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1 ioning on the liking for sweet beverages and caloric adjustment after their consumption in children.
2 troversial; the impact of these beverages on caloric adjustment needs clarification.
3 found between the clock hour of food intake, caloric amount, meal macronutrient composition, activity
4                                       At any caloric amount, vegetarians should optimize intakes of v
5 ent associations of beverages sweetened with caloric and low-calorie sweeteners with dietary quality
6  ad libitum buffet was used to measure total caloric and macronutrient intakes.
7  a predilection for conserving energy during caloric and protein deprivation and a profligate respons
8                              Very short-term caloric and saturated fat dietary restrictions do not le
9 aviors that function to maintain homeostatic caloric balance.
10        The DB group showed decreases in most caloric beverages and specifically reduced more desserts
11                     Decreasing the amount of caloric beverages consumed and simultaneously increasing
12 stly associated with ED, whereas the partial caloric compensation may consistently be guided by ED.
13  of the beverage flavor (eg, after reversal; caloric compensation score: 29%).
14 esidence into a validated microsimulation of caloric consumption, glycemic load, overweight/obesity p
15 reased IHTG and abdominal fat independent of caloric content and body weight gain, whereas increasing
16 on of signals related to sweet taste and the caloric content of food and may offer a pathway to novel
17 ronutrient-fortified flour without increased caloric content of the general food basket was introduce
18 ate consummatory behaviors regardless of the caloric content or biological relevance of the consumed
19 unt of iodine in infant formulas is based on caloric content, and the label must provide the iodine c
20 plications.We aimed to quantify the systemic caloric contribution of acid-citrate-dextrose regional a
21 can enable the practitioner to calculate the caloric cost associated with a specific protein source t
22                        They are activated by caloric deficiency and, when naturally or artificially s
23 tatory input to AgRP neurons is important in caloric-deficiency-induced activation, and is notable fo
24    Conversely, acute inhibition in mice with caloric-deficiency-induced hunger decreases feeding.
25  of POMC neurons is decreased selectively by caloric deficit and not altered by high-fat diet or stre
26                       Tolerating substantial caloric deficit by withholding PN until day 8 of critica
27 increase in AgRP neuron firing with mounting caloric deficit in afternoon vs morning recordings.
28 sed daily energy expenditure, resulting in a caloric deficit of approximately 1 kcal/day; however, to
29                                 Although the caloric deficits achieved by increased awareness, policy
30 tudies we have demonstrated that a high fat, caloric dense maternal diet structures the offspring's e
31                                  As food and caloric-dense liquid solutions were used, the data repor
32                  More happy tweets and lower caloric density of food tweets in a zip code were associ
33 pothalamus (ARC) are oppositely regulated by caloric depletion and coordinately stimulate and inhibit
34 ate-day fasting, and other forms of periodic caloric desistance are gaining popularity in the lay pre
35  (HFD) (20% of calories from FA) or a normal caloric diet (C group) (10% of calories from FA) for 16
36       The cellular processes that cause high caloric diet (HCD)-induced infertility are poorly unders
37 antly more compared with animals on a normal caloric diet (P < 0.05).
38 n body composition under a high fat and high caloric diet, although the weight of the mutant mice was
39 ibit any fertility decline compared with low caloric diet-fed males despite their resistance to the s
40  remarkable difference in resistance to high caloric diet-induced weight gain of the dab2-deleted mic
41 , because each participant ingested the same caloric dose, the change in plasma glucose depended upon
42                                              Caloric effects are currently under intense study due to
43 ch for solid state materials that show large caloric effects.
44  receiving minimal enteral feeds versus full caloric enteral nutrition.
45 ion was given as control nutrition to obtain caloric equivalence and minimize confounding.
46 besity is critical for adaptation to chronic caloric excess and maintenance of energy homeostasis via
47 ulation is a common manifestation of chronic caloric excess and obesity that is strongly associated w
48 s is insufficient to accommodate the chronic caloric excess and prevent adipocyte overloading.
49 sly unrecognized transcriptional response to caloric excess by reducing PNC1 expression.
50       Recent research indicates that chronic caloric excess can lead to hypothalamic microinflammatio
51  Successful adaptation to periods of chronic caloric excess is a highly coordinated event that is cri
52 or adaptive thermogenesis and dissipation of caloric excess through the activity of uncoupling protei
53                               During chronic caloric excess, adipose tissue expands primarily by enla
54 both reduced daily food intake and increased caloric expenditure, driven by an increase in whole-body
55  known as beiging or browning that regulates caloric expenditure.
56  in limiting adiposity in mice by increasing caloric expenditure.
57 by superior nutritional density or minimized caloric expenditure.
58 -derived signaling peptide AQEE-30 upon high caloric feeding.
59  complications compared with early full dose caloric feedings.
60 s essential towards optimization of its spin-caloric functionality for spintronics applications.
61                              The appropriate caloric goal for critically ill adults is unclear.
62   Pavlovian cues associated with junk-foods (caloric, highly sweet, and/or fatty foods), like the sme
63               Obesity typically is linked to caloric imbalance as a result of overnutrition.
64 ARC) promote homeostatic feeding at times of caloric insufficiency, yet they are rapidly suppressed b
65 -19.8%, p = 0.49) and in mean per capita SSB caloric intake (-13.3%, p = 0.56) from baseline to post-
66 p, individuals had lower protein (30.1%) and caloric intake (30.2%) (P = 0.01 and 0.02, respectively)
67  all patients with bvFTD had increased total caloric intake (mean, 1344 calories) compared with the A
68  sleep restriction, subjects increased daily caloric intake (P < 0.001) and fat intake (P = 0.024), i
69  proportion of patients with low protein and caloric intake (P = 0.02 and 0.01, respectively).
70 ration of cardiac aging under TRF, even when caloric intake and activity were unchanged.
71 resents a promising option for reducing both caloric intake and appetite in humans.
72 tones in postmenopausal women independent of caloric intake and BMI, primarily because of the amount
73    In the whole cohort, after adjustment for caloric intake and cardiovascular disease risk factors,
74 is affects host fitness owing to the loss of caloric intake and colonization resistance (protection f
75 -type mice, the lean AAV mice have increased caloric intake and do not develop age-related obesity or
76 t is the consequence of an imbalance between caloric intake and energy consumption.
77 matory pathways results in the uncoupling of caloric intake and energy expenditure, fostering overeat
78  a novel role of PDE10A in the regulation of caloric intake and energy homeostasis.
79 cues, and that maintaining a balance between caloric intake and expenditure may reduce striatal, insu
80 lso imply that maintaining a balance between caloric intake and expenditure over time may reduce stri
81 iposity, the effect of FGF21 on body weight, caloric intake and fat oxidation were significantly atte
82                         First, we determined caloric intake and food choice after bilateral administr
83 libitum breakfast test meal, and their total caloric intake and food preferences were measured.
84 e onset of weight gain in response to excess caloric intake and hyperinsulinemia; however, the mechan
85 e energy expenditure, GLP-1 action to reduce caloric intake and improve glucose control, and GIP acti
86                                              Caloric intake and meal-timing data were collected durin
87 ure secondary to exposure to excessive daily caloric intake and overnutrition.
88 of LHA glutamatergic neurons increased daily caloric intake and produced weight gain in mice that had
89 hotosynthesis constitute much of human daily caloric intake and provide the basis for high-energy bio
90  dietary switch changed the pattern of daily caloric intake and suppressed HFD-induced adipose macrop
91 low social rank is associated with increased caloric intake and weight gain.
92 hat a restricted HFD intake regimen inhibits caloric intake as a consequence of FA-induced VMH ketone
93 ne week of daily anodal tDCS reduced overall caloric intake by 14% in comparison with sham stimulatio
94            The evidence is accumulating that caloric intake can increase production of reactive oxyge
95 ivity, other breast cancer risk factors, and caloric intake controlled for (false discovery rate <0.2
96                                    Increased caloric intake correlated with atrophy in discrete neura
97                                         High caloric intake disrupted this interaction and decreased
98 restriction, exhibited a greater increase in caloric intake during sleep restriction (d = 0.62), and
99 ly differ in daily caloric intake, increased caloric intake during sleep restriction, or meal timing.
100            Animals exposed to 3 days of high caloric intake exhibited hyperinsulinemia without hyperg
101                                       Higher caloric intake further increases the risk of incident st
102                                       Proper caloric intake goals in critically ill surgical patients
103 ats lack compensatory mechanisms to increase caloric intake in response to a T3-induced increase in E
104                              Increased daily caloric intake is a major behavioral mechanism that unde
105 igenic gastric peptide hormone secreted when caloric intake is limited.
106                  Moderate differences in the caloric intake of meat products provided nontrivial redu
107  lower than those of nonvegetarians and that caloric intake of vegetarians is typically lower than th
108 hGH) gene, GH1, to assess the effect of high caloric intake on expression as well as the local chromo
109 hysical activity muted the effects of excess caloric intake on insulin levels, GH1 promoter hyperacet
110 ned from stores contribute to disparities in caloric intake over time.
111 iasis, but the role of physical activity and caloric intake remains poorly understood.
112                                              Caloric intake tended to decrease after DB administratio
113 the women were nulliparous, and median daily caloric intake was 1,840 cal (IQR 1,487-2,222).
114       A greater reduction in body weight and caloric intake was observed in response to IMC-H7 during
115                                              Caloric intake was similar in the two groups, with the t
116     PDE10A deficiency produces a decrease in caloric intake without affecting meal frequency, daytime
117  (500-kcal/d deficit from weight-maintaining caloric intake) and then randomly assigned to pioglitazo
118 ypercaloric diets (in 75% excess of habitual caloric intake) for 3 days, enriched in unsaturated FA (
119  time of scan, gender, ethnicity, education, caloric intake, and apolipoprotein genotype.
120 on cycle, provides a percentage of our daily caloric intake, and is a major driver in the renewable c
121                    Urinary creatinine, total caloric intake, and percentages of nutrient intake from
122       These changes were not due to aging or caloric intake, as neither these changes nor the MS were
123 e quality of dietary intake (particularly in caloric intake, dietary protein intake, dietary fiber in
124 whites did not significantly differ in daily caloric intake, increased caloric intake during sleep re
125                    These focused on reducing caloric intake, increasing physical activity, and behavi
126 sought to assess sex and race differences in caloric intake, macronutrient intake, and meal timing du
127 than women due to a larger increase in daily caloric intake, particularly during late-night hours.
128    Secondary outcomes included self-reported caloric intake, walking, and moderate physical activity.
129                                 Decreases in caloric intake, weight, and BMI correlated with activati
130 lation of LHA glutamatergic neurons enhances caloric intake.
131 eted their fat stores, despite having higher caloric intake.
132 er a meal and shows potential for decreasing caloric intake.
133 s of lactoferrin being partly independent of caloric intake.
134 in the first 6 days, and not used to augment caloric intake.
135 etofore assumed, simply triggered by reduced caloric intake.
136 ession, with adjustment for age, gender, and caloric intake.
137 gth z scores were negatively correlated with caloric intake.
138 eppers, is able to induce satiety and reduce caloric intake.
139  ghrelin plasma concentrations, satiety, and caloric intake.Women (n = 39) were more sensitive toward
140 ries that help in balancing food choice with caloric intake; however, this metabolic learning or memo
141 estricted feeding (TRF) regimen in which all caloric intakes occur consistently within </= 12 h every
142 an studies, we examine the relative roles of caloric load and perceived sweetness in driving metaboli
143 demonstrate a non-linear association between caloric load and reward and describe an unanticipated ro
144                                      Whereas caloric load was manipulated using the tasteless carbohy
145 es and (2) when sweetness is proportional to caloric load, greater metabolic responses are observed.
146 demonstrate a non-linear association between caloric load, metabolic response, and reinforcement pote
147 extent to which sweetness is proportional to caloric load.
148 volving food anticipatory activity (FAA) and caloric loading following food access.
149         Here, we report that optimization of caloric loading in B6 mice subject to HFS, characterized
150 urons are also sufficient to restore FAA and caloric loading of B6 mice subjected to HFS.
151 ver failure and 2 patients developed protein-caloric malnutrition treated by elongation of the common
152 aves a new pathway for developing novel spin-caloric materials.
153 y before and at 160 and 240 min after a high-caloric mixed meal.
154  to the sweet taste of sugar, but due to the caloric nature of the sweetener.
155 own to modify their behavior during times of caloric need, rapidly adapting to a consistently changin
156 ir mates benefit in ways that go well beyond caloric nutrition.
157 4% of products in the US food supply contain caloric or low-calorie sweeteners, or both.
158 and peripheral inflammatory responses due to caloric overload.
159  were also given intermittent access to high-caloric palatable food.
160 s in-line determination of NO and NO2 at the caloric power plant Durnrohr (Austria).
161  measurements reflect the sum of overlapping caloric processes involving binding-linked population sh
162      Among critically ill surgical patients, caloric provision across a wide acceptable range does no
163                       The optimum target for caloric provision remains elusive.
164 trolled for important socioeconomic factors, caloric purchases fell significantly from 2003 to 2011 (
165 ic purchases were driven more by declines in caloric purchases from beverages than food.
166             Results also indicated shifts in caloric purchases were driven more by declines in calori
167 ssion was associated with small increases in caloric purchases, in which a 1-percentage point increas
168  daily cycles of feeding and fasting without caloric reduction, sustains robust diurnal rhythms and c
169         The intervention emphasized moderate caloric reduction, the DASH (Dietary Approaches to Stop
170 cent evidence is shaping a picture where low caloric regimes and exercise may improve healthy senesce
171 receive either the standard calculated daily caloric requirement of 25-30 kcal . kg(-1) . d(-1) (euca
172 lity can be used to estimate the free-living caloric requirements of nonobese adults.
173 issive underfeeding (40 to 60% of calculated caloric requirements) or standard enteral feeding (70 to
174 l per day, P<0.001; 46+/-14% vs. 71+/-22% of caloric requirements, P<0.001).
175 avioral pattern that promotes acquisition of caloric resources to compensate for low social resources
176                                      Whether caloric restriction (CR) acts synergistically with RT to
177                                              Caloric restriction (CR) can increase longevity in roden
178 is unclear, but prior evidence suggests that caloric restriction (CR) can slow thymic aging by mainta
179                                      Because caloric restriction (CR) delays aging, at least in part,
180                                 Importantly, caloric restriction (CR) extends lifespan in several org
181                                              Caloric restriction (CR) extends the lifespan of flies,
182                                              Caloric restriction (CR) has SSRI-like effects on neural
183                                              Caloric restriction (CR) improves insulin sensitivity an
184                                              Caloric restriction (CR) is a dietary regimen known to p
185                                              Caloric restriction (CR) is commonly recommended for imp
186 e examined whether these features are due to caloric restriction (CR) or altered nutrient handling.
187 e to adjust their activities when faced with caloric restriction (CR) to deal with reduced energy int
188                                              Caloric restriction (CR) without malnutrition extends li
189                                              Caloric restriction (CR) without malnutrition increases
190  (iePPARgammaKO) to a two-week period of 25% caloric restriction (CR), following which iePPARgammaKO
191 ction of resting energy expenditure (REE) to caloric restriction (CR).
192 xtension of chronological life span (CLS) by caloric restriction (CR).
193                                              Caloric restriction alone also led to significant decrea
194 ve caloric restriction and aerobic exercise, caloric restriction alone, aerobic exercise alone, or us
195  for potential factors that are regulated by caloric restriction and act as caloric restriction mimet
196 tested the hypothesis that implementation of caloric restriction and aerobic exercise is feasible and
197 ts consented, 111 were randomized to receive caloric restriction and aerobic exercise, caloric restri
198 lycemia and promoting survival during severe caloric restriction and the requirement for ghrelin cell
199                                  In rodents, caloric restriction and young blood-induced revitalizati
200               Previous reports indicate that caloric restriction attenuates anxiety and other behavio
201                              We propose that caloric restriction attenuates behavioral and physiologi
202                                  In mammals, caloric restriction consistently results in extended lif
203 nth randomized controlled trial, comparing a caloric restriction diet arm (goal: 10% weight loss, N =
204                   In this study we find that caloric restriction dramatically rescues the motor incoo
205                          Gene expression and caloric restriction experiments in model organisms confi
206 mates, and exposure of Ghsr-null mice to 60% caloric restriction fails to elicit antidepressant-like
207 treatment with 2.24 mg/kg.d rapamycin or 40% caloric restriction for 9 weeks partially rescued cardio
208  Biase et al. independently demonstrate that caloric restriction from fasting and pharmacologic inhib
209 rift is a determinant of lifespan in mammals.Caloric restriction has been shown to increase lifespan
210 sely, lifespan-extending maneuvers including caloric restriction impose beneficial pleiotropic effect
211 , it is significantly prevented by long-term caloric restriction in aged mice.
212 eplacement of ghrelin blocked the effects of caloric restriction in beta1AR-deficient mice.
213 F-1 immobilization as a specific response to caloric restriction in C. elegans intestinal cells.
214 tion drift correlates with lifespan and that caloric restriction in mice and rhesus monkeys results i
215 sized by the hypoglycemia that is induced by caloric restriction in mouse models of deficient ghrelin
216                                        Also, caloric restriction increased apoptosis of DG subgranula
217                                              Caloric restriction inhibits hepatosteatosis, reduces Wn
218                                              Caloric restriction is known to up-regulate expression o
219 ller reductions in energy expenditure during caloric restriction is not known.
220                          Longevity-promoting caloric restriction is thought to trigger downregulation
221                                              Caloric restriction mimetic drugs have geroprotective ef
222 y systems, or longevity pathways targeted by caloric restriction mimetic drugs.
223                                              Caloric restriction mimetics (CRMs) mimic the biochemica
224 anism, effects, and organ specificity of the caloric restriction mimetics RSV and SRT.
225  regulated by caloric restriction and act as caloric restriction mimetics.
226 "habit," supporting the view that persistent caloric restriction mimics some aspects of addiction to
227                               The effects of caloric restriction on DNA methylation were detectable a
228 herefore determined the effects of aging and caloric restriction on the expression of FXR and TGR5 in
229 older patients with clinically stable HFPEF, caloric restriction or aerobic exercise training increas
230  normalization of weight was mediated not by caloric restriction or increased activity, but by increa
231 uggest that the anti-inflammatory effects of caloric restriction or ketogenic diets may be linked to
232                       Dietary habits such as caloric restriction or nutrients that mimic these effect
233 e on control diet or one of 2 diet regimens (caloric restriction or rapamycin) that altered protein t
234 6 y] men were recruited to either WL through caloric restriction or weight maintenance (WM) for 6 mo.
235 eater decreases in energy expenditure during caloric restriction predict less weight loss, indicating
236 s are decreased in the aging kidney and that caloric restriction prevents these age-related decreases
237                         We further show that caloric restriction rescues SIRT1 levels in transgenic M
238 red with depletions by exercise alone, acute caloric restriction results in rapid changes in appetite
239 ell-like features, whereas CtBP depletion or caloric restriction reverses gene repression and increas
240 how that both short-term fasting and chronic caloric restriction significantly reduce the percentage
241 to 30-year-old rhesus monkeys exposed to 30% caloric restriction since 7-14 years of age showed atten
242 seen in 2.7-3.2-year-old mice exposed to 40% caloric restriction starting at 0.3 years of age.
243 es with overfeeding, lose less weight during caloric restriction than those with a "spendthrift" phen
244 oluntary wheel running (HFD+Ex) and then 25% caloric restriction with exercise (Ex/CR), each for an a
245 ls of cAMP, thereby mimicking the effects of caloric restriction with respect to metabolic reprogramm
246  the weight-loss program included nutrition (caloric restriction) and psychological therapies.
247 nduced by dietary restriction (also known as caloric restriction).
248 en shown to decrease with age, increase with caloric restriction, and influence stress resistance.
249 t stimulate AMPK and PGC1alpha via exercise, caloric restriction, and medications result in stimulati
250 ditis elegans BAF-1 mobility is regulated by caloric restriction, food deprivation, and heat shock.
251       BHB levels are elevated by starvation, caloric restriction, high-intensity exercise, or the low
252 energy deficit (kilocalories per day) during caloric restriction, incorporating energy intake and was
253                               Starvation, or caloric restriction, is known to activate the transcript
254                    Metabolic factors such as caloric restriction, ketogenic diet, and hyperglycemia i
255 nd nutritional factors, such as exercise and caloric restriction, may exert their known health benefi
256  and induced in a VHR1 dependent manner upon caloric restriction, on non-fermentable carbon sources,
257 idative stress, exercise-induced adaptation, caloric restriction, osmotic stress, mechanical stress,
258 cations such as reduced carbohydrate intake, caloric restriction, structured exercise, and/or pharmac
259 by specific interventions: metabolic risk by caloric restriction, systemic inflammation by statins, p
260 F levels increase with physical activity and caloric restriction, thus BDNF may mediate some of the o
261 e after bariatric surgery independently from caloric restriction, whereas the level of WAT TGR5 prote
262 ergic nature of AgRP neurons is increased by caloric restriction, whether the GABAergic phenotype of
263 be more effective for reducing body fat than caloric restriction, which is currently the treatment of
264  in Ghsr-null mice exposed to either CSDS or caloric restriction, while the more highly active analog
265 ) localized to ghrelin cells is required for caloric restriction-associated ghrelin release and the e
266 th the FXR-TGR5 dual agonist INT-767 induced caloric restriction-like effects and reversed age-relate
267  LG isoflavone supplementation resulted in a caloric restriction-like gene expression profile for bot
268 nisms promotes longevity in a manner akin to caloric restriction.
269 groups followed a Mediterranean diet without caloric restriction.
270     Volunteers then underwent 6 weeks of 50% caloric restriction.
271 uenced by the energy expenditure response to caloric restriction.
272 armacologic agents that mimic the effects of caloric restriction.
273 n in different species by mechanisms akin to caloric restriction.
274  accumulation through a mechanism resembling caloric restriction.
275 own and delays age-related kidney disease is caloric restriction.
276 se and the changes in their capacities after caloric restriction.
277 glycemia and prevalent mortality upon severe caloric restriction.
278 reased the consumption of a palatable liquid caloric reward.
279  and is notable for its remarkable degree of caloric-state-dependent synaptic plasticity.
280                        Our findings show how caloric suppression of the guanylin-GUCY2C signaling axi
281 ctive mechanism against tissue damage during caloric surplus and that it is only when the maximum fat
282                                          The caloric surplus consisted of fat and sugar (high-fat-hig
283 ssues, leading to the deleterious effects of caloric surplus.
284 vioside and rebaudioside A, are natural, non-caloric sweet-tasting organic molecules, present in extr
285 verage was associated with more purchases of caloric-sweetened desserts or sweeteners, which accounte
286 urchases, sugar, and fat, and purchased more caloric-sweetened desserts/caloric sweeteners compared w
287 013, 68% (by proportion of calories) contain caloric sweeteners and 2% contain low-calorie sweeteners
288 nd purchased more caloric-sweetened desserts/caloric sweeteners compared with nonconsumers.
289 orie sweeteners and beverages sweetened with caloric sweeteners had poorer dietary quality, exhibited
290  The novel sweeteners can be utilised as non-caloric sweeteners in the production of low-calorie food
291                               Replacement of caloric sweeteners with lower- or no-calorie alternative
292 termined by a doubly labeled water study and caloric titration to weight maintenance.
293  quantified nitrogen (N), phosphorus (P) and caloric turnover rates for Lake Huron lake trout, and re
294 ed, caused by adaptive mechanisms maximizing caloric uptake and increasing intestinal, villi, and mic
295 li did not alter changes in host metabolism, caloric uptake, or inflammation but instead sustained si
296 ore swiftly than a flower type with the same caloric value but without nicotine.
297 ructural properties of fat rather than total caloric value determine intestinal sensing and the assig
298                           Accounting for the caloric value of RDAs for carbohydrate and fat, "flexibl
299 vidence that L-lactate, independently of its caloric value, serves as an astrocytic signalling molecu
300 eostasis favored detection and comparison of caloric value.

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