ライフサイエンスコーパス: calmodulin-dependent protein kinase II

1 s the autophosphorylation of CaMKII (calcium-calmodulin-dependent protein kinase II).

2 nt downstream enzymes calcineurin and Ca(2+)-calmodulin-dependent protein kinase II.

3 acellular calcium and activation of calcium, calmodulin-dependent protein kinase II.

4 ](i) was insensitive to inhibition of Ca(2+)-calmodulin-dependent protein kinase II.

5 a(2+)-mobilizing phospholipase C, and Ca(2+)/calmodulin-dependent protein kinase II.

6 ia S135 phosphorylation catalyzed by calcium/calmodulin-dependent protein kinase II.

7 pressed by inhibiting the activity of Ca(2+)/calmodulin-dependent protein kinase II.

8 sarcoplasmic reticulum and activated Ca(2+)/calmodulin-dependent protein kinase II.

9 dent on calcium influx and linked to calcium/calmodulin-dependent protein kinase II.

10 pendent on changes in [Ca(2+)](i) and Ca(2+)/calmodulin-dependent protein kinases II.

11 pines, (3) required activation of the Ca(2+)/calmodulin-dependent protein-kinase II, (4) was restrict

12 Inhibitors of calcium/calmodulin-dependent protein kinase II, a mitochondrial

13 Moreover, Carabin reduced Ca(2+)/calmodulin-dependent protein kinase II activation and pr

14 24.3.1 and acute chorea sera induced calcium/calmodulin-dependent protein kinase II activity in human

15 Inhibition of calcium/calmodulin-dependent protein kinase II activity, but not

16 id) receptor activation, calcium and calcium/calmodulin-dependent protein kinase II activity, but not

17 increase in oxidation-dependent calcium and calmodulin-dependent protein kinase II activity, which c

18 -d-aspartate receptor activation and calcium/calmodulin-dependent protein kinase II activity.

19 ng protein, but not the activation of Ca(2+)/calmodulin-dependent protein kinase II, Akt or mitogen-a

20 er brain regions, using Thy1-Cre and calcium/calmodulin dependent protein kinase II alpha-Cre for abl

21 bunit, Rpt6, by the plasticity kinase Ca(2+)/calmodulin-dependent protein kinase II alpha (CaMKIIalph

22 entanyl decreased the activity of the Ca(2+)/calmodulin-dependent protein kinase II alpha (CaMKIIalph

23 Alcohol-sensitive proteins included calcium/calmodulin-dependent protein kinase II alpha (CaMKIIalph

24 Ca(2+)/calmodulin-dependent protein kinase II alpha (CaMKIIalph

25 increasing dendritic translation of calcium calmodulin-dependent protein kinase II alpha and the alp

26 ice) by using the forebrain-specific calcium/calmodulin-dependent protein kinase II alpha promoter to

27 s with mRNA, e.g. encoding GluR1 and calcium calmodulin-dependent protein kinase II alpha.

28 , the amount of PSD-associated alpha-calcium calmodulin-dependent protein kinase II (alpha- CaMKII) l

29 is critically regulated by the alpha-calcium/calmodulin-dependent protein kinase II (alpha-CaMKII), a

30 Calcium/calmodulin-dependent protein kinase II-alpha (CaMKII-alp

31 FTO interacts with three isoforms of calcium/calmodulin-dependent protein kinase II: alpha, beta and

32 , a model of memory formation, requires Ca2+.calmodulin-dependent protein kinase II (alphaCaMKII) act

33 ing rely on the alpha-isoform of the calcium/calmodulin-dependent protein kinase II (alphaCaMKII) aut

34 The alpha isoform of the calcium/calmodulin-dependent protein kinase II (alphaCaMKII) has

35 The alpha-Ca(2+)/calmodulin-dependent protein kinase II (alphaCaMKII) is

36 Since alpha calmodulin-dependent protein kinase II (alphaCaMKII), a

37 tant for learning and memory, including Ca2+/calmodulin-dependent protein kinase II (alphaCaMKII), PK

38 oth Ca2+ entry-dependent involvement of Ca2+/calmodulin-dependent protein kinase II and Ca2+-independ

39 ling molecules, calcineurin, Ras, and Ca(2+)/calmodulin-dependent protein kinase II and implicates Ca

40 + with BAPTA, and by inhibition of both Ca2+-calmodulin-dependent protein kinase II and mitogen-activ

41 itive deficits via altered levels of calcium/calmodulin-dependent protein kinase II and N-methyl-D-as

42 ociated with reduced levels of total calcium/calmodulin-dependent protein kinase II and N-methyl-D-as

43 lated to higher activation of nuclear Ca(2+)/calmodulin-dependent protein kinase II and nuclear expor

44 ta4)alpha5 to alpha7 nAChR transition-Ca(2+)/calmodulin-dependent protein kinase II and p38 MAPK, and

45 ta4)alpha5 to alpha7 nAChR transition-Ca(2+)/calmodulin-dependent protein kinase II and p38 MAPK, and

46 o be independent of their effects on calcium/calmodulin-dependent protein kinase II and PKA, respecti

47 e to both Ca2+-dependent recruitment of Ca2+/calmodulin-dependent protein kinase II and protein kinas

48 Calmodulin-dependent protein kinase II and protein kinas

49 rrhythmic manifestations, related to Ca(2+) /calmodulin-dependent protein kinase II and ryanodine rec

50 it that mediates dephosphorylation of Ca(2+)/calmodulin-dependent protein kinase II and tyrosine hydr

51 nd is accompanied by altered CaMKII (calcium/calmodulin-dependent protein kinase II) and flotillin-1

52 4) is the failure to activate CaMKII (Ca(2+)/calmodulin-dependent protein kinase II) and retinal dege

53 mitogen-activated protein kinase, and Ca(2+)/calmodulin-dependent protein kinase II, and activators o

54 in cAMP, light-induced activation of Ca(2+) /calmodulin-dependent protein kinase II, and dopamine-ind

55 ellular protein mediators Homer1b/c, calcium/calmodulin-dependent protein kinase II, and the Alzheime

56 se element-binding protein, Thr286 of Ca(2+)/calmodulin-dependent protein kinase II, and Thr202/Tyr20

57 manner requiring cytosolic calcium, calcium/calmodulin-dependent protein kinase II, and toll-like re

58 c activation of protein kinase A and calcium/calmodulin-dependent protein kinase II, as well as synap

59 stabilization of postsynaptic CaMKII (Ca(2+)/calmodulin-dependent protein kinase II) at inhibitory sy

60 es and Thr-287 autophosphorylation of Ca(2+)/calmodulin-dependent protein kinase II beta (CaMKIIbeta)

61 sion of glutamate receptor 2 and beta Ca(2+)/calmodulin-dependent protein kinase II (betaCaMKII).

62 disrupted by inhibitors of calcineurin or Ca-calmodulin-dependent protein kinase II, but not PKC.

63 distinct downstream targets, including Ca2+/calmodulin-dependent protein kinase II, calcineurin, pro

64 a marker for cholinergic terminals; calcium/calmodulin-dependent protein kinase II (CaMK) was used a

65 Calcium/calmodulin-dependent protein kinase II (CAMK2) is one of

66 sphorylation sites in Cx36 and evidence that calmodulin dependent protein kinase II (CaMKII) may pote

67 This facilitated the Ca2+/calmodulin dependent protein kinase II (CaMKII)-dependen

68 glycerol lipase-alpha (DGLalpha) and calcium/calmodulin dependent protein kinase II (CaMKII).

69 e Cl(-) currents can be attributed to Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) activati

70 stically, this effect was mediated by Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) activati

71 calmodulin, but was not dependent on calcium/calmodulin-dependent protein kinase II (CaMKII) activati

72 Ca2+-calmodulin-dependent protein kinase II (CaMKII) activati

73 tate (NMDA) receptor activation, and Calcium/calmodulin-dependent protein kinase II (CaMKII) activati

74 Whereas increased Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) activity

75 ctivity in mutant cultures was lower, Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) activity

76 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) activity

77 ether this depends on protein kinase A or Ca/calmodulin-dependent protein kinase II (CaMKII) activity

78 tamine exposure transiently increases Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) alpha ex

79 y (SOCE) and sequential activation of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and Ca(2

80 Dys-/- had reduced expression of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and CaMK

81 haKAP is an anchoring protein for the Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and is e

82 reduced activation of PLCgamma-alpha-calcium/calmodulin-dependent protein kinase II (CaMKII) and PI3K

83 We show that phosphorylation by Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and Polo

84 In hypertrophy, Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and prot

85 kinase signaling pathways, including Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and prot

86 This site is a substrate for both Ca(2+)-calmodulin-dependent protein kinase II (CaMKII) and prot

87 phorylated at serine 409 (Ser-409) by Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and prot

88 The Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and the

89 hibitory peptide (mAIP) selective for Ca2+ / calmodulin-dependent protein kinase II (CaMKII) and U012

90 cription factor DeltaFosB and protein kinase calmodulin-dependent protein kinase II (CaMKII) are co-r

91 response-element-binding (CREB) and calcium-calmodulin-dependent protein kinase II (CAMKII) are crit

92 Ca(v)1 (L-type) channels and calmodulin-dependent protein kinase II (CaMKII) are key

93 Both alpha and beta isoforms of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) are pres

94 d the contribution of multifunctional Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) as a med

95 The Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) assemble

96 In this study, we show that Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) binds di

97 Calcium/calmodulin-dependent protein kinase II (CaMKII) binds to

98 Ca(2)(+)/calmodulin-dependent protein kinase II (CaMKII) blockers

99 e classic regulators of this current, Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) but not

100 urrents was diminished by inhibition of Ca2+/calmodulin-dependent protein kinase II (CaMKII) but was

101 During cellular Ca(2+) overload, the calmodulin-dependent protein kinase II (CaMKII) causes a

102 horylation at serine 73, a conserved calcium/calmodulin-dependent protein kinase II (CaMKII) consensu

103 ulation; (5) inhibiting either PKA or Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) during b

104 imulation; (5) inhibiting either PKA or Ca2+/calmodulin-dependent protein kinase II (CaMKII) during b

105 lum (SR) Ca(2+) release that involves Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) effects

106 activation of the multifunctional Ca(2+) and calmodulin-dependent protein kinase II (CaMKII) favors m

107 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) forms a

108 Calcium-calmodulin-dependent protein kinase II (CaMKII) has been

109 Calcium/calmodulin-dependent protein kinase II (CaMKII) has been

110 Calcium/calmodulin-dependent protein kinase II (CaMKII) has been

111 Calcium/calmodulin-dependent protein kinase II (CaMKII) has impo

112 The Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) has rece

113 Ca(2+)-calmodulin-dependent protein kinase II (CaMKII) hyperact

114 o signal neuronal cells and activate calcium calmodulin-dependent protein kinase II (CaMKII) in neuro

115 ctly associates with and targets the calcium/calmodulin-dependent protein kinase II (CaMKII) in pancr

116 nt studies examined the role of Ca-super(2+)/calmodulin-dependent protein kinase II (CaMKII) in this

117 iew, the functions of multifunctional Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) in VSM p

118 The extent of calcium/calmodulin-dependent protein kinase II (CaMKII) inactiva

119 Myocardial Ca2+/calmodulin-dependent protein kinase II (CaMKII) inhibiti

120 Further downstream, the Ca2+/calmodulin-dependent protein kinase II (CamKII) inhibito

121 The Ca2+/calmodulin-dependent protein kinase II (CaMKII) inhibito

122 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) inhibits

123 Calcium/calmodulin-dependent protein kinase II (CaMKII) interpre

124 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is a cen

125 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is a cri

126 The multifunctional Ca(2+) and calmodulin-dependent protein kinase II (CaMKII) is a dow

127 Ca2+/calmodulin-dependent protein kinase II (CaMKII) is a dow

128 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is a key

129 Ca(2+) /calmodulin-dependent protein kinase II (CaMKII) is a maj

130 Calmodulin (CaM) trapping by Ca2+/calmodulin-dependent protein kinase II (CaMKII) is a phe

131 Calcium/calmodulin-dependent protein kinase II (CaMKII) is a piv

132 Ca2(+)/calmodulin-dependent protein kinase II (CaMKII) is a ser

133 Ca(2+)-calmodulin-dependent protein kinase II (CaMKII) is a ser

134 Calcium/calmodulin-dependent protein kinase II (CaMKII) is a syn

135 The multifunctional Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is activ

136 It has been shown recently that Ca2+/calmodulin-dependent protein kinase II (CaMKII) is activ

137 e heart; however, the multifunctional Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is also

138 Ca(2+)-Calmodulin-dependent protein kinase II (CaMKII) is an ab

139 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is an en

140 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is an im

141 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is an im

142 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is centr

143 Here, we report that calcium/calmodulin-dependent protein kinase II (CaMKII) is const

144 Although Ca2+/calmodulin-dependent protein kinase II (CaMKII) is enric

145 Calcium/calmodulin-dependent protein kinase II (CaMKII) is essen

146 Here we show that the activity of calcium/calmodulin-dependent protein kinase II (CaMKII) is incre

147 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is known

148 w that Ca(2+)-dependent activation of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is requi

149 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is up-re

150 ed increased prefrontal cortex (PFC) calcium/calmodulin-dependent protein kinase II (CaMKII) levels,

151 Considerable evidence suggests that calcium/calmodulin-dependent protein kinase II (CaMKII) overacti

152 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) oxidatio

153 nhanced [(3) H]ryanodine binding and Ca(2+) /calmodulin-dependent protein kinase II (CaMKII) phosphor

154 ated Ca(2+) channels (VGCCs) leads to Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) phosphor

155 pid atrial pacing resulted in increased Ca2+/calmodulin-dependent protein kinase II (CaMKII) phosphor

156 Ca2+/calmodulin-dependent protein kinase II (CaMKII) phosphor

157 Calcium/calmodulin-dependent protein kinase II (CaMKII) phosphor

158 Calcium/calmodulin-dependent protein kinase II (CaMKII) plays a

159 Calcium/calmodulin-dependent protein kinase II (CaMKII) plays a

160 Calcium/calmodulin-dependent protein kinase II (CaMKII) plays a

161 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) plays an

162 The multifunctional Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) promotes

163 Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) promotes

164 nel activity reduced EGF receptor (EGFR) and calmodulin-dependent protein kinase II (CAMKII) signalin

165 is downstream of Dalpha7 nAChRs and Calcium/calmodulin-dependent protein kinase II (CaMKII) signalin

166 Ca2+/calmodulin-dependent protein kinase II (CaMKII) signalin

167 Ca(2+)/Calmodulin-dependent protein kinase II (CaMKII) signalin

168 treated wild-type C57BL/6 mice with calcium/calmodulin-dependent protein kinase II (CaMKII) specific

169 Here, we show that activated Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) strongly

170 Localization of the Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) to dendr

171 lored the role of the sole Drosophila Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) to media

172 esulted in compromised signaling from Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) to myosi

173 which in turn requires binding of the Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) to the N

174 protein GW182 increases expression of a Ca2+/calmodulin-dependent protein kinase II (CaMKII) translat

175 In contrast, when the calcium/calmodulin-dependent protein kinase II (CaMKII) was bloc

176 In contrast, when the calcium-calmodulin-dependent protein kinase II (CaMKII) was bloc

177 nificantly increased the activity of calcium/calmodulin-dependent protein kinase II (CaMKII) while re

178 teraction disrupts the association of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) with NR2

179 ought to determine how activation of calcium/calmodulin-dependent protein kinase II (CaMKII), a centr

180 CaMK2N2 are endogenous inhibitors of calcium/calmodulin-dependent protein kinase II (CaMKII), a key s

181 Calcium/calmodulin-dependent protein kinase II (CaMKII), a major

182 reduces FRET between the NMDARcd and calcium/calmodulin-dependent protein kinase II (CaMKII), a proce

183 examined the potential involvement of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), a signa

184 following: 1) that autophosphorylated Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), an impo

185 triggers the exchange of subunits in Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), an olig

186 previously that inositol trisphosphate, Ca2+/calmodulin-dependent protein kinase II (CaMKII), and pro

187 embranes, synGAP is phosphorylated by Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), another

188 ono pentanoic acid; the inhibitor of calcium/calmodulin-dependent protein kinase II (CaMKII), autocam

189 Emi2 degradation in MI depends not on Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), but on

190 One such kinase, Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), can tar

191 ed its effects on phosphorylation of calcium/calmodulin-dependent protein kinase II (CaMKII), extrace

192 Inhibition of the calcium/calmodulin-dependent protein kinase II (CaMKII), inhibit

193 vented by pharmacological blockade of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), it was

194 tream effector of WNT/Ca(2+) pathway, Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), led to

195 ylation was also attenuated by inhibitors of calmodulin-dependent protein kinase II (CaMKII), p38 MAP

196 athway is a kinase cascade involving calcium/calmodulin-dependent protein kinase II (CaMKII), p38alph

197 tors of transient spine expansion, including calmodulin-dependent protein kinase II (CaMKII), RhoA, a

198 d Ang II activate the multifunctional Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), the inh

199 ta(B) and delta(C) splice variants of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII), which d

200 scular smooth muscle (VSM) expresses calcium/calmodulin-dependent protein kinase II (CaMKII)-delta an

201 ators of myocardial excitability, and Ca(2+)/calmodulin-dependent protein kinase II (CaMKII)-dependen

202 lism regulates oocyte cell death via calcium/calmodulin-dependent protein kinase II (CaMKII)-mediated

203 Although many studies have focused on Ca(2+)/calmodulin-dependent protein kinase II (CaMKII)-mediated

204 he hypotheses that (1) inhibition of Ca(2+) /calmodulin-dependent protein kinase II (CAMKII)-mediated

205 o the model reveal that inclusion of Ca(2+) /calmodulin-dependent protein kinase II (CAMKII)-mediated

206 cium flux triggered the activation of Ca(2+)-calmodulin-dependent protein kinase II (CaMKII).

207 y NMDAR downstream signaling protein calcium/calmodulin-dependent protein kinase II (CaMKII).

208 equired cell-autonomous activation of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

209 age-gated Na(+) channel (Na(v)1.5) by Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

210 smic reticulum (ER) and activation of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

211 regulated through phosphorylation via Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

212 he decline in NMDAR responses through Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

213 hannels and requires the activity of calcium/calmodulin-dependent protein kinase II (CaMKII).

214 subsequent activation of presynaptic calcium/calmodulin-dependent protein kinase II (CaMKII).

215 the postsynaptic density, including calcium/calmodulin-dependent protein kinase II (CaMKII).

216 nase C (PKC), protein kinase A (PKA) or Ca2+/calmodulin-dependent protein kinase II (CaMKII).

217 c Ca2+ and concomitant activation of calcium/calmodulin-dependent protein kinase II (CaMKII).

218 NMDA receptor-dependent activation of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

219 est was mediated by the activation of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

220 ryanodine receptors or RyR2s) and the Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

221 lux (nSOC) and continuous activity of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

222 the caspase-2 prodomain by activated Ca(2+)/calmodulin-dependent protein kinase II (CaMKII).

223 tion of the calcium-sensitive kinase calcium-calmodulin-dependent protein kinase II (CaMKII).

224 Here, we show that endogenous calcium/calmodulin-dependent protein kinase II (CamkII, also kno

225 Calmodulin expression (60%), Ca(2+)/calmodulin-dependent protein kinase-II (CaMKII) autophos

226 Ca(2+)/calmodulin-dependent protein kinase-II (CaMKII) phosphor

227 or EGTA, or a specific inhibitor for calcium/calmodulin-dependent protein kinase-II (CaMKII).

228 tor DeltaFosB and the brain-enriched calcium/calmodulin-dependent protein kinase II (CaMKIIalpha) are

229 However, double knockdown of pygo and Ca2+/calmodulin-dependent protein kinase II caused additional

230 , inhibitors, and a dominant negative Ca(2+)/calmodulin-dependent protein kinase II construct block A

231 after birth using CaMKII-Cre (alpha-calcium/calmodulin-dependent protein kinase II-Cre) lines to rec

232 ice using the CaMKIIalpha-Cre (alpha-calcium/calmodulin-dependent protein kinase II-Cre) system to KD

233 The calcium/calmodulin-dependent protein kinase II delta (CAMK2D), w

234 nt and function, including Titin and calcium/calmodulin-dependent protein kinase II delta (Camk2d).

235 he mechanical effects of the kinases calcium/calmodulin-dependent protein kinase II delta (CaMKIIdelt

236 Ca(2+)/calmodulin-dependent protein kinase II delta (CaMKIIdelt

237 myocytes from arrhythmia-susceptible calcium calmodulin-dependent protein kinase II delta C (CaMKIIde

238 The multifunctional Ca(2+)/calmodulin-dependent protein kinase II delta(C) (CaMKIId

239 The multifunctional Ca(2+)/calmodulin-dependent protein kinase II delta-isoform (Ca

240 phosphorylation at Ser16 and CaMKII (Ca(2+)/calmodulin-dependent protein kinase II)-dependent phosph

241 2 expression can be upregulated in a calcium/calmodulin-dependent protein kinase II-dependent manner

242 at have the ryanodine receptor 2 calcium and calmodulin-dependent protein kinase II-dependent phospho

243 ng activation of NHE3 by ANG II via a Ca(2+)/calmodulin-dependent protein kinases II-dependent pathwa

244 the LTP kinase dependency from PKA to Ca2(+)/calmodulin-dependent protein kinase II during synapse ma

245 a(2+) must first mobilize actin-bound Ca(2+)/calmodulin-dependent protein kinase II, freeing it for s

246 ons that interfere with either calmodulin or calmodulin-dependent protein kinase II function.

247 Here, we show that calcium/calmodulin-dependent protein kinase II gamma (CAMK2gamma

248 Here, Ca(2+)/calmodulin-dependent protein kinase II gamma (CAMKIIgamm

249 Calcium/calmodulin-dependent protein kinase II gamma knockout mi

250 NMDA receptor signaling, we identify calcium/calmodulin-dependent protein kinase II gamma subunit (Ca

251 We identified a role for calcium/calmodulin-dependent protein kinases II gamma isoform (C

252 A null mutation of the Drosophila calcium/calmodulin-dependent protein kinase II gene (CaMKII) was

253 of the promoter for alpha subunit of Ca(2+)/calmodulin-dependent protein kinase II, greatly increase

254 hereas inhibiting protein kinase A or Ca(2+)/calmodulin-dependent protein kinase II had no effect.

255 ion and subsequent activation of calcium and calmodulin-dependent protein kinase II has a causal role

256 eam signaling protein, PKC-alpha, and Ca(2+)/calmodulin-dependent protein kinase II in endothelial ce

257 2B, also referred to as Pyk2) and of calcium/calmodulin-dependent protein kinase II in wild-type brai

258 rolled firing rate adaptation whereas Ca(2+)/Calmodulin-dependent protein kinase II induced a delayed

259 ia inhibition of ryanodine receptors, Ca(2+)/calmodulin-dependent protein kinase II inhibition, or by

260 90% (P<0.05) by STAT1 deficiency or calcium/calmodulin-dependent protein kinase II inhibition.

261 sphatase inhibitor okadaic acid and the Ca2+/calmodulin-dependent protein kinase II inhibitor KN62 bl

262 This was abolished by the Ca(2+)/calmodulin-dependent protein kinase II inhibitor KN93, s

263 Development of organ-specific calcium and calmodulin-dependent protein kinase II inhibitors may re

264 ignal-regulated kinase activators and Ca(2+)/calmodulin-dependent protein kinase II inhibitors showed

265 Ca(2+)/calmodulin-dependent protein kinase II inhibitors suppre

266 rom transgenic mice expressing a calcium and calmodulin-dependent protein kinase II inhibitory peptid

267 Calcium/calmodulin-dependent protein kinase II is a prototypical

268 Ca(2+)/calmodulin-dependent protein kinase II is a synapse-enri

269 hat increased RyR2 phosphorylation by Ca(2+)/calmodulin-dependent protein kinase II is both necessary

270 es including JNK, GSK3alpha/beta, and Ca(2+)/calmodulin-dependent protein kinase II is increased sign

271 1 pathway, phospho-alphaCaMKII (alpha Ca2(+)/calmodulin-dependent protein kinase II) level in the hip

272 se (phosphoinositide 3-kinase), CaMKII (Ca2+/calmodulin-dependent protein kinase II), MAPK (mitogen-a

273 midal neuron dendrites by activating calcium/calmodulin-dependent protein kinase II-mediated downstre

274 Genetic inhibition of Ca(2+)/calmodulin-dependent protein kinase II-mediated RyR2-S28

275 R2 by cAMP-dependent protein kinase A and by calmodulin-dependent protein kinase II modulates channel

276 re, we show that RNAs encoding alpha calcium calmodulin-dependent protein kinase II, neurogranin, and

277 ng postsynaptic density-95 and alpha-calcium/calmodulin-dependent protein kinase II, normally elicite

278 almodulin-dependent processes involving Ca2+/calmodulin-dependent protein kinase II or the protein ph

279 Oxidized Ca(2+)/calmodulin-dependent protein kinase II (ox-CaMKII) was s

280 p38 and Ca(2+)/calmodulin-dependent protein kinase II pathways were fou

281 our results suggest that Ca(2+)/calmodulin-dependent protein kinase II phosphorylation o

282 on synapsin I, two of which are known Ca(2+)/calmodulin-dependent protein kinase II phosphorylation s

283 ivity of the protein kinases CaMKII (calcium/calmodulin-dependent protein kinase II), PKA, and PKC, w

284 used 1 Hz optogenetic stimulation of calcium/calmodulin-dependent protein kinase II-positive principa

285 glutamate-mediated Ca(2+) signaling (Ca(2+)/calmodulin-dependent protein kinase II, PPP3CA, and VISL

286 ites by protein kinase A (Ser-7) and calcium-calmodulin-dependent protein kinase II (Ser-13) and at m

287 Finally, genetic ablation of the Ca(2+)/calmodulin-dependent protein kinase II site on RyR2 (S28

288 mice in which the S2814 Ca(2+)/calmodulin-dependent protein kinase II site on RyR2 is c

289 icity, but was absent in adult alpha-calcium/calmodulin-dependent protein kinase II;T286A (alphaCaMKI

290 Calcium and calmodulin-dependent protein kinase II, through phosphor

291 ignaling kinases protein kinase C and Ca(2+)/Calmodulin-dependent protein kinase II to AngII-mediated

292 In addition, phosphorylation of calcium/calmodulin-dependent protein kinase II was increased in

293 as phosphorylation of substrates for calcium/calmodulin-dependent protein kinase II was unchanged.

294 th cAMP-dependent protein kinase and calcium/calmodulin-dependent protein kinase II, whereas Ca(V)1.1

295 were labeled by using antibodies to calcium/calmodulin-dependent protein kinase II, whereas differen

296 mitochondrial recruitment of CaMKII (Ca(2+)/calmodulin-dependent protein kinase II), which decreases

297 In addition, the presence of Ca2+/calmodulin-dependent protein kinase II, which can bind u

298 turn, led to the phosphorylation of calcium/calmodulin-dependent protein kinase II, which promoted b

299 The inhibition of Ca(2+)/calmodulin-dependent protein kinase II with 1-[N,O-bis(5

300 Pharmacologic inhibition of calcium and calmodulin-dependent protein kinase II with 2.5 microM o