ライフサイエンスコーパス: calcium

1 ires the continued presence of extracellular calcium.

2 out the need to systemically monitor ionized calcium.

3 troducing Flower or by raising extracellular calcium.

4 ic seawater components such as magnesium and calcium.

5 ion in response to oscillations in cytosolic calcium.

6 Gateau di patate was particularly rich in calcium (343mg), both contributing 34% of the recommende

7 st common adverse effect was decreased blood calcium (68.9% vs 59.8%).

8 d by statin intensity (maximal: atorvastatin calcium, 80 mg/d, or rosuvastatin, 40 mg/d; submaximal:

9 ntly, experimental increase in intracellular calcium abolished Flunarizine's effect.

10 Calcium accumulation during triggering stimuli appears t

11 Large conductance calcium-activated (BK) channels are broadly expressed in

12 EM16A), also called anoctamin 1 (ANO1), is a calcium-activated chloride channel expressed widely mamm

13 Calcium-activated chloride channels (CaCCs) encoded by T

14 RATIONALE: Large-conductance calcium-activated potassium channels (BK) are composed o

15 Sustained contacts result in long-lasting calcium activity and NFAT translocation, a measure of fu

16 m are broadly activated during movement with calcium activity corresponding to movement rate.

17 s a periplasmic arabinogalactan glycoprotein-calcium (AGP-Ca(2+) ) capacitor with tip-localized AGPs

18 t work, we study the use of electrodeposited calcium alginate hydrogels as a biocompatible matrix in

19 Eu-doped calcium aluminate was synthesized via the low-cost self-

20 fication), offers some automatic features in calcium analysis.

21 significant increase in oxidation-dependent calcium and calmodulin-dependent protein kinase II activ

22 rine springs supply the vast majority of the calcium and carbonate ions required for supersaturation

23 edback loop involving elevated intracellular calcium and enhanced mGluR1 function, a mechanism likely

24 r luciferase profiles reflect their neuronal calcium and in some cases firing profiles in wake-behavi

25 Furthermore, we report that calcium and integrin-binding protein 2 binds to the comp

26 Amorphous solid granules containing calcium and phosphorus were pervasive in the mitochondri

27 involved in the regulation of intracellular calcium and proliferation, and preventing the increase o

28 r genes involved in vitamin D metabolism and calcium and renal phosphate transport associated with di

29 orrelated with the altering concentration of calcium and sulfur.

30 activity under substrate conditions (various calcium and/or ATP concentrations) promoting particular

31 homeostasis biomarkers (parathyroid hormone, calcium, and 25-hydroxyvitamin D), and annualized BMD re

32 ns, beta-carotene, vitamin C, vitamin D plus calcium, and multivitamins or multi-ingredient supplemen

33 in vertebrates, involving the deposition of calcium- and phosphate-containing hydroxyapatite (HA) mi

34 The large-conductance, calcium- and voltage-activated K(+)(BK) channel consists

35 The presence of divalent ions, particularly calcium, appears to be an important stimulus for Longus

36 domains G3 and G4, which, in the absence of calcium, are compacted but adopt an open conformation in

37 Ds), this mechanism led to the endolysosomal calcium as a critical target for development of anti-Ebo

38 Plasma membrane calcium ATPase 2 (PMCA2) is a calcium pump that plays im

39 inhibitors of the sarcoendoplasmic reticulum calcium ATPase and ryanodine receptor.

40 ound that SMOC-EC, lacking the extracellular calcium binding (EC) domain, inhibited BMP2 signaling, w

41 MPCs displayed increased expression of S100 calcium-binding A4 (S100A4), a protein linked to cancer

42 ps13 and its surprising binding partner, the calcium-binding centrin Cdc31, in trans-Golgi network (T

43 Vps13p must be in complex with the small calcium-binding protein Cdc31p to be active.

44 The calcium-binding protein S100A4 is expressed at elevated

45 ine kinase, myosin light chain, sarcoplasmic calcium-binding protein, and hemocyanin are the most rel

46 se to hyperglycemia, neutrophil-derived S100 calcium-binding proteins A8/A9 (S100A8/A9) interact with

47 Calcium-binding proteins such as parvalbumin and calbind

48 PA, vitamins B6, K1, and D3) as enhancers of calcium bioavailability according to recommended dietary

49 Calcium bursts and hypersecretion are reversed by mutati

50 Calcium (Ca(2+)) and transverse-tubule imaging of ventri

51 Calcium (Ca(2+)) is an essential second messenger requir

52 Ionic calcium (Ca(2+)) is an essential signal for axon guidanc

53 4526 caused an accumulation of intracellular calcium (Ca(2+)) over time and cell death.

54 icrodomains exhibit spontaneous increases in calcium (Ca(2+)), but the mechanisms and functional sign

55 (CML) proteins are major EF-hand-containing, calcium (Ca(2+))-binding proteins with crucial roles in

56 he ability to conduct divalent cations, like calcium (Ca(2+)).

57 similar with all three fertilizers, but the calcium (Ca) and magnesium (Mg) was higher with ORG and

58 lants had an irregular increase of cytosolic calcium ([Ca(2+) ]cyt ) after NaCl treatment.

59 phenotype is promoted by increased cytosolic calcium ([Ca(2+)]cyto), aerobic glycolysis, and mitochon

60 It is not known whether coronary artery calcium (CAC) assessment at baseline in individuals with

61 ne the predictive ability of coronary artery calcium (CAC) score vs age for incident ASCVD and how ri

62 ionships among adult height, coronary artery calcium (CAC) score, incident atherosclerotic cardiovasc

63 2 targets revealed that extinction recruited calcium/calmodulin (Ca(2+)/CaMK)-dependent protein kinas

64 induces autophosphorylation (activation) of calcium/calmodulin-dependent kinase 2 (CaMKII) and also

65 Scaffolding the calcium/calmodulin-dependent phosphatase 2B (PP2B, calci

66 In contrast, when the calcium/calmodulin-dependent protein kinase II (CaMKII)

67 cium, in turn, led to the phosphorylation of calcium/calmodulin-dependent protein kinase II, which pr

68 as dependent on calcium influx and linked to calcium/calmodulin-dependent protein kinase II.

69 , reduced cardiomyocyte apoptosis, fibrosis, calcium/calmodulin-dependent protein kinase IIdelta phos

70 Calcium/calmodulin-dependent protein kinase type IV (CaM

71 supplementation with vitamin D3 (1000 IU) or calcium carbonate (1200 mg elemental calcium) or both or

72 st, one hydrated and one anhydrous amorphous calcium carbonate (ACC); that these are formed in the ti

73 o built heritage (mortars, black crusts, and calcium carbonate formations).

74 X-ray diffraction analysis showed that calcium carbonate is formed into the germ structure to 2

75 Corrosion inhibitors can affect calcium carbonate precipitation and associated in situ a

76 ied Flunarizine - a well-known anti-migraine calcium channel (CC) blocker - being able to diminish in

77 g exon (exon 47) of the Cav2.1 voltage-gated calcium channel (VGCC) gene produces two major isoforms

78 rovide a management approach for adults with calcium channel blocker poisoning.

79 ients with sepsis, of which, 19,742 received calcium channel blocker treatments prior to the admissio

80 Use of calcium channel blocker was associated with a reduced 30

81 termine the association between prior use of calcium channel blockers and the outcome of patients adm

82 Moreover, antiepileptics and calcium channel blockers may provide repurposing opportu

83 ve SNc neurons differ substantially in their calcium channel composition and efficacy of excitatory i

84 INTERPRETATION: Four T-type calcium channel variants and 1 ABCB1 transporter variant

85 nodine receptor but not in the voltage-gated calcium channel, indicating that these phenotypes are ca

86 ccepted mechanism of action of nifedipine, a calcium-channel blocker clinically used in patients with

87 In particular, EV-associated annexin calcium channelling proteins, which form a nucleational

88 ctivation and inactivation of the underlying calcium channels and correctly identified the accepted m

89 scillatory calcium signals via voltage-gated calcium channels as a key component.

90 implications, with a focus on voltage-gated calcium channels as part of the disease process and as a

91 unctions of axonal and dendritic L-type like calcium channels likely operate synergistically to maxim

92 Calcium channels required for symbiosis signaling have b

93 us signalling, mediated via NMDAR and L-type calcium channels, results in rapid FOXP1 deSUMOylation.

94 ting the function of pre-synaptic UNC-2/CaV2 calcium channels.

95 ransients due to strong expression of T-type calcium channels.

96 ion of Gbetagamma subunits and activation of calcium channels.

97 In the absence of calcium chelating salts, these concentrations were signi

98 eating skim milk with soluble calcium salts, calcium chloride, calcium lactate, calcium gluconate and

99 0.5wt%) gelation was induced by potassium or calcium chloride.

100 inhibits contractility at high intracellular calcium concentration by disrupting the actin-myosin ATP

101 ed by a transient rise in intracellular free calcium concentration linked to a change in the structur

102 n more sensitive to changes in environmental calcium concentration.

103 At low luminal calcium concentrations, ouf8 had little detectable effec

104 ng a potential explanation for the different calcium content in tumor cells versus normal tissues.

105 bbons had larger global and ribbon-localized calcium currents.

106 3-D intracellular structures with models of calcium cycling, presenting the possibility to directly

107 n mechanisms via which it regulates invasion.Calcium dependent protein kinase 1 (CDPK1) plays an impo

108 Calcium Dependent Protein Kinases are key effectors of c

109 Calpain is a family of calcium-dependent endopeptidases, which plays an importa

110 lanthanide-dependent MDH (XoxF)-type, to the calcium-dependent MDH (MxaF)-type.

111 hormone-stimulated PLC activity, indicating calcium-dependent PLCs are not upregulated by alcohol.

112 high rates of auditory nerve firing, or that calcium-dependent processes involved in release are alte

113 inhibitors (BKIs) of Cryptosporidium parvum calcium-dependent protein kinase 1 (CpCDPK1) are leading

114 they demonstrate decreased expression of the calcium-dependent protein kinase C conventional subclass

115 The glycinocins are a class of calcium-dependent, acidic cyclolipopeptide antibiotics s

116 increased alkaline phosphatase activity and calcium deposition of encapsulated hASCs.

117 eased alkaline phosphates (ALP) activity and calcium deposition.

118 lular calcium, or chelation of intracellular calcium did not normalize the differences in hormone-sti

119 ults show that agonist-induced intracellular calcium dynamics can be modified by changing the levels

120 The model reproduces intracellular calcium dynamics during control pacing and reveals the h

121 of SERCA, IP3R, and RyR on the intracellular calcium dynamics of VSMC and to understand how variation

122 se structures tightly controls intracellular calcium dynamics.

123 lasticity is independent of calcium entry or calcium dynamics.

124 c2B - inhibition of release during sustained calcium elevations - depends on an overlapping protein d

125 dopt an open conformation in the presence of calcium, enabling actin binding and severing.

126 the present study, we report store-operated calcium entry (SOCE) as a novel target of TRPM7 kinase a

127 The store-operated calcium entry (SOCE) pathway is an important route for g

128 Interestingly, the store-operated calcium entry channel inhibitor (SK&F96365) also reduced

129 y, these alterations were found to be due to calcium entry into the mitochondria, because the swellin

130 mGluR-dependent plasticity is independent of calcium entry or calcium dynamics.

131 teolytic processing occurred normally during calcium excess.

132 ta supporting the role for NCX-9 in handling calcium exchange at the mitochondrion.

133 specificity by which the handling by NCLX of calcium exchange can map to neural circuit patterning an

134 ic Ca(2+) oscillations induced by the sodium/calcium exchanger NCX1/3 working in its reverse mode.

135 f piconewton integrin tension coincides with calcium flux.

136 We used high-throughput calcium-flux assays and patch clamp recordings of transi

137 response and those that release oscillatory calcium fluxes.

138 ols, we assigned 2378 models of voltage- and calcium-gated ion channels coded in NEURON to 211 cluste

139 um salts, calcium chloride, calcium lactate, calcium gluconate and calcium lactobionate, on the physi

140 the high-resolution 3-D spatial structure of calcium gradients and intracellular fluxes in both the c

141 (95% CI, 0.032 to 0.056) in the vitamin D3 + calcium group and 0.060 (95% CI, 0.048 to 0.076) in the

142 participants, 45 (3.89%) in the vitamin D3 + calcium group and 64 (5.58%) in the placebo group (diffe

143 essed whether people with genetically higher calcium had a higher risk of coronary artery disease (CA

144 ad to proteasome impairment, such as altered calcium handling and increased oxidative stress due to m

145 cardiac dysfunction, dampened intracellular calcium handling, alterations in cardiac morphology, and

146 Remarkable new roles for mitochondria in calcium handling, apoptosis, heme turnover, inflammation

147 er myofibril density and alignment, improved calcium handling, enhanced contractility, and more physi

148 rm black carbon [BC] and PM2.5 levels, serum calcium homeostasis biomarkers (parathyroid hormone, cal

149 se mitochondrial membrane potential controls calcium homeostasis, and AMP-activated protein kinase (A

150 s a critical role in cellular energetics and calcium homeostasis; however, how MAM is affected under

151 formed into the germ structure to 2.1 w/w of calcium hydroxide and 9h steeping time.

152 Calcium imaging and patch-clamp experiments show that G2

153 METHODS AND The CRESCENT trial (Calcium Imaging and Selective CT Angiography in Comparis

154 y performing perforated patch recordings and calcium imaging experiments in rats (male and female), w

155 however, combining an atlas with whole-brain calcium imaging has yet to be performed in vivo in adult

156 Using calcium imaging of cellular responses in awake mice, we

157 Using in vivo two-photon calcium imaging of layer 2/3 barrel cortex neurons expre

158 Furthermore, two-photon calcium imaging revealed that M2 ensemble activity also

159 However, the kinds of information that calcium imaging reveals is limited.

160 We report here that ratiometric calcium imaging reveals that Der p1 activates the human

161 evidence of "silencing", intracellular free calcium imaging showed that the cells were still viable.

162 Calcium imaging shows that the beta-cells in the embryon

163 f repetitive whisker stimulation and in vivo calcium imaging to assess tactile defensiveness and barr

164 Calcium imaging with cellular resolution typically requi

165 synaptic labeling, ultrastructural analysis, calcium imaging, optogenetics and behavioral analyses, w

166 Using calcium imaging, we found that these neuron types are no

167 antly assessed through behavioral assays and calcium imaging.

168 These results suggest that intraterminal calcium in old endbulbs may rise to abnormally high leve

169 To bind large amounts of calcium in vitro, calsequestrin must polymerize and then

170 The increased cytosolic calcium, in turn, led to the phosphorylation of calcium/

171 We used a genetically encoded calcium indicator and readily detected active calcium tr

172 that ICWs were initiated by an extracellular calcium influx across the cell membrane nearest to the j

173 is expressed in cardiomyocytes and decreases calcium influx across the L-type Ca(2+) channel.

174 ought transporters involved in mitochondrial calcium influx and efflux have recently been identified.

175 drenaline on TRPV1 channels was dependent on calcium influx and linked to calcium/calmodulin-dependen

176 anization of input fibres does not depend on calcium influx or dynamics.

177 s perturbations in firing through changes in calcium influx, and translates this into compensatory ch

178 7 synergistically enhanced degranulation and calcium influx.

179 reasing body mass index (BMI) on recommended calcium intakes.

180 The entry of calcium into mitochondria is central to metabolism, inte

181 ytes with LPS followed by treatment with the calcium ionophore A23187 resulted in the formation of PG

182 Signal transduction via calcium ions (Ca2+) represents a fundamental signaling p

183 Many have proposed that calcium ions binding to daptomycin is a precondition for

184 s solid hydrogel matrices by adding divalent calcium ions.

185 bone status including bone mineral density, calcium kinetics studies, and markers of bone remodeling

186 ith soluble calcium salts, calcium chloride, calcium lactate, calcium gluconate and calcium lactobion

187 ride, calcium lactate, calcium gluconate and calcium lactobionate, on the physico-chemical and rheolo

188 nome-wide association meta-analysis of serum calcium levels (N = up to 61079 individuals) and from th

189 Cytoplasmic calcium levels and transients increased upon mechanical

190 Elevated calcium levels in HFpEF are neither a result of an impai

191 A genetic predisposition to higher serum calcium levels was associated with increased risk of CAD

192 ion of Serca2, reduced endoplasmic reticulum calcium levels, and induction of the UPR.

193 Mitral annular calcium (MAC), commonly identified by cardiac imaging, i

194 ons in EDTA-treated samples unless exogenous calcium/magnesium was added at the time of anti-IgE stim

195 rant of genetic or chemical perturbations of calcium-mediated signalling, but abolished in null mutan

196 and for BCMA and transmembrane activator and calcium-modulator and cyclophilin ligand (TACI).

197 In vivo calcium monitoring, dynamics of PDF projections, ArcLigh

198 IU) or calcium carbonate (1200 mg elemental calcium) or both or neither.

199 Removal of extracellular calcium, or chelation of intracellular calcium did not n

200 Calcium orchestrates the activity of several kinases and

201 s expressed in astrocytes tripled astrocytic calcium oscillation frequency in both the preBotzinger c

202 lating inositol 1,4,5-trisphosphate-mediated calcium oscillations and the up-regulation of the transc

203 tions in membrane potential accompanying the calcium oscillations have no significant effect on the p

204 ased during hypercapnic challenge, increases calcium oscillations in the chemosensitive parafacial re

205 significant effect on the properties of the calcium oscillations.

206 lly modulate effects of EFS-induced cellular calcium oscillations.

207 te increased susceptibility to mitochondrial calcium overload in LRRK2-driven neurodegeneration, and

208 hanisms contribute to the increased risk for calcium oxalate stone formation observed in patients wit

209 e whether common variants in 7 vitamin D and calcium pathway genes (VDR, GC, DHCR7, CYP2R1, CYP27B1,

210 Together, these data implicate astroglial calcium pathways as potential targets for stroke therapy

211 y rescued the hypercalciuric and lower serum calcium phenotype in Ksp-cre;Pth1r(fl/fl) mice, emphasiz

212 e exposed to a PPi-stabilized supersaturated calcium phosphate (CaP) solution containing 0 to 0.06 mg

213 at bran, oat bran and white bean had a lower calcium:phosphate ratio than barley bran and red kidney

214 unction experiments show that Pth4 can alter calcium/phosphorus levels and affect expression of genes

215 Calcium/PKCalpha-dependent activation of NUAK1 supports

216 Calcium plays a key role in determining the specificity

217 zation was dependent on C5aR1, intracellular calcium, protein kinase C, and calmodulin, and downstrea

218 y functionally connect them to the transient calcium pulses observed in restricted areas in the formi

219 ulatory factor 1 (NHERF1) interacts with the calcium pump PMCA2 and the tyrosine kinase receptor ErbB

220 lasma membrane calcium ATPase 2 (PMCA2) is a calcium pump that plays important roles in neuronal func

221 (n = 15), spondyloarthropathy (n = 15), and calcium pyrophosphate deposition disease (CPPD) (n = 15)

222 One software, CalQuo (Calcium Quantification), offers some automatic features

223 RET), confocal microscopy, and intracellular calcium quantitation.

224 Barium-to-calcium ratios (Ba/Ca) and carbon isotopes (delta(13)C)

225 ar signaling via second messengers-cytosolic calcium, reactive oxygen species, and nitric oxide.

226 The role of calcium regulators in governing these processes is becom

227 In addition, the network indicates calcium-related genes play key roles in P. euphratica re

228 risphosphate receptor (ITPR2), the principle calcium release channel in hepatocytes.

229 ring inhibition of UTP-induced intracellular calcium release in 1321N1 astrocytoma cells stably trans

230 abapentin impairs the T-cell receptor-driven calcium response and cytokine production associated with

231 ) differentiates cells that release a single calcium response and those that release oscillatory calc

232 Although cavitation-induced calcium responses are believed to be important for modul

233 yes has led to the routine quantification of calcium responses in non-excitable cells.

234 thyl-d-aspartic acid receptor (NMDAR)-driven calcium responses in single spines, we provide a spatial

235 thout influencing NMDA-induced intracellular calcium responses.

236 Calcium retention increases with increasing body mass in

237 he effects of heating skim milk with soluble calcium salts, calcium chloride, calcium lactate, calciu

238 Coronary artery calcium score was more likely than age to provide higher

239 ctivity were attributable to the activity of calcium-sensing receptors (CaSRs), which appear to be fu

240 Advances in fluorescence microscopy and calcium sensitive dyes has led to the routine quantifica

241 studies suggest that it enhances myofilament calcium sensitivity and alters calpain-mediated cTnI pro

242 probability of TMEM16A without changing its calcium sensitivity.

243 gulation of dStim, the endoplasmic reticular calcium sensor and a principal component of SOCE in the

244 e of STIM2, suggesting STIM1 is the dominant calcium sensor required for classical short-term neutrop

245 ined unknown if, and how, calcineurin B-like calcium sensors (CBLs) and CBL-interacting protein kinas

246 depends on spatiotemporal patterning of the calcium signal and decoding it by multiple, tunable, and

247 of the probe for these two species triggered calcium signaling and intracellular protein translocatio

248 P+ cells and glucose-responsive synchronized calcium signaling as well as expression of the transcrip

249 pendent Protein Kinases are key effectors of calcium signaling in malaria parasite.

250 Calcium signaling leads to a promotion of complex format

251 roliferation, and preventing the increase of calcium signaling rescues the cell-cell junctional defec

252 mmune evasion, increased stemness, increased calcium signaling, transformation, and novel E-cadherin-

253 Because LR depends on calcium signaling, we examined the effects of NAFLD on e

254 lly, glucose triggers KATP channel-dependent calcium signaling, which promotes HDAC5 phosphorylation

255 phagocytic capacity and effects on neuronal calcium signaling.

256 haft of neurons, triggering an inhibition of calcium signaling.

257 cid sequence and (3) its ability to activate calcium signalling and/or ERK1/2 phosphorylation via PAR

258 ion of acidic stores with other parts of the calcium signalling apparatus in cardiac myocytes is unkn

259 s which suggest a central role for perturbed calcium signalling in DYT2 dystonia.

260 Calcium signalling silencing is a part of the mechanisms

261 spines, we provide a spatial map of synaptic calcium signals along dendritic arbors of hippocampal ne

262 Hence EGFR elicits PLCgamma1-calcium signals to facilitate formation of a subset of C

263 twork refinement, which includes oscillatory calcium signals via voltage-gated calcium channels as a

264 Additionally, despite larger presynaptic calcium signals, we observed fewer evoked spikes with lo

265 Key properties of the calcium signatures are not intuitive, exemplifying the i

266 further used to highlight the criticality in calcium spark propagation in relation to inter-dyad dist

267 roach, we show that Gaussian processes model calcium spike rates with high fidelity and perform bette

268 ntaining newly formed synapses via dendritic calcium spike-dependent mechanisms.

269 us to quantitatively describe the timing of calcium spikes.

270 modulation, induced antioxidant activity and calcium spiking.

271 ng the forms and quantities of mitochondrial calcium stores is needed.

272 nd CASR) modify the effects of vitamin D3 or calcium supplementation on colorectal adenoma recurrence

273 nt (eg, comprehensive geriatric assessment), calcium supplementation, and vitamin D supplementation (

274 o significant interactions of genotypes with calcium supplementation.

275 24, and Cav1.2-S1928, and leads to a reduced calcium time to transient 50% decay.

276 alcium indicator and readily detected active calcium transients although no spontaneous contractions

277 to generate the hCMP, which began generating calcium transients and beating synchronously within 1 da

278 puts, which can then trigger large dendritic calcium transients due to strong expression of T-type ca

279 instead of IP3, also potentiated K20-induced calcium transients in the presence of beta-estradiol, in

280 nd relaxation and the peak amplitudes of the calcium transients increased significantly over the next

281 -related genetic and chemical factors in the calcium transients of the ASH sensory neuron.

282 This potentiation of depolarization-induced calcium transients was blocked by the IP3 antagonist, an

283 to inflammatory stimuli, migrate and undergo calcium transients, and robustly phagocytose CNS substra

284 We report that nuclei display spontaneous calcium transients, and that changes in the activity pat

285 Using pre-steady-state charge (calcium) translocation and steady-state ATPase activity

286 Inhibition of the sarcoendoplasmic reticulum calcium trasport ATPase (SERCA) pump activity with thaps

287 tretch-activated channel Piezo1 and involves calcium-triggered ERK signalling.

288 The mitochondrial calcium uniporter (MCU) is a highly selective ion channe

289 Mitochondrial calcium uniporter (MCU), which is the core channel subun

290 be corrected by silencing the mitochondrial calcium uniporter (MCU).

291 reversed by the inhibition of mitochondrial calcium uniporter (MCU).

292 The mitochondrial calcium uniporter is a Ca(2+)-activated Ca(2+) channel c

293 Local calcium upstroke was delayed (23.9+/-4.9 versus 10.3+/-1

294 To obtain a unified picture of mitochondrial calcium utilization, a parallel advance in understanding

295 nitude speedups in spatiotemporally demixing calcium video data into estimates of single-cell neural

296 Intracellular calcium was higher after blue/green, and could be inhibi

297 ease in viscosity nor a reduction in soluble calcium was responsible, leading to the conclusion that

298 Thus, silencing calcium waves in the auditory thalamus induces Rorbeta u

299 MPK) is regulated, in part, by intracellular calcium, we postulated that AMPK participates in STING a

300 modify the association between the intake of calcium with vitamin D (CaD) and fracture risk.Data from