ライフサイエンスコーパス: calcium ionophore

1 induced by sperm (but twice that achieved by calcium ionophore).

2 oted after treatment with cAMP and A23187 (a calcium ionophore).

3 n buffer using phorbol myristate actetate or calcium ionophore.

4 when both cell populations were activated by calcium ionophore.

5 ls more slowly in response to treatment with calcium ionophore.

6 ssion in mast cells stimulated with PMA plus calcium ionophore.

7 15-EET when the cells were stimulated with a calcium ionophore.

8 enerated by activating washed platelets with calcium ionophore.

9 occurred when sporozoites were treated with calcium ionophore.

10 e nuclear envelope upon stimulation with the calcium ionophore.

11 lon mAb, or stimulated with combined PMA and calcium ionophore.

12 the impaired relaxation to acetylcholine or calcium ionophore.

13 ts and breast cancer cells when treated with calcium ionophore.

14 nduced upon treatment with phorbol ester and calcium ionophore.

15 of polychlorinated biphenyls, and A23187, a calcium ionophore.

16 pid calcium influx induced by 1 and 5 microM calcium ionophore.

17 ither serum deprivation or by treatment with calcium ionophore.

18 to phenylephrine and enhanced relaxations to calcium ionophore.

19 th was induced by glucocorticoid (6-48 h) or calcium ionophore.

20 elaxant responses to acetylcholine (ACh) and calcium ionophore.

21 tored after treatment with phorbol ester and calcium ionophore.

22 and phosphorylation decrease in response to calcium ionophore.

23 tosis stimulated by cytokine deprivation and calcium ionophore.

24 n cells in response to interleukin-1beta and calcium ionophore.

25 d by treatment with either phorbol esters or calcium ionophores.

26 als delivered through the FcepsilonRI or via calcium ionophores.

27 gher concentrations, presumably by acting as calcium ionophores.

28 demonstrated augmented maximal relaxation to calcium ionophore (32+/-4.5% versus 17.4+/-7.4%; n=6; P<

29 necessary for inhibition of NHE3 activity by calcium ionophore 4-Br-A23187.

30 l; 17.8 microm) and then stimulated with the calcium ionophore, (5 microm).

31 flammatory agonist N-formyl peptides (FMLP), calcium ionophore A(23187), or phorbol mirystate acetate

32 This synthesis was enhanced by the calcium ionophore A-23187, by IL-1beta, or by supplying

33 hyl ether)-N-N' -tetraacetic acid (EGTA) and calcium ionophore (A-23187) on phosphorus uptake was stu

34 Additionally, the calcium ionophore, a reagent that promotes nitric oxide

35 upon stimulation with lipopolysaccharide and calcium ionophore A21387, as compared with that in untre

36 topical application of bradykinin (-59%) or calcium ionophore A23187 (-49%) and by systemic hypercap

37 ent with staurosporine (20-100 ng/mL) or the calcium ionophore A23187 (0.5 microM).

38 m the lens is tightly regulated, neither the calcium ionophore A23187 (1 microM) nor depolarization b

39 orophenylhydrazone (CCCP, 10 microM) and the calcium ionophore A23187 (10 microM), abolished TPP+ acc

40 al conditions and after stimulation with the calcium ionophore A23187 (10(-6) M).

41 he receptor-independent endothelial agonist, calcium ionophore A23187 (10(-7) mol/L and 3 x 10(-7) mo

42 te flow (from 0 to 25 microL/min) and to the calcium ionophore A23187 (5 x 10(-8) to 10(-6) mol/L), n

43 Ch, 100+/-2% versus 75+/-2%, P<0.05) and the calcium ionophore A23187 (92+/-2% versus 72+/-3%, P<0.05

44 ther recombinant TNF alpha supernatants from calcium ionophore A23187 (CaI)-stimulated mast cells or

45 rmal livers in response to acetylcholine and calcium ionophore A23187 (P < .0001).

46 h phorbol 12,13-dibutyrate together with the calcium ionophore A23187 also promoted ubiquitination an

47 hat increase intracellular calcium (i.e. the calcium ionophore A23187 and thapsigargin) or protein ki

48 Calcium ionophore A23187 and the muscarinic cholinergic

49 ted degranulation of storage granules by the calcium ionophore A23187 caused release of ET into the c

50 Treatment of infected host cells with the calcium ionophore A23187 causes the parasites to undergo

51 The calcium ionophore A23187 destabilized YY1 in cultured my

52 tracellular cyclic AMP or treatment with the calcium ionophore A23187 do not cause SPR phosphorylatio

53 Activation of cPLA(2)alpha by the calcium ionophore A23187 enhanced PPARdelta binding to P

54 MC-9 cells were stimulated with calcium ionophore A23187 for 15 min in the presence or t

55 Although the calcium ionophore A23187 increased the expression of CD1

56 Interestingly, the calcium ionophore A23187 induced accumulation of a polyu

57 ists such as collagen, ADP, epinephrine, and calcium ionophore A23187 induced RAFTK phosphorylation.

58 inhibited ACTH-induced SAPK activity and the calcium ionophore A23187 induced SAPK activity 3-fold.

59 The calcium ionophore A23187 induced SFK phosphorylation in

60 of intact aortic endothelial cells with the calcium ionophore A23187 leads to the rapid disruption o

61 of HUVECs or isolated mice aortas to either calcium ionophore A23187 or bradykinin significantly inc

62 en Jurkat cells were treated with either the calcium ionophore A23187 or thapsigargin, z-VAD failed t

63 e also show that treatment of LNCaP with the calcium ionophore A23187 or the phorbol ester phorbol 12

64 Addition of calcium ionophore A23187 produced a rapid release of 20-

65 Treatment with the calcium ionophore A23187 recapitulated the force-induced

66 ytes with LPS followed by treatment with the calcium ionophore A23187 resulted in the formation of PG

67 nase C by TPA or increases in [Ca2+]i by the calcium ionophore A23187 stimulated p130(Cas) phosphoryl

68 hed using the chemotherapy drug VP-16 or the calcium ionophore A23187 to induce apoptosis in Burkitt'

69 Adding calcium ionophore A23187 to Mphi inoculated with Mtb fur

70 oethyl ether)-N,N'-tetraacetic acid (EGTA) + calcium ionophore A23187 treatments resulted in reduced

71 Receptor-independent relaxation to calcium ionophore A23187 was not significantly impaired

72 to acetylcholine and to a lesser extent the calcium ionophore A23187 were highly variable and correl

73 Basal and stimulated (by the calcium ionophore A23187) secretion of NO and intracellu

74 in, 0.1 U/ml; collagen, 4 microg/ml; and the calcium ionophore A23187, 1 microM) results in shedding

75 Stimulation of these vessels with calcium ionophore A23187, a known secretagogue of Weibel

76 ivated with bacterial lipopolysaccharide and calcium ionophore A23187, and biosynthesis was blocked b

77 l exocytosis by progesterone, but not by the calcium ionophore A23187, and elicited a concomitant red

78 Relaxations to acetylcholine, the calcium ionophore A23187, and nitroglycerin, as well as

79 responsiveness of several cell lines to the calcium ionophore A23187, bacterial lipopolysaccharide (

80 demonstrate that induction of ER stress with calcium ionophore A23187, brefeldin A, or tunicamycin is

81 nstitutively produced and are induced by the calcium ionophore A23187, but not by heat shock.

82 o induced by thapsigargin, okadaic acid, and calcium ionophore A23187, but not by phenobarbital or th

83 sphorylation of Pyk2 was also induced by the calcium ionophore A23187, by phorbol myristate acetate,

84 re increased or decreased in C cells, by the calcium ionophore A23187, by physiologic concentrations

85 stimulation with histamine, thrombin or the calcium ionophore A23187, cPLA2-alpha relocated to intra

86 ylation of T-753 in beta3 is reversed by the calcium ionophore A23187, demonstrating that these effec

87 e following treatment of thymocytes with the calcium ionophore A23187, ionomycin, or forskolin.

88 s pathways induced by five stimuli; PMA, the calcium ionophore A23187, nigericin, Candida albicans an

89 PGI2 synthesis stimulated by bradykinin, the calcium ionophore A23187, or arachidonic acid.

90 uch as sphingomyelinase and phospholipase C, calcium ionophore A23187, or heat resulted in the dramat

91 During platelet activation with thrombin, calcium ionophore A23187, or phorbol 12-myristate 13-ace

92 doleyl maleimide, cyclic nucleotide analogs, calcium ionophore A23187, or phorbol 12-myristate 13-ace

93 -19 cells with tunicamycin, brefeldin A, the calcium ionophore A23187, or thapsigargin increased the

94 peptide all induced Rap1 activation, as did calcium ionophore A23187, phorbol ester, forskolin, 8-br

95 stion using cross-adaptation to H2O2 and the calcium ionophore A23187, stable DSCR1 (Adapt78) transfe

96 effect of endothelin-1 was also mimicked by calcium ionophore A23187, suggesting the importance of C

97 Treatment with the calcium ionophore A23187, which increases Ca(2+)-calmodu

98 mulated by FcepsilonRI cross-linking and the calcium ionophore A23187.

99 lease of superoxide anion in response to the calcium ionophore A23187.

100 %; P = 0.1), as were relaxation responses to calcium ionophore A23187.

101 cked ERK activation evoked by Ang II and the calcium ionophore A23187.

102 ficiently as wt TGF-alpha in response to the calcium ionophore A23187.

103 rences in responses to acetylcholine and the calcium ionophore A23187.

104 regulated within minutes by phorbol ester or calcium ionophore A23187.

105 enhanced endothelium-dependent relaxation to calcium ionophore A23187.

106 pecially following degranulation, induced by calcium ionophore A23187.

107 ther enhanced by IKKgamma in the presence of calcium ionophore A23187.

108 ells upon stimulation of the CESS cells with calcium ionophore A23187.

109 riene B(4) (LTB(4)) when stimulated with the calcium ionophore A23187.

110 of granule exocytosis are induced by PMA and calcium ionophore A23187.

111 Ca(2+) channels, but not that induced by the calcium ionophore A23187.

112 er than that observed after stimulation with calcium ionophore A23187.

113 nd ERK activation, which was reversed by the calcium ionophore A23187.

114 e approximately 50% of the levels induced by calcium ionophore A23187.

115 Treatment with calcium ionophores A23187 or thapsigargin markedly inhib

116 es caused by prostaglandin E2, theophylline, calcium-ionophore A23187, 8-bromoadenosine 3':5'-cyclic

117 Pro-BTC shedding was activated by calcium ionophore (A23187) and by the metalloprotease ac

118 ivation by epidermal growth factor (EGF) and calcium ionophore (A23187) on IL-8 and COX-2 reporter ge

119 ximal membrane signaling, and with PMA and a calcium ionophore (A23187) to bypass membrane-mediated s

120 helium-dependent vasorelaxation induced by a calcium ionophore (A23187) was increased in both groups

121 2) activity of CD34(+) cells stimulated with calcium ionophore (A23187) was of similar magnitude to t

122 veolar lavage and stimulated in vitro with a calcium ionophore (A23187), and the concentrations of le

123 However, both spontaneous and calcium ionophore (A23187)-initiated AR were unaffected.

124 -O-tetradecanoylphorbol-13-acetate; TPA) and calcium ionophore (A23187)-mediated induction of PGE2 sy

125 H2O+.OH)], interferon-gamma (IFN-gamma), and calcium ionophore (A23187).

126 es induced by not only ethanol but also by a calcium ionophore (A23187).

127 mast cells (MC) on stimulation with PMA and calcium ionophore (A23187).

128 age to C2C12 skeletal muscle cells using the calcium ionophore, A23187 and the mitochondrial uncouple

129 TPA (1 microM) caused 45% and the calcium ionophore, A23187, 11% of maximal tyrosine phosp

130 Interestingly, treatment with the calcium ionophore, A23187, and the PKC activator, PMA, r

131 ) can also inhibit the light response, and a calcium ionophore, A23187, can substitute for light.

132 Treatment of endothelial cells with a calcium ionophore, A23187, decreased alpha-actinin-4-eNO

133 Incubation of cells with thapsigargin or the calcium ionophore, A23187, had no effect on SOK-1 activi

134 ssion of calpain or exposure of cells to the calcium ionophore, A23187, increased CCTalpha degradatio

135 okine, interleukin-1beta (IL-1beta), and the calcium ionophore, A23187, induce an increase in C-1-P l

136 sside for 7 d or the single application of a calcium ionophore, A23187, mimicked effects of glucose,

137 Stimulation of platelets with the calcium ionophore, A23187, resulted in complete proteoly

138 At physiological pH, treatment with the calcium ionophore, A23187, resulted in increased GFAP-IR

139 nts, induced by exposure of the cells to the calcium ionophore, A23187, were resolved at the level of

140 capable of exocytosis upon activation by the calcium ionophore, A23187.

141 se constructs are strongly responsive to the calcium ionophore, A23187.

142 activator, phorbol myristate acetate, or the calcium ionophore, A23187; such mast cells can rapidly r

143 03 for AUC; P = .0002 for PT) and similar to calcium-ionophore activated platelets (AUC of 332 +/- 15

144 y platelets and leukotriene B4 released from calcium ionophore-activated blood were markedly reduced,

145 tase did not influence matrix release by the calcium ionophore-activated chondrocytes.

146 In contrast to cytolytic IL-4 release from calcium ionophore-activated eosinophils, eotaxin and RAN

147 on at lipid bodies, and priming for enhanced calcium ionophore-activated LTC(4) release.

148 yte is artificially activated by exposure to calcium ionophore, after which PB2 is biopsied and colle

149 ibited by a combination of phorbol ester and calcium ionophore, agents that bypass proximal signaling

150 can be induced by stimulation of T cells by calcium ionophore alone and requires the calcineurin-dep

151 ly, however, in contrast to the IL-2 gene, a calcium ionophore alone was sufficient to induce IL-21 g

152 xposure to tumor necrosis factor alpha or to calcium ionophore also was prevented by removal of unest

153 erence to fibronectin in response to PMA and calcium ionophore and allows higher saturation densities

154 s in response to inflammatory agonists (e.g. calcium ionophore and ATP).

155 ggregates was significantly augmented by the calcium ionophore and prevented by the inhibitor cystami

156 The concept is established with lithium and calcium ionophores and accompanied by a response model t

157 Calcium ionophores and ionomycin also activated ERK and

158 y Ni2+ exposure because Cap43 was induced by calcium ionophores and its induction was attenuated by b

159 ostimulation of WEHI-231 and CH31 cells with calcium ionophores and protein kinase C agonists partial

160 Furthermore, ionomycin (calcium ionophore) and TPA augmented the enhancer activi

161 TM cells is regulated by a corticosteroid, a calcium ionophore, and a component of aqueous humor, sug

162 hed carborane, a recently introduced grafted calcium ionophore, and an MMA-DMA polymer matrix.

163 The calcium ionophore, and bradykinin, which are known to ac

164 activated by growth factors, phorbol ester, calcium ionophore, and hypertonic stress.

165 tussis toxin-insensitive, can be mimicked by calcium ionophore, and is inhibited by treatment with cy

166 pertussis toxin treatment, is not induced by calcium ionophore, and is insensitive to cytochalasin D.

167 Ionomycin, a calcium ionophore, and thapsigargin, an inhibitor of cal

168 s significantly enhanced by stimulation with calcium ionophore, and was drastically reduced by cyclos

169 vates a Gs-coupled beta-adrenergic receptor, calcium ionophores, and phorbol 12-myristate 13-acetate,

170 cipitates with NF-ATc in nuclear extracts of calcium ionophore- and Dex-treated cells.

171 nduced by a combination of phorbol ester and calcium ionophore at the highest level among the inducib

172 mpared the response of human erythrocytes to calcium ionophore at various temperatures in the range o

173 mical calcium pump based on a fast diffusive calcium ionophore-based membrane is reported.

174 , gamma irradiation, UV irradiation, and the calcium ionophore beauvericin.

175 ith microtubule antagonists, using the caged-calcium ionophore Br-A23187, was capable of inducing the

176 istate 13-acetate plus phytohemagglutinin or calcium ionophore but not to anti-CD3/anti-CD28 costimul

177 We also discovered that BWSV and the calcium ionophore calcimycin stimulate FM1-43 destaining

178 Calcium ionophores, calmodulin antagonists, and tyrosine

179 ast, activation of platelets by thrombin and calcium ionophore caused release of both APP and Abeta i

180 Ionomycin, a calcium ionophore, causes rapid mitochondrial accumulati

181 m entry from intracellular stores or through calcium ionophore channels activated secretion, though n

182 d from endogenous esterified lipid stores by calcium ionophore (CI) calcimycin (A-23187).

183 fied CD33(+) CML cells from 15 patients with calcium ionophore (CI) consistently resulted in de novo

184 blood monocytes or dendritic cells (DC) with calcium ionophore (CI) led to the rapid (18 hour) acquis

185 Cs, additional overnight culture with either calcium ionophore (CI) or interferon gamma (IFN-gamma),

186 In contrast, calcium ionophore (CI) treatment induced rapid and consi

187 We have shown previously that calcium ionophore (CI) treatment of various myeloid orig

188 d DCs on culture with Bryo-1 alone, Bryo-1 + calcium ionophore (CI), but not CI alone exhibited morph

189 in response to bacterial LPS, TNF-alpha, or calcium ionophore (CI).

190 ring of cytoplasmic Ca(2+) blocks egress and calcium ionophores circumvent the need for a reduction o

191 nly after stimulation with phorbol ester and calcium ionophore could these Th2-like cells be induced

192 ctivation of endogenous calcineurin with the calcium ionophore decreased p38 phosphorylation and incr

193 d oocytes could be artificially activated by calcium ionophore, demonstrating that the oocytes were f

194 lation of mouse astrocytes with ionomycin, a calcium ionophore, enhanced the production of anandamide

195 1 cells pretreated with thapsigargin but not calcium ionophore exhibited increased Duox-1 mRNA expres

196 Following stimulation with phorbol ester and calcium ionophore, expression of the bovine gene was det

197 Incubation of DCs with A23187 calcium ionophore for 48 h induced an expression of matu

198 Additional treatment of the cells with calcium ionophore further enhanced the hydrolysis rate a

199 Conversely, calcium ionophore greatly enhanced SR-A up-regulation by

200 NFAT activation, whereas and ionomycin, two calcium ionophores, had synergistic effects with vanadiu

201 orate and calcium ionophore IV (ETH 5234) or calcium ionophore I (ETH 1001).

202 bol ester alone, whereas B-2 cells require a calcium ionophore in addition to phorbol ester to trigge

203 , porcine aqueous humor, dexamethasone, or a calcium ionophore in cells pretreated with dexamethasone

204 ways with a combination of phorbol ester and calcium ionophore in clinorotation resulted in full expr

205 modified merocyanine photoacid polymer and a calcium ionophore in plasticized poly(vinyl chloride).

206 Nitrite generation after stimulation with calcium ionophore increased in Ad.CMVeNOS veins (1420.0+

207 atment of nonprimed macrophages with ATP and calcium ionophore induced a rapid release of MV that wer

208 n with EGTA suppressed p38 activation, while calcium ionophore induced p38 activity.

209 Moreover, although Raf activation or calcium ionophores induced little c-Fos expression, we o

210 Activation of cPLA(2) by the calcium ionophore, induced a dose-dependent increase of

211 tact T cells with the BTP compounds prior to calcium ionophore-induced activation of CaN caused NFAT

212 we report that dexamethasone (Dex) inhibits calcium ionophore-induced activation of the human IL-4 p

213 Evaluation of 3-oxa analogues 4 and 5 in calcium ionophore-induced acute skin inflammation model

214 denced by their distinct effects on LPS- and calcium ionophore-induced DC differentiation, on LPS-ind

215 thermore, tamoxifen pretreatment accelerated calcium ionophore-induced death by more than 20 min, sug

216 ponse to LPS, but had little or no effect on calcium ionophore-induced differentiation.

217 e same promoter region was also required for calcium ionophore-induced gene expression.

218 ar concentrations, both LTRAs also inhibited calcium ionophore-induced leukotriene (leukotriene B(4)

219 Addition of the calcium ionophore ionomycin alone induced the translocat

220 The calcium ionophore ionomycin also induced a rapid hyperpo

221 vated by phorbol 12-myristate 13-acetate and calcium ionophore ionomycin and was blocked by the NFAT

222 In this study, using the calcium ionophore ionomycin and/or PMA on Jurkat T cells

223 When the cells were treated with the calcium ionophore ionomycin in the nominal absence of ex

224 calcium signaling, and 2) treatment with the calcium ionophore ionomycin or cotransfection with activ

225 Exposure of human erythrocytes to the calcium ionophore ionomycin rendered them susceptible to

226 Treatment with the calcium ionophore ionomycin resulted in increased GlcAT-

227 ing MARK2 activity blocks the ability of the calcium ionophore ionomycin to promote neurite outgrowth

228 After stimulation with the calcium ionophore ionomycin, 2-AG levels in MGL-silenced

229 plus TNF-alpha, or PMA (with or without the calcium ionophore ionomycin, or TNF-alpha), with differe

230 h elevated extracellular potassium, with the calcium ionophore ionomycin, or with thyrotropin releasi

231 vate the nitric oxide pathway, including the calcium ionophore ionomycin, the nitric oxide donor S-ni

232 measured before and after application of the calcium ionophore ionomycin, which promotes the inflow o

233 er this mucin is secreted in response to the calcium ionophore ionomycin.

234 by constitutively active calcineurin or the calcium ionophore ionomycin.

235 The same is true of the calcium ionophore ionomycin.

236 y-5-methyl-4-isoxalonepropionic acid, or the calcium ionophore ionomycin.

237 ecreased upon exposure of the parasites to a calcium ionophore (ionomycin), to an inhibitor of the V-

238 aling pathways using phorbol ester (PMA) and calcium ionophore (ionomycin).

239 The calcium ionophore, ionomycin, induced chondrogenesis thr

240 cium ATPase inhibitor, thapsigargin, and the calcium ionophore, ionomycin, were used to deplete store

241 n SH-SY5Y human neuroblastoma cells with the calcium ionophore, ionomycin.

242 Second, exposure of macrophages to the calcium ionophores, ionomycin or A23187, mimicked recept

243 ar sites where, following stimulation with a calcium ionophore, it can be acted upon by cytosolic pho

244 is[3,5-bis(trifluoromethyl)phenyl]borate and calcium ionophore IV (ETH 5234) or calcium ionophore I (

245 A23187, a calcium ionophore known to block furin maturation, also

246 he endoplasmic reticulum to the Golgi, or, a calcium ionophore known to inhibit the autoactivation of

247 Treatment of cells with the calcium ionophore, leading to inhibition of calcium-depe

248 nt of beta cell lines (betaTC1 and HIT) with calcium ionophore led to a significant elevation in acti

249 t desensitization of NPR-B, and ionomycin, a calcium ionophore, mimics the AVP effect.

250 FK506 overcame the antiadipogenic effect of calcium ionophore on the differentiation of 3T3-L1 pread

251 Addition of a calcium ionophore or 100 microm NMDA decreased phospho-E

252 at observed for typical NO synthase agonists calcium ionophore or acetylcholine.

253 When Prss31-null MCs were activated with a calcium ionophore or by their high affinity IgE receptor

254 ne, without any alteration in sensitivity to calcium ionophore or hydrogen peroxide.

255 ndothelial cells were stimulated by either a calcium ionophore or thapsigargin to produce NO.

256 tosolic calcium with adrenergic stimulation, calcium ionophore, or calcium-containing pipette solutio

257 Activation with LPS, PMA, and calcium ionophore, or ligation of CD40 alone or in combi

258 , cytokines with platelet activating factor, calcium ionophore, or phorbol myristate acetate, develop

259 agents (the chemotherapeutic drug etoposide, calcium ionophore, or UV irradiation) and the withdrawal

260 cers of ER stress, including tunicamycin and calcium ionophore, or when a nonpolymerogenic alpha(1)-a

261 Treatment with calcium ionophores overcame agatoxin inhibition in a cal

262 pulse-chase analysis of tissue explants, to calcium ionophore perturbation of intracellular calcium

263 rine B82L fibroblasts that were treated with calcium ionophore, phorbol ester, or epidermal growth fa

264 Functionally, spermatozoa exposed to calcium ionophore, phorbol ester, or H(2)O(2) exhibited

265 macrophages with diverse agonists including calcium ionophores, phorbol myristate acetate (PMA), oka

266 Stimulation of VSM cells with ionomycin, a calcium ionophore, resulted in activation of CaMKIIdelta

267 treatment resulted in enhanced IFN-gamma and calcium ionophore-stimulated arachidonic acid release su

268 lipid body numbers correlated with increased calcium ionophore-stimulated LTC(4) production; and as d

269 caveolae-enriched membranes (2.23-fold), and calcium ionophore-stimulated NO production (1.56-fold).

270 Calcium ionophore-stimulated platelets also recapitulate

271 to study the effect of the glucocorticoid on calcium-ionophore-stimulated 5-lipoxygenase product and

272 sis and killing of S. pneumoniae and reduced calcium-ionophore-stimulated leukotriene B(4) synthesis

273 ility to generate leukotriene (LT) C(4) upon calcium ionophore stimulation but had little effect on L

274 e in vivo and in humans to phorbol ester and calcium ionophore stimulation in vitro in the face of lo

275 on of the biodetector ring relaxation during calcium ionophore stimulation of Ad.CMVeNOS veins.

276 was duplicated by incubating the cells with calcium ionophores such as ionomycin.

277 ) with Mos and restarted the cell cycle with calcium ionophore; the 30 minute cycle was converted int

278 eficient sperm acrosome-react in response to calcium ionophore, they do not acrosome-react in respons

279 failure of microvesiculation in response to calcium ionophore, thrombin, and collagen stimulation.

280 background levels of thrombin in response to calcium ionophore, thrombin, or combined thrombin plus c

281 cts was observed in U87MG cells treated with calcium ionophore to activate the calcium-dependent calp

282 In human erythrocytes, the ability of the calcium ionophore to cause susceptibility depends on tem

283 Furthermore, the ability of calcium ionophore to induce increased membrane order and

284 d with arachidonic acid were stimulated with calcium ionophore to initiate leukotriene biosynthesis.

285 +)](i) was significantly lower compared with calcium ionophore-treated cells, suggesting that a susta

286 ity to phagocytose apoptotic lymphocytes and calcium ionophore-treated erythrocytes but had no effect

287 semia, we studied the role of PS exposure in calcium ionophore-treated normal RBC adherence to human

288 Calcium ionophore treatment of chondrocytes, in a dose-d

289 Calcium ionophore treatment of eggs did not cause associ

290 We have found that calcium ionophore treatment of Rat-1 Elk-1, MCF-7 Elk-1,

291 ot Ras, protein activation following PMA and calcium ionophore treatment.

292 uption, TF pathway inhibitor inhibition, and calcium ionophore treatment.

293 ts, and the amount of adducts increased with calcium ionophore treatment.

294 Leaf infiltration with calcium ionophore triggered high-level PR gene expressio

295 (P = .0001) and by exposing intact cells to calcium ionophore using the cGMP reporter cell assay (P

296 sms, a simplified model of apoptosis using a calcium ionophore was applied.

297 ulted in equivalent up-regulation, whereas a calcium ionophore was relatively ineffective.

298 .) production evoked by either bradykinin or calcium ionophore was unaffected by PEG-catalase.

299 to bradykinin and acetylcholine but not the calcium ionophore were shifted significantly to the left

300 as well as reactive oxygen species (ROS) and calcium ionophore, whereas knockdown of RIP3 and MLKL bl