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1 eavage ends in wild-type but not Col1a1(r/r) calvariae.
2 nto the subcutaneous tissues overlying mouse calvariae.
3 lonal cell line, 2T3, derived from the mouse calvariae.
4 ae vs. an increase in Alx4 in Ambn-deficient calvariae.
5 months in long bones and vertebrae, but not calvariae.
7 n-deficient osteoblasts from neonatal murine calvariae and found that the absence of bgn caused less
11 ormation in organ cultures of neonatal mouse calvariae, and a neutralizing antibody to hPDGF-BB block
12 ed by sequential digestion of neonatal mouse calvariae, and cultured with fetal calf serum (10% for o
13 Critical-sized defects were created in rat calvariae, and GBR procedures were performed with a coll
15 brae in one strain) and cortical bone in the calvariae (bone mineral density was decreased on average
16 extreme ends of the osteogenic fronts of the calvariae, facilitating expansion of the skull and closu
17 ns, we detect empty lacunae in osteocytes in calvariae from Col1a1(r/r) mice at age 2 weeks, increasi
18 d periosteal proliferation were increased in calvariae from PTH-treated +/+ mice, whereas in r/r mice
24 Injection of recombinant MIP-1alpha over calvariae of normal mice evoked a striking increase in o
26 e osteoprogenitor cell line derived from the calvariae of transgenic mice containing the SV40 T-antig
28 sts cultured on dentine or explants of mouse calvariae prelabeled with (45)Ca, we could not detect si
30 were harvested from fetal Swiss Webster mice calvariae prior to osteoblast differentiation and calcif
31 n, and a reduction in Alx4 in Amel-deficient calvariae vs. an increase in Alx4 in Ambn-deficient calv
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