ライフサイエンスコーパス: calcium mobilization

1 red intermediate signaling events, including calcium mobilization.

2 blocked estrogen-induced PI3K activation and calcium mobilization.

3 negative cooperativity for agonist-mediated calcium mobilization.

4 ed kinases, and AKT, and blocked BCR-induced calcium mobilization.

5 , significantly increased carbachol-mediated calcium mobilization.

6 o significantly increased carbachol-mediated calcium mobilization.

7 of mammalian cells and mediate IP(3)-induced calcium mobilization.

8 l adhesion and is inefficient in stimulating calcium mobilization.

9 n a measure of membrane potential and not in calcium mobilization.

10 via the Galphai/phospholipase C pathway for calcium mobilization.

11 eading to activation of protein kinase C and calcium mobilization.

12 PAR4mediated aggregation are independent of calcium mobilization.

13 t, and its coligation with the BCR inhibited calcium mobilization.

14 d nonhemopoietic compartments via effects on calcium mobilization.

15 , to scavengers of ROS, and to inhibitors of calcium mobilization.

16 tive mutant strongly inhibited BCR-dependent calcium mobilization.

17 g each step leading to Rho activation and/or calcium mobilization.

18 cRgamma phosphorylation, Syk activation, and calcium mobilization.

19 totoxic T lymphocyte antigen-4 (CTLA-4), and calcium mobilization.

20 ated platelet shape change and intracellular calcium mobilization.

21 a chain phosphorylation, Syk activation, and calcium mobilization.

22 ptor gamma-chain signaling and intracellular calcium mobilization.

23 diated platelet alphaIIbbeta3 activation and calcium mobilization.

24 Ten-fold increases in IP3 caused saturated calcium mobilization.

25 eutrophil respiratory burst, chemotaxis, and calcium mobilization.

26 ly or largely eliminated dopamine-stimulated calcium mobilization.

27 st, SKF83959 appeared to be an antagonist of calcium mobilization.

28 increased elastase release and intracellular calcium mobilization.

29 protein-coupled receptors and leads to rapid calcium mobilization.

30 r signaling networks that critically rely on calcium mobilization.

31 ted activation of PLCgamma1 and that inhibit calcium mobilization.

32 Inhibitors of calcineurin and calcium mobilization abolished proline oxidase-mediated

33 ists leukotriene D(4) and UDP signal through calcium mobilization, acting on separate receptors, and

34 However, CD22 negative regulation of calcium mobilization after B cell antigen receptor ligat

35 f surface immunoglobulin M and impairment of calcium mobilization after BCR engagement in vitro.

36 Indeed, we investigated the degree of calcium mobilization after FasL stimulation and found th

37 n inhibition of tyrosine phosphorylation and calcium mobilization after GIT1 knockdown with antisense

38 or a kinase-dead mutant (GRK2-K220R) reduced calcium mobilization after receptor activation, whereas

39 addition, overexpression of DGKeta enhanced calcium mobilization after stimulating muscarinic recept

40 Anergic T cells showed impaired calcium mobilization after TCR triggering and were unabl

41 However, calcium mobilization alone by ionomycin was insufficient

42 d in a significant increase in intracellular calcium mobilization, an effect that was both delayed an

43 Validation was conducted using both calcium mobilization and [(125)I]-orexin-A competition b

44 tly inhibited CP55,940-induced intracellular calcium mobilization and [(35)S]GTP-gamma-S binding whil

45 antagonist of chemerin induced intracellular calcium mobilization and a much higher potency against t

46 an affect second messenger systems including calcium mobilization and a plethora of kinases to alter

47 an affect second messenger systems including calcium mobilization and a plethora of kinases within ne

48 through a mechanism dependent upon cytosolic-calcium mobilization and a significant decline in GRP78/

49 ; G(q) protein and pertussis toxin-sensitive calcium mobilization and activation of a nonselective ca

50 ed we demonstrate that LPI is able to induce calcium mobilization and activation of Akt and extracell

51 lation and was needed for optimal TCR-driven calcium mobilization and activation of the kinase Erk.

52 am signaling events, including intracellular calcium mobilization and activation of the Ras-extracell

53 rs did not affect fMLF-induced intracellular calcium mobilization and Akt phosphorylation.

54 her, Pak1(-/-) BMMCs demonstrated diminished calcium mobilization and altered depolymerization of cor

55 s well as reductions in chemokine-stimulated calcium mobilization and altered ERK and Akt activation.

56 ase C but activated phospholipase Cgamma and calcium mobilization and augmented these signals as well

57 Pharmacological evaluation in calcium mobilization and binding assays revealed the imp

58 this response is dependent on intracellular calcium mobilization and calmodulin activation.

59 Ghrelin increased intracellular calcium mobilization and cAMP levels in oral epithelial

60 mately reduced cancer cell functions such as calcium mobilization and cellular chemotaxis.

61 Furthermore, the calcium mobilization and chemotactic responses of LPS-st

62 NA and protein, in association with impaired calcium mobilization and chemotactic responses to peptid

63 mor cell line 66.1 resulted in intracellular calcium mobilization and chemotaxis in vitro.

64 nting cell function to T helper cells and DC calcium mobilization and chemotaxis to histamine.

65 ic Src family enzymes, Ly-49D/DAP12-mediated calcium mobilization and cytokine production by CD45 nul

66 This was linked to a similar enhancement in calcium mobilization and cytoskeletal rearrangement attr

67 ary for MC activation, including Ag-mediated calcium mobilization and cytoskeletal reorganization, po

68 y different FcalphaRI-mediated intracellular calcium mobilization and degranulation in rat basophilic

69 mma (PLCgamma) is a ubiquitous gatekeeper of calcium mobilization and diacylglycerol-mediated events

70 PR120S receptors elicited both intracellular calcium mobilization and DMR responses in human embryoni

71 +)CD21(low) subpopulation exhibits decreased calcium mobilization and does not efficiently differenti

72 king of GPR120S and GPR120L receptors, using calcium mobilization and dynamic mass redistribution (DM

73 n epidermal melanocytes (HEMs) to UVA causes calcium mobilization and early melanin synthesis.

74 recise organization is essential for optimal calcium mobilization and efficient and effective muscle

75 n the present study, we have used cell-based calcium mobilization and electrophysiological assays to

76 Stimulation with those CCR6 ligands leads to calcium mobilization and elevated active RhoA, phosphory

77 de corresponding to its V3-like loop induced calcium mobilization and enhanced thrombin-mediated plat

78 onal EP(1) receptor coupled to intracellular calcium mobilization and EP(2) receptor coupled to cAMP

79 This results in impaired TCR-mediated calcium mobilization and Erk activation.

80 inating biased agonism between intracellular calcium mobilization and ERK1/2 phosphorylation.

81 2 and S1P3 receptors activated intracellular calcium mobilization and extracellular signal-regulated

82 as functional tests of H3 receptor-mediated calcium mobilization and GTPgammaS binding.

83 d or toll-like receptor signaling to improve calcium mobilization and improve keratinocyte differenti

84 ation, this chimeric receptor induced robust calcium mobilization and increase of adenylate cyclase a

85 Kynurenic acid elicits calcium mobilization and inositol phosphate production i

86 ssessed significant in vivo activity on bone calcium mobilization and intestinal calcium transport.

87 signals that induce changes in intracellular calcium mobilization and maintain tolerance by preventin

88 gnaling pathway in melanocytes that leads to calcium mobilization and melanin synthesis and may under

89 tromal cell-derived factor-1 (SDF-1)-induced calcium mobilization and migration and do not localize t

90 cPLA(2)alpha is regulated by calcium mobilization and mitogen-activated protein kinas

91 , and downstream signaling events, including calcium mobilization and mitogen-activated protein kinas

92 is pathway is dependent on protein kinase C, calcium mobilization and mitogen-activated protein kinas

93 Apical ATP release with subsequent calcium mobilization and PKA activation are involved in

94 atelet responses by inhibiting intracellular calcium mobilization and PKC activation through G(q) pat

95 strate PLCgamma2 to enhance collagen-induced calcium mobilization and platelet activation.

96 roprotection by decreasing glutamate-induced calcium mobilization and preventing apoptotic gene expre

97 10(8) cells, thrombin), is externalized via calcium mobilization and protease-activated receptors-1

98 e variants on their effects on AT1R-mediated calcium mobilization and provides molecular understandin

99 Likewise, calcium mobilization and radioligand binding assays foun

100 Intracellular calcium mobilization and receptor antagonist studies rev

101 TCR-CD28 linked calcium mobilization and subsequent uptake by mitochondr

102 GPR30 by estrogen resulted in intracellular calcium mobilization and synthesis of phosphatidylinosit

103 CRAMP also induced calcium mobilization and the activation of MAPK in monoc

104 ritical for the CXCR2-mediated intracellular calcium mobilization and the resultant migration and inf

105 y secreted nucleotides plays a major role in calcium mobilization and the subsequent calcium-dependen

106 n cells was found to prolong mGluR5-mediated calcium mobilization and to also potentiate mGluR5-media

107 tdIns(3,4,5)P3, leading to the inhibition of calcium mobilization and to the attenuation of mast cell

108 ormal BCR signal transduction as measured by calcium mobilization and tyrosine phosphorylation.

109 otion of cell proliferation, cell migration, calcium mobilization, and activation of ERK1/2.

110 sponses, including activation of G proteins, calcium mobilization, and activation of mitogen-activate

111 Effects of the drugs on DC chemotaxis, calcium mobilization, and antigen-presenting cell functi

112 n IL-4-stimulated monocytes, are elevated on calcium mobilization, and are detected at increased leve

113 In human PBMC, RvE1 partially induced calcium mobilization, and blocked subsequent stimulation

114 ns-4,5-P(2) synthesis, PLCgamma2 activation, calcium mobilization, and cell proliferation.

115 hK2 is required for murine MC degranulation, calcium mobilization, and cytokine and leukotriene produ

116 s enhanced platelet activation, monitored by calcium mobilization, and endothelial activation, monito

117 e 5'-O-(3-[(35)S]thio)-triphosphate binding, calcium mobilization, and extracellular signal-regulated

118 f the Syk-Akt-S6kinase signaling pathway and calcium mobilization, and inhibits SHP-1 activity upon B

119 re measured, including platelet aggregation, calcium mobilization, and integrin activation.

120 nd CCK(2)) receptors using receptor binding, calcium mobilization, and internalization studies.

121 RP1 has since been implicated in suppressing calcium mobilization, and its expression is misregulated

122 Cell structural integrity and viability, calcium mobilization, and nitric oxide generation were d

123 rtery endothelial organization and function, calcium mobilization, and nitric oxide generation were w

124 , caspase-1, cathepsin-mediated degradation, calcium mobilization, and potassium efflux but not caspa

125 al-regulated kinase 1/2 (ERK1/2) activation, calcium mobilization, and receptor endocytosis] in the s

126 rotein q, the activation of phospholipase C, calcium mobilization, and the release of the inhibitory

127 ation, C5a- and UTP-stimulated intracellular calcium mobilization, and the stimulation of cAMP format

128 augment phospholipase Cgamma1 activation and calcium mobilization, and to phosphorylate NFkappaB and

129 lling activity, measured by anti-IgM-induced calcium mobilization, and with increased expression of C

130 , including phosphatidylinositol hydrolysis, calcium mobilization, arachidonic acid release, mitogen-

131 (PLD) and protein kinase C (PKC) as well as calcium mobilization are essential signals for degranula

132 ncluding that of phospholipase C-gamma2, and calcium mobilization are impaired in IgA-presenting WIP

133 phospho-phospholipase Cgamma1 induction and calcium mobilization are largely unaffected.

134 actin rearrangement, MTOC polarization, and calcium mobilization are not.

135 he same enhancing effect on carbachol-evoked calcium mobilization as overexpressed DGKeta, and overex

136 Static adhesion also caused calcium mobilization, as did the subsequent onset of flo

137 ive vesicle-associated membrane proteins and calcium mobilization, as well as PI3K, MAPK, and IkappaB

138 sted for OX1 and OX2 receptor activity using calcium mobilization assay in recombinant cell lines.

139 ng a cell-based high-throughput fluorescence calcium mobilization assay.

140 ist (efficacy ca. 50%) in a 5-HT(2)-mediated calcium mobilization assay.

141 ovel D2 dopamine receptor modulators using a calcium mobilization assay.

142 MMPIP acts as a noncompetitive antagonist in calcium mobilization assays in cells coexpressing mGluR7

143 meric receptors were evaluated in functional calcium mobilization assays to compare orthosteric agoni

144 , Brilliant Black BN is commonly used within calcium mobilization assays to quench extracellular fluo

145 ced the Emax of the agonist curve in the CB1 calcium mobilization assays, confirming their negative a

146 g GTP-gamma-S accumulation and intracellular calcium mobilization assays.

147 restin-2 recruitment, cAMP accumulation, and calcium mobilization assays.

148 and determined their potency in OX1 and OX2 calcium mobilization assays.

149 ed them responsive to CXCL17, as measured by calcium-mobilization assays.

150 with Fluo-4 resulted in a rapid increase in calcium mobilization associated cell fluorescence simila

151 s desensitization of a receptor(s) linked to calcium mobilization because a second application of SP-

152 osphorylation was dependent on intracellular calcium mobilization, because BAPTA-AM blocked DRAK2 kin

153 -induced global tyrosine phosphorylation and calcium mobilization but impaired the Gab2-PI3K pathway

154 s required for CXCR3-mediated chemotaxis and calcium mobilization but is not required for ligand bind

155 d found that knockdown of CK1alpha increased calcium mobilization but not ERK activation.

156 internalization, increased PI production and calcium mobilization by LTD4, and significantly attenuat

157 tion assays but marked loss of intracellular calcium mobilization by peptide agonists.

158 this hypothesis, we disrupted intracellular calcium mobilization by treating immortalized mouse M-1

159 control B cell activation was determined by calcium mobilization, by tyrosine phosphorylation of sig

160 Since platelet intracellular calcium mobilization [Ca(t)]i controls granule release,

161 ular tyrosine phosphorylation, intracellular calcium mobilization, CD69 surface expression, interleuk

162 d defects in chemokine-induced intracellular calcium mobilization, chemotaxis, and homing, whereas Gn

163 ining the PDZ motif) inhibited intracellular calcium mobilization, chemotaxis, and transepithelial mi

164 edly enhanced CysLT-stimulated intracellular calcium mobilization compared with endogenous expression

165 Calcium mobilization correlated with the secretion of ly

166 lonRI, evidenced by enhanced Erk activation, calcium mobilization, degranulation, and cytokine produc

167 esponses in leukocytes including chemotaxis, calcium mobilization, degranulation, and gene expression

168 The ligand-selectivity profile of calcium mobilization-dependent signaling and prolactin s

169 estigate the molecular mechanisms regulating calcium mobilization during Fas-mediated apoptosis, we h

170 onist (EC50, 802 nM) yet potently stimulated calcium mobilization (EC50, 7 nM) in 5-HT2A receptor-exp

171 termined by mRNA knockdown and intracellular calcium mobilization experiments.

172 memory T cells, characterized by diminished calcium mobilization, expression of CD25, and IL-10 prod

173 and by blocking tyrosine phosphorylation and calcium mobilization following BCR cross-linking.

174 ns, was ubiquitinated, and was able to block calcium mobilization following BCR cross-linking.

175 t not R2.60(190)/E6.53(364) was critical for calcium mobilization for all three peptides.

176 ese phmSG cells also demonstrated functional calcium mobilization, formation of epithelial monolayer

177 e vitamin D [1,25(OH)(2)D], which can elicit calcium mobilization from bone when intestinal calcium a

178 stimulation with ionomycin, and BCR-induced calcium mobilization from internal stores is decreased.

179 through voltage-gated calcium channels or on calcium mobilization from intracellular stores.

180 e in inositol trisphosphate (IP3) to trigger calcium mobilization from stores and elevation of [Ca(2+

181 f type II apoptotic cell death that involves calcium mobilization from the ER and that this step is m

182 lls (U937) manifested by immediate cytosolic-calcium mobilization, GADD153 and GADD34 protein inducti

183 nced expression of Itk leads to increases in calcium mobilization, granule release, and cytotoxicity

184 atelet functions such as fibrinogen binding, calcium mobilization, granule secretion, aggregation, an

185 ssays that we have explored, including cAMP, calcium mobilization, guanosine 5'-[gamma-thio]triphosph

186 Although the role of cADPR in modulating calcium mobilization has been extensively examined, its

187 teracting protein, and molecules upstream of calcium mobilization, i.e., activated ZAP70 and phosphol

188 triphosphate) incorporation; IC50 = 210 nm), calcium mobilization (IC50 = 71 nm), monocyte chemotaxis

189 ncluding PLCgamma1 tyrosine phosphorylation, calcium mobilization, IL-2 secretion, and T-cell prolife

190 ar calcium pumps with thapsigargin increases calcium mobilization in 125Tg B cells.

191 nosine, ADP, ATP, UDP, and UTP all stimulate calcium mobilization in bone marrow-derived mast cells w

192 tion, phosphate absorption in the intestine, calcium mobilization in bone, and calcium reabsorption i

193 the amplitude and duration of injury-induced calcium mobilization in cells at the leading edge.

194 dose-response curves assaying intracellular calcium mobilization in cells heterologously expressing

195 We conclude that disruption of calcium mobilization in cells that are normally growth-i

196 We also show that CD38 and cADPR modulate calcium mobilization in chemokine-stimulated DCs and are

197 itol phosphate accumulation or intracellular calcium mobilization in Chinese hamster ovary (CHO) cell

198 suggesting that distinct mechanisms regulate calcium mobilization in each of the two transitional B c

199 led that ethanol inhibited glutamate-induced calcium mobilization in endothelial cells, affected plas

200 f the compounds was supported by analysis of calcium mobilization in HL-60 cells transfected with hum

201 The reduced calcium mobilization in HPKO platelets is associated wit

202 They elicit robust, FPR-dependent calcium mobilization in human and mouse leukocytes and t

203 These agonists produce robust calcium mobilization in human melanoma cell lines which

204 pounds inhibit PAR-4 induced aggregation and calcium mobilization in human platelets and reduce 12-HE

205 Furthermore, OMPT-induced intracellular calcium mobilization in mammalian cells is efficiently i

206 tes TRPA1 expression, which is functional in calcium mobilization in mast cells.

207 eil dematin as a novel regulator of internal calcium mobilization in platelets affecting multiple sig

208 In BSDL(-/-) mice, calcium mobilization in platelets and thrombus formation

209 llagen, and vWF are known to induce in vitro calcium mobilization in platelets.

210 es for their ability to induce intracellular calcium mobilization in RBL-2H3 cells transfected with h

211 Likewise, Quin-C1 selectively stimulates calcium mobilization in RBL-FPRL1 cells, a response that

212 t has been implicated in taste buds, elicits calcium mobilization in Receptor (Type II) taste cells.

213 from the BLT2(-/-) mice showed intracellular calcium mobilization in response to 12(S)-hydroxyheptade

214 h that initiates and maintains intracellular calcium mobilization in response to B cell antigen recep

215 tion, reduced Erk activation, and attenuated calcium mobilization in response to BCR engagement.

216 LTE(4) enhanced calcium mobilization in response to PGD(2) in Th2 cells

217 s that disrupted dimer formation had reduced calcium mobilization in response to the PAR4 agonist pep

218 ignaling molecules, cytokine production, and calcium mobilization in response to this toxin.

219 e BCR signalosome is pivotal to BCR-mediated calcium mobilization in the cytosol.

220 nsPEF and the purinergic agonist UTP induced calcium mobilization in the presence and absence of extr

221 n of DGKeta produced no additional effect on calcium mobilization in the presence of BIM I.

222 ctivation threshold by targeting TCR-induced calcium mobilization in thymocytes and peripheral T cell

223 s inhibited trout C3a-mediated intracellular calcium mobilization in trout leukocytes.

224 ), both of which have been shown to modulate calcium mobilization in vitro.

225 reduced phospholipase Cgamma activation and calcium mobilization, indicating a defect in calcium sig

226 Chemotaxis and calcium mobilization induced by all three CXCR3 ligands

227 Tyrosine phosphorylation and calcium mobilization induced by BCR engagement is dimini

228 Platelet aggregation and calcium mobilization induced by the PAR4 thrombin recept

229 PACE4/PC5A decreased calcium mobilization induced by thrombin stimulation.

230 new insight into the molecular basis of bone calcium mobilization induced by vitamin D.

231 Medium-chain FFAs elicit calcium mobilization, inhibit 3',5'-cyclic AMP productio

232 B cell receptor (BCR) inhibited BCR-mediated calcium mobilization, intracellular tyrosine phosphoryla

233 We hypothesize that calcium mobilization is a potential key molecular initia

234 In addition, calcium mobilization is enhanced in RAW264.7 macrophages

235 trant KD, and we identify that regulation of calcium mobilization is fundamental to the underlying im

236 Similarly, calcium mobilization is Galpha(q)-mediated and independe

237 n this article, we confirm that BCR-mediated calcium mobilization is reduced in Cd20(-)/(-) murine B

238 diated activation of RhoGTPase signaling and calcium mobilization, leading to the regulation of kerat

239 to G(o)- but not G(q)-mediated intracellular calcium mobilization, leading us to investigate the role

240 echanism through which reduced intracellular calcium mobilization may be altering cellular proliferat

241 The thrombin-induced calcium mobilization may play an important permissive ro

242 Cyclic ADP-ribose (cADPR) is a calcium mobilization messenger important for mediating a

243 cle progression, PLC-gamma2 phosphorylation, calcium mobilization, NF-ATp dephosphorylation, and Erk

244 p23/p21 ratio shift, ZAP-70 phosphorylation, calcium mobilization, NFAT activation, and transient IL-

245 signals downstream of this enzyme including calcium mobilization, NFAT, NF-kappaB, and Ras-ERK-AP-1

246 Intracellular calcium mobilization occurs within milliseconds of pulse

247 chanistically, loss of Cbl-b rescues reduced calcium mobilization of anergic T cells, which was attri

248 f PAR-1 or PAR-2 by agonist peptides induced calcium mobilization, only PAR-1 activation affected bar

249 ta had no detectable effect on intracellular calcium mobilization or endothelial cell viability.

250 pter protein, despite the inability to cause calcium mobilization or TCR polarization.

251 in intracellular signaling pathways, such as calcium mobilization, phosphatidylinositol 3-kinase-AKT

252 Calcium mobilization, phosphorylation of Syk, phospholip

253 IM of FcgammaRIIb and suppressed BCR-induced calcium mobilization, proliferation, and costimulatory m

254 lls resulted in an increase in intracellular calcium mobilization, protein kinase C activation, actin

255 se Cgamma2 activity leading to activation of calcium mobilization, Rap1 and protein kinase C activati

256 of phospholipase Cgamma2, and intracellular calcium mobilization, reflecting competency of BCR signa

257 wound healing regulatory pathway, along with calcium mobilization, regulates CCR6-directed epithelial

258 Forced calcium mobilization rescued the TNFalpha secretion defe

259 cement in the magnitude of the intracellular calcium mobilization response to the group I metabotropi

260 as observed in dose-dependent chemotaxis and calcium mobilization responses.

261 s, dual inhibition of the P2Y12 receptor and calcium mobilization result in a complete inhibition of

262 ng both phosphoinositide (PI) production and calcium mobilization stimulated by LTD4 yet had almost n

263 500 microM) had no significant effect on the calcium mobilization stimulated by the nonselective aden

264 Importantly, Galphai immunoprecipitation and calcium mobilization studies indicated that nuclear CXCR

265 Intracellular calcium mobilization studies using a human epithelial ce

266 Calcium mobilization studies were done utilizing a fluor

267 tion, a detailed analysis of the dynamics of calcium mobilization suggests a possible inhibitory role

268 sphorylation but does not alter C5a-mediated calcium mobilization, supporting its role in modulation

269 taTCR dimerization also alter ligand-induced calcium mobilization, TCR accumulation at the site of pM

270 wed a preference of AEME for the M2 subtype; calcium mobilization tests revealed partial agonist effe

271 ls had lower glutamate-induced intracellular calcium mobilization than did normal RGC-5 cells, and th

272 Like other PAR1 activators, C4a induced calcium mobilization through the PAR1/Galphaq/PLCbeta si

273 ring with "capacitative" or "store-operated" calcium mobilization, thus raising the possibility that

274 We monitored calcium mobilization to assess activation of the laser-t

275 WT neutrophils in vitro along with increased calcium mobilization upon activation and expression of e

276 ative responses to anti-CD40, and diminished calcium mobilization upon stimulation with BCR-dependent

277 reas GPCR135 signaling could be converted to calcium mobilization using a Gqi5 or Galpha16 G-proteins

278 emporal and spatial pattern of VEGF-mediated calcium mobilization using donor dequenching FRET method

279 d the Fc region of chicken IgY and to induce calcium mobilization via association with the common gam

280 utamate receptor subtype 5 (mGlu5) activates calcium mobilization via binding of glutamate, the major

281 ival promotion by OXB required intracellular calcium mobilization via inositol-1,4,5-triphosphate and

282 Calcium mobilization was activated when a mouse fibrobla

283 Cis-UCA-induced calcium mobilization was blocked with a selective 5-HT2A

284 In contrast, bone calcium mobilization was equal to that produced by 1alph

285 BCR-induced calcium mobilization was intact after Mef2c deletion, bu

286 Glutamate-induced intracellular calcium mobilization was measured by using ratiometric c

287 image unstimulated and stimulated platelets, calcium mobilization was monitored as a reporter of plat

288 dicating that P2Y2R-stimulated intracellular calcium mobilization was not involved.

289 Intracellular calcium mobilization was noted at early time points usin

290 Increased intracellular calcium mobilization was observed in B cell lines from h

291 XCR4 signaling in HEK293 cells revealed that calcium mobilization was primarily negatively regulated

292 eness, respiratory burst, degranulation, and calcium mobilization were conducted in human peripheral

293 The mechanisms involved in this calcium mobilization were investigated in the present st

294 iptional changes indicative of intracellular calcium mobilization were significantly overrepresented

295 Agonists that elicit distinct modes of calcium mobilization were used.

296 ed [3H]inositol phosphate (IP) formation and calcium mobilization with EC50 values of 8.5 nM and 0.8

297 19 and CCL21 induce G protein activation and calcium mobilization with equal potency.

298 er-1177, whereas inhibition of intracellular calcium mobilization with TMB-8 blunted Thr-495 dephosph

299 ibit platelet accumulation resulted in rapid calcium mobilization within the endothelium.

300 its IP(3) (inositol-1,4,5-trisphosphate) and calcium mobilization, without inhibiting Ang II binding