ライフサイエンスコーパス: calcium-activated

1 ne a signaling pathway from chloride channel calcium-activated 1 (CLCA1) to MAPK13 that is responsibl

2 ctrometry identified murine chloride channel calcium-activated 1 (mCLCA1) as the 10.1.1 Ag, a 90-kDa

3 s, Western blot analysis of chloride channel calcium activated 3 (CLCA3) expression in lung homogenat

4 se include mucin Muc5b, the chloride channel calcium-activated 3 (Clca3), macrophage inflammatory pro

5 Lowering the activity of Gelsolin, a known calcium-activated actin filament-severing protein, also

6 chment), which regulates voltage-independent calcium-activated action potentials at the neuronal syna

7 w that the functional expression of the slow calcium-activated afterhyperpolarizing current in pyrami

8 ensor proteins in the activation of the slow calcium-activated afterhyperpolarizing current.

9 Both calcium-activated and stretch-activated tensions increas

10 significantly from caspases because they are calcium-activated, arginine-specific peptidases that do

11 tress axis-regulated exon (STREX)-containing calcium-activated big potassium (BKCa) channel splice va

12 Calcium-activated binding of S100A4 to CD16A, promoted b

13 Large conductance calcium-activated (BK) channels are broadly expressed in

14 This increase in cytosolic calcium activated by postsynaptic Gs-coupled CRF recepto

15 emonstrated that the increase in cytoplasmic calcium activated Ca(2+) dependent potassium currents an

16 amine receptor (amitraz metabolite), and the calcium-activated calcium channel (diamides).

17 ha-mediated Ser648 phosphorylation abrogated calcium-activated calmodulin (CaM) binding to the regula

18 e backbone and side-chain methyl dynamics of calcium-activated calmodulin in complex with a peptide c

19 cing it into a peptide that is recognized by calcium-activated calmodulin.

20 Proteolytic processing of PKCalpha by calcium-activated calpain activates pathological cardiac

21 Moreover, these findings identify calcium-activated calpains as powerful modulators of cel

22 gulation of RyR1 by unoxidized CaM, in which calcium-activated CaM acts to enhance the calcium sensit

23 ficantly affect the high-affinity binding of calcium-activated CaM to the CaM-binding sequence of RyR

24 igh-affinity association of both apo-CaM and calcium-activated CaM with RyRp.

25 norganic phosphate [Pi] were investigated in calcium-activated cardiac myofibrils.

26 One candidate subpopulation expresses the calcium activated cation channel TRPM5 (transient recept

27 Calcium-activated CCTalpha nuclear import is mediated by

28 ial cells (PDECs) have been shown to express calcium activated chloride channels (CaCCs) and there is

29 TMEM16A (ANO1) is a calcium-activated chloride channel (CaCC) expressed in s

30 eins with unknown function 16 (TMEM16A) is a calcium-activated chloride channel (CaCC) important for

31 ver, the molecular identity of the olfactory calcium-activated chloride channel (CaCC) is unknown.

32 Here, we show that the recently identified calcium-activated chloride channel (CaCC) TMEM16A is exp

33 te that TMEM16A, an evolutionarily conserved calcium-activated chloride channel (CaCC), regulates cyt

34 inhibit self-cleavage prevent activation of calcium-activated chloride channel (CaCC)-mediated chlor

35 TMEM16A (ANO1) functions as a calcium-activated chloride channel (CaCC).

36 s cell metaplasia, depends on members of the calcium-activated chloride channel (CLCA) gene family.

37 Calcium-activated chloride channel (CLCA) proteins were

38 BEST1 is presumed to assemble into a calcium-activated chloride channel and be involved in ch

39 The calcium-activated chloride channel ANO1 regulates multip

40 To date, only five mutations in the proposed calcium-activated chloride channel ANO5/TMEM16E gene hav

41 The calcium-activated chloride channel anoctamin 1 (ANO1) is

42 s in the ANO5 gene, which encodes a putative calcium-activated chloride channel belonging to the Anoc

43 EM16A), also called anoctamin 1 (ANO1), is a calcium-activated chloride channel expressed widely mamm

44 Human bestrophin-1 (hBest1) is a calcium-activated chloride channel from the retinal pigm

45 16A is the major constituent of the vascular calcium-activated chloride channel in rat pulmonary arte

46 inhibited the expression of Gob5, a putative calcium-activated chloride channel involved in the regul

47 Calcium-activated chloride channel regulator 1 (CLCA1) a

48 nes, including IL13, IL5, periostin (POSTN), calcium-activated chloride channel regulator 1 (CLCA1),

49 ), which revealed an increased expression of calcium-activated chloride channel regulator 1 (CLCA1).

50 ANO1 is a calcium-activated chloride channel that is frequently ov

51 Human Bestrophin 1 (hBest1) is a calcium-activated chloride channel that regulates neuron

52 Inducible expression of either CFTR or the calcium-activated chloride channel TMEM16A attenuated th

53 The calcium-activated chloride channel TMEM16A is a member o

54 The calcium-activated chloride channel TMEM16A is overexpres

55 KEY POINTS: The calcium-activated chloride channel TMEM16A provides a pa

56 Targeted disruption of the calcium-activated chloride channel TMEM16A, also known a

57 A selective inhibitor of the calcium-activated chloride channel TMEM16A, N-((4-methox

58 ANO1, a calcium-activated chloride channel, is highly expressed

59 propose that ANO2 constitutes the olfactory calcium-activated chloride channel.

60 y was performed to detect ANO4 activity as a calcium-activated chloride channel.

61 Calcium-activated chloride channels (CaCC) with similar

62 Calcium-activated chloride channels (CaCCs) are key play

63 Calcium-activated chloride channels (CaCCs) are major re

64 Calcium-activated chloride channels (CaCCs) are widely e

65 Calcium-activated chloride channels (CaCCs) encoded by T

66 Calcium-activated chloride channels (CaCCs) play importa

67 r components of the physiologically relevant calcium-activated chloride channels (CaCCs) present in m

68 Bestrophin calcium-activated chloride channels (CaCCs) regulate the

69 TMEM16A forms calcium-activated chloride channels (CaCCs) that regulat

70 TMEM16A and TMEM16B are calcium-activated chloride channels (CaCCs) with importa

71 enuated inward currents carried by TRPC2 and calcium-activated chloride channels (CACCs).

72 hippocampal neuronal signaling that involves calcium-activated chloride channels (CaCCs).

73 n absence of expression/activity of reported calcium-activated chloride channels (TMEM16A, Bestrophin

74 an intrinsic property observed in endogenous calcium-activated chloride channels and could be relevan

75 ng pathway in the VNO and the requirement of calcium-activated chloride channels currents to mediate

76 Recently, calcium-activated chloride channels have been shown to c

77 Evidence is provided for a role for calcium-activated chloride channels such as TMEM16a in G

78 r the analyses of physiological functions of calcium-activated chloride channels that contain TMEM16A

79 Calcium-activated chloride channels TMEM16A and TMEM16B

80 gative effect through interaction with other calcium-activated chloride channels, such as hBest2, 3,

81 ffects of calcium signaling on CFTR or other calcium-activated chloride channels; here, we investigat

82 vertebrate olfactory signal transduction, a calcium-activated chloride conductance serves as a major

83 ide secretion, which consists, in part, of a calcium-activated chloride conductance.

84 re, our analysis suggests that activation of calcium-activated chloride conductances by intracellular

85 rowth, primarily due to HIF-1alpha-dependent calcium-activated chloride secretion.

86 ient for HIF-1alpha almost completely lacked calcium-activated chloride secretion.

87 TMEM16A was found recently to be a calcium-activated Cl(-) channel (CaCC).

88 lso found to strongly inhibit the intestinal calcium-activated Cl(-) channel TMEM16A by a voltage-ind

89 ost two decades, it has been postulated that calcium-activated Cl(-) channels (CaCCs) play a role in

90 The chloride channel calcium-activated (CLCA) family are secreted proteins th

91 Although a weaker activator than zinc, calcium activated CPE in vitro.

92 a potential feed-forward mechanism in which calcium-activated CPK32 activates CNGC18, further promot

93 the mean intracellular activity of calpains, calcium-activated cysteine proteases that are known to c

94 Calpains, calcium-activated cysteine proteases, have been shown to

95 The neuronal signal is transmitted through calcium-activated dense core vesicle neurosecretion.

96 A new study reveals that a calcium-activated endoribonuclease of the EndoU protein

97 microarrays, we found that chloride channel, calcium-activated, family member 1 (CLCA1), periostin, a

98 force of diaphragm strips, absolute maximal calcium activated force, and maximal specific calcium-ac

99 Compared with control, maximal calcium-activated force and calcium sensitivity declined

100 alcium activated force, and maximal specific calcium-activated force of permeabilized diaphragm fiber

101 ss spectrometric analysis of the products of calcium-activated hydrolysis of endogenous mitochondrial

102 , as a result of the excessive activation of calcium-activated hyperpolarizing conductances.

103 ium influx accompanying such signaling opens calcium-activated ion channels for feedback regulation.

104 By aligning distantly related calcium-activated ion channels in the TMEM16 family and

105 The calcium activated K(+) channel KCa3.1 plays an important

106 est in the pharmacology of large conductance calcium-activated K (BK) channels.

107 The Slack (sequence like a calcium-activated K channel) and Slick (sequence like an

108 ses of ion channels, including examples from calcium-activated K(+) (BK(Ca)), voltage-activated K(+)

109 idal neurons revealed that large-conductance calcium-activated K(+) (BK) channel open probability was

110 Large conductance, calcium-activated K(+) (BK) channels are important regul

111 Large-conductance voltage- and calcium-activated K(+) (BK) channels are key physiologic

112 ons, FMRP interaction with large-conductance calcium-activated K(+) (BK) channels, specifically their

113 Large-conductance calcium-activated K(+) (BK-type) channels, abundantly di

114 Voltage-dependent and calcium-activated K(+) (MaxiK, BK) channels are widely e

115 The calcium-activated K(+) channel KCa3.1 plays an important

116 tigated two candidate ethanol effectors, the calcium-activated K(+) channel SLO-1 and gap junctions,

117 excitotoxicity: anti-apoptotic Bcl-2, and a calcium-activated K(+) channel, SK2.

118 1-2.3) and intermediate-conductance (KCa3.1) calcium-activated K(+) channels are critically involved

119 ctance (KCa3.1) and small-conductance (KCa2) calcium-activated K(+) channels are gated by calcium bin

120 (KCa2) and intermediate-conductance (KCa3.1) calcium-activated K(+) channels are voltage-independent

121 mediated by activation of small conductance calcium-activated K(+) channels in PDGFRalpha(+) cells,

122 Under noisy conditions, calcium-activated K(+) current (I(AHP)) improved efficie

123 assium efflux through voltage gated (Kv) and calcium activated (K(Ca)) potassium channels.

124 goal of this study was to determine whether calcium-activated kinases such as calcium/calmodulin-dep

125 l increase in phosphorylation likely tied to calcium-activated kinases.

126 n synaptic transmission properties, LTD, and calcium-activated membrane channels of hippocampal CA1 p

127 id, an antagonist of a previously identified calcium activated non-selective cation channel (I(CAN)).

128 while flufenamic acid, an antagonist for the calcium-activated non-selective cation conductance (ICAN

129 ity, while flufenamic acid, a blocker of the calcium-activated non-selective cation conductance, abol

130 ion further excites the cell by recruiting a calcium-activated non-selective cation current (ICAN) ca

131 Calcium-activated non-specific cation (CAN) channels con

132 d synaptic efficacy, and a small-conductance calcium-activated nonselective cation channel, TMEM16F,

133 nigral plateau potentials are mediated by a calcium-activated nonselective cation conductance (I(CAN

134 re abolished in low-sodium buffer and by the calcium-activated nonselective cation conductance blocke

135 lar calcium and appeared to be mediated by a calcium-activated nonselective cation current (I(CAN)).

136 carinic afterdepolarization is mediated by a calcium-activated nonselective cation current, suggestin

137 ained responses were found to be mediated by calcium-activated nonselective cationic current induced

138 Here we show that calcium activated NOX-independent NETosis is fast and me

139 tinguished DBQD type 1 and identified CANT1 (calcium activated nucleotidase 1) mutations as responsib

140 Human calcium-activated nucleotidase (CAN) exists as both a me

141 Human soluble calcium-activated nucleotidase 1 (hSCAN-1) represents a

142 plications for engineering the soluble human calcium-activated nucleotidase for clinical applications

143 sponses to herbivory can be separated into a calcium-activated oxidative response and a K(+) -depende

144 The calcium-activated phosphatase calcineurin is regulated b

145 cium channels but not on the activity of the calcium-activated phosphatase calcineurin, and was oppos

146 The calcium pathway involved the calcium-activated phosphatase calcineurin, which stabili

147 TRPM4 is a calcium-activated, phosphatidylinositol-4,5-bisphosphate

148 tructure-function relationship of this novel calcium-activated phosphoryl transfer enzyme.

149 rylation was phenocopied only by deletion of calcium-activated PKC-2.

150 of (1) sensory nerves and large-conductance calcium activated potassium (BKCa) channels, and (2) nit

151 Large-conductance calcium-activated potassium (BK(Ca)) channels were studi

152 ulting from a reduction in large-conductance calcium-activated potassium (BK) and subthreshold-activa

153 lcium channel CaV1.3 and the big conductance calcium-activated potassium (BK) channel are preferentia

154 alternative splices of the large-conductance calcium-activated potassium (BK) channel have been the s

155 The large-conductance calcium-activated potassium (BK) channel lacks a classic

156 The large conductance, voltage and calcium-activated potassium (BK) channel negatively regu

157 ract with the auxiliary beta4 subunit of the calcium-activated potassium (BK) channel; this interacti

158 ction in the expression of large-conductance calcium-activated potassium (BK) channels and Kv3.3 volt

159 ncreases in cytoplasmic Ca(2+), voltage- and calcium-activated potassium (BK) channels and their modu

160 Large conductance voltage- and calcium-activated potassium (BK) channels are highly exp

161 Large conductance voltage- and calcium-activated potassium (BK) channels are important

162 The large-conductance, calcium-activated potassium (BK) channels help eliminate

163 Calcium-activated potassium (BK) channels play a central

164 MRP's interaction with the large-conductance calcium-activated potassium (BK) channels that modulate

165 h nonmammals and mammals, large-conductance, calcium-activated potassium (BK) channels underlie a pri

166 spines act by suppressing large-conductance calcium-activated potassium (BK) channels, and this effe

167 of ion channels, including large conductance calcium-activated potassium (BK) channels.

168 The appearance of large-conductance, calcium-activated potassium (BK) current is a hallmark o

169 Large-conductance, voltage- and calcium-activated potassium (BK, or K(Ca)1.1) channels a

170 ABSTRACT: Large conductance, calcium-activated potassium (BKCa ) channels have numero

171 acemaking is caused by reduced activation of calcium-activated potassium (K(Ca)) channels and was rev

172 We investigated the role of calcium-activated potassium (K(Ca)) channels in this dys

173 larization potential, a voltage signature of calcium-activated potassium (Kca) channel activity.

174 ting through nitric oxide synthase (NOS) and calcium-activated potassium (KCa) channels in young adul

175 ctivation of nitric oxide synthase (NOS) and calcium-activated potassium (KCa) channels.

176 firing through their close association with calcium-activated potassium (KCa) channels.

177 Small-conductance calcium-activated potassium (KCa2) channels have also be

178 High-conductance calcium-activated potassium (Maxi-K) channels are presen

179 Large conductance calcium-activated potassium (MaxiK) channels play a pivo

180 are occluded by apamin, a small-conductance calcium-activated potassium (SK(Ca)) channel blocker.

181 vivo, and were mediated by small-conductance calcium-activated potassium (SK) channel and CB1 cannabi

182 Small conductance calcium-activated potassium (SK) channels are required f

183 um conductance mediated by small-conductance calcium-activated potassium (SK) channels in rat MNTB pr

184 +) conductance mediated by small-conductance calcium-activated potassium (SK) channels in the MNTB ne

185 Pharmacological inhibition of calcium-activated potassium (SK) channels increases the

186 Small-conductance calcium-activated potassium (SK) channels mediate a pota

187 Small-conductance calcium-activated potassium (SK) channels mediate medium

188 Small-conductance calcium-activated potassium (SK) channels play an import

189 Small-conductance calcium-activated potassium (SK) channels regulate actio

190 increased contribution of small-conductance calcium-activated potassium (SK) channels.

191 Small conductance calcium-activated potassium (SK2/K(Ca)2.2) channels are

192 Large-conductance calcium-activated potassium BK channels are widely expre

193 nique splice variant of a large conductance, calcium-activated potassium channel (BK channel).

194 ET-cGMP-S (50 muM), and the high-conductance calcium-activated potassium channel (BK(Ca) channel) inh

195 The Drosophila large-conductance calcium-activated potassium channel (dSlo) binds to and

196 Large-conductance calcium-activated potassium channel (KCa1.1; BK, Slo1, M

197 is associated with reduced small-conductance calcium-activated potassium channel (SK) currents and de

198 inding and also normalized large-conductance calcium-activated potassium channel activity.

199 ized protein levels of the large conductance calcium-activated potassium channel and the water channe

200 ely prolonged by small-conductance (SK-type) calcium-activated potassium channel blockers in normally

201 fication of a role for the large conductance calcium-activated potassium channel brings new thinking

202 nergic neuron burst firing by decreasing the calcium-activated potassium channel current (SK), as wel

203 rther demonstrate that the activation of the calcium-activated potassium channel is sufficient to ind

204 The intermediate conductance calcium-activated potassium channel KCa3.1 contributes t

205 The calcium-activated potassium channel KCa3.1 controls diff

206 The calcium-activated potassium channel KCa3.1 is critically

207 The intermediate-conductance calcium-activated potassium channel KCa3.1 is expressed

208 members of the small-intermediate family of calcium-activated potassium channel proteins.

209 We found that the calcium-activated potassium channel SK3 and the G protei

210 eterminant of its anthelmintic effect is the calcium-activated potassium channel SLO-1.

211 ated potassium channel and large-conductance calcium-activated potassium channel, respectively).

212 ing peptide (GRP) and the small conductance, calcium-activated potassium channel, SK2.

213 releasing peptide and the small conductance, calcium-activated potassium channel, SK2.

214 g protein) modulates the Drosophila SLOWPOKE calcium-activated potassium channel.

215 um efflux from RBCs is the Gardos channel, a calcium-activated potassium channel.

216 These findings identify calcium-activated potassium channelopathy as a cause of

217 he presence of functional large-conductance, calcium-activated potassium channels (BK channels) on th

218 RATIONALE: Large-conductance calcium-activated potassium channels (BK) are composed o

219 Voltage- and calcium-activated potassium channels (BK) are important

220 Large-conductance calcium-activated potassium channels (BK) are potent neg

221 Mammalian large-conductance, voltage- and calcium-activated potassium channels (BK, K(Ca)1.1) are

222 Large conductance calcium-activated potassium channels (BK, MaxiK, Slo) ha

223 howed several significant results, including calcium-activated potassium channels (GO:0016286; P=2.30

224 ctifier channels (I(KV)) and noninactivating calcium-activated potassium channels (I(BK,steady)), and

225 co-localization of intermediate-conductance calcium-activated potassium channels (IKCa) and IP3 rece

226 e colocalization of intermediate-conductance calcium-activated potassium channels (IKCa) and TRPV4 ch

227 voltage-gated potassium channels (K(v)) and calcium-activated potassium channels (K(Ca)).

228 Large conductance voltage- and calcium-activated potassium channels (MaxiK, BK(Ca)) are

229 Small conductance calcium-activated potassium channels (SK channels) are p

230 (HVA) (N- and P/Q-type) calcium channels and calcium-activated potassium channels (SKKCa).

231 e established by the interactions between BK calcium-activated potassium channels and an L-type calci

232 mechanisms underlying seizure generation (BK calcium-activated potassium channels and interneuron-exp

233 d after acute dissociation, we found that BK calcium-activated potassium channels and Kv2 channels bo

234 This work shows that BK calcium-activated potassium channels and Kv2 voltage-act

235 f N-type voltage-gated calcium channels with calcium-activated potassium channels in DCN neurons.

236 f calcium-calmodulin-dependent kinase II and calcium-activated potassium channels in mediating these

237 Calcium-activated potassium channels regulate AHP and ex

238 d is abolished by blocking small conductance calcium-activated potassium channels with apamin.

239 ent is reduced by blocking large conductance calcium-activated potassium channels with iberiotoxin, a

240 For large-conductance calcium-activated potassium channels, data are satisfact

241 Administration of an activator of calcium-activated potassium channels, SKA-31, partially

242 cellular calcium that gated surface-membrane calcium-activated potassium channels.

243 ncreases the expression of small-conductance calcium-activated potassium channels.

244 I(AHP) was occluded by previous blockade of calcium-activated potassium channels.

245 ropic acetylcholine receptors and associated calcium-activated potassium channels.

246 enced by the activation of small-conductance calcium-activated potassium channels.

247 ue to an increase in the open probability of calcium-activated potassium channels.

248 ation of small- and intermediate-conductance calcium-activated potassium channels.

249 d by a nonselective cation conductance and a calcium-activated potassium conductance (SK), respective

250 , we show that the excessive activation of a calcium-activated potassium conductance disrupts the ace

251 The slow afterhyperpolarization (sAHP) is a calcium-activated potassium conductance with critical ro

252 endritic activation of the small conductance calcium-activated potassium current, SK.

253 hyperpolarization that is mediated by a slow calcium-activated potassium current.

254 perpolarizations that are mediated by a slow calcium-activated potassium current.

255 Pretreatment with blockers of calcium-activated potassium currents (I(KCa)) reproduced

256 -clamp) that changes in the leak, sodium and calcium-activated potassium currents are central to thes

257 plasticity mediated by reductions in BK-type calcium-activated potassium currents in spontaneously fi

258 and a reduction in the fast-inactivating and calcium-activated potassium currents.

259 c outward currents through small-conductance calcium-activated potassium SK2 channels.

260 he contribution of the SK (small-conductance calcium-activated potassium) channel to neuronal functio

261 r firing range, and of SK (small-conductance calcium-activated potassium) currents, which were essent

262 a2, Myl3, and Myom1, myofibril proteins; and calcium-activated potassium-channel gene activity (KCNMB

263 ovary cells' plasma membrane gave rise to a calcium-activated, potassium-selective activity in patch

264 Length-tension relationships in maximally calcium activated preparations are relatively shallow an

265 c mouse model to investigate the role of the calcium-activated protease calpain in the pathogenesis o

266 tic cleavage of p35 to a p25 fragment by the calcium-activated protease calpain or by phosphorylation

267 y calpeptin and MDL-28170, inhibitors of the calcium-activated protease calpain.

268 ted the consequences of physiologic calpain (calcium-activated protease) activity in cultured cardiom

269 ins220 was found to occur through calpain, a calcium-activated protease.

270 Although participation of calpain calcium-activated proteases in post-necrotic myocardial

271 We report that calpastatin, an inhibitor of calcium-activated proteases of the calpain family, funct

272 animal models, the dysregulated activity of calcium-activated proteases, calpains, contributes direc

273 could cleave filamin A through activation of calcium-activated proteases, such as calpains.

274 The calcium activated protein, calcium/calmodulin-dependent

275 We have found that the calcium-activated protein for secretion (CAPS) protein i

276 Additionally, the synaptic protein UNC-31 [calcium-activated protein for secretion (CAPS)] acts thr

277 We investigated the role of the calcium-activated protein phosphatase calcineurin in sev

278 Here we show that the calcium-activated protein phosphatase calcineurin is als

279 cataracts, and opacification may result from calcium-activated proteolysis.

280 suggest CXCL12 regulates restitution through calcium-activated Pyk2 localized to active focal adhesio

281 Together, these results demonstrate that calcium-activated removal of RNA from membranes by Xendo

282 entify a unique mechanism of virus-mediated, calcium-activated signaling that initiates autophagy and

283 blocked by apamin, a selective antagonist of calcium-activated SK channels.

284 yed rectifier, A-type, and small-conductance calcium-activated (SK) potassium and HCN) and two recept

285 e of reduced activation of small-conductance calcium-activated (SK) potassium channels.

286 ntaining ionotropic receptors and associated calcium-activated (SK2) potassium channels, providing th

287 ce was mediated via intermediate conductance calcium-activated (SK4) potassium channels.

288 eartbeat, cardiac contractility results from calcium-activated sliding of actin thin filaments toward

289 The calcium-activated slow afterhyperpolarization (sAHP) is

290 t KCNQ channels might partially underlie the calcium-activated slow afterhyperpolarization (sAHP), a

291 om KCNQ-deficient mice, we observed that the calcium-activated slow afterhyperpolarization current (I

292 We focus on calcium-activated small conductance (SK) potassium chann

293 ndependent NETosis is fast and mediated by a calcium-activated small conductance potassium (SK) chann

294 The calcium-activated small conductance potassium channel SK

295 holinergic receptors functionally coupled to calcium-activated, small conductance (SK2) potassium cha

296 nge likely used during flight), stretch- and calcium-activated tension contributed 80% and 20%, respe

297 We found that EGF and extracellular calcium activated the C-terminus of ERalpha and the acti

298 Muscle cell preparations were calcium activated to yield 50% maximal force, after whic

299 he determinants of alpha-KTx specificity for calcium-activated versus voltage-dependent potassium cha

300 Our study links calpain and dysferlin in the calcium-activated vesicle fusion of membrane repair, pla