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1 , confirming a decrease in resorption of the calcified cartilage.
2  at the boundary of the subchondral bone and calcified cartilage.
3 comes susceptible following focal changes in calcified cartilage.
4 re present in resorption lacunae in areas of calcified cartilage and in subchondral bone immediately
5  and spectra were successfully obtained from calcified cartilage and subchondral bone for the first t
6 ng of the subchondral bone plate, comprising calcified cartilage and underlying subchondral bone.
7  in subchondral bone immediately adjacent to calcified cartilage, as previously described.
8                                     Bone and calcified cartilage can be fossilized and preserved for
9 f tissues (uncalcified cartilage [UCC] only, calcified cartilage [CC] and subchondral bone [bone] [CC
10  to upward invasion by vascular canals or to calcified cartilage erosion may contribute to biomechani
11 ed to the massive accumulation of unresorbed calcified cartilage in a large area below the growth pla
12 LN also inhibited vascular invasion into the calcified cartilage in rats with OA and blocked osteocla
13           There were large discrete areas of calcified cartilage in the hypertrophic zone, few signs
14                                          The calcified cartilage layer was thicker (1.5-fold) in part
15 e is aggressive resorption of the underlying calcified cartilage leading to an extraordinary phenotyp
16 % of these atherosclerotic lesions developed calcified cartilage-like metaplasia.
17 ineralized tissues suggests that bone and/or calcified cartilage provide signals that are critical fo
18                                              Calcified cartilage remodeling due to upward invasion by
19 were analyzed by 3-dimensional histology for calcified cartilage, subchondral bone, and subchondral b
20 gnificantly reduced OARSI scores and CC/TAC (calcified cartilage to total articular cartilage), but i

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