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1 on is medical therapy with beta-blockers and calcium antagonists.
2 nt to adenosine and moderately responsive to calcium antagonists.
3 s achieved with ACE inhibitors compared with calcium antagonists.
4 eviously unexplored with heart-rate-lowering calcium antagonists.
5 angina per week with beta-blockers than with calcium antagonists.
6 is association was also true for long-acting calcium antagonists.
7 ts the albumin-decreasing effects of certain calcium antagonists.
8 ze the albumin-decreasing effects of certain calcium antagonists.
10 ly prescribed antihypertensive drugs, 3 were calcium antagonists, 3 were angiotensin-converting enzym
12 , double-blind crossover study comparing the calcium antagonist amlodipine (10 mg once daily) versus
13 placebo, (2) beta-blocker (acebutolol), (3) calcium antagonist (amlodipine), (4) diuretic (chlorthal
14 e a diuretic (chlorthalidone; n = 13,860), a calcium antagonist (amlodipine; n = 8174), an angiotensi
16 meta-analysis was to compare the effects of calcium antagonists and other antihypertensive drugs on
19 rom 2 or 3 different classes (beta-blockers, calcium antagonists, and long-acting nitrates) lasting a
20 newer vasodilating beta-blocking agents and calcium antagonists appear to be metabolically neutral.
21 , the large available database suggests that calcium antagonists are inferior to other types of antih
23 1,500 primary care sites to receive either a calcium antagonist-based (verapamil) or beta-blocker/diu
24 y artery disease (CAD) treated with either a calcium antagonist-based or a noncalcium antagonist-base
27 n the basis of these data, the longer-acting calcium antagonists cannot be recommended as first-line
28 tensin-converting enzyme inhibitors (ACEIs), calcium antagonists (CCBs) and alpha-blockers in prevent
31 ) and degranulation (P-selectin expression), calcium antagonists enhancing degranulation, and beta-bl
33 ect sustained-release preparations and newer calcium antagonists have on the clinical endpoints of my
36 studied in combination with beta-blockers or calcium antagonists in a large patient population with s
38 re associated with fewer adverse events than calcium antagonists in randomized trials of patients who
39 in-converting enzyme inhibitors, niacin, and calcium antagonists may also contribute to plaque passiv
40 e suggested that, compared with other drugs, calcium antagonists may be associated with a higher risk
44 reduction 11.2/8.5 mm Hg; p < 0.00001), and calcium antagonists (mean BP reduction 11.0/8.1 mm Hg; p
45 blocker monotherapy, patients on long-acting calcium antagonists (n = 136) had no increased risk of a
46 .41-1.43]), whereas patients on short-acting calcium antagonists (n = 27) were at significantly great
47 ors, or clonidine (n=15,044), those assigned calcium antagonists (n=12,699) had a significantly highe
48 We aimed to investigate the effect of the calcium antagonist nifedipine on long-term outcome in pa
49 the effects of isradipine, a dihydropyridine calcium antagonist, on the course of the nephropathy wer
50 ts received first-line treatment of either a calcium antagonist or beta-blocker followed by angiotens
51 this reason, recent attention has focused on calcium antagonists or angiotensin-converting enzyme inh
53 assigned intermediate-acting or long-acting calcium antagonists or other antihypertensive drugs and
54 cause of adverse events less often than were calcium antagonists (OR, 0.72; 95% CI, 0.60-0.86; P<.001
55 ifferent for treatment with beta-blockers vs calcium antagonists (OR, 0.97; 95% confidence interval [
62 considered in the context of other trials of calcium antagonists, these data indicate that the effect
67 th earlier findings, the use of short-acting calcium antagonists was associated with increased risk o
68 el hydrochlorothiazide and a dihydropyridine calcium antagonist were added, if needed, to achieve blo
71 The differences between beta-blockers and calcium antagonists were most striking for nifedipine (O
72 hibitors, angiotensin receptor blockers, and calcium antagonists yielded a lower incidence of diabete
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