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1  of the molecular and cellular mechanisms of calcium pyrophosphate dihydrate and apatite crystal form
2 the pathologic matrix mineralization seen in calcium pyrophosphate dihydrate and basic calcium phosph
3  the cellular responses to monosodium urate, calcium pyrophosphate dihydrate and basic calcium phosph
4 The pathologic matrix mineralization seen in calcium pyrophosphate dihydrate and basic calcium phosph
5                  Calcium crystals, including calcium pyrophosphate dihydrate and basic calcium phosph
6                                              Calcium pyrophosphate dihydrate and basic calcium phosph
7 concerning clinical and etiologic aspects of calcium pyrophosphate dihydrate and basic calcium phosph
8 ystals of calcium oxalate, monosodium urate, calcium pyrophosphate dihydrate and cystine trigger casp
9 ved in the pathogenesis of monosodium urate, calcium pyrophosphate dihydrate and hydroxyapatite cryst
10             In addition to monosodium urate, calcium pyrophosphate dihydrate, and apatite crystals, a
11                                              Calcium pyrophosphate dihydrate (CPPD) and basic calcium
12        Calcium deposition diseases caused by calcium pyrophosphate dihydrate (CPPD) and basic calcium
13                             Microcrystals of calcium pyrophosphate dihydrate (CPPD) and monosodium ur
14                  Familial autosomal dominant calcium pyrophosphate dihydrate (CPPD) chondrocalcinosis
15 tion, and increased concentrations promoting calcium pyrophosphate dihydrate (CPPD) crystal depositio
16                                              Calcium pyrophosphate dihydrate (CPPD) crystal depositio
17  characterization of the role of NTPPHase in calcium pyrophosphate dihydrate (CPPD) crystal depositio
18 he pathologic mineralization that results in calcium pyrophosphate dihydrate (CPPD) crystal formation
19 In this study, following our earlier work on calcium pyrophosphate dihydrate (CPPD) crystal-induced m
20       Monosodium urate monohydrate (MSU) and calcium pyrophosphate dihydrate (CPPD) crystals cause ac
21                             The formation of calcium pyrophosphate dihydrate (CPPD) crystals in artic
22 luding nanoscale silicon dioxide (NanoSiO2), calcium pyrophosphate dihydrate (CPPD) crystals, and mur
23  Articular calcium-containing crystals cause calcium pyrophosphate dihydrate (CPPD) deposition diseas
24 of fluid containing synthetic or native BCP, calcium pyrophosphate dihydrate (CPPD), or monosodium ur
25 ed spectroscopy revealed crystals resembling calcium pyrophosphate dihydrate (CPPD).
26 of ePPi while excess levels of ePPi leads to calcium pyrophosphate dihydrate crystal deposition (CPPD
27  load is a likely source of pyrophosphate in calcium pyrophosphate dihydrate crystal deposition disea
28                                  In familial calcium pyrophosphate dihydrate crystal deposition disea
29 hic arthropathy of the atlantoaxial joint in calcium pyrophosphate dihydrate crystal deposition disea
30 c pyrophosphate in cartilage matrix leads to calcium pyrophosphate dihydrate crystal deposits.
31                            Current models of calcium pyrophosphate dihydrate crystal formation are le
32 view, the author discusses various models of calcium pyrophosphate dihydrate crystal formation from e
33 l for studying the major factors involved in calcium pyrophosphate dihydrate crystal formation.
34                                              Calcium pyrophosphate dihydrate crystals are common comp
35        Progress in understanding why and how calcium pyrophosphate dihydrate crystals form in articul
36 young animals might promote the formation of calcium pyrophosphate dihydrate crystals in aged cartila
37                               Elimination of calcium pyrophosphate dihydrate crystals may occur extra
38                        Tumoral deposition of calcium pyrophosphate dihydrate crystals may occur in si
39                                The effect of calcium pyrophosphate dihydrate crystals on the progress
40                                  Addition of calcium pyrophosphate dihydrate crystals to a lapine men
41 osition of either basic calcium phosphate or calcium pyrophosphate dihydrate crystals, remains unclea
42 sphate (PPi) that promotes the deposition of calcium pyrophosphate dihydrate crystals.
43         A discussion of ANKH as the familial calcium pyrophosphate dihydrate deposition disease gene
44  radiographic techniques to the diagnosis of calcium pyrophosphate dihydrate deposition disease holds
45 nt as a definitive rheumatic disease such as calcium pyrophosphate dihydrate deposition disease or as
46 nts were identified that segregated with the calcium pyrophosphate dihydrate deposition disease pheno
47 t literature reminds us of the propensity of calcium pyrophosphate dihydrate deposition disease to mi
48               In genetic studies of familial calcium pyrophosphate dihydrate deposition disease, a re
49 as a potential positional candidate gene for calcium pyrophosphate dihydrate deposition disease, and
50 rs: craniometaphyseal dysplasia and familial calcium pyrophosphate dihydrate deposition disease.
51 evalence and significance of extra-articular calcium pyrophosphate dihydrate deposits, and demonstrat
52 play radiographically detectable crystals of calcium pyrophosphate dihydrate in their joint spaces.
53 lysis supports the role of ANKH mutations in calcium pyrophosphate dihydrate-induced arthritis.
54 ATD5 cells were basic calcium phosphate, not calcium pyrophosphate dihydrate, underlying the signific

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