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1        Only 1 ACS occurred in the absence of calcified plaque.
2 o human peripheral arteries with substantial calcified plaque.
3 traclass correlation coefficients, >0.99) of calcified plaque.
4 ied plaque was noted or EBT- if there was no calcified plaque.
5 with fewer mixed plaques and more often only calcified plaques.
6 scrimination between soft, intermediate, and calcified plaques.
7  (n = 10), mild (n = 10), or severe (n = 10) calcified plaques.
8 ; 95% CI, 0.15-0.85) and more often had only calcified plaques (38% versus 16%; ORadjusted=3.57; 95%
9  American patients had a lower prevalence of calcified plaque (39 [26%] vs 68 [45%] white patients, P
10      The diagnostic accuracy of CT to detect calcified plaque (83% versus 92%), necrotic core (80% ve
11              Attenuated plaques evolved into calcified plaques after stent implantation.
12 e assessed the characteristics of individual calcified plaques and their relationship to other parame
13  92% and 88% for any plaque, 95% and 91% for calcified plaque, and 91% and 89% for noncalcified plaqu
14 cified plaque, a mixture of noncalcified and calcified plaque, and calcified plaque were significantl
15 t or supplements with any of our measures of calcified plaque, and no greater mortality risk was obse
16 is, macrophage area, necrotic core area, and calcified plaque area was evaluated by using recursive p
17 ile mixed plaque at coronary CT angiography, calcified plaque at intravascular US, and lipid-rich pla
18                                              Calcified plaque at the proximal stent edge (relative ri
19 for uptake was significantly associated with calcified plaque burden (P < 0.0001) and cardiovascular
20              Tracer uptake was compared with calcified plaque burden and cardiovascular risk factors.
21  Younger diabetic individuals appear to have calcified plaque burden comparable to that of older indi
22  women 50 to 59 years old at enrollment, the calcified-plaque burden in the coronary arteries after t
23 he number of proximal segments with mixed or calcified plaques (C-index 0.64, p < 0.0001) and the num
24 The presence and severity of coronary artery calcified plaque (CAC) differs markedly between individu
25 l atherosclerosis imaging of coronary artery calcified plaque (CAC) to the primary prevention of coro
26 d computed tomography measurement of carotid calcified plaque (CarCP) and coronary calcified plaque (
27 ied plaque (CCP) and with or without carotid calcified plaque (CarCP) measured by electrocardiogram-g
28 est for an association among coronary artery calcified plaque, carotid artery calcified plaque, carot
29 nary artery calcified plaque, carotid artery calcified plaque, carotid IMT, and ACR while adjusting f
30  subjects with multiple (> or =3) individual calcified plaques, CC was heterogeneous within individua
31 are with or without the presence of coronary calcified plaque (CCP) and with or without carotid calci
32 raphy showed a better agreement with ICA for calcified plaques compared with SR coronary CT angiograp
33      Both spatial distribution and amount of calcified plaque contribute to risk for CHD.
34 arotid calcified plaque (CarCP) and coronary calcified plaque (CorCP).
35                Characteristics of individual calcified plaques, especially calcium concentration (CC)
36 may reflect that the pathological process of calcified plaque formation and progression is the same i
37 f coronary artery calcium, mixed plaque, and calcified plaque; higher CCL2 levels were associated wit
38  plaque (HR, 58.06; P = .005) or exclusively calcified plaque (HR, 32.94; P = .02).
39 n after CAS, particularly in patients with a calcified plaque in the carotid bulb, but is easily trea
40      Albuminuria is strongly associated with calcified plaque in the coronary and carotid arteries in
41                                              Calcified plaque in the coronary arteries is a marker fo
42 supplements and measures of subclinical CVD (calcified plaque in the coronary artery, carotid artery,
43 e standard deviation of CC of all individual calcified plaques in a subject.
44 ified volume, and mineral mass of individual calcified plaques in each subject.
45 of HRM (defined as noncalcified or partially calcified plaques) in the LMCA.
46                         The CC of individual calcified plaques is independent of age and sex but hete
47                   These data illustrate that calcified plaque limited intravascular drug delivery, an
48   Accurate quantification of calcium in each calcified plaque may require that the threshold be set i
49 respecified endpoints were non-calcified and calcified plaque measures and high risk plaque features
50 e, a proximal segment with either a mixed or calcified plaque or a stenosis >50% is equivalent to a 5
51 6 to 3.26], p < 0.0001) or the presence of a calcified plaque (OR 1.89 [range 1.25 to 2.84], p < 0.00
52 ) and of any plaque; noncalcified, mixed, or calcified plaque; or stenosis on coronary CT angiography
53 s, age was the only independent predictor of calcified plaque (p = 0.02) and remodeling (p = 0.005).
54 stprocessing techniques enhanced accuracy of calcified plaque quantification by reducing effects of t
55 1, P=0.002; fibrous plaque: r=0.54, P<0.001; calcified plaque: r=0.59, P<0.001; total plaque: r=0.62,
56                                          For calcified plaque, sensitivity was 94% (33 of 36) and spe
57                                Comparison of calcified plaque sizes determined with OCT with those de
58          However, statin therapy reduced non-calcified plaque volume and high-risk coronary plaque fe
59                        The prevalence of non-calcified plaque was 100%, 62%, and 77%, respectively, a
60 and 77%, respectively, and the prevalence of calcified plaque was 71%, 92%, and 85%, respectively, in
61  was good, and agreement for the presence of calcified plaque was high (kappa = 0.92, MESA; kappa = 0
62                                  The area of calcified plaque was measured at histopathologic examina
63  and HDLC > or =35 mg/dl; and 2) EBT+ if any calcified plaque was noted or EBT- if there was no calci
64 therosclerosis was regarded as definite if a calcified plaque was seen in the wall of an artery and p
65 correction factor was applied, the volume of calcified plaque was statistically better quantified wit
66                             CC of individual calcified plaques was independent of subject age (P=0.76
67                  Coronary and carotid artery calcified plaque were measured using fast-gated helical
68 re of noncalcified and calcified plaque, and calcified plaque were significantly higher among men wit
69                      Negative remodeling and calcified plaque with rare plaque ruptured were common i
70 nel-volume CT by comparing measured areas of calcified plaque with respect to the reference standard

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