コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
4 he costameric protein plasma membrane Ca(2+)/calmodulin-dependent ATPase (PMCA), and that its deletio
5 er, they were associated with reduced Ca(2+)/calmodulin-dependent auto-phosphorylation of eEF2 kinase
6 ave characterized two common forms of Ca(2+)/calmodulin-dependent endocytosis, i.e., slow clathrin-de
7 zures in zydeco mutants, suggesting a Ca(2+)/calmodulin-dependent glial signaling pathway underlies g
10 sible for dBest1 activation is likely Ca(2+)/calmodulin dependent kinase II (CaMKII), because specifi
11 ymbioses using three candidate genes: Ca(2+)/Calmodulin-Dependent Kinase (CCaMK), which plays a centr
12 autophosphorylation (activation) of calcium/calmodulin-dependent kinase 2 (CaMKII) and also that inh
13 The Ca(2+) and redox-sensing enzyme Ca(2+) /calmodulin-dependent kinase 2 (CaMKII) is a crucial and
14 mmon signaling pathway including the calcium/calmodulin-dependent kinase Doesn't Make Infection3 (DMI
17 dynamin-related protein 1 (Drp1), by Ca(2+)/calmodulin-dependent kinase II (CaMKII) at a serine 616
20 onses were decreased by inhibition of Ca(2+)/calmodulin-dependent kinase II (CaMKII) in myocytes from
21 n resulted in increased expression of Ca(2+)/calmodulin-dependent kinase II (CaMKII) in the forebrain
23 glucagon-induced calcium signaling, calcium/calmodulin-dependent kinase II (CaMKII) phosphorylates O
24 rum show that synapsin is a target of Ca(2+) calmodulin-dependent kinase II (CaMKII) phosphorylation
25 a putative regulator of calmodulin and Ca2+/calmodulin-dependent kinase II (CaMKII) signaling, exclu
29 NK1 in vitro Inhibiting PKC, JNK, or calcium/calmodulin-dependent kinase II activity prevented the ef
30 y-dependent phosphorylation event on calcium-calmodulin-dependent kinase II alpha (CaMKIIalpha) at se
31 horylation directly through JNK1 and calcium/calmodulin-dependent kinase II and also by inducing expr
32 pulations suggest the involvement of calcium-calmodulin-dependent kinase II and calcium-activated pot
33 ed RyRs have a distinct modulation by Ca(2+)/calmodulin-dependent kinase II and reactive oxygen speci
35 of coupled RyRs was abolished by the Ca(2+)/calmodulin-dependent kinase II blockers autocamtide-2-re
36 a substrate for protein kinase D and Ca(2+)/calmodulin-dependent kinase II in vitro and identified S
37 m signaling, autoinhibition is reinforced by calmodulin-dependent kinase II phosphorylation of serine
38 FIP2 expression also increases alpha-calcium/calmodulin-dependent kinase II protein expression, and t
39 ts Nav1.5 and its regulatory protein calcium/calmodulin-dependent kinase II to the intercalated disc.
40 ude local synthesis of APP and alpha-calcium/calmodulin-dependent kinase II, a kinase that can phosph
41 e for ankyrin-dependent targeting of calcium/calmodulin-dependent kinase II-delta; however, betaIV-sp
45 utophagy via a pathway that included calcium/calmodulin-dependent kinase kinase beta (CaMKKbeta), AMP
46 the cytoplasm, thereby activating a calcium/calmodulin-dependent kinase kinase-beta and 5' adenosine
47 ns of the excitatory neuron-specific Ca(2+) /calmodulin-dependent kinase subunit alpha (CaMKIIalpha)
48 ongation factor 2 kinase (eEF2K) is a Ca(2+)/calmodulin-dependent kinase that regulates translation e
50 ese cells, and in cells where calmodulin and calmodulin-dependent kinase were blocked pharmacological
53 rborization through interactions with Ca(2+)/calmodulin-dependent kinases (CaMKs) in rat hippocampal
55 pendent on the relative activities of Ca(2+)-calmodulin-dependent myosin light chain kinase (MLCK) an
56 endent on the relative activities of Ca(2+) /calmodulin-dependent myosin light chain kinase (MLCK) an
58 ght chain (RLC) is phosphorylated by Ca(2+) /calmodulin-dependent myosin light chain kinase and depho
59 ends on the respective activities of Ca(2+) /calmodulin-dependent myosin light chain kinase and myosi
62 These observations show that calcium- and calmodulin-dependent PDEs (PDE1A and PDE1C) and PDE3A mo
64 otein phosphatase 2A isoform and the calcium-calmodulin-dependent phosphatase calcineurin (CaN) activ
65 n 1 (RCAN1) controls the activity of calcium/calmodulin-dependent phosphatase calcineurin (CaN), whic
66 esults also provide evidence that the Ca(2+)/calmodulin-dependent phosphatase calcineurin plays a rol
67 k1 signaling negatively regulates the Ca(2+)/calmodulin-dependent phosphatase, calcineurin, to enable
68 BAA signaling by calcineurin, a calcium- and calmodulin-dependent phosphatase, enables homeostatic ba
70 h and is dephosphorylated by the Ca(2+)- and calmodulin-dependent phosphoprotein phosphatase calcineu
71 ranslational control mechanism is the Ca(2+)/calmodulin-dependent phosphorylation of eukaryotic elong
73 er brain regions, using Thy1-Cre and calcium/calmodulin dependent protein kinase II alpha-Cre for abl
74 A protein with similarities to the Ca(2+)/ calmodulin dependent protein kinase II_association domai
77 Psi Here, we characterize a role for calcium/calmodulin-dependent protein kinase (CaMK) I in the regu
78 f D-myo-inositol 1,4,5-trisphosphate/Ca(2+) /calmodulin-dependent protein kinase (CaMK) I. gamma-Amin
79 II could also induce the phosphorylation of calmodulin-dependent protein kinase (CaMK) II and cAMP r
80 ostretrieval bilateral inhibition of calcium/calmodulin-dependent protein kinase (CaMK) II in dorsal
81 +)-dependent binding of S100B to the calcium/calmodulin-dependent protein kinase (CaMK)-type domain o
82 2 model of Huntington disease and the Ca(2+)/calmodulin-dependent protein kinase (CaMK)/p25 double-tr
83 wn that the cytoplasmically oriented calcium/calmodulin-dependent protein kinase (CaMK)Ialpha regulat
84 gnaling in the inner ear is the type II Ca2+/calmodulin-dependent protein kinase (CaMK-II), which is
85 ore than 20 years, we have known that Ca(2+)/calmodulin-dependent protein kinase (CaMKII) activation
86 and the ensuing activation of the Ca(2+) and calmodulin-dependent protein kinase (CaMKII) are require
88 lation of RyR2 phosphorylation at the Ca(2+)/calmodulin-dependent protein kinase (CaMKII) site (S2814
90 prevent the arrhythmias induced by a Ca(2+) -calmodulin-dependent protein kinase (CaMKII)-dependent l
92 crease its activity in BTICs, whereas Ca(2+)-calmodulin-dependent protein kinase 2 (CAMK2) inhibited
93 hanistically, CaMKK2 signals through Ca(2+) /calmodulin-dependent protein kinase 4 (CaMKIV) to contro
95 d dysregulation of Na and Ca handling and Ca/calmodulin-dependent protein kinase and are especially p
96 ined increase in cytosolic Ca(2+), activated calmodulin-dependent protein kinase and the calpain-casp
97 cannabinoid type 1 (CB1) receptor and Ca(2+)/calmodulin-dependent protein kinase beta, activates AMP-
99 plex dephosphorylates and inactivates Ca2(+)/calmodulin-dependent protein kinase I (CaMKI), an upstre
102 is critically regulated by the alpha-calcium/calmodulin-dependent protein kinase II (alpha-CaMKII), a
106 sion of glutamate receptor 2 and beta Ca(2+)/calmodulin-dependent protein kinase II (betaCaMKII).
108 e Cl(-) currents can be attributed to Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) activati
109 stically, this effect was mediated by Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) activati
110 tate (NMDA) receptor activation, and Calcium/calmodulin-dependent protein kinase II (CaMKII) activati
111 tamine exposure transiently increases Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) alpha ex
112 y (SOCE) and sequential activation of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and Ca(2
113 reduced activation of PLCgamma-alpha-calcium/calmodulin-dependent protein kinase II (CaMKII) and PI3K
116 phorylated at serine 409 (Ser-409) by Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and prot
117 hibitory peptide (mAIP) selective for Ca2+ / calmodulin-dependent protein kinase II (CaMKII) and U012
118 cription factor DeltaFosB and protein kinase calmodulin-dependent protein kinase II (CaMKII) are co-r
121 ulation; (5) inhibiting either PKA or Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) during b
122 imulation; (5) inhibiting either PKA or Ca2+/calmodulin-dependent protein kinase II (CaMKII) during b
123 lum (SR) Ca(2+) release that involves Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) effects
128 ctly associates with and targets the calcium/calmodulin-dependent protein kinase II (CaMKII) in pancr
129 iew, the functions of multifunctional Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) in VSM p
133 e heart; however, the multifunctional Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is also
136 Here we show that the activity of calcium/calmodulin-dependent protein kinase II (CaMKII) is incre
137 w that Ca(2+)-dependent activation of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is requi
138 Considerable evidence suggests that calcium/calmodulin-dependent protein kinase II (CaMKII) overacti
140 nhanced [(3) H]ryanodine binding and Ca(2+) /calmodulin-dependent protein kinase II (CaMKII) phosphor
144 nel activity reduced EGF receptor (EGFR) and calmodulin-dependent protein kinase II (CAMKII) signalin
145 is downstream of Dalpha7 nAChRs and Calcium/calmodulin-dependent protein kinase II (CaMKII) signalin
146 treated wild-type C57BL/6 mice with calcium/calmodulin-dependent protein kinase II (CaMKII) specific
149 esulted in compromised signaling from Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) to myosi
150 which in turn requires binding of the Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) to the N
151 protein GW182 increases expression of a Ca2+/calmodulin-dependent protein kinase II (CaMKII) translat
154 nificantly increased the activity of calcium/calmodulin-dependent protein kinase II (CaMKII) while re
155 ought to determine how activation of calcium/calmodulin-dependent protein kinase II (CaMKII), a centr
156 CaMK2N2 are endogenous inhibitors of calcium/calmodulin-dependent protein kinase II (CaMKII), a key s
157 reduces FRET between the NMDARcd and calcium/calmodulin-dependent protein kinase II (CaMKII), a proce
158 following: 1) that autophosphorylated Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), an impo
159 triggers the exchange of subunits in Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), an olig
160 embranes, synGAP is phosphorylated by Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), another
161 ono pentanoic acid; the inhibitor of calcium/calmodulin-dependent protein kinase II (CaMKII), autocam
162 vented by pharmacological blockade of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), it was
163 tream effector of WNT/Ca(2+) pathway, Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), led to
164 athway is a kinase cascade involving calcium/calmodulin-dependent protein kinase II (CaMKII), p38alph
165 tors of transient spine expansion, including calmodulin-dependent protein kinase II (CaMKII), RhoA, a
166 scular smooth muscle (VSM) expresses calcium/calmodulin-dependent protein kinase II (CaMKII)-delta an
167 ators of myocardial excitability, and Ca(2+)/calmodulin-dependent protein kinase II (CaMKII)-dependen
168 lism regulates oocyte cell death via calcium/calmodulin-dependent protein kinase II (CaMKII)-mediated
169 Although many studies have focused on Ca(2+)/calmodulin-dependent protein kinase II (CaMKII)-mediated
170 he hypotheses that (1) inhibition of Ca(2+) /calmodulin-dependent protein kinase II (CAMKII)-mediated
171 o the model reveal that inclusion of Ca(2+) /calmodulin-dependent protein kinase II (CAMKII)-mediated
181 tor DeltaFosB and the brain-enriched calcium/calmodulin-dependent protein kinase II (CaMKIIalpha) are
183 ites by protein kinase A (Ser-7) and calcium-calmodulin-dependent protein kinase II (Ser-13) and at m
185 id) receptor activation, calcium and calcium/calmodulin-dependent protein kinase II activity, but not
186 increase in oxidation-dependent calcium and calmodulin-dependent protein kinase II activity, which c
189 Alcohol-sensitive proteins included calcium/calmodulin-dependent protein kinase II alpha (CaMKIIalph
190 ling molecules, calcineurin, Ras, and Ca(2+)/calmodulin-dependent protein kinase II and implicates Ca
191 itive deficits via altered levels of calcium/calmodulin-dependent protein kinase II and N-methyl-D-as
192 ociated with reduced levels of total calcium/calmodulin-dependent protein kinase II and N-methyl-D-as
193 lated to higher activation of nuclear Ca(2+)/calmodulin-dependent protein kinase II and nuclear expor
194 o be independent of their effects on calcium/calmodulin-dependent protein kinase II and PKA, respecti
195 rrhythmic manifestations, related to Ca(2+) /calmodulin-dependent protein kinase II and ryanodine rec
196 es and Thr-287 autophosphorylation of Ca(2+)/calmodulin-dependent protein kinase II beta (CaMKIIbeta)
197 However, double knockdown of pygo and Ca2+/calmodulin-dependent protein kinase II caused additional
199 nt and function, including Titin and calcium/calmodulin-dependent protein kinase II delta (Camk2d).
200 he mechanical effects of the kinases calcium/calmodulin-dependent protein kinase II delta (CaMKIIdelt
202 myocytes from arrhythmia-susceptible calcium calmodulin-dependent protein kinase II delta C (CaMKIIde
204 the LTP kinase dependency from PKA to Ca2(+)/calmodulin-dependent protein kinase II during synapse ma
208 A null mutation of the Drosophila calcium/calmodulin-dependent protein kinase II gene (CaMKII) was
209 ion and subsequent activation of calcium and calmodulin-dependent protein kinase II has a causal role
210 eam signaling protein, PKC-alpha, and Ca(2+)/calmodulin-dependent protein kinase II in endothelial ce
211 2B, also referred to as Pyk2) and of calcium/calmodulin-dependent protein kinase II in wild-type brai
212 rolled firing rate adaptation whereas Ca(2+)/Calmodulin-dependent protein kinase II induced a delayed
213 ia inhibition of ryanodine receptors, Ca(2+)/calmodulin-dependent protein kinase II inhibition, or by
214 Development of organ-specific calcium and calmodulin-dependent protein kinase II inhibitors may re
215 ignal-regulated kinase activators and Ca(2+)/calmodulin-dependent protein kinase II inhibitors showed
217 rom transgenic mice expressing a calcium and calmodulin-dependent protein kinase II inhibitory peptid
220 es including JNK, GSK3alpha/beta, and Ca(2+)/calmodulin-dependent protein kinase II is increased sign
222 on synapsin I, two of which are known Ca(2+)/calmodulin-dependent protein kinase II phosphorylation s
223 ignaling kinases protein kinase C and Ca(2+)/Calmodulin-dependent protein kinase II to AngII-mediated
225 as phosphorylation of substrates for calcium/calmodulin-dependent protein kinase II was unchanged.
226 Pharmacologic inhibition of calcium and calmodulin-dependent protein kinase II with 2.5 microM o
227 stabilization of postsynaptic CaMKII (Ca(2+)/calmodulin-dependent protein kinase II) at inhibitory sy
228 1 pathway, phospho-alphaCaMKII (alpha Ca2(+)/calmodulin-dependent protein kinase II) level in the hip
229 mitochondrial recruitment of CaMKII (Ca(2+)/calmodulin-dependent protein kinase II), which decreases
230 phosphorylation at Ser16 and CaMKII (Ca(2+)/calmodulin-dependent protein kinase II)-dependent phosph
232 ng protein, but not the activation of Ca(2+)/calmodulin-dependent protein kinase II, Akt or mitogen-a
233 mitogen-activated protein kinase, and Ca(2+)/calmodulin-dependent protein kinase II, and activators o
234 ellular protein mediators Homer1b/c, calcium/calmodulin-dependent protein kinase II, and the Alzheime
235 c activation of protein kinase A and calcium/calmodulin-dependent protein kinase II, as well as synap
236 a(2+) must first mobilize actin-bound Ca(2+)/calmodulin-dependent protein kinase II, freeing it for s
237 glutamate-mediated Ca(2+) signaling (Ca(2+)/calmodulin-dependent protein kinase II, PPP3CA, and VISL
239 turn, led to the phosphorylation of calcium/calmodulin-dependent protein kinase II, which promoted b
240 2 expression can be upregulated in a calcium/calmodulin-dependent protein kinase II-dependent manner
241 at have the ryanodine receptor 2 calcium and calmodulin-dependent protein kinase II-dependent phospho
243 used 1 Hz optogenetic stimulation of calcium/calmodulin-dependent protein kinase II-positive principa
247 FTO interacts with three isoforms of calcium/calmodulin-dependent protein kinase II: alpha, beta and
248 AT-C24 DAT) and thereby contained the Ca(2+)-calmodulin-dependent protein kinase IIalpha (CaMKIIalpha
249 a synapse-enriched protein kinase, Ca(2)(+)/calmodulin-dependent protein kinase IIalpha (CaMKIIalpha
250 is study, we investigated the role of Ca(2+)/calmodulin-dependent protein kinase IIalpha (CaMKIIalpha
252 ceptors, p600 associates with the calmodulin.calmodulin-dependent protein kinase IIalpha complex.
253 d cardiomyocyte apoptosis, fibrosis, calcium/calmodulin-dependent protein kinase IIdelta phosphorylat
254 as a direct inhibitor of CaMKIIdelta (Ca(2+)/calmodulin-dependent protein kinase IIdelta) activity, a
255 n vascular smooth muscle (VSM) cells, Ca(2+)/calmodulin-dependent protein kinase IIdelta2 (CaMKIIdelt
256 Here, we present evidence that the calcium/calmodulin-dependent protein kinase IV (CaMK4) is increa
257 ive oxygen species (ROS) production, calcium/calmodulin-dependent protein kinase IV (CaMKIV) activati
258 describe a novel mechanism in which calcium/calmodulin-dependent protein kinase IV (CaMKIV), through
260 KN93, a small-molecule inhibitor of calcium/calmodulin-dependent protein kinase IV, targeted to CD4(
261 is regulated by the classical nuclear Ca(2+)/calmodulin-dependent protein kinase IV-CREB/CREB-binding
262 re, we report that the expression of Ca(2+) /calmodulin-dependent protein kinase kinase 2 (CaMKK2) is
263 ally reduced by the application of a calcium/calmodulin-dependent protein kinase kinase 2 inhibitor (
264 istic target of rapamycin complex 1, calcium/calmodulin-dependent protein kinase kinase 2, and protei
265 e expression of constitutively active Ca(2+)/calmodulin-dependent protein kinase kinase alpha (caCaMK
266 AMPK activation by aa is mediated by Ca(2+)/calmodulin-dependent protein kinase kinase beta (CaMKKbe
267 PK kinases liver kinase B1 (LKB1) and Ca(2+)/calmodulin-dependent protein kinase kinase beta (CaMKKbe
270 events were blocked by inhibition of Ca(2+)/calmodulin-dependent protein kinase kinase beta, an upst
272 altered calcium signaling, transduced by the calmodulin-dependent protein kinase kinase cascade, medi
274 alcineurin, Akt/protein kinase B, and Ca(2+)/calmodulin-dependent protein kinase signaling pathways i
275 O)-1, glutathione reductase (GSR)-1, calcium/calmodulin-dependent protein kinase type (CAMK)-IV, cAMP
277 ger, phospholamban, calcineurin, and calcium/calmodulin-dependent protein kinase type II (CaMKII) wer
278 -bisphosphate binding, protein kinase C- and calmodulin-dependent protein kinase type II phosphorylat
279 to remodeling pathways (e.g., Akt and Ca(2+)/calmodulin-dependent protein kinase type II) and develop
282 ngation factor 2 kinase (eEF2K), an atypical calmodulin-dependent protein kinase, phosphorylates and
285 y AMPA-type glutamate receptors and required calmodulin-dependent protein kinase-mediated phosphoryla
287 r new protein synthesis and required calcium/calmodulin-dependent protein kinases and the nuclear cal
289 he mechanistic role of the family of calcium/calmodulin-dependent protein kinases in mediating these
292 pines, (3) required activation of the Ca(2+)/calmodulin-dependent protein-kinase II, (4) was restrict
293 or somatostatin-positive interneurons and of calmodulin-dependent, protein kinase-positive, principal
295 ongation factor 2 kinase (eEF2K), a Ca(2)(+)/calmodulin dependent serine/threonine kinase that phosph
296 h GluN2B and with the adaptor protein Ca(2+)/calmodulin-dependent serine protein kinase and kinesin K
297 G1 (discs-large homolog 1) and CASK (calcium/calmodulin-dependent serine protein kinase) interact at
298 ; membrane protein, palmitoylated 3; calcium/calmodulin-dependent serine protein kinase; membrane-ass
299 ere we provide evidence that the calcium and calmodulin-dependent serine/threonine protein kinase typ
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。