ライフサイエンスコーパス: calcium homeostasis

1 (NSP4) induces dramatic changes in cellular calcium homeostasis.

2 f AD, including altered lipid metabolism and calcium homeostasis.

3 actin-mediated, and disrupted intracellular calcium homeostasis.

4 ular system protein, is a major regulator of calcium homeostasis.

5 e force-frequency curve, suggesting improved calcium homeostasis.

6 are important in the regulation of cellular calcium homeostasis.

7 l function and survival, i.e., intracellular calcium homeostasis.

8 ell proliferation, cell differentiation, and calcium homeostasis.

9 ered markers of mitochondrial biogenesis and calcium homeostasis.

10 resiliency factor through its modulation of calcium homeostasis.

11 le membrane fragility, but also dysregulated calcium homeostasis.

12 eticulum Ca(2+) ATPase (SERCA) that disrupts calcium homeostasis.

13 and has been suggested to have functions in calcium homeostasis.

14 iating calcium spike activity and regulating calcium homeostasis.

15 cers of energy and play an important role in calcium homeostasis.

16 lism plays in the development of diseases of calcium homeostasis.

17 ation through increased load on dysregulated calcium homeostasis.

18 ochondria cooperate with the SER to maintain calcium homeostasis.

19 , impairs cell cycle progression and affects calcium homeostasis.

20 r barrier function, resulting in the loss of calcium homeostasis.

21 he endoplasmic reticulum and plays a role in calcium homeostasis.

22 eoclasts are essential for bone dynamics and calcium homeostasis.

23 trophy (DMD) include, among others, abnormal calcium homeostasis.

24 a mechanism involving the modulation of the calcium homeostasis.

25 not required for viability, reproduction, or calcium homeostasis.

26 min, a drug known to perturb ER ceramide and calcium homeostasis.

27 t through the affected channels and disrupts calcium homeostasis.

28 gin and clotrimazole (CLT), which disrupt ER calcium homeostasis.

29 id hormone (PTH), the principal regulator of calcium homeostasis.

30 ics of dark adaptation through modulation of calcium homeostasis.

31 al cells is tightly controlled during normal calcium homeostasis.

32 ranes that, among other functions, modulates calcium homeostasis.

33 te important processes such as apoptosis and calcium homeostasis.

34 lastic cells to regulate bone remodeling and calcium homeostasis.

35 cardiac muscle by maintaining cardiomyocyte calcium homeostasis.

36 ractile dysfunction through dysregulation of calcium homeostasis.

37 response to acute hypoxia and regulation of calcium homeostasis.

38 tion pathway and regulation of intracellular calcium homeostasis.

39 l, we arrive at a simple dynamical model for calcium homeostasis.

40 ients has been reported to result in altered calcium homeostasis.

41 remodeling and as an essential regulator of calcium homeostasis.

42 regulators in cardiac energy metabolism and calcium homeostasis.

43 ultures, SMA astrocytes exhibited defects in calcium homeostasis.

44 urther insights into the function of WFS1 in calcium homeostasis.

45 The kidney has a major role in extracellular calcium homeostasis.

46 hich allow for longitudinal monitoring of ER calcium homeostasis.

47 ation influx and disruption of intracellular calcium homeostasis.

48 MTC1 and TMTC2 as ER proteins involved in ER calcium homeostasis.

49 identify a new role for AP2 in extracellular calcium homeostasis.

50 als within a cell including dysregulation of calcium homeostasis.

51 optimization of the mechanisms that regulate calcium homeostasis.

52 o PPT1-deficiency increases ROS and disrupts calcium homeostasis activating caspase-9 and (ii) caspas

53 and gene expression of proteins important in calcium homeostasis after 4 wk.

54 viously reported disruption of mitochondrial calcium homeostasis after chronic doxorubicin administra

55 apparatus) proteins, although disturbance of calcium homeostasis also seems to be important.

56 PS is also involved in the regulation of calcium homeostasis, although the precise site of its ac

57 Here we investigated abnormal calcium homeostasis and altered exocytosis in SOD1G93A a

58 a link between myocardial isoform switching, calcium homeostasis and altered metabolism in the develo

59 response to mefloquine-induced disruption of calcium homeostasis and appropriate control agents were

60 ic hematopoiesis through maintenance of bone calcium homeostasis and are consistent with the concept

61 clude tissues involved in the maintenance of calcium homeostasis and bone development and remodeling.

62 Parathyroid hormone (PTH) is central to calcium homeostasis and bone maintenance in vertebrates,

63 yroid hormone (PTH) is the major mediator of calcium homeostasis and bone remodeling and is now known

64 The objective was to determine whether calcium homeostasis and bone turnover are affected by hi

65 es MTM1 mutations do not dramatically affect calcium homeostasis and calcium release mediated through

66 he major organelle involved in intracellular calcium homeostasis and calcium signaling, including cal

67 atory axis may be linked to dysregulation of calcium homeostasis and cardiac arrhythmias.

68 ion channels/pumps that alter intracellular calcium homeostasis and cause renin-independent aldoster

69 yzed the sequential steps leading to altered calcium homeostasis and cell death in response to activa

70 c stimulation, implicating its importance in calcium homeostasis and cell survival.

71 ttenuate vitamin D signaling associated with calcium homeostasis and cellular growth processes.

72 l or cell-matrix interaction, Rho signaling, calcium homeostasis and copper-binding/sensitive activit

73 -term OVL did not increase the alteration in calcium homeostasis and did not deplete muscle cell prog

74 wth with activation involving the effects on calcium homeostasis and downstream effects involving the

75 Vitamin D is known as a key player in calcium homeostasis and electrolyte and blood pressure r

76 M as redox sensors that function to modulate calcium homeostasis and energy metabolism in response to

77 ng the mechanisms involved in the control of calcium homeostasis and have provided evidence for a rol

78 pointes arrhythmias (TdP), while maintaining calcium homeostasis and hemodynamics.

79 lso support a role for ICS1 (SA) in iron and calcium homeostasis and identify components of SA cross

80 In this work, we develop a model for calcium homeostasis and identify integral feedback contr

81 whose actions include those associated with calcium homeostasis and immune responses as well as cell

82 Mutant neurons show a dysregulation of calcium homeostasis and increased vulnerability to stres

83 ption of high-protein diets does not disrupt calcium homeostasis and is not detrimental to skeletal i

84 combined lead to disruption of intracellular calcium homeostasis and isoproterenol-induced arrhythmia

85 ctor, which is shown to play a major role in calcium homeostasis and keratinocyte differentiation.

86 Our data suggest that impaired calcium homeostasis and mitochondrial dysfunction in PS1

87 We investigated calcium homeostasis and mitochondrial function in PINK1-

88 These data signify the essential role of calcium homeostasis and NCX1h in establishing rhythmic c

89 findings link mutations in TRPV4 to altered calcium homeostasis and peripheral neuropathies, implyin

90 nnel dOrai in the same pathway that promotes calcium homeostasis and phagocytosis.

91 oid hormone-related protein (PTHrP) in fetal calcium homeostasis and placental calcium transport was

92 cts of the anti-Abeta antibody aducanumab on calcium homeostasis and plaque clearance in aged Tg2576

93 of oxygen radicals as well as alteration in calcium homeostasis and possibly alteration in contracti

94 mpal neurons mimics the defects exhibited in calcium homeostasis and presynaptic function.

95 Overexpression of Bcl-2 normalizes ER calcium homeostasis and prevents calcium-mediated apopto

96 with broad functions that regulate cellular calcium homeostasis and reduce oxidative stress.

97 her module encloses many genes important for calcium homeostasis and signaling and contains SCA genes

98 hat sigma-1 receptor activation can regulate calcium homeostasis and signaling in RGCs, likely by dir

99 The overrepresentation of genes involved in calcium homeostasis and signaling may indicate an import

100 developments in the context of intracellular calcium homeostasis and signaling.

101 BP-D28k plays a critical role in maintaining calcium homeostasis and skeletal mineralization and sugg

102 teraction patterns with proteins involved in calcium homeostasis and sphingolipid metabolism could in

103 ynamic organ constantly remodeled to support calcium homeostasis and structural needs.

104 These findings establish a role for ERO1 in calcium homeostasis and suggest that modifying the lumen

105 ctively, our findings show that PS regulates calcium homeostasis and synaptic function via RyR and su

106 lications for the long-term monitoring of ER calcium homeostasis and the development of therapeutic a

107 Whereas the role of the mitochondrial calcium homeostasis and the Mitochondria Cacium Uniporte

108 onstrate that modulation of dynamic cellular calcium homeostasis and TXNIP suppression present viable

109 ding neurofilament filled swellings, loss of calcium homeostasis, and accumulation of reactive oxygen

110 ndoplasmic reticular stress, a disruption of calcium homeostasis, and activation of capase-4.

111 se mitochondrial membrane potential controls calcium homeostasis, and AMP-activated protein kinase (A

112 such as energy and intermediate metabolism, calcium homeostasis, and apoptosis.

113 rtant role in, for example, lipid synthesis, calcium homeostasis, and apoptotic signaling.

114 anscriptional regulation of key contraction, calcium homeostasis, and conduction genes.

115 neurotransmitter signaling, perturbations in calcium homeostasis, and damage-associated molecular pat

116 These channels couple lipid metabolism, calcium homeostasis, and electrophysiological properties

117 urite retraction, loss of synapses, aberrant calcium homeostasis, and imbalanced neurotransmitter rel

118 adical production, stabilization of cellular calcium homeostasis, and inhibition of apoptosis.

119 of basic mechanisms of electrical activity, calcium homeostasis, and metabolism.

120 ith the ER during phospholipid biosynthesis, calcium homeostasis, and mitochondrial fission.

121 servation of erythrocyte membrane integrity, calcium homeostasis, and osmotic resistance through an a

122 Regulation of K(+) channels, calcium homeostasis, and protein kinase C (PKC) activati

123 Mitochondria provide ATP, maintain calcium homeostasis, and regulate apoptosis.

124 ve been implicated in synaptic transmission, calcium homeostasis, and structural function and thus ma

125 transduction, regulation of gene expression, calcium homeostasis, and the maintenance of a unique ext

126 ed in several cellular processes, apoptosis, calcium homeostasis, and transcriptional regulation.

127 egulation of de novo sphingolipid synthesis, calcium homeostasis, and unfolded protein response.

128 ents), this study suggests that disorders of calcium homeostasis are associated with fatal and nonfat

129 an heart, myocyte function and intracellular calcium homeostasis are closely coupled.

130 transcriptomic analysis revealed changes in calcium homeostasis as a potential mechanism.

131 channels TRPV5 and TRPV6 play vital roles in calcium homeostasis as Ca(2+) uptake channels in epithel

132 rant beta-adrenergic signaling and depressed calcium homeostasis, associated with an imbalance of pro

133 Calcium homeostasis balances passive calcium leak and ac

134 Four constraints were imposed: calcium homeostasis before activation; stable in zero ex

135 In line with calcium homeostasis being a potential pathway mis-regula

136 rm black carbon [BC] and PM2.5 levels, serum calcium homeostasis biomarkers (parathyroid hormone, cal

137 tes, mutations in the presenilins also alter calcium homeostasis, but the mechanism linking presenili

138 oupled receptor family C, regulates systemic calcium homeostasis by activating G(q)- and G(i)-linked

139 s (CaR) contribute to regulation of systemic calcium homeostasis by activation of G(q)- and G(i)-link

140 The kidneys contribute to calcium homeostasis by adjusting the reabsorption and ex

141 Binding of these antibodies disrupted calcium homeostasis by both complement-dependent and com

142 is used to examine the regulation of ROS and calcium homeostasis by local, subcellular X-ROS signalin

143 tabolism and therefore in the maintenance of calcium homeostasis by vitamin D.

144 Mutations that affect calcium homeostasis (Ca(2+)) in rod photoreceptors are l

145 Here we show that modulation of cellular calcium homeostasis can mitigate cytokine- and ER stress

146 Disorders of calcium homeostasis caused by CaR mutations may therefor

147 The cod1Delta mutant disrupted cellular calcium homeostasis, causing increased transcription of

148 The epidermal adrenergic signal controls calcium homeostasis, cell growth, differentiation, motil

149 Thus, although alterations in intracellular calcium homeostasis contribute to glucocorticoid-induced

150 through modulation of SERCA and maintaining calcium homeostasis could be a therapeutic aim for bette

151 line in mechanisms controlling intracellular calcium homeostasis could contribute to altered neuronal

152 Our data thus show disturbed calcium homeostasis coupled with mitochondrial dysfuncti

153 r endothelial cells, TRPV4 channels regulate calcium homeostasis, cytoskeletal signalling and the org

154 ypercalcemia type 3 (FHH3), an extracellular calcium homeostasis disorder affecting the parathyroids,

155 oid plaques in an acute setting and restores calcium homeostasis disrupted in a mouse model of AD upo

156 ty, injuring sarcolemmal membranes, altering calcium homeostasis due to effects on the sarcoplasmic r

157 d the hypothesis that alterations in cardiac calcium homeostasis due to sepsis underlie the observed

158 nflux at afferent ribbon synapses influences calcium homeostasis during long-lasting cholinergic inhi

159 robust model to understand dysregulation of calcium homeostasis during virus infections.

160 ing low-dose caffeine to mimic the defective calcium homeostasis encountered under these conditions.

161 on, modulation of endoplasmic reticulum (ER) calcium homeostasis, ER stress signaling, autophagy, rea

162 form a novel signaling module that controls calcium homeostasis following T cell activation.

163 g TORC2, mitochondrial oxidative stress, and calcium homeostasis for autophagy regulation.

164 usly expressed membrane protein essential in calcium homeostasis for many cells including those in ma

165 eal a regulatory role of the PLN pentamer in calcium homeostasis, going beyond the previously hypothe

166 Calcium homeostasis has also been shown to be perturbed

167 Downstream of mGluR1, dysregulation of calcium homeostasis has been hypothesized as a key patho

168 ogenesis, cell proliferation, apoptosis, and calcium homeostasis have been attributed to its N termin

169 s that molecules responsible for maintaining calcium homeostasis have multiple roles.

170 s a critical role in cellular energetics and calcium homeostasis; however, how MAM is affected under

171 mic reticulum (ER) stress, including loss of calcium homeostasis, hypoxia and oxidative stress.

172 n up by neurons causing: (i) deregulation of calcium homeostasis, (ii) endoplasmic reticulum-calcium

173 ite degeneration, loss of synapses, aberrant calcium homeostasis, imbalanced neurotransmitter release

174 in vivo, we quantitatively imaged astrocytic calcium homeostasis in a mouse model of Alzheimer's dise

175 arrhythmia syndrome associated with abnormal calcium homeostasis in a mouse model.

176 te the effect of sigma-1 receptor ligands on calcium homeostasis in a retinal ganglion cell line (RGC

177 n of caspase-12 activation and its effect on calcium homeostasis in an ER stress-induced model of apo

178 um Ca(2+)-ATPase (SERCA), thereby regulating calcium homeostasis in cardiac muscle.

179 turbance in endoplasmic reticulum-associated calcium homeostasis in cultured embryonic motor neurons

180 Our data implicate a central role for calcium homeostasis in excystation.

181 n D3 (1,25(OH)2D3) plays an integral role in calcium homeostasis in higher organisms through its acti

182 n contractility despite normal intracellular calcium homeostasis in intact cardiomyocytes and resulte

183 t manner, demonstrating the critical role of calcium homeostasis in maintaining embryonic cardiac fun

184 levels of ion channels involved in cellular calcium homeostasis in mouse cortical microglial cells i

185 ntry (SOCE) is thought to primarily regulate calcium homeostasis in neurons.

186 nserved role of dematin in the regulation of calcium homeostasis in other cell types will be discusse

187 glutamate receptor signalling and disrupted calcium homeostasis in PNs form a common, early pathophy

188 bellar ataxia type 2 (SCA2) mouse model that calcium homeostasis in PNs is disturbed across a broad r

189 hus, TRPC2 functions as a major regulator of calcium homeostasis in rat thyroid cells.

190 ous system, where it regulates intracellular calcium homeostasis in response to excitatory signaling.

191 t the NFkappaB activity may help to maintain calcium homeostasis in SGNs.

192 Thus, APP contributes to calcium homeostasis in situations of metabolic stress.

193 lays an important role in maintaining ER and calcium homeostasis in T lymphocytes.

194 pocalcemic hormone important for maintaining calcium homeostasis in teleost fish.

195 elation between IP(3)R elevation and altered calcium homeostasis in terms of either kinetics or dose

196 K protein or by thapsigargin, which disrupts calcium homeostasis in the endoplasmic reticulum.

197 iquitin-proteasome system and alterations of calcium homeostasis in the neuronal loss observed during

198 se may alter glutamate neurotransmission and calcium homeostasis in the retina, which may have implic

199 The hypertension-induced alterations of calcium homeostasis in the soleus muscle of SHRs occurre

200 a simple and sensitive method to monitor ER calcium homeostasis in vitro or in vivo by analyzing cul

201 onstrate that senile plaques impair neuritic calcium homeostasis in vivo and result in the structural

202 harmacological approach to restore cytosolic calcium homeostasis in vivo, we administered the clinica

203 udy provide evidence for redox regulation of calcium homeostasis in yeast cells.

204 Recent work suggests that HCV also alters ER calcium homeostasis, inducing oxidative stress.

205 Cellular stresses such as disruption of calcium homeostasis, inhibition of protein glycosylation

206 ellular stress signals such as disruption of calcium homeostasis, inhibition of protein glycosylation

207 t 9-fold from the target organs required for calcium homeostasis (intestine, bone, kidney, and parath

208 Because calcium homeostasis is a biomarker of muscle function, w

209 al changes, supports the view that disturbed calcium homeostasis is an early feature of Parkinson's d

210 strongly suggests that imbalance in cellular calcium homeostasis is an important factor leading to CD

211 A disturbance of calcium homeostasis is believed to play an important rol

212 To address whether the role of CaBP-D28k in calcium homeostasis is compensated by CaBP-D9k, we gener

213 Calcium homeostasis is critical for cardiac myocyte func

214 Endoplasmic reticulum (ER) calcium homeostasis is disrupted in diverse pathologies,

215 Precise regulation of calcium homeostasis is essential for many physiological

216 Calcium homeostasis is essential for the normal function

217 grity, and active adaptation at the level of calcium homeostasis is not mechanistic in protection.

218 y support the hypothesis that maintenance of calcium homeostasis is one function of complex gangliosi

219 e mechanism by which glucocorticoids disrupt calcium homeostasis is unknown.

220 Altered neuronal calcium homeostasis is widely hypothesized to underlie c

221 ave shown that ion homeostasis, particularly calcium homeostasis, is critical to limiting tissue dama

222 osphatase (SERCA)2a, a critical regulator of calcium homeostasis, is known to be decreased in heart f

223 lcitonin (CT), a peptide hormone involved in calcium homeostasis, is transiently expressed in the rec

224 uggest that mutant LRRK2 causes a deficit in calcium homeostasis, leading to enhanced mitophagy and d

225 PC1 disease pathogenesis that causes altered calcium homeostasis, leading to the secondary storage of

226 Blocking KCa1.1 disturbs calcium homeostasis, leading to the sustained phosphoryl

227 enes involved in cell adhesion and motility, calcium homeostasis, lipid transport and metabolism, and

228 ght to occur include a loss of intracellular calcium homeostasis, loss of energy supply to the cell,

229 Modulators of intracellular calcium homeostasis, MAPTAM, and TMB-8 blocked ookinete

230 Together, the results indicate that altered calcium homeostasis may be a key early event in basal to

231 and suggest that disruption of intracellular calcium homeostasis may be an early pathogenic event lea

232 Altered glutamatergic neurotransmission and calcium homeostasis may contribute to retinal neural cel

233 ta suggest that BI-1, through its actions on calcium homeostasis, may confer affective resiliency in

234 licated in many neuronal processes including calcium homeostasis, membrane excitability, synaptic tra

235 been used to study metabolism, mitochondrial calcium homeostasis, mitochondrial membrane potential, a

236 the MPT differentially affect mitochondrial calcium homeostasis: mitoK(ATP) channels suppress calciu

237 Calcium homeostasis modulator 1 (CALHM1) expression was

238 Here we show that calcium homeostasis modulator 1 (CALHM1), a voltage-gate

239 CALHM1 (calcium homeostasis modulator 1) forms a plasma membrane

240 This strategy identified CALHM1 (calcium homeostasis modulator 1), a gene modulating AD a

241 vated by Ca(2+) signaling via opening of the calcium-homeostasis modulator-1 (CALHM1) channel.

242 Targeted STIM deletion impairs calcium homeostasis, NFAT activation, and growth of smoo

243 n implicated as one of the causes of altered calcium homeostasis observed during human heart failure.

244 Derangements in calcium homeostasis occur during sepsis that is sensitiv

245 pigment pyocyanin, which is known to disrupt calcium homeostasis of host cells.

246 show that blocking this channel perturbs the calcium homeostasis of the cells and inhibits the prolif

247 understanding of the regulation of bone and calcium homeostasis on Earth.

248 se observations suggest an important role of calcium homeostasis on the Apaf-1-dependent apoptotic pa

249 dentified have functions related to neuronal calcium homeostasis or central nervous system developmen

250 nd this effect was not due to alterations in calcium homeostasis or myosin light chain phosphorylatio

251 D3 [1,25(OH)2D3] to perform its function in calcium homeostasis, or it is activated by lithocholic a

252 The disruption of intracellular calcium homeostasis plays a central role in the patholog

253 Calcium homeostasis plays a major role in maintaining ne

254 these results establish a link between TgA1, calcium homeostasis, polyP storage and virulence.

255 rticoid-induced alterations in intracellular calcium homeostasis promote apoptosis, but the mechanism

256 etoxification enzymes, antioxidant proteins, calcium homeostasis proteins, growth factors, neuron-spe

257 ive oxygen species production and signaling, calcium homeostasis, regulated cell death, and heme bios

258 ; consistently, tissue-specific induction of calcium homeostasis-related genes and suppression of gro

259 tabolic derangements (particularly involving calcium homeostasis), renal failure, and possibly, morta

260 phenotypes, such as alkaline pH sensitivity, calcium homeostasis, respiratory defects, and cell wall

261 oA dehydrogenase, and uncoupling protein 3), calcium homeostasis (sarcoplasmic reticulum Ca(2+)-ATPas

262 naptic cistern may play an essential role in calcium homeostasis, serving as sink or source, dependin

263 fiber growth (GDF8), structure (Actg1), and calcium homeostasis (Stim1 and Jph1).

264 and rcy1), all were found to have defective calcium homeostasis, supporting a correlation with amiod

265 tions at the level of glial gene expression, calcium homeostasis, swelling, and volume regulation.

266 oid hormone (PTH), an important regulator of calcium homeostasis, targets most of its complex actions

267 t that PMCAs may play a more complex role in calcium homeostasis than generally recognized.

268 min D is a secosteroid with known effects on calcium homeostasis that has recently been shown to have

269 ole for alpha 3 gap junctions in maintaining calcium homeostasis that in turn is required to control

270 oncentrations induce a stochastic failure of calcium homeostasis that precedes both mitochondrial dep

271 to the generation of ATP and maintenance of calcium homeostasis, the effective delivery of mitochond

272 nes are known to play a contributory role in calcium homeostasis, the entire caste of key components

273 itially thought to play a restricted role in calcium homeostasis, the pleiotropic actions of vitamin

274 aused a phenotype-dependent dysregulation of calcium homeostasis; the resting intracellular calcium o

275 Vpr disturbs calcium homeostasis through downregulation of endogenous

276 hyroid hormone (PTH) regulates extracellular calcium homeostasis through the type 1 PTH receptor (PTH

277 structures in vertebrates and helps maintain calcium homeostasis throughout life.

278 well as core biological processes mediating calcium homeostasis, tissue integrity, cornification, an

279 fficking of ion channel subunits involved in calcium homeostasis to and from the plasma membrane.

280 e currently no treatments targeting cellular calcium homeostasis to combat type 1 diabetes.

281 er se, is the key effector of Drp1, altering calcium homeostasis to modulate neuronal morphology and

282 data link defects in neuronal intracellular calcium homeostasis to the vulnerability of central auto

283 ecies, electron transport chain function and calcium homeostasis trigger altered mitochondrial dynami

284 to explore the ability of BI-1 to influence calcium homeostasis under basal conditions and also foll

285 These results suggest that disruption of calcium homeostasis underlies isoflurane-induced caspase

286 by which PS regulates synaptic function and calcium homeostasis using acute hippocampal slices from

287 have shown that mefloquine disrupts neuronal calcium homeostasis via liberation of the endoplasmic re

288 ion due to its ability to stabilize cellular calcium homeostasis via N-methyl-D-aspartate-receptor an

289 upts mitochondrial and endoplasmic reticulum calcium homeostasis via ryanodine receptor (RyR) activat

290 etabolite, NAADP(+), regulates intracellular calcium homeostasis via the 2-pore channel, ryanodine re

291 Parathyroid hormone (PTH) regulates calcium homeostasis via the type I PTH/PTH-related pepti

292 gland VDR content is an important factor in calcium homeostasis, vitamin D metabolism, and the treat

293 ne permeability and consequent disruption of calcium homeostasis were implicated in cellular degenera

294 In contrast to the regulation of calcium homeostasis, which has been extensively studied

295 heimer's disease (AD), disturb intracellular calcium homeostasis, which in turn activates the calcium

296 dergo increased oxidative stress and altered calcium homeostasis, which may contribute to myofiber lo

297 utophagic/lysosomal system and intracellular calcium homeostasis, which underlie vulnerability to neu

298 Calcitriol, a regulator of calcium homeostasis with antitumor properties, is degrad

299 hat PTHrP is an important regulator of fetal calcium homeostasis with its predominant effect being on

300 suggesting a dysregulation of intracellular calcium homeostasis within the muscle or an alteration o