ライフサイエンスコーパス: calcium oscillation

1 lly modulate effects of EFS-induced cellular calcium oscillations.

2 the active phase) of the various regimes of calcium oscillations.

3 population of cells maintained synchronized calcium oscillations.

4 cells acts on purinergic receptors to induce calcium oscillations.

5 Cx43-null neurospheres displayed spontaneous calcium oscillations.

6 in the IP3 receptor slow or eliminate these calcium oscillations.

7 +)] spikes and exerts a positive feedback on calcium oscillations.

8 ns in which sperm extracts elicit pronounced calcium oscillations.

9 n turn coincided with the termination of the calcium oscillations.

10 not individual cells and monolayers, exhibit calcium oscillations.

11 lso been reported to occur in the absence of calcium oscillations.

12 emonstration that it acts upstream of oocyte calcium oscillations.

13 ciated with the rapid onset of intracellular calcium oscillations.

14 significant effect on the properties of the calcium oscillations.

15 ions and is activated via CaM binding during calcium oscillations.

16 MI1 and improved its efficiency in mediating calcium oscillations.

17 easing the frequency and/or amplitude of the calcium oscillations.

18 9593) mimicked the effect of Dyn-A (1-13) on calcium oscillations.

19 oncentration and the frequency of alpha-cell calcium oscillations.

20 exclusively involved in calcium entry during calcium oscillations.

21 gher agonist concentration for intracellular calcium oscillations.

22 egg laying, in part by interfering with HSN calcium oscillations.

23 erative discharges of stored calcium, termed calcium oscillations [1].

24 M) was appropriate for the interpretation of calcium oscillations (~125-850 nM), while that of EF-han

25 calcium influx as an essential component of calcium oscillations [2].

26 d as a positive regulator of sperm-triggered calcium oscillations, a finding that may apply to other

27 Rapid cellular calcium oscillations activate gene expression hours late

28 Calcium oscillations activate nodule development; we wan

29 d indefinitely with (delta)90 cyclin B1, the calcium oscillations also continue indefinitely.

30 P/Q-, and T-type VGCCs are not required for calcium oscillations, although inhibitors of these chann

31 the diversity in damping characteristics of calcium oscillations among cells.

32 Calcium oscillation amplitude and frequency control gene

33 n, attenuating SST2 effects on intracellular calcium oscillation and internalization.

34 o different calcium responses: intra-nuclear calcium oscillations and a calcium influx at the root ha

35 olerant B cells, self-antigen stimulated low calcium oscillations and activated NF-AT and ERK/pp90rsk

36 es insights into mechanisms of intracellular calcium oscillations and capacitative calcium entry.

37 TLC-S induced global calcium oscillations and extended calcium transients as

38 calcium channels is critical for alpha-cell calcium oscillations and glucagon secretion at low gluco

39 of dexamethasone were sufficient to inhibit calcium oscillations and interleukin-2 mRNA after weak T

40 Calcium oscillations and maturation were typically lacki

41 Here, we show that PAWP triggers calcium oscillations and pronuclear formation in human a

42 y connexin-36 coordinates intracellular free calcium oscillations and pulsatile insulin release in is

43 n36 gap junctions coordinate glucose-induced calcium oscillations and pulsatile insulin secretion acr

44 A model for the mechanism of neuronal calcium oscillations and the reason for their synchrony

45 from the endoplasmic reticulum, resulting in calcium oscillations and the secretion of luteinizing ho

46 lating inositol 1,4,5-trisphosphate-mediated calcium oscillations and the up-regulation of the transc

47 Calcium oscillations and traveling calcium waves have be

48 hat includes five genes required to generate calcium oscillations and two genes required for the perc

49 nsor, as a key link between LPS-induced ROS, calcium oscillations, and endothelial cell (EC) dysfunct

50 l, enhances glucose-stimulated intracellular calcium oscillations, and enhances insulin secretion fro

51 alcium uptake explained our results, causing calcium oscillations, AP amplitude alternans, and TWA th

52 In addition, the calcium oscillations appear to serve as a bidirectional

53 In the absence of TMEM24, calcium oscillations are abolished, leading to a defect

54 d elevated CDK activity, the sperm-triggered calcium oscillations are again prolonged.

55 Most important, sperm-induced calcium oscillations are blocked by coinjection of a com

56 Secondly, although calcium oscillations are caused by the stochastic openin

57 In animal systems, comparable calcium oscillations are decoded by calmodulin (CaM)-dep

58 ET technology have been used to describe how calcium oscillations are decoded through phase-locked os

59 We have found that these calcium oscillations are dependent on an influx of extra

60 re fertilized by sperm, a distinct series of calcium oscillations are generated which serve as the es

61 vide genetic evidence that stimulus-specific calcium oscillations are necessary for stomatal closure.

62 Here we demonstrate that these calcium oscillations are regulated by kappa opioid recep

63 lic calcium concentration (calcium spikes or calcium oscillations) are a common mode of signal transd

64 onse to ABA is unlinked to the generation of calcium oscillations, as the pp2aB'1 mutant displays a n

65 Baseline spontaneous calcium oscillations assessed by fluorescent confocal mi

66 While the calcium oscillations associated with cells directly expo

67 The generation of calcium oscillations at fertilisation and during mitosis

68 o other oocytes that display sperm-triggered calcium oscillations at fertilisation.

69 wering SERCA level will enable intracellular calcium oscillations at low agonist concentrations where

70 Furthermore, the calcium oscillation attenuates the histamine-induced cyt

71 In the presence of 4-AP, synchronized calcium oscillations become independent of NMDA receptor

72 imulation, for example, not only altered the calcium oscillations but also facilitated osteodifferent

73 , were also able to produce local and global calcium oscillations (but at higher concentrations than

74 re-operated calcium channels is critical for calcium oscillations, but calcium entry through voltage-

75 Regulation of calcium oscillation by external physical stimulation cou

76 e report the manipulation of sperm-triggered calcium oscillations by cyclin-dependent kinase (CDK) ac

77 ubsystem can account for these complex islet calcium oscillations by modifying the relative contribut

78 Information embedded in calcium oscillation can provide molecular cues for cell

79 was also able to prevent the suppression of calcium oscillations caused by Dyn-A (1-13).

80 otif regulates damping of G-protein-mediated calcium oscillations consistent with experimental data.

81 n culture likely results from the decline in calcium oscillations/contractile activity and mitofusin

82 The data show that the calcium oscillations contribute a significant fraction o

83 Cytosolic calcium oscillations control signaling in animal cells,

84 , and computational modeling reveal that the calcium oscillation depends on the number of neighboring

85 he ATP stimulus increases the propensity for calcium oscillations, despite large cell-to-cell variabi

86 ve of mitochondria to cause dysregulation of calcium oscillations during prolonged stimulation.

87 Calcium oscillations evoked by physiological concentrati

88 This architecture explains why calcium oscillations evoked by synchronized periodic act

89 Calcium oscillations exert physiological control on mito

90 is initiated by sperm-mediated intracellular calcium oscillations, followed by activation of metaphas

91 olonies, both communication channels lead to calcium oscillations following the stimulation by extern

92 s expressed in astrocytes tripled astrocytic calcium oscillation frequency in both the preBotzinger c

93 n is important for setting the physiological calcium oscillation frequency.

94 cord neurons in culture exhibit spontaneous calcium oscillations from day in vitro (DIV) 6 through D

95 The role of cytosolic calcium oscillation has long been recognized in the regu

96 Intracellular calcium oscillations have fascinated scientists for deca

97 These intracellular calcium oscillations have long fascinated biologists as

98 tions in membrane potential accompanying the calcium oscillations have no significant effect on the p

99 Here we report novel intraciliary calcium oscillations (ICOs) at the LRO that connect cili

100 lular reactive oxygen species production and calcium oscillation in rice roots.

101 agonist dynorphin (Dyn)-A (1-13) suppressed calcium oscillations in a concentration-dependent manner

102 s a statistical description of heterogeneous calcium oscillations in a dynamic environment.

103 scle had decreased AHR, and the frequency of calcium oscillations in CD148-deficient ASM was substant

104 factor, found in sperm extracts, that causes calcium oscillations in cells; thus, the protein was nam

105 Conversely, cold and abscisic acid elicited calcium oscillations in det3, and stomatal closure occur

106 sed calcium imaging to monitor intracellular calcium oscillations in GnRH-1 neurons maintained in nas

107 hways underlying muscarinic receptor-induced calcium oscillations in human embryonic kidney (HEK293)

108 acute GABA(A) receptor antagonism decreased calcium oscillations in individual GNRH-1 cells as well

109 Here we report that insulin induces calcium oscillations in isolated rat hepatocytes, and th

110 revealed that PHE caused dramatic cytosolic calcium oscillations in NST neurones.

111 For example, the calcium oscillations in oocytes and embryos occur during

112 CCaMK) is essential in the interpretation of calcium oscillations in plant root cells for the establi

113 t developing murine cortical neurons exhibit calcium oscillations in response to direct activation of

114 whereas a mix of CO4 and Myc-LCOs activated calcium oscillations in rice trichoblasts.

115 symbiosis signaling pathway, with resultant calcium oscillations in root epidermal cells of Medicago

116 Our data suggest that calcium oscillations in the cell cycle may be linked to

117 ased during hypercapnic challenge, increases calcium oscillations in the chemosensitive parafacial re

118 ly multiple factors control the frequency of calcium oscillations in the egg after fertilization and

119 the defecation motor program in C. elegans, calcium oscillations in the pacemaker (intestine), which

120 tes, and (iii) ryanodine receptor-2-mediated calcium oscillations increased fusion activity in HEK293

121 J suppressed Plcg2 expression and downstream calcium oscillations indirectly by a TGF-beta/PLCgamma2/

122 the Gbetagamma translocation rate regulates calcium oscillations induced by G-protein-coupled recept

123 During the higher-frequency sinusoidal calcium oscillations induced by higher doses of ACh, NAD

124 During the transient cytosolic calcium oscillations induced by intermediate doses of AC

125 s termed oscillin because it correlated with calcium oscillation-inducing activity in mammalian eggs.

126 r sperm 33-kDa protein that co-migrated with calcium oscillation-inducing activity was recently descr

127 Previous studies have shown that these calcium oscillations involve the activation of NMDA rece

128 Mg(2+) also suppressed thimerosal-induced calcium oscillations (IP(3)R-dependent).

129 cium-EGTA Krebs solution suggesting that the calcium oscillation is mediated principally by intracell

130 ee eggs show that the general pattern of the calcium oscillations is identical in monospermic and pol

131 e early Nod factor signaling associated with calcium oscillations is limited to the root surface, the

132 ling systems, the frequency of intracellular calcium oscillations is physiologically important.

133 and the number of cells showing synchronous calcium oscillations is reduced.

134 ted to determine whether the second phase of calcium oscillations is required to reactivate the MPF a

135 The nature of the calcium oscillations is similar for LCOs produced by rhi

136 The KOR-mediated inhibition of spontaneous calcium oscillations may therefore be a consequence of p

137 le model for intercellular communication via calcium oscillations, motivated in part by a recent expe

138 old, and external calcium elicited cytosolic calcium oscillations of differing amplitudes and frequen

139 As one varies the frequency of calcium oscillations of the donor cell, the sensor cell

140 lacking the phosphatase CD45 did not exhibit calcium oscillations or ERK/pp90rsk activation, nor did

141 oscillations may be in phase with cytosolic calcium oscillations or out of phase.

142 While an altered calcium oscillation pattern may be an indicator for hMSC

143 In the present study we characterized the calcium oscillation profiles in hMSCs before and after s

144 malian cortex, where low-frequency (0.01 Hz) calcium oscillations refine topographic maps.

145 The appearance of spontaneous calcium oscillations, reflecting synchronous neuronal ac

146 rrelated with the onset and cessation of the calcium oscillations required for subsequent cleavage, a

147 izocilpine maleate (MK-801), also suppressed calcium oscillations, revealing a dependence on glutamat

148 2-myristate 13-acetate induced low amplitude calcium oscillations, slower translocation of cPLA(2)alp

149 erlying global plasma membrane PI(4,5)P2 and calcium oscillations spatially regulates actin dynamics,

150 ABA that impairs mycorrhizal factor-induced calcium oscillations, suggesting different modes of acti

151 Calcium oscillations suppress mitochondrial movements al

152 n the presence of full-length cyclin B1, the calcium oscillations terminate when cyclin B1 levels fal

153 ocytes or eggs to generate multiple wavelike calcium oscillations that arise at least in part from th

154 ar factor kappaB (NF-kappaB) proteins and of calcium oscillations that became apparent 1-3 d after TN

155 of symbiotic associations in plants requires calcium oscillations that must be decoded to invoke down

156 This work provides a model for decoding calcium oscillations that uses differential calcium bind

157 channel blocker nifedipine did not suppress calcium oscillations, the N-type calcium channel blocker

158 s are not required for fertilization-induced calcium oscillations, they do play a critical role in de

159 exhibit numerous oscillatory behaviors from calcium oscillations to circadian rhythms that recur dai

160 ate with rhizobia, showed Nod factor-induced calcium oscillations to S. fredii NGR234 Nod factors, bu

161 m channels (VGCCs), since diltiazem inhibits calcium oscillations under all conditions.

162 Calcium oscillations were activated in rice atrichoblast

163 These spontaneous calcium oscillations were blocked by 1 muM tetrodotoxin,

164 In RyR1-expressing cells, regenerative calcium oscillations were observed in response to caffei

165 ricted to central regions, compartmentalized calcium oscillations were sometimes observed.

166 spiking but also suppresses the frequency of calcium oscillations when applied at lower concentration

167 zation, are involved in both events, whereas calcium oscillations, which correlate with global fluctu

168 modulin and CCaMK would substantially mirror calcium oscillations, which typically have a 90 s period

169 riods match closely with those of intraislet calcium oscillations, while the slower oscillations are

170 ation in substrate phosphorylation follows a calcium oscillation with a lag of approximately 10 s.

171 d with CaMKII inhibition contributes both to calcium oscillations within astrocytes and ultimately co