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1 of the molecular and cellular mechanisms of calcium pyrophosphate dihydrate and apatite crystal form
2 the pathologic matrix mineralization seen in calcium pyrophosphate dihydrate and basic calcium phosph
3 the cellular responses to monosodium urate, calcium pyrophosphate dihydrate and basic calcium phosph
4 The pathologic matrix mineralization seen in calcium pyrophosphate dihydrate and basic calcium phosph
7 concerning clinical and etiologic aspects of calcium pyrophosphate dihydrate and basic calcium phosph
8 ystals of calcium oxalate, monosodium urate, calcium pyrophosphate dihydrate and cystine trigger casp
9 ved in the pathogenesis of monosodium urate, calcium pyrophosphate dihydrate and hydroxyapatite cryst
15 tion, and increased concentrations promoting calcium pyrophosphate dihydrate (CPPD) crystal depositio
17 characterization of the role of NTPPHase in calcium pyrophosphate dihydrate (CPPD) crystal depositio
18 he pathologic mineralization that results in calcium pyrophosphate dihydrate (CPPD) crystal formation
19 In this study, following our earlier work on calcium pyrophosphate dihydrate (CPPD) crystal-induced m
22 luding nanoscale silicon dioxide (NanoSiO2), calcium pyrophosphate dihydrate (CPPD) crystals, and mur
23 Articular calcium-containing crystals cause calcium pyrophosphate dihydrate (CPPD) deposition diseas
24 of fluid containing synthetic or native BCP, calcium pyrophosphate dihydrate (CPPD), or monosodium ur
26 of ePPi while excess levels of ePPi leads to calcium pyrophosphate dihydrate crystal deposition (CPPD
27 load is a likely source of pyrophosphate in calcium pyrophosphate dihydrate crystal deposition disea
29 hic arthropathy of the atlantoaxial joint in calcium pyrophosphate dihydrate crystal deposition disea
32 view, the author discusses various models of calcium pyrophosphate dihydrate crystal formation from e
36 young animals might promote the formation of calcium pyrophosphate dihydrate crystals in aged cartila
41 osition of either basic calcium phosphate or calcium pyrophosphate dihydrate crystals, remains unclea
44 radiographic techniques to the diagnosis of calcium pyrophosphate dihydrate deposition disease holds
45 nt as a definitive rheumatic disease such as calcium pyrophosphate dihydrate deposition disease or as
46 nts were identified that segregated with the calcium pyrophosphate dihydrate deposition disease pheno
47 t literature reminds us of the propensity of calcium pyrophosphate dihydrate deposition disease to mi
49 as a potential positional candidate gene for calcium pyrophosphate dihydrate deposition disease, and
51 evalence and significance of extra-articular calcium pyrophosphate dihydrate deposits, and demonstrat
52 play radiographically detectable crystals of calcium pyrophosphate dihydrate in their joint spaces.
54 ATD5 cells were basic calcium phosphate, not calcium pyrophosphate dihydrate, underlying the signific
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