (CREB) signaling.

96 fying the stimulatory effects of the drug on cAMP response element binding protein (CREB) signalling.

97 genic potential of EGb 761 and its effect on cAMP response element binding protein (CREB) were examin

98 ootshock) activates the transcription factor cAMP response element binding protein (CREB) within the

99 altered function of the transcription factor cAMP response element binding protein (CREB) within the

100 ils display increased phosphorylation of the cAMP response element binding protein (CREB), a major tr

101 te (cGMP) leading to increased levels of the cAMP response element binding protein (CREB), a transcri

102 levels was associated with activation of the cAMP response element binding protein (CREB), an essenti

103 displayed attenuated phosphorylation of ERK, cAMP response element binding protein (CREB), and dopami

104 ed binding site for the transcription factor cAMP response element binding protein (CREB), and we dem

105 in striatal MSNs and resultant activation of cAMP response element binding protein (CREB), in rat pri

106 phosphomimetic mutant S386/396E bound to the cAMP response element binding protein (CREB)-binding pro

107 ptor rho coactivator 1alpha (PGC-1alpha) and cAMP response element binding protein (CREB)-binding pro

108 The starvation-inducible coactivator cAMP response element binding protein (CREB)-cAMP-regula

109 /calmodulin kinase-I (CaMKI), which triggers cAMP response element binding protein (CREB)-dependent W

110 This photic resetting involves cAMP response element binding protein (CREB)-mediated up

111 Chronic exposure to addictive drugs enhances cAMP response element binding protein (CREB)-regulated g

112 For example, the cAMP response element binding protein (CREB)-regulated t

113 slocation that generates an unusual chimeric cAMP response element binding protein (CREB)-regulated t

114 ) response element, which complexes with the cAMP response element binding protein (CREB).

115 eptors, adenyl cyclase, protein kinase A and cAMP response element binding protein (CREB).

116 rophage-derived IL-10 resulted in epithelial cAMP response element-binding protein (CREB) activation

117 to increased Erk1/2 MAP-kinase signaling and cAMP response element-binding protein (CREB) activation

118 ha) activation by free fatty acid (FFA), and cAMP response element-binding protein (CREB) activation

119 y- and anhedonia-like symptoms and decreased cAMP response element-binding protein (CREB) activity in

120 extracellular signal-regulated kinase (ERK)/cAMP response element-binding protein (CREB) and Akt/nuc

121 trigger the cAMP-mediated phosphorylation of cAMP response element-binding protein (Creb) and dephosp

122 phosphorylation of the transcription factor, cAMP response element-binding protein (CREB) and increas

123 kers of antidepressant action: activation of cAMP response element-binding protein (CREB) and inducti

124 cAMP response element-binding protein (CREB) and nuclear

125 s process occurred through the activation of cAMP response element-binding protein (CREB) and peroxis

126 TX phosphorylated the cAMP response element-binding protein (CREB) and recruit

127 genic program through the phosphorylation of cAMP response element-binding protein (CREB) and the dep

128 tors is mediated by the transcription factor cAMP response element-binding protein (CREB) and the rec

129 he activated PKA subsequently phosphorylated cAMP response element-binding protein (CREB) at Ser-133

130 For example, deficits in cAMP response element-binding protein (CREB) binding pro

131 Activation of glial cAMP response element-binding protein (CREB) by MBP-prim

132 R) and the fasting transcriptional activator cAMP response element-binding protein (CREB) coordinatel

133 t CaMKK2/CaMKIV-dependent phosphorylation of cAMP response element-binding protein (CREB) correlates

134 ar mechanisms of transcriptional control via cAMP response element-binding protein (CREB) during indu

135 s a leucine-zipper-containing protein of the cAMP response element-binding protein (CREB) family.

136 cAMP response element-binding protein (CREB) has been im

137 The transcription factor cAMP response element-binding protein (CREB) has been im

138 dulin-dependent protein kinase (CaMK) II and cAMP response element-binding protein (CREB) in cultured

139 demonstrated that E2 rapidly phosphorylates cAMP response element-binding protein (CREB) in GnRH neu

140 ssion of FTO delays the dephosphorylation of cAMP response element-binding protein (CREB) in human ne

141 reasing activity of the transcription factor cAMP response element-binding protein (CREB) in young ad

142 The cAMP response element-binding protein (CREB) is a highly

143 The cAMP response element-binding protein (CREB) is a nuclea

144 Phosphorylation of cAMP response element-binding protein (CREB) is also sig

145 The activity-regulated transcription factor cAMP response element-binding protein (CREB) is an essen

146 The transcription factor cAMP response element-binding protein (CREB) is required

147 diated signaling proteins, phosphoryation of cAMP response element-binding protein (CREB) kinase, pp9

148 P actions are dependent on activation of the cAMP response element-binding protein (CREB) pathway and

149 nd that the p90 ribosomal S6 kinase 2 (RSK2)-cAMP response element-binding protein (CREB) pathway is

150 ic administration of rolipram also increased cAMP response element-binding protein (CREB) phosphoryla

151 -WT, exhibited higher basal PKA activity and cAMP response element-binding protein (CREB) phosphoryla

152 hallenge in drug-naive mice increases Ser133 cAMP response element-binding protein (CREB) phosphoryla

153 Furthermore, all the PICs increased cAMP response element-binding protein (CREB) phosphoryla

154 Furthermore, increases in ERK1/2 and cAMP response element-binding protein (CREB) phosphoryla

155 1 knock-out mice demonstrate a deficiency in cAMP response element-binding protein (CREB) phosphoryla

156 Low picomolar PregS similarly activates cAMP response element-binding protein (CREB) phosphoryla

157 blasts have been identified and culminate in cAMP response element-binding protein (CREB) regulation

158 Many growth regulatory stimuli promote cAMP response element-binding protein (CREB) Ser(133) ph

159 n part via the PKA-mediated induction of the cAMP response element-binding protein (CREB) signaling p

160 Previously, we reported that expression of cAMP response element-binding protein (CREB) target gene

161 IP) and whole-genome sequencing) to identify cAMP response element-binding protein (CREB) targets fol

162 t uncovered that HDAC2 is a direct target of cAMP response element-binding protein (CREB) that is act

163 We previously demonstrated that the cAMP response element-binding protein (CREB) transcripti

164 endent protein kinase II (CaMKII) and Ca(2+)/cAMP response element-binding protein (CREB) transcripti

165 mitogen-activated protein kinase (MAPK), and cAMP response element-binding protein (CREB) transcripti

166 scription factors of the NFkappaB family and cAMP response element-binding protein (CREB) was assesse

167 The transcription factor cAMP response element-binding protein (CREB) within the

168 ression of targets of protein kinase A (PKA)-cAMP response element-binding protein (CREB), a pathway

169 Here, we use outbred rats to investigate if cAMP response element-binding protein (CREB), a transcri

170 on of Arc and phosphorylation of cofilin and cAMP response element-binding protein (CREB), a transcri

171 n cortisol production and phosphorylation of cAMP response element-binding protein (CREB), a transcri

172 inhibition did not affect phosphorylation of cAMP response element-binding protein (CREB), a well-est

173 ntisense to mRNA for a transcription factor, cAMP response element-binding protein (CREB), and by an

174 llular signal-regulated kinase 1/2 (ERK1/2), cAMP response element-binding protein (CREB), and microt

175 G1 also enhanced cAMP response element-binding protein (CREB), as well as

176 ased mRNA of ERK2 and its downstream targets cAMP response element-binding protein (CREB), BDNF, c-Fo

177 ylation and activity of transcription factor cAMP response element-binding protein (CREB), binding si

178 HCV increased activation of cAMP response element-binding protein (CREB), CCAAT/enha

179 CpG dinucleotides found in cAMP response element-binding protein (CREB), CREB/c-Jun

180 enhanced phosphorylation of a CaN substrate, cAMP response element-binding protein (CREB), in the bra

181 Phosphorylated cAMP response element-binding protein (CREB), nuclear NF

182 egulation of prosurvival signaling (i.e. the cAMP response element-binding protein (CREB)-Bdnf cascad

183 Here, we show that high glucose induced cAMP response element-binding protein (CREB)-binding pro

184 ssion restored normal ryanodine receptor and cAMP response element-binding protein (CREB)-dependent g

185 ffects of HT-0712 on memory formation and on cAMP response element-binding protein (CREB)-regulated g

186 duction by inducing the dephosphorylation of cAMP response element-binding protein (CREB)-regulated t

187 ription factor mediator of Ca(2+)-signals is cAMP response element-binding protein (CREB).

188 n hypoxic adaptation, such as HIF-1alpha and cAMP response element-binding protein (CREB).

189 via activation of the transcription factor, cAMP response element-binding protein (CREB).

190 y and the phosphorylation of MAPK kinase and cAMP response element-binding protein (CREB).

191 downstream cascade adenylyl cyclase-cAMP-PKA-cAMP response element-binding protein (CREB).

192 y and the phosphorylation of MAPK kinase and cAMP response element-binding protein (CREB).

193 Erk-1/2, which led to the activation of the cAMP response element-binding protein (CREB).

194 n of ID1 by prostaglandin E2 was mediated by cAMP response element-binding protein (CREB).

195 synthases and reduced phosphorylation of the cAMP response element-binding protein (CREB).

196 SCO rapidly enhanced the phosphorylation of cAMP response element-binding protein (CREB).

197 he cAMP pathway and accompanied by increased cAMP response-element binding protein (CREB) activity.

198 m channels couple membrane depolarization to cAMP response-element-binding protein (CREB)-dependent t

199 ption factor cyclic adenosine monophosphate (cAMP) response element binding protein (CREB) and the ne

200 F1 to the GH promoter along with cyclic AMP (cAMP) response element binding protein (CREB) binding pr

201 ation of the cyclic adenosine monophosphate (cAMP) response element binding protein (CREB) throughout

202 factor (ATF)-adenosine 3',5'-monophosphate (cAMP) response element-binding protein (CREB) family tra

203 of DNA repair adenosine 3',5'-monophosphate (cAMP) response element-binding protein (CREB)-binding pr

204 l-regulating kinase 1 (ASK1) and cyclic AMP (cAMP) response element-binding protein (CREB).

205 cAMP-response element binding protein (CREB) is a nuclea

206 intracellular stores, causing an increase in cAMP-response element binding protein (CREB) phosphoryla

207 itical role in memory formation, such as the cAMP-response element binding protein (CREB), have been

208 tes the formation of Abeta by activating the cAMP-response element binding protein (CREB), which in t

209 ed a novel intracellular signaling molecule, cAMP-response element binding protein (CREB), which serv

210 the induction of a persistent activation of cAMP-response element binding-protein (CREB) and C/EBPbe

211 phosphorylation of the transcription factor cAMP-response element-binding protein (CREB) as well as

212 he murine RGS2 promoter that is critical for cAMP-response element-binding protein (CREB) binding and

213 A cAMP-response element-binding protein (CREB) binding seq

214 AT/enhancer-binding protein (C/EBPbeta), and cAMP-response element-binding protein (CREB) have been i

215 nt modulator (CREM) and down-regulation of p-cAMP-response element-binding protein (CREB) in activate

216 inase A activity, thus reducing both phospho-cAMP-response element-binding protein (CREB) levels and

217 to G transition under basal/nicotine-induced/cAMP-response element-binding protein (CREB) overexpress

218 -3 expression via the protein kinase C (PKC)-cAMP-response element-binding protein (CREB) pathway.

219 tracellular cAMP production was impaired and cAMP-response element-binding protein (CREB) phosphoryla

220 es by CO(2) gave a corresponding increase in cAMP-response element-binding protein (CREB) phosphoryla

221 The two cell lines differed in cAMP-response element-binding protein (CREB) phosphoryla

222 hrough a cAMP-dependent binding of FGFR1 and cAMP-response element-binding protein (CREB) to a conser

223 nstrate that RNS60 induced the activation of cAMP-response element-binding protein (CREB) via the PI

224 that activation of the transcription factor cAMP-response element-binding protein (CREB), a common m

225 The activation of cAMP-response element-binding protein (CREB), but not NF

226 attenuating the expression of phosphorylated cAMP-response element-binding protein (CREB), c-Fos, and

227 e endogenous Galphas promoter is occupied by cAMP-response element-binding protein (CREB), Egr-1, and

228 However, phosphorylation of cAMP-response element-binding protein (CREB), the cAMP-r

229 ost notable throughout was the high level of cAMP-response element-binding protein (CREB)-response el

230 sociated with overexpression and activity of cAMP-response element-binding protein (CREB).

231 ognized by the cellular transcription factor cAMP-response element-binding protein (CREB).

232 ults in decreased cAMP signaling and reduced cAMP-response-element binding protein (CREB) activation,

233 lin-dependent protein kinase type (CAMK)-IV, cAMP-response-element-binding protein (CREB) and brain-d

234 phosphorylation of the transcription factor "cAMP response element-binding protein" (CREB) are also a

235 e classical nuclear calcium-CaMKIV-CREB/CBP (cAMP-response element-binding protein/CREB-binding prote

236 33 (miR-433) inhibition of expression of the cAMP response element-binding protein CREB1 represses he

237 showed rapid and protracted asbestos-induced cAMP response element binding protein (CREB1) phosphoryl

238 phokinase A (PKA)-mediated activation of the cAMP-response element binding protein (CREB1) to transcr

239 llular outputs: p38-dependent growth arrest, cAMP response element-binding protein-dependent cell sur

240 Inhibition of cAMP response element-binding protein-dependent signalin

241 n and mouse islet alpha cells by GS/cAMP/PKA/cAMP-response element-binding protein-dependent activati

242 They also showed reduced phosphorylated cAMP response element-binding protein expression within

243 cient neuronal cell lines have reduced CREB (cAMP response element-binding protein) expression and in

244 protein, which induced the expression of the cAMP-response element-binding protein family repressor c

245 sured by phospho-GSK3beta (p-GSK3beta) and p-cAMP-response element-binding protein formation.

246 nscription levels of specific members of the cAMP Response Element Binding protein gene family.

247 sponse element-binding protein (ChREBP), and cAMP response element-binding protein, hepatocyte specif

248 xtracellular signal-regulated kinase and the cAMP response element binding protein in SC.

249 n neurons, and in the efficient signaling to cAMP-response element-binding protein in neurons.

250 quinpirole reduced phosphorylation of CREB (cAMP response element-binding protein) in dysbindin down

251 ound an enduring reduction in phosphorylated cAMP-response element binding protein levels in the NAcS

252 Silencing of cAMP-response element-binding protein, LRP1B or GPR6 exp

253 lin-dependent protein kinase IV (CaMKIV) and cAMP response element binding protein mediate the Ca(2)(

254 kinase 2 (SIK2) is an important regulator of cAMP response element-binding protein-mediated gene expr

255 t not PDE4B ablation significantly prolonged cAMP-response element-binding protein-mediated transcrip

256 ated by the transcription factors CREB/CREM (cAMP response element-binding protein/modulator) is link

257 sing a repressor of the transcription factor cAMP response element-binding protein or a calcium/calmo

258 transcription factor c-Jun, but not that of cAMP response element-binding protein or NF-kappaB.

259 the GcgR cDNA restored hepatic GcgR, phospho-cAMP response element binding protein (P-CREB), and phos

260 we found that PKA-induced phosphorylation of cAMP-response element-binding protein ((P)CREB) and EPAC

261 rylation status of their respective targets, cAMP response element-binding protein, p38, and extracel

262 ted cyclin A2 promoter activity via the cAMP-cAMP response element binding protein pathway.

263 ndent of the canonical cAMP/Protein Kinase A/cAMP response element-binding protein pathway downstream

264 s --> adenylate cyclase --> cAMP --> PKA --> cAMP response element-binding protein pathway mediating

265 pe, we administered rolipram to activate the cAMP response element-binding protein pathway, which led

266 n of the cAMP-dependent protein kinase (PKA)/cAMP response element-binding protein pathway.

267 lpha production via the protein kinase A and cAMP response-element-binding protein pathway, decreased

268 in acetylated histone H3 (AcH3) and phospho-cAMP response element binding protein (pCREB) associatio

269 rmal rats, riluzole increased phosphorylated cAMP response element binding protein (pCREB) expressing

270 Tax, in complex with phosphorylated cAMP response element binding protein (pCREB), strongly

271 ed kinase-1 (pMSK1) at Ser376 and Thr581 and cAMP response element-binding protein (pCREB) at Ser133

272 ippocampi had lower levels of phosphorylated cAMP response element-binding protein (pCREB), an activi

273 phosphorylation of the transcription factor cAMP response element-binding protein (pCREB).

274 hippocampal neurogenesis and phosphorylated cAMP response element-binding protein (pCREB).

275 f hyperglucagonemia and reduction of hepatic cAMP response element-binding protein, phoshoenolpyruvat

276 Consistently, leptin promotes cAMP response element-binding protein phosphorylation in

277 coupling we characterized the dependence of cAMP response element-binding protein phosphorylation, a

278 ments of hippocampal synaptic plasticity and cAMP response element-binding protein phosphorylation.

279 and pEC50 of ACEA-induced Galphas-dependent cAMP response element-binding protein phosphorylation.

280 rol of both H3-Ser10 and promoter-associated cAMP-response element-binding protein phosphorylation.

281 ns are essential for the regulation of CREB (cAMP response element-binding protein) phosphorylation a

282 acellular signal-regulated kinase) and CREB (cAMP response element-binding protein) phosphorylation.

283 ta-mediated increases in PKA activity, CREB (cAMP-response element-binding protein) phosphorylation,

284 Here we tested the hypothesis that CREB (cAMP response element-binding protein) provides a cell-s

285 nal activators, Tax and phosphorylated CREB (cAMP-response element-binding protein), recruited the p3

286 eting human Apolipoprotien C3 (Apoc3), CREB (cAMP Response Element Binding Protein) Regulated Transcr

287 ecretase activates the transcription factor, cAMP response element-binding protein, regulating miR-21

288 activity and elevated STMN Ser-63 and CREB (cAMP-response element-binding protein) Ser-133 phosphory

289 At the transcriptional level, cAMP response element-binding protein serves as a prime

290 providing the first evidence that enhancing cAMP response element binding protein signaling can alle

291 we reveal a previously unappreciated role of cAMP response element binding protein signaling in RTT p

292 Consequently, the cAMP-->PKA-->cAMP response element-binding protein signaling axis is

293 ed inactivation of the cAMP/protein kinase A/cAMP-response element-binding protein signaling pathway,

294 d phosphoprotein, M(r) 32,000, Ser133 of the cAMP-response element-binding protein, Thr286 of Ca(2+)/

295 morphine suppressed binding of phospho-CREB (cAMP response element binding protein) to Bdnf promoters

296 otein-1, CCAAT/enhancer-binding protein, and cAMP response element-binding protein transcription fact

297 ceptor-gamma accompanied with ATF2 and CREB (cAMP-response element-binding protein) was enhanced acro

298 Each induces phosphorylation of cAMP response element-binding protein, which binds to th

299 genic inhibition of the transcription factor cAMP response element-binding protein, which disrupts st

300 phosphorylation of the transcription factor cAMP-response element-binding protein, which induced the