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1 ecreasing the L-type Ca current (with 50 mum cadmium chloride).
2 tinal synaptic transmission was blocked with cadmium chloride.
3 ated using Balb/c-3T3 cells transformed with cadmium chloride.
4 either IT nor LTS was sensitive to 50 microM cadmium chloride.
5 m arsenate, antimony potassium tartrate, and cadmium chloride.
6 ed by culturing the cells in the presence of cadmium chloride and analyzed by spectra, transducin act
8 ed to hemin, hydrogen peroxide, paraquat, or cadmium chloride, and they were hypersensitive to cytoto
12 subcutaneously administered a combination of cadmium chloride (CdCl2) and methylmercury (II) chloride
13 nds to amino acid limitation and exposure to cadmium chloride (CdCl2) and sodium arsenite (NaAsO2).
14 The voltage-gated calcium channel blocker cadmium chloride did not alter the evoked increase in in
16 gh the dialysis probe of tetrodotoxin (TTX), cadmium chloride or magnesium chloride produced no reduc
17 gnature is indicated by our demonstration of cadmium chloride- or Fas ligand-induced apoptosis resist
18 eritoneally on day 9.0 of gestation, 4 mg/kg cadmium chloride produces forelimb defects (predominantl
19 ts demonstrate that the exposure of cells to cadmium chloride resulted in cytotoxicity and cell death
20 lines, HCT15, PLC/PR/5, HeLa, and Chang, to cadmium chloride resulted in cytotoxicity and cell death
21 by the voltage-gated calcium channel blocker cadmium chloride that blocks both neurotransmission to t
22 re exposed ad libitum for 40 days to 100 ppm cadmium chloride through their diet, or an identical die
23 nitroquinoline-N-oxide (4-NQO), benomyl, and cadmium chloride, which could be fully recovered by rein
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