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1 ne 25-hydroxyvitamin D level, and additional calcium intake.
2 , particularly in settings of habitually low calcium intake.
3 reduced risk even in participants with lower calcium intake.
4 time from blood collection to diagnosis, or calcium intake.
5 o consider the influences of body weight and calcium intake.
6 the interaction between vitamin D status and calcium intake.
7 observed with weight loss at normal or high calcium intake.
8 ccretion in boys requires additional dietary calcium intake.
9 ed calcium supplements to ensure an adequate calcium intake.
10 ge of threshold values in part due to varied calcium intake.
11 ascular risks associated with high levels of calcium intake.
12 ium, and 0.97 (95% CI: 0.87, 1.09) for total calcium intake.
13 gen, physical activity, smoking, and current calcium intake.
14 iability in calcium balance than does actual calcium intake.
15 cause most studies have focused attention on calcium intake.
16 y during lactation, even in women with a low calcium intake.
17 nt predictor of bone mineral density than is calcium intake.
18 on without requiring an increase in maternal calcium intake.
19 ved during lactation reflect inadequacies in calcium intake.
20 compared with girls who continued their low calcium intake.
21 % CI: 0.94, 1.02) for a 300-mg/d increase in calcium intake.
22 r mortality risk was observed with increased calcium intake.
23 on depended on circulating 25(OH)D and total calcium intake.
24 res, and this association may be modified by calcium intake.
25 mg/d) (P < 0.05), with no effects of race or calcium intake.
26 reasing body mass index (BMI) on recommended calcium intakes.
27 regnant adolescents and adult women with low calcium intakes.
28 ss at recommended or higher than recommended calcium intakes.
29 gnancy and early lactation in women with low calcium intakes.
30 akes and inversely associated with fiber and calcium intakes.
31 f even those individuals who have inadequate calcium intakes.
33 ke (<700 mg/d; RR for a 300-mg/d increase in calcium intake: 0.82; 95% CI: 0.76, 0.88) but was weakly
35 signed to examine relations between maternal calcium intake, 25-hydroxyvitamin D [25(OH)D] status, an
38 leton, but there is some indication that low calcium intakes adversely influence the effect of dietar
39 correlation between total hip BMD and dairy calcium intake after adjustment for age, race, and weigh
40 versus 1st (median = 211 mg/d) quartiles of calcium intake after adjustment for potential confounder
42 ine whether acute or chronic increased dairy calcium intakes alter postprandial whole-body fat oxidat
46 ociation between self-reported supplementary calcium intake and AMD was stronger in older than younge
47 e study was to evaluate the relation between calcium intake and balance in healthy children aged 1-4
48 stratifications by compliance-adjusted total calcium intake and by final stature or metacarpal total
49 rt on the evaluation of the relation between calcium intake and calcium absorption and retention.
51 was used to examine the association between calcium intake and DeltaBW or DeltaWC adjusted for concu
53 res), there was no association between total calcium intake and hip fracture risk [pooled risk ratio
55 rb calcium with age limits adaptation to low calcium intake and is thought to lead to secondary hyper
62 The findings from epidemiologic studies of calcium intake and risk of stroke have been conflicting.
65 nsistent dose-response relationships between calcium intake and risks for total stroke or stroke mort
67 s offer long-term feasibility for increasing calcium intake and serum 25-hydroxyvitamin D concentrati
68 rceived lactose intolerance as it relates to calcium intake and specific health problems that have be
70 e processes that are independent of maternal calcium intake and that provide the calcium necessary fo
75 ates of bone calcium turnover in relation to calcium intakes and circulating concentrations of parath
78 ed the association between dairy product and calcium intakes and prostate cancer risk in the Physicia
83 ciations with absorption were found for age, calcium intake, and estrogen status, no association was
84 tween fractional calcium absorption, dietary calcium intake, and risk for fracture have never been st
85 bsorption efficiency were menopausal status, calcium intake, and serum estradiol and serum 1,25(OH)(2
86 ight, physical activity, menopause duration, calcium intake, and the interaction between calcium and
87 atston score decreased with increasing total calcium intake, and the trend was not significant after
89 pregnant teens, populations with inadequate calcium intake, and women at risk of developing PIH, may
90 Gambian women were adapted to a low dietary calcium intake, and/or obesity, high gestational weight
91 between self-perceived lactose intolerance, calcium intakes, and physician-diagnosed health conditio
93 and n10, respectively) in rural Gambia where calcium intakes are low with little seasonality in UVB-e
95 etention than did whites (P < 0.0001) at all calcium intakes as a result of significantly greater net
96 o be dependent on, or at least augmented by, calcium intakes at or above currently recommended levels
97 ling for known determinants (including daily calcium intake, average daily time spent in moderate-to-
98 with HIV infection should emphasize adequate calcium intakes because of the importance of this age pe
102 g to be more beneficial for women with lower calcium intakes, but evidence for this interaction was n
104 ance [defined as calcium output (Y) equal to calcium intake (C)] at intakes of 741 mg/d [95% predicti
107 n subgroup analyses, women with higher total calcium intake (diet plus supplements) at baseline were
108 to either a normal (1 g/d) or high (1.8 g/d) calcium intake during 6 mo of energy restriction [weight
109 date, the molecular mechanism that leads to calcium intake during CTL differentiation and function h
110 re are firm data that demonstrate that a low calcium intake during lactation does not lead to impaire
111 (n = 15) were significantly associated with calcium intake during pregnancy (y = -3.53 + 0.107x; R(2
112 Evidence suggests that increased maternal calcium intake during pregnancy may result in lower offs
113 exist on the intergenerational influence of calcium intake during pregnancy on offspring blood press
116 n in the early postpartum period, and higher calcium intakes during pregnancy appeared to be protecti
118 hird, and fourth quartiles, respectively, of calcium intake for women (n=380;P<0.0009); a similar inv
120 isms for the relation between an increase in calcium intake from calcium carbonate or dairy and weigh
121 lerant respondents had a significantly lower calcium intake from dairy foods and reported having a si
123 this study, we examined associations between calcium intake from diet and supplements and measures of
124 ot support a substantial association between calcium intake from diet or supplements and CVD risk in
125 In light of the evidence available to date, calcium intake from food and supplements that does not e
126 een vitamin D and all vascular measures, and calcium intake from phosphate binders weakly correlated
129 significantly lower (P < 0.05) average daily calcium intakes from dairy foods than did those without
133 icant (P = 0.2) time x vitamin D treatment x calcium intake grouping interaction effect on the mean s
135 wer mortality, whereas for men, supplemental calcium intake >/=1000 mg/d may be associated with highe
137 ociated with risk in populations with a high calcium intake (>/=700 mg/d; corresponding RR: 1.03; 95%
138 articipants in the highest quintile of total calcium intake (>1767 mg/d) than for participants in the
145 elation between changes in fat mass gain and calcium intake in preschool children, who typically cons
147 ol studies that examined differences in mean calcium intake in the case compared with the control gro
151 = 0.74) with an increasing quartile of total calcium intake in women and 4.32, 4.39, 4.19, and 4.37 (
152 dy does not support the hypothesis that high calcium intake increases coronary artery calcification,
153 nal calcium absorption in the setting of low calcium intake increases the risk for hip fracture.
155 hat WL decreases TFCA and suggest that, when calcium intake is 1.2 g/d, either 10 or 63 mug vitamin D
160 from prospective cohort studies suggest that calcium intake is not significantly associated with hip
162 and adolescence, when controlled for current calcium intake, is associated with adult bone mass (ie,
164 hips between total, dietary, or supplemental calcium intake levels and cardiovascular mortality and h
165 luded milligrams of dietary and supplemental calcium intake, likelihood of meeting national calcium a
166 were of similar magnitudes in subjects with calcium intakes <700 mg/d (and even <550 mg/d) compared
167 When examined one at a time, very low calcium intake (<60% of EAR), very low 25(OH)D (<12 ng/m
168 n populations with a low to moderate average calcium intake (<700 mg/d; RR for a 300-mg/d increase in
169 (PTH) (>62 pg/mL) accompanied by a very low calcium intake [<60% of the Estimated Average Requiremen
171 lactation in adolescents with habitually low calcium intake may adversely affect maternal bone mass.
172 40 years, investigators have suggested that calcium intake may be associated with alveolar bone reso
176 t that supplementing maternal midgestational calcium intake may lower offspring blood pressure, thus
178 ar exercise and adherence with vitamin D and calcium intake may reduce the risk of hip fracture.
180 improved calcium balance; therefore, greater calcium intakes may minimize bone loss across pregnancy
181 ancy in Gambian women with very low habitual calcium intakes may not result in lower offspring blood
182 calcium retention was calculated as dietary calcium intake minus the calcium excreted in the feces a
183 y (n = 125) who were stratified according to calcium intakes [moderate-low (<700 mg/d) or high (>1000
184 ne calcium intake (n = 23,504) and change in calcium intake (n = 19,615) were associated with weight
185 te linear regression to examine how baseline calcium intake (n = 23,504) and change in calcium intake
188 fect of 6 wk of WL at 2 different amounts of calcium intake [normal (NlCa): 1 g/d; high (HiCa): 1.8 g
190 inear modeling of balance data showed that a calcium intake of approximately 470 mg/d led to calcium
191 following American diets are met by a daily calcium intake of approximately 470 mg/d, which suggests
193 eal 13- to 14-y-old overweight girls who had calcium intakes of </=600 mg/d in a 12-mo randomized con
194 During the development of peak bone mass, calcium intakes of <1 g/d are associated with lower bone
197 cts was 79.1 +/- 25.6 g/d and the mean total calcium intakes of the supplemented and placebo groups w
199 lead exposure, breastfeeding practices, and calcium intake on breast milk lead levels over the cours
200 synergistic effects of vitamin D status and calcium intake on calcium absorption; 2) effects of calc
201 systematic reviews to examine the effects of calcium intake on cardiovascular disease (CVD) among gen
203 mixed models to estimate effects of maternal calcium intake on offspring systolic blood pressure.
205 intake on calcium absorption; 2) effects of calcium intake on vitamin D status; and 3) largely obser
206 t support the hypothesis that an increase in calcium intake or dairy consumption is associated with l
207 al calcium metabolic stress, rather than low calcium intake or insufficient vitamin D, has an adverse
208 s in early pregnancy, rather than suboptimal calcium intake or insufficient vitamin D, influences the
209 healthy populations who do not have very low calcium intakes or serum 25-hydroxyvitamin D concentrati
210 drugs (OR 0.4, 95% CI 0.2-0.9) or had higher calcium intakes (OR 0.4, 95% CI 0.2-0.9) if they had no
212 tive association was evident between VO+ and calcium intake (P </= 0.002) and between VO+ and African
215 associations were positive for magnesium and calcium intake (p=0.016) after adjusting for demographic
217 rsonal and family history of fracture, total calcium intake, past use of hormone therapy, BMD, or sum
219 ed race, sex, height, weight, energy intake, calcium intake, physical activity measured by accelerome
220 dings provide new longitudinal evidence that calcium intake predicts bone mineral status during middl
221 research is required to define whether a low calcium intake prior to or during pregnancy can have del
222 05, P = 0.5) or total (dietary + supplement) calcium intake (r = -0.02, P = 0.8 and r = -0.06, P = 0.
223 sorption was inversely associated with total calcium intake (r = -0.18, P = 0.030), dietary fiber int
228 for men and women, we determined the dietary calcium intake required to maintain neutral calcium bala
229 ion during pregnancy of adolescents with low calcium intake results in higher lumbar spine bone mass
230 ntation of pregnant Gambian women with a low calcium intake results in lower maternal bone mineral co
232 ndicate that small children may benefit from calcium intakes similar to those recommended for older c
233 Results stratified by baseline vitamin D and calcium intake, solar irradiance, and other factors were
236 rticularly marked in populations in whom low calcium intake, stunting, and delayed puberty are common
237 Underreporters reported lower dairy and calcium intakes than did plausible reporters; the result
239 espectively; P = 0.0094), and men had higher calcium intakes than did women (735 and 655 mg/d, respec
241 er, crossover metabolic study with 3 dietary calcium intakes; the magnesium dietary intake was fixed
242 ing the current recommendations for adequate calcium intake through diet alone or with supplements.
245 elations of previous milk intake and current calcium intake to current bone mineral measures were inv
246 icient to modify current recommendations for calcium intake to protect skeletal health with respect t
247 .The aim was to determine whether increasing calcium intake to recommended amounts with dairy foods i
248 rmine the effect of the doubling of habitual calcium intake to the recommended intake from dairy or c
249 e of the study was to assess the relation of calcium intake to the risk of hip fracture on the basis
250 activity score, smoking, alcohol use, total calcium intake, total vitamin D intake, caffeine from no
251 ong women in the highest quartile of dietary calcium intake versus the lowest (p for trend = 0.0006).
252 ent for age, body mass index, energy intake, calcium intake, vitamin D intake, smoking status, physic
260 ompanied by insufficient 25(OH)D or very low calcium intake was associated with a 2- to 3-fold increa
264 zards regression models, postdiagnosis total calcium intake was inversely associated with all-cause m
267 uent hip fracture; among women whose dietary calcium intake was less than 400 mg/d, those who had fra
268 calcium primarily from the diet, whose total calcium intake was lower than that in those who obtained
269 cohorts and in the whole group (P = 0.029); calcium intake was not a significant determinant of VO+
277 of protein in the diet, the protein source, calcium intake, weight loss, and the acid/base balance o
278 s for the lowest to the highest quintiles of calcium intake were 1.00, 0.82, 0.73, 0.67, and 0.74 (P
279 th low fractional calcium absorption and low calcium intake were at greatest risk for subsequent hip
281 nd the variations in these associations with calcium intake were studied in a community-dwelling coho
283 ther age nor sex affected the estimates when calcium intakes were expressed as mg/d or as mg kg body
284 fractures since age 45 years and low dietary calcium intake, were associated with increased risks of
285 es, but not nondairy calcium or supplemental calcium intakes, were also positively associated with to
287 When 25(OH)D was insufficient, even a high calcium intake (which equaled or exceeded the Recommende
288 he 9 case-control studies that examined mean calcium intake, which had appropriate data for the meta-
289 idence of a vitamin D sparing effect of high calcium intake, which has been referred to by some autho
290 e bone during weight loss at the recommended calcium intake, which may be explained by sufficient amo
292 colon adenoma to evaluate the association of calcium intake with early stages of colorectal tumor dev
293 ssociation of habitual dairy, magnesium, and calcium intake with insulin sensitivity at baseline and
294 orted RRs and 95% CIs for the association of calcium intake with stroke incidence or mortality were e
295 ciations of supplemental, dietary, and total calcium intakes with all-cause, CVD-specific, and cancer
296 on the relation of magnesium, potassium, and calcium intakes with stroke risk are inconsistent, and t
298 avidae with insufficient 25(OH)D or very low calcium intake without elevated PTH or with elevated PTH
299 oods in adolescent girls with habitually low calcium intakes would decrease body fat gain compared wi
300 d, for example, are raised on relatively low calcium intakes yet have less osteoporosis than those wh
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