ライフサイエンスコーパス: calcium-free

1 ibitor-bound furin; and the respective (iii) calcium-free and (iv) calcium-bound forms.

2 a stable, noncovalent homodimer in both its calcium-free and -bound states.

3 iously reported CaM structures and resembles calcium-free apo-calmodulin (apo-CaM), despite the zinc

4 Structural comparison with calcium-free apo-CaM, calcium-loaded CaM, and a cross-li

5 When calcium-free artificial cerebrospinal fluid was used as

6 an alpha7-nAChR subunit-specific antagonist, calcium-free artificial cerebrospinal fluid, or a cockta

7 e Premier Perspective Database, the use of a calcium-free balanced crystalloid for replacement of flu

8 ation with 0.9% NaCl versus Plasma-Lyte A, a calcium-free balanced crystalloid solution, hypothesizin

9 The plateau potential was abolished in calcium-free buffer, as well as by nickel or cadmium.

10 In calcium-free buffer, there was no difference in the ATP-

11 ation dispersion for the nine methionines in calcium-free calmodulin (apo-CaM).

12 studies from our laboratory have shown that calcium-free calmodulin and calcium bound calmodulin pro

13 Our data suggest that the binding sites for calcium-free calmodulin and calcium-bound calmodulin are

14 We now demonstrate that both calcium-free calmodulin and calcium-bound calmodulin bin

15 channel, ryanodine receptor, is activated by calcium-free calmodulin and inhibited by calcium-bound c

16 destroys the ability of the peptide to bind calcium-free calmodulin, but not calcium-bound calmoduli

17 Dialysis anticoagulation with calcium-free citrate-containing dialysate and calcium re

18 icoagulation of the dialysis circuit using a calcium-free citrate-containing dialysate, with calcium

19 Furthermore, calcium-free conditions in the absence of anti-Fas antib

20 e effect of hypoxia on MKP-1 persisted under calcium-free conditions.

21 ium-sensing dyes under calcium-saturated and calcium-free conditions.

22 from kinetic separation of calcium-bound and calcium-free cross-bridge pools.

23 ctroscopic characterization of GCaMP2 in the calcium-free dark state, and in two calcium-bound bright

24 ated stimulation with chemical agonists in a calcium-free environment and this sensitisation is very

25 XE991 did not enhance mEPSCs frequency in a calcium-free external medium.

26 Nominally calcium-free external solution immediately and reversibl

27 gnals could still be triggered by TLC-S in a calcium-free external solution.

28 ted by exposure to thapsigargin/ionomycin in calcium-free external solution.

29 o statistically significant difference among calcium-free external solutions containing different imp

30 ese oscillations were abolished in nominally calcium-free extracellular medium and in 1 muM tetrodoto

31 Axonal injury in a calcium-free extracellular solution resulted in no chang

32 anisms of spontaneous neuronal bursting in a calcium-free extracellular solution.

33 a higher three-photon cross-section for the calcium-free form of Indo-1 than for the calcium-bound f

34 s the solution NMR structure of the inactive calcium-free form of the protein.

35 s the conformational entropy observed in the calcium-free form.

36 solin by releasing the tail latch that locks calcium-free gelsolin in a conformation unable to bind a

37 The tightly packed architecture of calcium-free gelsolin, seen from both SAXS and x-ray cry

38 rapeutic advances include the development of calcium-free intestinal phosphate binders, calcimimetics

39 incubated on ever-decreasing volumes of cold calcium-free KCl closed on the lowest volume (0.2 cm(3))

40 l-like calcium-binding linker, and an EC9-10 calcium-free linker that alters the linear arrangement o

41 Continued incubation of 5AHSmyc cells in calcium-free media induced substantial apoptotic DNA fra

42 en regulated release was prevented by use of calcium-free media, glucose-stimulated IAPP-LI release w

43 TCR-mediated apoptosis can be induced in calcium-free media, indicating that extracellular calciu

44 on of intracellular calcium did not occur in calcium-free media, indicating that the increase in intr

45 n regulated secretion is prevented by use of calcium-free media, suggesting that IAPP secretion occur

46 ed to prevent spontaneous store depletion in calcium-free media.

47 ce of intracellular calcium chelators and in calcium-free media.

48 The reactions also occurred in calcium-free media.

49 ation of calcium to retinal slices bathed in calcium-free media.

50 or if the mild irritant was administered in calcium-free media.

51 Incubation of arteries in calcium-free medium almost abolished the synergistic eff

52 Calcium-free medium blocked not only the increase in AA

53 Culturing cells in calcium-free medium or with the protein kinase Cdelta in

54 oxia or 1-h pretreatment of enterocytes with calcium-free medium resulted in increased internalizatio

55 in vitro in control medium or in a nominally calcium-free medium with high magnesium, glutamate recep

56 calcium channel blockers and/or a nominally calcium-free medium with high magnesium.

57 In SMC pretreated with calcium-free medium, baseline [Ca2+]i fell by about 60 n

58 using a dual emission microfluorometry in a calcium-free medium, that the 17beta-estradiol-stimulate

59 Using dual emission microfluorometry in a calcium-free medium, the 17beta-estradiol-stimulated rel

60 (52 +/- 6 to 297 +/- 26 nM, P: < 0.001) in a calcium-free medium.

61 activation, we used diluted Sendai virus in calcium-free medium.

62 eport a helical reconstruction of TMV in its calcium-free, metastable assembling state at 3.3 A resol

63 Simulations of calcium-free monomeric OMPLA, of the Ca(2+)-bound dimer,

64 Here we report the crystal structure of the calcium-free N-terminal half of adseverin (iA1-A3) and t

65 odimers, suggesting that S100A4 can exist as calcium-free or calcium-bound dimers in vivo.

66 A modified cocktail of CDCs in calcium-free PBS, 100 U/mL of heparin, and 250 microg/mL

67 Although TTX (1 microm) or a calcium-free perfusate both caused reductions in the pow

68 rhythm was further reduced on combination of calcium-free perfusate with octanol (1 mM) and was aboli

69 DA mitochondria were not dependent upon ATP, calcium, free radical species, JNK, or caspase3/PKC path

70 ough the toxin is applied and washed away in calcium-free saline.

71 ter H. capsulatum yeasts were subjected to a calcium-free shock, exogenously added CBP allowed yeasts

72 previous replica-exchange molecular dynamics calcium-free simulations as a control, we reached three

73 We show that dimeric calcium-free sNUCB1 binds to expressed Galpha(i1) and th

74 the present experiments demonstrated that in calcium-free solution (magnesium replacement) zinc can p

75 mitter release has been reported to occur in calcium-free solution and has been attributed either to

76 The response persisted in calcium-free solution and when nickel chloride (4 mM) wa

77 Also, after prestimulation by AngII, a calcium-free solution completely reversed the effects of

78 release as assayed by spontaneous release in calcium-free solution or by the application of hyperosmo

79 noradrenaline and blocked by perfusion with calcium-free solution.

80 synaptic transmission globally using TTX or calcium-free solutions led to a 40-120% increase in the

81 show that hemichannels open upon exposure to calcium-free solutions when they are either heterologous

82 reduced by tetrodotoxin and were blocked in calcium-free solutions.

83 east 10 min after washout of the compound in calcium-free solutions.

84 spontaneous bursting behavior of neurons in calcium-free solutions.

85 -dimensional structures are available in the calcium-free state (e.g., S100B and S100A1).

86 Here we present the calcium-bound and calcium-free structures of the N- and C-terminal domains

87 elected mutations increased the half-life of calcium-free subtilisin at elevated temperature by 15,00

88 hich is independently stable (proR9) and two calcium-free subtilisin mutants.

89 ially randomizing 12 amino acid positions in calcium-free subtilisin.

90 Assuming that calcium-free SYT rings are physiologically relevant, the

91 Treatment for 10 d with calcium-free TPN restored the nocturnal rise in serum PT

92 when introduced into subtilisin BPN' and two calcium-free versions of subtilisin.