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1 maining adaptation process is independent of calcium entry.
2 appear to depend explicitly on the route of calcium entry.
3 esicles (SVs) that are distal to the site of calcium entry.
4 binding of annexins to membranes at sites of calcium entry.
5 nto signalosomes, resulting in BCR-activated calcium entry.
6 type channels contribute modestly to overall calcium entry.
7 to investigate the mechanism of PACAP-evoked calcium entry.
8 ediated signaling or store depletion-induced calcium entry.
9 4 mM) was added to suppress channel-mediated calcium entry.
10 to synchronous EPSC amplitude, activity, and calcium entry.
11 otide-dependent pathways coupled to neuronal calcium entry.
12 rform other roles unrelated to voltage-gated calcium entry.
13 l Ca(2+), also referred to as store-operated calcium entry.
14 zation of T cells using KCl does not lead to calcium entry.
15 coupled to intracellular calcium release and calcium entry.
16 to contribute to action potential-dependent calcium entry.
17 n including decreased chemotaxis and reduced calcium entry.
18 rane calcium influx channels or capacitative calcium entry.
19 anilloid receptor 1 triggered store operated calcium entry.
20 showing the critical role of TRPM2-mediated calcium entry.
21 to membrane depolarization and voltage-gated calcium entry.
22 olved in agonist-stimulated and capacitative calcium entry.
23 ings and processes including mitogenesis and calcium entry.
24 tivates STIM1/Orai1-dependent store-operated calcium entry.
25 ts deficiency had no effect on TCR-initiated calcium entry.
26 release is associated with CaV and not TRPV1 calcium entry.
27 acute loss of Orai1-dependent store-operated calcium entry.
28 stitute the core machinery of store-operated calcium entry.
29 STIM2) are key modulators of store-operated calcium entry.
30 tore depletion, KSR2 is required for optimal calcium entry.
31 (1) subunit important for excitation-coupled calcium entry?
32 ator (ORAI) pathway controls store-dependent calcium entry, a major mechanism of physiological calciu
33 s as a negative regulator of agonist-induced calcium entry (ACE) that suppresses surface accumulation
35 ular calcium stores activates store-operated calcium entry across the plasma membrane in many cells.
36 sting that the oscillations were mediated by calcium entry across the plasma membrane in response to
40 ted calcium channels (VGCCs) allow for rapid calcium entry and are expressed during early neural stag
42 o pH 6.4 caused inhibition of Orai1-mediated calcium entry and conferred capability for PDGF to evoke
43 NA and proteins and abrogated store-operated calcium entry and I(CRAC) in VSMC; control shRNA was tar
44 sigargin was used to activate store-operated calcium entry and increase cytosolic calcium in endothel
47 alcium channels (Cav) are the major route of calcium entry and regulate multiple functions such as co
49 findings demonstrate the close apposition of calcium entry and release sites and the dynamics of such
50 Ps), particularly the falling phase, affects calcium entry and small changes in calcium influx can pr
52 In particular, reduced spike height reduces calcium entry and subsequent calcium-activated potassium
53 Tonic synapses are specialized for sustained calcium entry and transmitter release, allowing them to
54 1 substantially reduced thapsigargin-induced calcium entry, and more modestly diminished the frequenc
55 idual oligomers larger than trimers inducing calcium entry as they cross the cell membrane, a result
56 is a rare genetic disorder of store-operated calcium entry, associated with a complex syndrome includ
61 nstrate that cholinergic excitation promotes calcium entry at subthreshold membrane potentials to rap
63 redict human genes involved in mitochondrial calcium entry based on clues from comparative physiology
66 ular calcium or inhibition of store-operated calcium entry blocked DIR, but the L-type calcium channe
67 d STIM2 punctum formation and enhanced basal calcium entry but decreased sarco/endoplasmic reticulum
68 MEM20 (POST), does not affect store-operated calcium entry but does reduce plasma membrane Ca(2+) pum
69 llular calcium or inhibition of capacitative calcium entry by 2-APB prevented ciglitazone-induced EGF
70 -1, -2, and -3 enhanced thapsigargin-induced calcium entry by 50-150% in cells stably overexpressing
74 ed receptor expression was due to attenuated calcium entry, cells were incubated with the calcium che
75 as recently been shown to exhibit myocardial calcium entry channel blocking activity, substantially h
77 sing a mouse model in which the gene for the calcium entry channel protein, Orai1, has been deleted.
78 n in stores, and Orai-1, the calcium-induced calcium entry channel, are colocalized with SP, in the s
79 nt receptor potential vanilloid 4 (Trpv4), a calcium-entry channel, is expressed in normal cholangioc
80 ntrolling cell function, can be generated by calcium entry channels activated by plasma membrane depo
82 ithelial P2X(4) receptors serve as ATP-gated calcium entry channels that induce a sustained increase
84 culum calcium release-induced store-operated calcium entry contributes to intracellular calcium incre
88 reas streptomycin antagonized TRPM8-mediated calcium entry, downregulated UCP-1 expression, and mitig
92 ads to a complete loss of excitation-coupled calcium entry during KCl depolarization and a more rapid
93 d to the mitochondrial matrix, we found that calcium entry during pacemaking created a basal mitochon
94 ed CPK32 activates CNGC18, further promoting calcium entry during the elevation phase of Ca(2+) oscil
96 sms of action is to block excitation-coupled calcium entry (ECCE) in both adult mouse flexor digitoru
97 mational coupling, termed excitation-coupled calcium entry (ECCE) is triggered by the alpha(1s)-DHPR
98 ypothesize that under conditions of enhanced calcium entry, elevation of intracellular calcium will r
99 ize, indicating that a separate mechanism of calcium entry exists, corresponding to cell loss at the
101 tsynaptic spiking alone and that it requires calcium entry following synaptic NMDA receptor activatio
102 microns away from the wound, allowing direct calcium entry from extracellular fluid into damaged cell
105 esults indicate that the P2Y6/store-operated calcium entry/IL-8 axis is involved in MSU crystal-induc
106 (s) involved in regulation of store operated calcium entry in Darier's disease (DD) is not known.
107 c Cav-1, we show that Cav-1 is essential for calcium entry in endothelial cells and governs the local
108 mbrane potential but abolishes mitochondrial calcium entry in intact and permeabilized cells, and att
109 nnels, thereby inhibiting glucose-stimulated calcium entry in isolated mouse pancreatic beta cells.
110 CaT1 also contributes to store-operated calcium entry in Jurkat T-lymphocytes and prostate cance
114 wanted cross-talk between pathways, sites of calcium entry in neurons are localized to specific membr
115 of the proteasome is dependent upon external calcium entry in part through N-methyl-D-aspartate recep
117 pen with fast kinetics and carry substantial calcium entry in response to individual action potential
118 this study, we show that RBCs exhibit robust calcium entry in response to mechanical stretch and that
124 nificant deficits in BCR triggering-mediated calcium entry in the cytosol, which correlates with impa
127 a(2+) channels (LTCC) are the main route for calcium entry in vascular smooth muscle cells (VSMC).
128 t effect on endogenous, thapsigargin-induced calcium entry in wild-type cells (HEK-293, COS1), in HEK
129 erning why other known routes of significant calcium entry, in particular, VGCCs, are not similarly t
130 and DeltaE9 cells showed larger capacitative calcium entry indicating a direct effect on Ca2+ influx
131 es not affect the time course of presynaptic calcium entry, indicating that the reduced Ca(influx) re
132 nous TRPC1 or TRPC4 inhibited store-operated calcium entry, indicating they are part of the native SO
136 lutamate modulation of H(+) flux arises from calcium entry into cells with subsequent activation of t
138 dies described here show that HBx stimulates calcium entry into cells, resulting in an increased plat
147 ved as one of the most important players for calcium entry into presynaptic endings responsible for t
148 pecifically, we show that CpG/SR-B1 triggers calcium entry into primary B lymphocytes via phospholipa
149 uration CS responses in Purkinje cells, more calcium entry into Purkinje cells, larger synaptic depre
151 Here we have shown an additional route for calcium entry into T cells-through the low-voltage-activ
152 y, these alterations were found to be due to calcium entry into the mitochondria, because the swellin
153 hondrial calcium uniporter (MCU) facilitates calcium entry into the mitochondrial matrix to stimulate
156 A unique mechanism called store-operated calcium entry is activated when ER calcium is depleted,
159 t that, under normal conditions, the role of calcium entry is to sustain [Ca(2+)](i) oscillations.
160 hat pairing IP3 with climbing fiber-mediated calcium entry leads to a large calcium release transient
161 e show that the activation of Orai1-mediated calcium entry leads to enhancing focal adhesion turnover
162 f extracellular calcium through capacitative calcium entry may be an unrecognized component that prov
163 activity-dependent regulation of presynaptic calcium entry may contribute to homeostatic regulation o
166 led that caspase-3 activation, extracellular calcium entry, mitochondrial membrane permeability, and
167 ly demonstrated that neuronal store-operated calcium entry (nSOC) in hippocampal neurons is regulated
172 hat requires store depletion (store-operated calcium entry or SOCE) and a second that is independent
174 cium channel CaV1.3 constitutes an important calcium entry pathway implicated in the regulation of sp
179 on conductance (I(CAN)) that is activated by calcium entry predominantly through L-type calcium chann
180 as a predominant mediator of store-operated calcium entry, proliferation, and cytokine production in
182 in naive CD8(+) T cells, and is critical for calcium entry required for their proper function during
183 posed to form nonselective receptor-operated calcium entry (ROCE) cation channels that are activated
186 e in cell proliferation rate, store-operated calcium entry (SOCE) amplitude, cationic channel TRPC6,
188 gnaling pathways initiated by store-operated calcium entry (SOCE) are known to regulate neutrophil ac
189 the present study, we report store-operated calcium entry (SOCE) as a novel target of TRPM7 kinase a
190 Our previous studies implied store-operated calcium entry (SOCE) as the major pathway for this Ca(2+
192 ers ER morphology and affects store-operated calcium entry (SOCE) by decreasing STIM1 puncta formatio
193 sed to form Ca(2+)-selective, store-operated calcium entry (SOCE) channels that are activated by stor
194 allowed a novel "permissive" store-operative calcium entry (SOCE) following the initial platelet-acti
195 model wherein STIM1-mediated store-operated calcium entry (SOCE) governs the Ca(2+) signaling requir
196 ing intracellular calcium and store-operated calcium entry (SOCE) in fast- and slow-twitch muscle fib
197 rine neutrophils show loss of store-operated calcium entry (SOCE) in response to both soluble ligands
198 nnel subunit that facilitates store operated calcium entry (SOCE) in T cells and is necessary for for
209 Transcriptional regulation by Store-operated Calcium Entry (SOCE) is well studied in non-excitable ce
212 ent advances in understanding store-operated calcium entry (SOCE) regulation, the fundamental questio
213 etion of ER calcium activates store-operated calcium entry (SOCE) through activation of the ER calciu
214 e of [Ca(2+)]cyt elevation is store-operated calcium entry (SOCE) through plasmalemmal calcium channe
215 ogical studies suggested that store-operated calcium entry (SOCE), a calcium refilling mechanism resp
216 ntrations in GC cells through store-operated calcium entry (SOCE), and then mediated Ca(2+)-dependent
217 ological characteristics with store-operated calcium entry (SOCE), is required to maintain baseline [
218 ed Ca(2+) signaling, known as store-operated calcium entry (SOCE), occurs downstream of immunorecepto
219 amatically inhibited (52-68%) store-operated calcium entry (SOCE), whereas suppression of TRPC4 or TR
230 tified two main components of store-operated calcium entry (SOCE): the endoplasmic reticulum-localize
231 , and indicate that inhibitors of ADPR-gated calcium entry, such as 8Br-ADPR, have the potential to b
233 olecular mechanism underlying store-operated calcium entry that replenishes ER stores in mouse Muller
234 tions augmented colbalt influx, a marker for calcium entry that selectively occurs through calcium pe
236 rimary pathway for so-called "store-operated calcium entry" - the cellular entry of calcium induced b
241 nductances generated by firing revealed that calcium entry through ICAT controls the emergence of the
242 ent with physiological findings showing that calcium entry through L-type calcium channels in pyramid
244 vious work has shown that activity-dependent calcium entry through L-type channels elevates perinucle
246 drugs approved for human use can antagonize calcium entry through L-type channels, these results poi
248 on at the neuromuscular junction, suggesting calcium entry through presynaptic N-type calcium channel
251 in the sarcoplasmic reticulum, by subsequent calcium entry through store-operated channels, and by in
252 the ductus arteriosus at birth is related to calcium entry through store-operated channels, encoded b
256 ion of the TRPM2 pore mutant E960D, in which calcium entry through TRPM2 is abolished, also resulted
257 reases in glutamate release, suggesting that calcium entry through TRPV1 channels may trigger glutama
258 action-potential duration, which would limit calcium entry through voltage-dependent calcium channels
260 alian motor nerve endings by reducing Ca(2+) calcium entry through voltage-gated calcium channels or,
262 ls is critical for calcium oscillations, but calcium entry through voltage-gated channels has much le
263 ine is associated with a reduction in Ca(2+) calcium entry through voltage-gated P/Q Ca(2+) channels
264 ntaining receptors on syntaxin-1 opening and calcium entry to enhance probability of vesicle fusion.
265 of neurotransmitters and drugs that inhibit calcium entry, transmitter release and nociception throu
266 xygenation status resulting in extracellular calcium entry, uncouples RGS9-2 from R7BP, triggering it
269 lly-uncoupled canine wedge model of enhanced calcium entry, using I(Ks) blockade with beta-adrenergic
270 maintain [Ca(2+)]i at extremely low levels; calcium entry usually occurs briefly, and within seconds
271 excitable cells are profoundly influenced by calcium entry via both store-operated and store-independ
272 to ER-plasma membrane junctions, leading to calcium entry via Ca(2+) release-activated Ca(2+) (CRAC)
273 ion was sensitive to apamin, phase-locked to calcium entry via Cav2.2 channels, and necessary for pre
274 These data suggest that during hypoxia, calcium entry via CRAC channels leads to AMPK activation
275 s expressing C4958S- or C4961S-RyR1 triggers calcium entry via ECCE that resembles that for wild-type
276 ients induced by tetanic stimulation rely on calcium entry via La(3+)- and nifedipine-sensitive calci
279 s well as thapsigargin, a known activator of calcium entry via store-operated channels, all increased
280 angiotensin to induce pericytes to contract, calcium entry via VDCCs serves to enhance the contractil
281 ansmission depends critically on presynaptic calcium entry via voltage-gated calcium (Ca(V)) channels
282 how that burst-induced depression depends on calcium entry via voltage-gated channels, is blocked by
291 selective blockers indicated that the lethal calcium entry was via reverse operation of a sodium-calc
292 UTP dose-dependent increase in capacitative calcium entry when calcium was added to the extracellula
293 es calcium-dependent inactivation and limits calcium entry, whereas CaBP1 blocks calcium-dependent in
294 affecting the cytoskeleton near the site of calcium entry, whereas calcium-dependent dendritic growt
295 ude, and shape of the AP falling phase alter calcium entry, which can affect neurotransmitter release
298 rs through a process known as store-operated calcium entry, which is initiated by calcium sensor prot
299 asmic reticulum (ER) induced large sustained calcium entry, which was blocked by SOC inhibitors, but
300 subunits, and observe the diffusive wave of calcium entry within the dendritic spine that follows ch
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