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1 of a protein specifically in response to ER calcium depletion.
2 chaperones, including CRT, is induced by ER calcium depletion.
3 ote phagocytic uptake of cells subject to ER calcium depletion.
4 tight junctions from disassembly induced by calcium depletion.
5 ong dimer that was unable to dissociate upon calcium depletion.
6 ion of AJC by using a model of extracellular calcium depletion.
7 contacts and adhesive dimers in response to calcium depletion.
8 shock and tunicamycin treatments), and upon calcium depletion.
9 nectin-1, over the entire cell surface after calcium depletion.
10 be infected by HSV and PRV, before and after calcium depletion.
11 e infected by HSV-1 and PRV were enhanced by calcium depletion.
12 onsible for only a portion of the effects of calcium depletion.
13 s required for maximal induction by ATF6 and calcium depletion.
14 PD098059 (a MAPK kinase (MEK) inhibitor) and calcium depletion.
15 y which grp78 transcription is stimulated by calcium depletion.
17 pe III secretion system (T3SS) is induced by calcium depletion and is positively regulated by the Exs
19 nslocated to contractile F-actin rings after calcium depletion, and siRNA-mediated depletion of GEF-H
29 ing store-operated calcium entry activation, calcium depletion from the endoplasmic reticulum trigger
30 lated during 30 min periods of extracellular calcium depletion in a region enriched with presynaptic
33 Finally, there is a strong synergy between calcium depletion in the ER and sterile IL-6, as well as
34 ine storage in the acidic compartment led to calcium depletion in these organelles, which then result
35 We further show that Ecadherin mutations and calcium depletion induce structural alterations that pro
37 for a chaperone and that heat treatment and calcium depletion induced the formation of calnexin dime
39 determine whether disruption of junctions by calcium depletion influenced the susceptibility of epith
40 trastructural analysis revealed that luminal calcium depletion is accompanied by increased accumulati
41 ar the resting level, whereas a threshold of calcium depletion is required for STIM1 activation [4].
42 calcium refilling mechanism responsive to ER calcium depletion, is enhanced in SOD1G93A astrocytes.
43 ate for the first time that specific luminal calcium depletion leads to a significant decrease in end
44 icing and BiP expression, suggesting that ER calcium depletion may be one factor contributing to ER s
46 TIM2 are calcium-sensing molecules that link calcium depletion of the endoplasmic reticulum with open
50 pletions in sulfur and possible aluminum and calcium depletions, relative to ordinary chondrites, may
52 8 induction by ATF6 in cells subjected to ER calcium depletion stress mediated by thapsigargin (Tg) t
54 tricular myocytes was not increased by SR/ER calcium depletion, suggesting that not all secreted prot
56 olymerized as [Ca(2+)] went down: fully when calcium depletion was maximal (a condition achieved with
58 A change in conformation of desmoglein 1 by calcium depletion was shown, with immunofluorescence and
61 ients, including gap junction inhibition and calcium depletion, were also found to disrupt the develo
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