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1 g protein) modulates the Drosophila SLOWPOKE calcium-activated potassium channel.
2 um efflux from RBCs is the Gardos channel, a calcium-activated potassium channel.
3 e Drosophila slowpoke gene encodes a BK-type calcium-activated potassium channel.
4 ither large- (BK) or small- (SK) conductance calcium-activated potassium channels.
5 o mediate efferent inhibition via associated calcium-activated potassium channels.
6 current, and higher concentrations activated calcium-activated potassium channels.
7 ropic acetylcholine receptors and associated calcium-activated potassium channels.
8 enced by the activation of small-conductance calcium-activated potassium channels.
9 ue to an increase in the open probability of calcium-activated potassium channels.
10 ation of small- and intermediate-conductance calcium-activated potassium channels.
11 cellular calcium that gated surface-membrane calcium-activated potassium channels.
12 ncreases the expression of small-conductance calcium-activated potassium channels.
13  I(AHP) was occluded by previous blockade of calcium-activated potassium channels.
14 urkinje neurons via decreased recruitment of calcium-activated potassium channels.
15 paradoxically mimics the effects of blocking calcium-activated potassium channels.
16 nels is to provide calcium for activation of calcium-activated potassium channels.
17 ium influx couples to large conductance (BK) calcium-activated potassium channels.
18 inding and also normalized large-conductance calcium-activated potassium channel activity.
19 ized protein levels of the large conductance calcium-activated potassium channel and the water channe
20 e established by the interactions between BK calcium-activated potassium channels and an L-type calci
21 mechanisms underlying seizure generation (BK calcium-activated potassium channels and interneuron-exp
22 d after acute dissociation, we found that BK calcium-activated potassium channels and Kv2 channels bo
23                      This work shows that BK calcium-activated potassium channels and Kv2 voltage-act
24 results in the opening of large-conductance, calcium-activated potassium channels, and an attendant h
25                     Here we demonstrate that calcium-activated potassium channels are activated exclu
26  P/Q-type voltage-gated calcium channels and calcium-activated potassium channels are required for no
27 he cloning and characterization of two novel calcium-activated potassium channel beta subunits, hKCNM
28 nique splice variant of a large conductance, calcium-activated potassium channel (BK channel).
29                           A high-conductance calcium-activated potassium channel (BK KCa) was charact
30 ET-cGMP-S (50 muM), and the high-conductance calcium-activated potassium channel (BK(Ca) channel) inh
31  the function of both the large conductance, calcium-activated potassium channel (BK) and specific me
32                            Large-conductance calcium-activated potassium channels (BK channels) are a
33 he presence of functional large-conductance, calcium-activated potassium channels (BK channels) on th
34                           Large conductance, calcium-activated potassium channels (BK(Ca) or maxi-K)
35                 RATIONALE: Large-conductance calcium-activated potassium channels (BK) are composed o
36                                 Voltage- and calcium-activated potassium channels (BK) are important
37                            Large-conductance calcium-activated potassium channels (BK) are potent neg
38 fects of ethanol (EtOH) on large-conductance calcium-activated potassium channels (BK) in cell bodies
39    Mammalian large-conductance, voltage- and calcium-activated potassium channels (BK, K(Ca)1.1) are
40                            Large conductance calcium-activated potassium channels (BK, MaxiK, Slo) ha
41 as recombinant cell lines (large-conductance calcium-activated potassium channel, BK(Ca)).
42 rt through cGMP-mediated activation of large calcium-activated potassium channels (BKCa).
43 as limited by activation of putative BK-type calcium-activated potassium channels (blocked by 250 mic
44 reductions in the sensitivity to the BK-type calcium-activated potassium channel blocker iberiotoxin.
45 y the CYP inhibitor, SKF525A, but not by the calcium-activated potassium channel blocker, charybdotox
46 ely prolonged by small-conductance (SK-type) calcium-activated potassium channel blockers in normally
47 fication of a role for the large conductance calcium-activated potassium channel brings new thinking
48                                   Inhibiting calcium-activated potassium channels by charybdotoxin or
49 vessels to adenosine; however, inhibition of calcium-activated potassium channels by iberiotoxin had
50 owed that slowpoke mutations, which affect a calcium-activated potassium channel, cause severe song a
51 mino acid level with three small conductance calcium-activated potassium channels cloned from brain.
52 nergic neuron burst firing by decreasing the calcium-activated potassium channel current (SK), as wel
53 ntial (AP) repolarization; small-conductance calcium-activated potassium channel currents generate an
54                        For large-conductance calcium-activated potassium channels, data are satisfact
55             The Drosophila large-conductance calcium-activated potassium channel (dSlo) binds to and
56 ates the fibroblast intermediate conductance calcium-activated potassium channel, FIK, a positive reg
57 hKCa4, encoding an intermediate conductance, calcium-activated potassium channel from a human lymph n
58 tional regulation of the Drosophila slowpoke calcium-activated potassium channel gene is complex.
59 a2, Myl3, and Myom1, myofibril proteins; and calcium-activated potassium-channel gene activity (KCNMB
60 howed several significant results, including calcium-activated potassium channels (GO:0016286; P=2.30
61                  An intermediate conductance calcium-activated potassium channel, hIK1, was cloned fr
62 ctifier channels (I(KV)) and noninactivating calcium-activated potassium channels (I(BK,steady)), and
63  co-localization of intermediate-conductance calcium-activated potassium channels (IKCa) and IP3 rece
64 e colocalization of intermediate-conductance calcium-activated potassium channels (IKCa) and TRPV4 ch
65 ane blockers of the intermediate conductance calcium-activated potassium channel, IKCa1, have therape
66 f N-type voltage-gated calcium channels with calcium-activated potassium channels in DCN neurons.
67 f calcium-calmodulin-dependent kinase II and calcium-activated potassium channels in mediating these
68                                  The role of calcium-activated potassium channels in the regulation o
69 sistent with native intermediate conductance calcium-activated potassium channels, including the eryt
70                 In addition, the activity of calcium-activated potassium channels increased markedly.
71 nel inhibitor), and iberiotoxin (100 nmol/L, calcium-activated potassium channel inhibitor).
72 rther demonstrate that the activation of the calcium-activated potassium channel is sufficient to ind
73             The opening of large-conductance calcium-activated potassium channels is a common endogen
74  voltage-gated potassium channels (K(v)) and calcium-activated potassium channels (K(Ca)).
75  muscle, we investigated the contribution of calcium-activated potassium channels (KCa) and endotheli
76                            Large-conductance calcium-activated potassium channel (KCa1.1; BK, Slo1, M
77                 The intermediate conductance calcium-activated potassium channel KCa3.1 contributes t
78                                          The calcium-activated potassium channel KCa3.1 controls diff
79                                          The calcium-activated potassium channel KCa3.1 is critically
80                 The intermediate-conductance calcium-activated potassium channel KCa3.1 is expressed
81                            Small conductance calcium-activated potassium channels link elevations of
82                            Large-conductance calcium-activated potassium channels (maxi-K channels) h
83               Large conductance voltage- and calcium-activated potassium channels (MaxiK, BK(Ca)) are
84 ctivating A-current or the large conductance calcium-activated potassium channel-mediated fast spike
85                Because the large conductance calcium-activated potassium channel mSlo is sensitive to
86                   The X-ray structure of the calcium-activated potassium channel MthK, which was crys
87 ne for hSK4, a novel human small conductance calcium-activated potassium channel, or SK channel, has
88 hese findings suggest that the maturation of calcium-activated potassium channels, particularly the a
89  members of the small-intermediate family of calcium-activated potassium channel proteins.
90        Cloned SK channels (small conductance calcium-activated potassium channel) recapitulate these
91                                              Calcium-activated potassium channels regulate AHP and ex
92 ated potassium channel and large-conductance calcium-activated potassium channel, respectively).
93  which selectively block the small and large calcium-activated potassium channels, respectively, resu
94            BK large conductance voltage- and calcium-activated potassium channels respond to elevatio
95                            Small conductance calcium-activated potassium channels show a distinct pha
96 is associated with reduced small-conductance calcium-activated potassium channel (SK) currents and de
97                            Small conductance calcium-activated potassium channels (SK channels) are p
98 rated by the activation of small-conductance calcium-activated potassium channels (SK channels).
99          The activation of small-conductance calcium-activated potassium channels (SK) has a profound
100 s were analyzed in native (small-conductance calcium-activated potassium channel, SK(Ca)) as well as
101 ing peptide (GRP) and the small conductance, calcium-activated potassium channel, SK2.
102 releasing peptide and the small conductance, calcium-activated potassium channel, SK2.
103                            We found that the calcium-activated potassium channel SK3 and the G protei
104            Administration of an activator of calcium-activated potassium channels, SKA-31, partially
105           Apamin-sensitive small conductance calcium-activated potassium channels (SKCa1-3) mediate t
106 triarylmethane-insensitive small conductance calcium-activated potassium channel SKCa3 into IKCa1 ren
107 (HVA) (N- and P/Q-type) calcium channels and calcium-activated potassium channels (SKKCa).
108 eterminant of its anthelmintic effect is the calcium-activated potassium channel SLO-1.
109 to, but distinct from, the small conductance calcium-activated potassium channel subfamily, which is
110 egulating voltage-gated calcium channels and calcium-activated potassium channels that together contr
111 ium was similar to that of small conductance calcium-activated potassium channels, the slope factor d
112 he contribution of the SK (small-conductance calcium-activated potassium) channel to neuronal functio
113 botropic glutamate receptors or postsynaptic calcium-activated potassium channels, two conditions tha
114                                              Calcium-activated potassium channels were analyzed in na
115 tent with the SK class of small-conductance, calcium-activated potassium channels, which contribute t
116 d is abolished by blocking small conductance calcium-activated potassium channels with apamin.
117 ent is reduced by blocking large conductance calcium-activated potassium channels with iberiotoxin, a

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