1 ert-butylcalix[4]arene tetraethyl ester, and calcimycin.

2 hartreusins, cephalosporins, holomycins, and calcimycin.

3 s caffeine and 4-chloro-3-ethylphenol (CEP), calcimycin (a Ca2+ ionophore) and carbonyl cyanide m-chl

4 ified lipid stores by calcium ionophore (CI) calcimycin (A-23187).

5             Treatment of N/N1003A cells with calcimycin, a calcium mobilizer, activates the RAF/MEK/E

6  EPS with the pharmacological Ca(2+) carrier calcimycin (A23187) because contraction induces marked i

7                                              Calcimycin also activates p38 kinase and JNK2, which are

8 erated by raising intracellular Ca(2+) using calcimycin, and increased AR breakdown was reversed with

9                           Caffeine, CEP, and calcimycin increased resting [Ca2+]i to a greater extent

10     Moreover, overexpression of p53 enhances calcimycin-induced apoptosis, whereas inhibition of p53

11 reas inhibition of p53 expression attenuates calcimycin-induced apoptosis.

12 it ERK1/2 activation, and greatly attenuates calcimycin-induced apoptosis.

13 -cultured rat lenses, the peptides inhibited calcimycin-induced epithelial cell apoptosis.

14 e, but intact cells showed no differences in calcimycin-induced onset of mPT, irrespective of energiz

15                          The wild-type-like, calcimycin-induced response in csp null mutants indicate

16                     De-energization hastened calcimycin-induced swelling in control and partially-exp

17         Activation of RAF/MEK/ERK pathway by calcimycin leads to distinct up-regulation of p53.

18 )-stimulated increases in IL-6 mRNA, neither calcimycin- nor EPS-induced IL-6 mRNA expression is unde

19 mPT's voltage-sensing properties elicited by calcimycin or H2O2 in human fibroblasts exhibiting parti

20 scovered that BWSV and the calcium ionophore calcimycin stimulate FM1-43 destaining and quantal relea

21  inhibited phorbol myristate acetate (PMA) + calcimycin-stimulated IFN-gamma and IL-2 (but not IL-13)