ライフサイエンスコーパス: calcium oxalate

1 rinary stones, which largely are composed of calcium oxalate.

2 Most kidney stones are composed primarily of calcium oxalate.

3 e of indinavir and other substances, such as calcium oxalate.

4 ned in 11 patients: calcium phosphate (55%), calcium oxalate (18%), mixed calcium phosphate and oxala

5 n measurements show that, after consumption, calcium oxalate also interferes with the conversion of p

6 (70-71 U(Slope)), struvite (56-60 U(Slope)), calcium oxalate and calcium phosphate (17-59 U(Slope)),

7 Polycrystalline calcium oxalate and calcium phosphate calculi were found

8 done therapy, the supersaturation ratios for calcium oxalate and calcium phosphate fell by 25% and 35

9 us, the effect of potassium citrate on urine calcium oxalate and calcium phosphate supersaturation an

10 Furthermore, calcium oxalate and calcium phosphate supersaturation we

11 late, and phosphate levels lead to increased calcium oxalate and calcium phosphate supersaturation.

12 stals in renal stones, which are composed of calcium oxalate and calcium phosphate.

13 lower urine oxalate and supersaturation for calcium oxalate and uric acid.

14 cant genetic influence are the excretions of calcium, oxalate and citrate.

15 Most kidney stones consist of calcium oxalate, and higher urinary oxalate increases th

16 ves: amorphous calcium carbonate cystoliths, calcium oxalates, and silica phytoliths.

17 r handling of lithogenic substrates, such as calcium, oxalate, and phosphate, and of inhibitors of cr

18 uding gender, diet, and urinary excretion of calcium, oxalate, and uric acid.

19 um phosphate (BCP)-associated syndromes, and calcium oxalate arthritis.

20 Because aluminum citrate blocks calcium oxalate binding and toxicity in human kidney cel

21 treatment strategies target the formation of calcium oxalate but not its interaction with kidney tiss

22 e composition (uric acid, cystine, struvite, calcium oxalate, calcium phosphate, brushite), and 20 we

23 Approximately 80% of stones are composed of calcium oxalate (CaOx) and calcium phosphate (CaP); 10%

24 ost common form of renal stone disease, with calcium oxalate (CaOx) being the predominant constituent

25 inhibitors and appear capable of inhibiting calcium oxalate (CaOx) crystallization in vitro.

26 onolayers and exposed to oxalate (Ox) and/or calcium oxalate (CaOx) crystals to investigate cellular

27 e diet of the GHS rats leads to formation of calcium oxalate (CaOx) kidney stones.

28 1 and TNFR2 in human and murine kidneys with calcium oxalate (CaOx) nephrocalcinosis-related CKD comp

29 Nephrocalcinosis, acute calcium oxalate (CaOx) nephropathy, and renal stone dise

30 nephron of the kidney is supersaturated with calcium oxalate (CaOx), which crystallizes in the tubule

31 n culture (cIMCD) and selectively adsorbs to calcium oxalate (CaOx).

32 on, plasma oxalate concentration, and kidney calcium oxalate crystal deposition.

33 for 2 wk to induce hyperoxaluria and kidney calcium oxalate crystal deposition.

34 mplanted rats had a 43 to 88% rate of kidney calcium oxalate crystal deposition.

35 ormally high oxalate production resulting in calcium oxalate crystal formation and deposition in the

36 on were visually screened for alterations in calcium oxalate crystal formation.

37 showed no evidence for reduced inhibition of calcium oxalate crystal growth, so low inhibition of gro

38 UAP) is a urinary glycoprotein that inhibits calcium oxalate crystallization in vitro.

39 Their calcium oxalate crystallization inhibitory activity was

40 UAP exhibited a strong calcium oxalate crystallization inhibitory activity.

41 Oxalate-producing plants accumulate calcium oxalate crystals (CaOx(c)) in the range of 3-80%

42 Calcium oxalate crystals are widespread among animals an

43 Calcium oxalate crystals can also dissolve after renal c

44 eir detrimental effects on a chewing insect, calcium oxalate crystals do not negatively affect the pe

45 In combination, calcium oxalate crystals in leaves can act as a biochemi

46 sors give rise to oxalic acid (OxA) found in calcium oxalate crystals in specialized crystal idioblas

47 control rats, and resisted the formation of calcium oxalate crystals in their nephrons.

48 ae feeding on wild-type plants with abundant calcium oxalate crystals suffer significantly reduced gr

49 ts of M. truncatula with decreased levels of calcium oxalate crystals were used to assess the defensi

50 hat these cells may be more resistant to the calcium oxalate crystals.

51 s the nucleation, growth, and aggregation of calcium oxalate crystals.

52 oradiography for incorporation of (14)C into calcium oxalate crystals.

53 nderstanding of excess urinary excretions of calcium, oxalate, cystine, and uric acid.

54 Seven different classes of calcium oxalate defective mutants were identified that e

55 w a clear feeding preference for tissue from calcium oxalate-defective (cod) mutant lines cod5 and co

56 The calcium oxalate deficient 5 (cod5) mutant of Medicago tr

57 Oxalosis, or calcium oxalate deposition in the tissues, may develop i

58 All species examined have abundant calcium oxalate deposits around the veins.

59 either calcium oxalate monohydrate (COM) or calcium oxalate dihydrate (COD).

60 Crystals of calcium oxalate dihydrate can also nucleate directly on

61 e cell surface, calcium oxalate monohydrate, calcium oxalate dihydrate, and hydroxyapatite crystals a

62 ories based on their Raman spectrum: type I, calcium oxalate dihydrate, and type II, calcium hydroxya

63 ings into question the hypothesized roles of calcium oxalate formation in supporting tissue structure

64 Plants accumulate crystals of calcium oxalate in a variety of shapes, sizes, amounts,

65 alate leading to the deposition of insoluble calcium oxalate in the kidney.

66 provide a different therapeutic approach to calcium oxalate-induced injury.

67 Calcium oxalate is the most abundant insoluble mineral f

68 Calcium oxalate is the predominant component in 70-80% o

69 unmasking of which occurs in the hereditary calcium oxalate kidney stone disease primary hyperoxalur

70 evelopment of hyperoxaluria and/or recurrent calcium oxalate kidney stone disease.

71 inary oxalate is an important determinant of calcium oxalate kidney stone formation.

72 actors for inhibiting the crystallisation of calcium oxalate kidney stones in susceptible individuals

73 ariety of factors influence the formation of calcium oxalate kidney stones, including gender, diet, a

74 In the ectopic biomineralization of calcium oxalate kidney stones, the competition between c

75 member 1) in two unrelated individuals with calcium oxalate kidney stones.

76 ates of microcrystals, most commonly contain calcium oxalate monohydrate (COM) as the primary constit

77 be used to switch the surface morphology of calcium oxalate monohydrate (COM) back and forth, result

78 Effects of oxalate exposure on calcium oxalate monohydrate (COM) crystal adherence to t

79 t study was undertaken to identify potential calcium oxalate monohydrate (COM) crystal-binding protei

80 Here we examine this phenomenon in calcium oxalate monohydrate (COM) crystallization, a mod

81 a monomer, is known to inhibit the growth of calcium oxalate monohydrate (COM) crystals in renal tubu

82 The attachment of calcium oxalate monohydrate (COM) crystals to renal tubu

83 The nucleation and growth of calcium oxalate monohydrate (COM) crystals were studied

84 Interaction of calcium oxalate monohydrate (COM) crystals with renal ce

85 ury during exposure to oxalate ions (Ox) and calcium oxalate monohydrate (COM) crystals.

86 uman kidney stones are composed primarily of calcium oxalate monohydrate (COM) crystals.

87 alate kidney stones, the competition between calcium oxalate monohydrate (COM) formation and its inhi

88 which crystallizes in the tubules as either calcium oxalate monohydrate (COM) or calcium oxalate dih

89 Studies of calcium oxalate monohydrate (COM) precipitation at Langm

90 of these proteins in the crystallization of calcium oxalate monohydrate (COM), the most prominent co

91 Calcium oxalate monohydrate (COM), which plays a functio

92 Some calcium oxalate monohydrate (whewellite) was formed with

93 Calcium oxalate monohydrate and hydroxyapatite (calcium

94 correlated with a corresponding increase in calcium oxalate monohydrate crystal attachment to the ce

95 zed in vitro inhibitor of hydroxyapatite and calcium oxalate monohydrate crystal formation, but it is

96 Calcium oxalate monohydrate crystals are responsible for

97 Once present on the cell surface, calcium oxalate monohydrate, calcium oxalate dihydrate,

98 is regularly supersaturated with respect to calcium oxalate monohydrate, the most common solid phase

99 atography, and fractions were incubated with calcium oxalate monohydrate.

100 eraction with the positively charged face of calcium-oxalate monohydrate leads to formation of a pept

101 By imaging the growth of calcium-oxalate monohydrate under the influence of aspar

102 Here we report that crystals of calcium oxalate, monosodium urate, calcium pyrophosphate

103 s unclear why men have a higher incidence of calcium oxalate nephrolithiasis than women.

104 ance in primary hyperoxaluria and idiopathic calcium oxalate nephrolithiasis.

105 an increased incidence of hyperoxaluria and calcium oxalate nephrolithiasis.

106 and enteric hyperoxaluria or even idiopathic calcium oxalate nephrolithiasis.

107 igher urinary oxalate increases the risk for calcium oxalate nephrolithiasis.

108 Precipitation of insoluble manganese and calcium oxalate occurred under colonies growing on agar

109 The urinary relative supersaturations of calcium oxalate (P = 0.03) and brushite (P = 0.002) were

110 re discussed with respect to fine control of calcium oxalate precipitation and the concept of crystal

111 How and why many plants form crystals of calcium oxalate remain largely unknown.

112 ne glycol toxicity, apparently by inhibiting calcium oxalate's interaction with, and retention by, th

113 The equilibrium parameters for calcium oxalate solubility in tissue culture media were

114 COM crystals dispersed in saturated aqueous calcium oxalate solutions attached preferentially to the

115 time during immersion in the supersaturated calcium oxalate solutions.

116 Idiopathic calcium oxalate stone formation is a multifactorial dise

117 hanisms contribute to the increased risk for calcium oxalate stone formation observed in patients wit

118 Idiopathic calcium oxalate stone formation results from an interact

119 ading intestinal flora with a higher risk of calcium oxalate stone formation, possibly opening the do

120 ody size may be an important risk factor for calcium oxalate stone formation.

121 surgeries have further increased the risk of calcium oxalate stone formation.

122 absorption resulting in an increased risk of calcium oxalate stone formation.

123 reduction in the risk for being a recurrent calcium oxalate stone former.

124 involving 247 adult patients with recurrent calcium oxalate stones and 259 age-, gender-, and region

125 authors randomly assigned 99 persons who had calcium oxalate stones for the first time to a low anima

126 Thirty-six calcium oxalate stones were scanned in an anthropomorphi

127 A6 develop hyperoxalemia, hyperoxaluria, and calcium-oxalate stones as a result of a defect in intest

128 to increased risk include increased urinary calcium oxalate supersaturation, while urinary citrate,

129 logic studies with patients at high risk for calcium oxalate urolithiasis showed a direct correlation

130 ted with hyperoxaluria, especially recurrent calcium oxalate urolithiasis.

131 xalate, thereby preventing hyperoxaluria and calcium oxalate urolithiasis.

132 lacking Slc26a6 develop a high incidence of calcium oxalate urolithiasis.

133 with an increased risk of hyperoxaluria and calcium-oxalate urolithiasis.

134 uria, and of subsequent nephrocalcinosis and calcium-oxalate urolithiasis.

135 In addition, calcium oxalate was formed from the calcium present in t