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1 wn about the genetic mechanisms that control cambial activity and the differentiation of secondary xy
2 n, our findings suggest that reinitiation of cambial activity and transdifferentiation of xylem paren
3 t cytokinins function as major regulators of cambial activity, these trees displayed stimulated cambi
4 pecies can be linked to a distinct period of cambial activity, we applied a bi-weekly pinning to six
6 nal activity of the gene is highest in root, cambial and shoot meristems and immature tissues of the
7 involving a polyphagan wood-borer consuming cambial and wood tissues of the conifer Ningxiaites spec
8 tected in developing embryos and procambial, cambial, and vascular cells of cotyledons, leaves, roots
9 l cytokinin content and signaling level, the cambial auxin concentration and auxin-responsive gene ex
11 l activity, these trees displayed stimulated cambial cell division activity resulting in dramatically
12 onal pathway that regulates both the rate of cambial cell division and woody tissue organization.
13 and CLE41 and a receptor kinase PXY controls cambial cell divisions; however, the pathway regulating
14 uring vascular development, the meristematic cambial cells divide down their long axis in a highly or
15 ghest concentration in the actively dividing cambial cells, cytokinins peak in the developing phloem
16 estingly, in addition to showing an elevated cambial cytokinin content and signaling level, the cambi
18 in VC activity and prolonged maintenance of cambial-derived cells in a meristematic state was crucia
19 onal significance of cytokinin signaling for cambial development, we engineered transgenic Populus tr
20 t regulation of the transcriptome underlying cambial growth and wood formation comprises numerous mod
24 atively short (three to four months maximum) cambial growth season corresponded to the core of the ra
25 ainfall event occurring after the end of the cambial growth season did not induce xylem initiation or
26 Our results show that the onset and end of cambial growth was synchronous between trees, but was no
28 ase, the symmetry of the defects implies (1) cambial orientation is controlled by a vector quantity a
30 and consequently for normal stem elongation, cambial proliferation, and xylem fiber differentiation.
31 hat the mRNA is present predominantly in the cambial region in stems, leaves, and roots and in the va
32 auxin- and cytokinin-regulated induction and cambial region localization, encourage us to suggest tha
33 tration of indole-3-acetic acid (IAA) in the cambial region of a tree branch and the radial expansion
34 In addition, the gene expression in the bark/cambial region was up-regulated in spring and fall when
35 RpALN gene was highly expressed in the bark/cambial region, but had no detectable expression in the
36 nd a cle41 mutant with altered regulation of cambial stem cell maintenance and differentiation, that
37 onal to the mass of IAA per unit area on the cambial surface, and (3) IAA is transported basipetally
38 ary growth, ARK2 is expressed broadly in the cambial zone and in terminally differentiating cell type
39 iming of cambium formation, the width of the cambial zone and inhibition of cambial daughter cell dif
42 reid lignification, and reprogramming of the cambial zone to form traumatic resin ducts in Pseudotsug
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