ライフサイエンスコーパス: callosum

1 e matter of cerebrum, cerebellum, and corpus callosum).

2 atter tracts (forceps minor, anterior corpus callosum).

3 cts of interest (fornix, cingulum and corpus callosum).

4 ntation within white matter, e.g. the corpus callosum.

5 hemispheric communication through the corpus callosum.

6 llar vermis and lobules V and VI, and corpus callosum.

7 rizone, and OPCs were sorted from the corpus callosum.

8 FA, MD, RD, and AD), primarily in the corpus callosum.

9 EDF was capable of infiltration along corpus callosum.

10 related to myelination and increased corpus callosum.

11 predominantly in the splenium of the corpus callosum.

12 ant axonal bundles within the rostral corpus callosum.

13 ed numbers of myelinated axons in the corpus callosum.

14 ardation (MR) and malformation of the corpus callosum.

15 rhemispheric connecting fibers of the corpus callosum.

16 ental retardation and agenesis of the corpus callosum.

17 appeared progressive, and a prominent corpus callosum.

18 us, as well as in the splenium of the corpus callosum.

19 associated with the evolution of the corpus callosum.

20 myelinating injury to the adult mouse corpus callosum.

21 hology of the sensorimotor cortex and corpus callosum.

22 well as in the frontal aspect of the corpus callosum.

23 matory microglia only (p=0.01) in the corpus callosum.

24 rain MRI of the patient showed a thin corpus callosum.

25 the main neocortical commissure, the corpus callosum.

26 adult-onset spastic ataxia and a thin corpus callosum.

27 glial progenitors in the hypoperfused corpus callosum.

28 , the number of OLs is reduced in the corpus callosum.

29 the fornix and the cingulum bundle or corpus callosum.

30 rs from four different sectors of the corpus callosum.

31 tracts), and the anterior body of the corpus callosum.

32 uction in adulthood in the cortex and corpus callosum.

33 required for development of the human corpus callosum.

34 ways and the splenium and genu of the corpus callosum.

35 he hippocampal dentate gyrus, and the corpus callosum.

36 bilateral prefrontal cortexes via the corpus callosum.

37 ed myelination of the optic nerve and corpus callosum.

38 l progenitors toward the demyelinated corpus callosum.

39 the anterior part of the genu of the corpus callosum.

40 or greater advanced maturation of the corpus callosum.

41 from the ventricular region into the corpus callosum.

42 al connectivity in anterior/posterior corpus callosum.

43 measures of the corticospinal tract and mid-callosum.

44 ic commissural bridges traversing the corpus callosum.

45 ntal white matter and the genu of the corpus callosum.

46 iled to cross the midline to form the corpus callosum.

47 P < .05) in posterior portions of the corpus callosum.

48 ncrease in unmyelinated fibers in the corpus callosum.

49 the superior cerebellar peduncle and corpus callosum.

50 ter organization, particularly in the corpus callosum.

51 tion of the axonal projections in the corpus callosum.

52 eved with FA index measurement of the corpus callosum (51%).

53 included intracranial calcifications, corpus callosum abnormalities, abnormal cortical formation, cer

54 viduals with isolated agenesis of the corpus callosum (ACC) without intellectual disability.

55 in the structural organization of the corpus callosum accounts for these differences.

56 or protein aggregates in axons of the corpus callosum after traumatic brain injury as compared to Sar

57 25 participants with agenesis of the corpus callosum (AgCC) and 21 matched neurotypical individuals.

58 ance in patients with agenesis of the corpus callosum (AgCC) and found reduced laterality (i.e., grea

59 tigated the effect of agenesis of the corpus callosum (AgCC), one of the most common brain malformati

60 characterized by a high prevalence of corpus callosum agenesis (32/80; 40%), and mild to severe cereb

61 s, such as autism spectrum disorders, corpus callosum agenesis, Joubert syndrome, Kallmann syndrome,

62 isorder, intellectual disability, and corpus callosum agenesis.

63 l cases exhibited partial or complete corpus callosum agenesis.

64 provide support for the hypothesis that the callosum aids in functional specialization throughout ne

65 ent in frontal and temporal lobes and corpus callosum (all p values <0.01).

66 However, it is unclear whether corpus callosum alterations are related to the underlying famil

67 Except for the genu of the corpus callosum, an increase in AD values was also found in the

68 From these, sections of the corpus callosum and adjacent parasaggital cortex were examined

69 ion anisotropy of the splenium of the corpus callosum and adjacent parietal white matter (P < .05).

70 in one hemisphere and transected the corpus callosum and anterior commissure in two macaques.

71 Neuroimaging reveals a thin corpus callosum and anterior commissure, and hypoplastic to abs

72 recessive inheritance pattern of thin corpus callosum and axonal Charcot-Marie-Tooth disease in three

73 reditary spastic paraplegia with thin corpus callosum and axonal peripheral neuropathy (SPG7/PGN, SPG

74 d MD in more extended portions of the corpus callosum and beyond (eg, corona radiata and inferior lon

75 leads to insufficient angiogenesis in corpus callosum and catastrophic axon loss.

76 crostructural comparison, we measured corpus callosum and centrum semiovale volumes on MRI.

77 ealed strong microglial activation in corpus callosum and cingulum along with severe astrogliosis and

78 jor clinically relevant white matter (corpus callosum and corticospinal tract) and deep grey matter (

79 model for focal demyelination of the corpus callosum and in multiple sclerosis lesions in humans, in

80 or temporal atrophy and reduced FA in corpus callosum and inferior frontal-occipital fasciculus.

81 te matter injury was prominent in the corpus callosum and internal capsule on day 3 and then partiall

82 We examined the corpus callosum and its subregion volumes and their relationshi

83 ation spread into the splenium of the corpus callosum and motor cortex white matter.

84 of anxiety and hypomyelination in the corpus callosum and optic nerve, providing in vivo evidence tha

85 ced number of myelinated axons in the corpus callosum and optic nerves.

86 amage to the genu and splenium of the corpus callosum and parahippocampal tract bilaterally (P < .05)

87 reditary spastic paraplegia with thin corpus callosum and peripheral axonal neuropathy, and account f

88 attenuated white matter growth of the corpus callosum and pons relative to nondrinkers.

89 and also increased cell death in the corpus callosum and reduced cell division in the mouse subventr

90 n addition, the measurements from the corpus callosum and superior cerebellar peduncles revealed a hi

91 Although both the corpus callosum and superior cerebellar peduncles were of great

92 the posterior midbody/isthmus of the corpus callosum and that fractional anisotropy in this region w

93 r mean diffusivity in the genu of the corpus callosum and the anterior thalamic tracts.

94 uroblasts in the subventricular zone, corpus callosum and the peri-infarct area 7 days after stroke b

95 teins regulating the formation of the corpus callosum and their respective developmental functions.

96 the hippocampus, degeneration of the corpus callosum, and ataxia and seizures.

97 not Nova1-/- mice had agenesis of the corpus callosum, and axonal outgrowth defects specific to ventr

98 sent with spastic paraparesis, a thin corpus callosum, and cognitive impairment.

99 developmental delay, agenesis of the corpus callosum, and enlargement of the third cerebral ventricl

100 rus, cortex of the temporal lobes and corpus callosum, and fractional anisotropy (FA) index measureme

101 lic commissures (anterior commissure, corpus callosum, and hippocampal commissure) along with an enla

102 telencephalic commissures (anterior, corpus callosum, and hippocampal), an enlarged posterior commis

103 FP+ cells were evident in the cortex, corpus callosum, and hippocampus.

104 stage around the striatum, within the corpus callosum, and in posterior white matter tracts.

105 y higher in the medial cortex, in the corpus callosum, and in the thalamus than in the corresponding

106 onnect auditory and motor structures, corpus callosum, and in tracts interconnecting cortical and sub

107 developmental delay, thinning of the corpus callosum, and seizures.

108 lformation (microcephaly, agenesis of corpus callosum, and simplified gyration), and severe encephalo

109 nodes of Ranvier in the optic nerve, corpus callosum, and spinal cord of young adult mice or rats.

110 normally proportioned cerebellum, and corpus callosum anomalies.

111 s included genu, body and splenium of corpus callosum, anterior and superior corona radiata, superior

112 s in the neocortex results in lack of corpus callosum, anterior commissure, and corticospinal tract f

113 M tracts, including internal capsule, corpus callosum, anterior commissure, and fimbria hippocampi, w

114 and MPF in all anatomical structures (corpus callosum, anterior commissure, internal capsule, thalamu

115 new astrocytes from the SVZ into the corpus callosum appears to be balanced by astroglial apoptosis,

116 y, developmental abnormalities of the corpus callosum, arachnoid cyst, abnormalities of the septum pe

117 Brain malformations involving the corpus callosum are common in children with developmental disab

118 We found significantly increased corpus callosum area and thickness in children with autism spec

119 matter tracts, including the anterior corpus callosum as well as bilateral internal and external caps

120 ctional anisotropy alterations in the corpus callosum) as a shared feature of ASD, ADHD, and OCD.

121 ippocampus hypoplasia and agenesis of corpus callosum, as well as neuromuscular and behavioral altera

122 fate-mapping, we demonstrate that new corpus callosum astrocytes are continuously generated from nest

123 These nestin fate-mapped corpus callosum astrocytes are uniformly postmitotic, express g

124 apoptosis, because overall numbers of corpus callosum astrocytes remain constant during normal adulth

125 e particularly robust in the anterior corpus callosum at the 6 and 12 month time points.

126 lable in 7 of the patients, revealing corpus callosum atrophy (7/7 [100%]) and periventricular white

127 rements of brain biparietal diameter, corpus callosum, basal ganglia and thalami, and cerebellum were

128 .90x10(-5)) and posterior part of the corpus callosum (beta=-15.3 muL per risk allele, p=1.23x10(-5))

129 terminalis, genu and splenium of the corpus callosum, bilateral anterior and posterior limbs of inte

130 d white matter areas (splenium of the corpus callosum, bilateral superior-parietal lobe, bilateral an

131 Corpus callosum bisections demonstrated that premotor cortex he

132 reater amounts of white matter in the corpus callosum, but did not control for length of training or

133 Lesions in the corpus callosum (CC) are important radiological clues to the di

134 The corpus callosum (CC) connects the left and right cerebral hemis

135 The corpus callosum (cc) contains nitric oxide (NO)-producing neuro

136 esponses to these unseen stimuli, the corpus callosum (CC) dynamically recruited areas in the visual

137 While microstructural alterations in corpus callosum (CC) have been identified as a consistent featu

138 The corpus callosum (CC) is one of the most commonly reported areas

139 The corpus callosum (CC) is the largest fiber tract in the mammalia

140 authors examined the relationship of corpus callosum (CC) morphology and organization to hand prefer

141 ols the formation of the layer II/III corpus callosum (CC) projections through the developmental tran

142 been often reported to have a smaller corpus callosum (CC) than control subjects.

143 dths of the frontal horn (FH) and the corpus callosum (CC) were not significantly different between t

144 D) and radial diffusivity (RD) in the corpus callosum (CC), superior longitudinal fasciculus (LF), co

145 tter (WM) damage, particularly to the corpus callosum (CC).

146 the two cerebral hemispheres via the corpus callosum (CC).

147 ffected males showing ID, agenesis of corpus callosum, cerebellar hypoplasia, microcephaly and ichthy

148 ion, brain atrophy, dysgenesis of the corpus callosum, cerebellar vermis hypoplasia, and facial dysmo

149 sed volume and abnormal signal), thin corpus callosum, cerebellar vermis hypoplasia, optic nerve hypo

150 s of the brain, such as the amygdala, corpus callosum, cerebellum, and gyrnecephalic index, all indic

151 ant decrease in FA in the genu of the corpus callosum characterized both conditions.

152 from alterations in white matter (in corpus callosum, cingulum bundle, corona radiata, and superior

153 control groups in subdivisions of the corpus callosum, cingulum, and fornix were measured as indicato

154 imaging abnormalities observed in the corpus callosum, cingulum, and temporal lobe likely constitute

155 ber of WM tracts, particularly in the corpus callosum, cingulum, bilateral superior and inferior long

156 ia (2 x 10(5) cells transplanted into corpus callosum) compared with WT microglia toward microinjecte

157 ident in the anterior portions of the corpus callosum connecting left and right frontal lobes.

158 creased white matter integrity in the corpus callosum connecting these regions, suggesting an involve

159 The corpus callosum connects cerebral hemispheres and is the larges

160 acrostructural variability within the corpus callosum, consistent with differential effects on cross-

161 sis that congenital disruption of the corpus callosum constitutes a major risk factor for developing

162 n those without along the body of the corpus callosum (corrected for multiple testing).

163 veral brain structures, including the corpus callosum, cortex, and striatum, and the corpus callosum

164 sALS group, encompassing parts of the corpus callosum, corticospinal tracts and superior longitudinal

165 anterior corona radiata (d=0.40) and corpus callosum (d=0.39), specifically its body (d=0.39) and ge

166 Remarkably, all seven genes showed corpus callosum defects, including thicker (Atg16l1, Coro1c, Dm

167 poral lobe epilepsy, microcephaly and corpus callosum deficiency, and by postnatal Day 21, microcepha

168 ing of an axonal subpopulation of the corpus callosum derived from the anterior cingulate cortex.

169 Our findings define a new stage in corpus callosum development and demonstrate that neocortical pr

170 The role that corpus callosum development has on the hemispheric specializati

171 identifying new proteins integral to corpus callosum development that will provide new insights into

172 sly-identified proteins in aspects of corpus callosum development, and identifies new candidates in u

173 y hypomyelination, microcephaly, thin corpus callosum, developmental delay, intellectual disability,

174 nce that the presence of any residual corpus callosum differentiated those who exhibited current auti

175 However, postnatal lesions of the corpus callosum do not precipitate social behavioral problems i

176 , but not ALSFRS-R, correlated with anterior callosum DTI measures.

177 on in the hippocampal fimbria and the corpus callosum during development, and that this is through th

178 g directional axonal growth, triggers corpus callosum enlargement due to the errant CB1 cannabinoid r

179 or greater delayed maturation of the corpus callosum; every additional 10 days of human milk use wer

180 ructural integrity of the body of the corpus callosum (FA, beta = 0.01 [P = .01]; RD, beta = -0.02 [P

181 s a congenital condition in which the corpus callosum fails to develop; such individuals exhibit loca

182 ty in the forceps minor, the anterior corpus callosum, fascicles in the temporal lobe, and the tracts

183 al atrophy of the dorsal cingulum and corpus callosum fibers, which we interpret as a consequence of

184 developmental mechanisms involved in corpus callosum formation have provided insights into the possi

185 mispheric connectivity by controlling corpus callosum formation remains unclear.

186 damage that involved the body of the corpus callosum, fornix, and main anterior-posterior pathways (

187 In slices of corpus callosum from mice subjected to a demyelination protocol

188 corticospinal tracts, to include the corpus callosum; frontal, sensory, and premotor cortices; thala

189 s suggest novel dual mechanism of the corpus callosum function in spatial attention and have broader

190 First, whereas FA in anterior corpus callosum (genu) correlated with word-matching BPA, FA in

191 The corpus callosum has been implicated in the pathogenesis of schi

192 le (dorsomedial) lying closest to the corpus callosum has the most complete set of commissural connec

193 These findings suggest that the corpus callosum helps to drive language lateralization.

194 al abnormalities and the absence of a corpus callosum; his immune deficit was fully corrected by hema

195 milies in which members affected with corpus callosum hypoplasia (CCH) lacked syndromic features and

196 howed subtle abnormalities, including corpus callosum hypoplasia and ventriculomegaly.

197 ected individuals have cerebellar and corpus callosum hypoplasia, abnormal myelination of the central

198 ovement disorders, behavior problems, corpus callosum hypoplasia, and epilepsy.

199 size of the anterior branches of the corpus callosum, i.e., forceps minor (CCFM), and this neuropath

200 s, respectively; abnormalities of the corpus callosum in 16 of 17 (94%) and 22 of 28 (78%) infections

201 ore and after surgical section of the corpus callosum in 22 patients with medically refractory epilep

202 ttention scores were related to AD in corpus callosum in a cluster sized 716 voxels.

203 ferior frontal gyrus white matter and corpus callosum in addition to the corticospinal tracts and mea

204 significantly lower integrity in the corpus callosum in bipolar subjects.

205 l organization of the splenium of the corpus callosum in low-risk infants, but this association was n

206 ndings suggest a notable role for the corpus callosum in maintaining stable functional communication

207 structure, supporting a role for the corpus callosum in mediating functional asymmetry.

208 uantitative proteomic analysis of the corpus callosum in mice mutant for Dcx.

209 t brain injury in the hippocampus and corpus callosum in rats with vascular dementia.

210 c axonal injury in the cerebellum and corpus callosum in those soldiers with pituitary dysfunction th

211 nect the cerebral hemispheres via the corpus callosum, integrating cortical information and playing k

212 malities of the cerebellum, cingulum, corpus callosum, internal capsule, thalamus, basal forebrain, o

213 myelinated fibers projecting from the corpus callosum into the cortex were lost.

214 alities with sparing of the U fibers, corpus callosum involvement with sparing of the outer blades, a

215 Agenesis of the corpus callosum is a common brain malformation that can occur e

216 Agenesis of the corpus callosum is a congenital condition in which the corpus c

217 The number of oligodendrocytes in the corpus callosum is established in childhood and remains stable

218 ain imaging studies indicate that the corpus callosum is smaller in older children and adults with au

219 The corpus callosum is the largest fibre tract in the brain, connec

220 The corpus callosum is the principal cerebral commissure connecting

221 The development of the corpus callosum is under tight genetic control, as demonstrated

222 Fourth, white matter such as corpus callosum, known to contain negligible numbers of NPY2 re

223 esults in abnormal development of the corpus callosum, lateral ventricles, and hippocampus.

224 l diffusivity in all divisions of the corpus callosum, left fornix, and subgenual cingulum compared w

225 FA values, including the splenium of corpus callosum, left posterior corona radiate/posterior thalam

226 Section of the corpus callosum markedly reduced interhemispheric functional co

227 adults, our findings suggest that the corpus callosum may be larger in infants who go on to develop a

228 gether, our findings suggest that the corpus callosum may have a dual inhibitory and excitatory funct

229 proximal mechanisms by which absence of the callosum might generate autistic symptomatology?

230 py data from the anterior body of the corpus callosum of 13 patients with systemic lupus erythematosu

231 cal uptake was found in the posterior corpus callosum of a TBI subject.

232 of myelin, were also detected in the corpus callosum of adult HdhQ250 mice.

233 nation induced by lysolecithin in the corpus callosum of adult mice.

234 elination phase of the CPZ model, the corpus callosum of Cav1.2(KO) animals presented a significant d

235 llosum, cortex, and striatum, and the corpus callosum of Cav1.2(KO) animals showed an important decre

236 ate nucleus, subthalamic nucleus, and corpus callosum of multiple system atrophy, and in all regions

237 ower microstructural integrity in the corpus callosum of non-demented elderly individuals, and this m

238 differentiation were impaired in the corpus callosum of Olig1-null mice, resulting in hypomyelinatio

239 athy, cognitive impairment and a thin corpus callosum on brain MRI.

240 rounding the anterior crossing of the corpus callosum on E18 as well as the persistence of large numb

241 hy 101LL mice with PrP plaques in the corpus callosum or (ii) brain extracts from mice overexpressing

242 evant to limited brain areas like the corpus callosum, or multiple orientations but without the abili

243 regions, and lower FA in the body of corpus callosum, posterior superior longitudinal fasciculus and

244 ir siblings, mainly restricted to the corpus callosum, posterior thalamic radiations, and left superi

245 we found that increased volume of the corpus callosum predicted good receptive language outcome, but

246 ional connectivity in relation to the corpus callosum presents a case in point.

247 d action potentials in the myelinated corpus callosum projections of Msh2-null mice were smaller than

248 The volumetric assessment of the corpus callosum proved to be a useful tool in discriminating be

249 me subcortical regions, including the corpus callosum, putamen, and cerebellum.

250 and microstructural integrity of the corpus callosum, represented the most promising candidate trait

251 .4% and 203.0% in the hippocampus and corpus callosum, respectively.

252 IFOF), genu (GCC) and splenium of the corpus callosum (SCC), posterior limbs of the internal capsules

253 ctional connectivity before and after corpus callosum section in rhesus monkeys.

254 pathy with or without agenesis of the corpus callosum (SLC12A6).

255 iori regions of interest: splenium of corpus callosum (SPCC) and posterior limb of internal capsule (

256 th word-matching BPA, FA in posterior corpus callosum (splenium-occipital) correlated with face-match

257 radiata, right tapetum, and bilateral corpus callosum, statistically moderates whether sleep spindles

258 and a large cluster that covered the corpus callosum, superior and medial frontal gyrii, as well as

259 n the subcortical gray matter nuclei, corpus callosum, superior temporal gyrus, and pre- and postcent

260 trogliosis significantly increased in corpus callosum (TBI = 6.7 +/- 0.69, Sham = 2.5 +/- 0.38; P = 0

261 hite matter tracts of the genu of the corpus callosum that connect the two hemispheres of the prefron

262 structure (FA) within a region of the corpus callosum that projects to the SMA within each hemisphere

263 nnectivity in individuals in whom the corpus callosum (the major commissure between the hemispheres)

264 ith the volumetric measurement of the corpus callosum - the values were 73% and 71%, respectively, an

265 repair deficiency is agenesis of the corpus callosum, the cause of which has not been established.

266 long the body and the splenium of the corpus callosum, the left cingulum, and the anterior part of th

267 Why do humans born without the corpus callosum, the major interhemispheric commissure, lack th

268 axons than in wild-type mice and, in corpus callosum, the myelin is thinner than in controls.

269 n the genu, body, and splenium of the corpus callosum, the right posterior limb of the internal capsu

270 (RLIC), the body and splenium of the corpus callosum, the superior and posterior corona radiata, and

271 tricular zone-subventricular zone and corpus callosum there is reduced OPC production from RGCs in LX

272 Reduced corpus callosum thickness confirmed trend-level observations fr

273 ver, there was a 25% reduction in the corpus callosum thickness with survival >1 year post-injury.

274 erhemispheric lipoma, agenesis of the corpus callosum, tibial hemimelia, preaxial polydactyly of the

275 The corpus callosum, two projection tracts, and five association tr

276 sotropy (FA) index measurement of the corpus callosum using diffusion tensor imaging.

277 Surprisingly, intercortical tracts-corpus callosum, ventral hippocampal, and anterior commissures-

278 average, but lower right caudate and corpus callosum volume, relative to 22q-del carriers.

279 anisotropy within the splenium of the corpus callosum was found in each NDD group, compared with the

280 nal corticospinal tract and bilateral corpus callosum was increased but sensorimotor CBF was decrease

281 Interestingly, the corpus callosum was markedly thinner, a characteristic we also

282 otropy in the body and isthmus of the corpus callosum was negatively correlated with the composite me

283 Absence of the corpus callosum was noted at screening prenatal head ultrasonog

284 White matter integrity in corpus callosum was reduced in BD1 patients only.

285 otropy in the body and isthmus of the corpus callosum was shown to mediate this association.

286 Luxol fast blue staining of the corpus callosum was significantly greater in the BCCAO rats tre

287 thinner (Kif21b and Wdr89), or absent corpus callosum (Wdr47), revealing a common role for WDR genes

288 bution volume (VT), determined in the corpus callosum, we calculated the binding potential (receptor

289 For each sector of the callosum, we obtained very similar conduction delays reg

290 ipsilateral cortical regions and the corpus callosum were significantly heritable, ranging from tota

291 hizencephaly and abnormalities of the corpus callosum were the most often developmental disorders acc

292 mainly in the uncinate, cingulum and corpus callosum, whereas responders were indistinguishable from

293 ssociations were found in the genu of corpus callosum which accounted for short-term memory binding i

294 parts of cingulum bundle and body of corpus callosum), which showed both increased WMV and decreased

295 ucleus, and cerebellum but not in the corpus callosum, which served as reference region for nonspecif

296 ularly significant with regard to the corpus callosum, whose development undergoes several dynamic st

297 In WT mice, microglia expanded in the corpus callosum with age, whereas aged Trem2(-/-) mice had fewe

298 ter with sparing of the U fibers, the corpus callosum with sparing of the outer blades, the basis pon

299 ontal cortex to 0.46 mL cm(-3) in the corpus callosum, with intermediate VT values in subcortical str

300 The corpus callosum, with its approximately 200 million axons, rema