ライフサイエンスコーパス: cancellous

1 Fourteen patients received a cancellous allograft (CAN group) and the other 14 receiv

2 controls received an intrasocket mineralized cancellous allograft (socket group), and 12 patients rec

3 raft (CAN group) and the other 14 received a cancellous allograft mixed with PRP (PRP group).

4 ine if PRP combined with a rapidly resorbing cancellous allograft would enhance the regenerative resu

5 s treated with BG, such as mineralized human cancellous allograft, were more likely to have greater v

6 te that solvent-preserved, mineralized human cancellous allograft, with or without collagen membrane,

7 Lrp4 deficiency promoted progressive cancellous and cortical bone gain in both mutants, altho

8 DBM formulated with hyaluronic acid (HY) and cancellous and cortical bone granules from the same dono

9 antibodies increased bone formation and thus cancellous and cortical bone mass in skeletally mature r

10 In conclusion, Notch2(Q2319X) mice exhibit cancellous and cortical bone osteopenia, enhanced osteoc

11 ntrols; and at 1 month of age they exhibited cancellous and cortical bone osteopenia.

12 eases the density, area and strength of both cancellous and cortical bone.

13 A solvent-preserved, mineralized human cancellous bone allograft (MBA) was recently developed.

14 Sinuses were grafted with mineralized cancellous bone allograft, anorganic bovine bone matrix,

15 orphometric analysis of tetracycline-labeled cancellous bone and dual-energy x-ray absorptiometry, re

16 Cancellous bone and other natural cellular solids have a

17 ne is rooted in the trajectory hypothesis of cancellous bone architecture.

18 We show that the more ductile surfaces of cancellous bone are a result of reduced accumulation of

19 creased bone density, serum osteocalcin, and cancellous bone area along with trabecular narrowing.

20 Cancellous bone area was lower in the patients with CF (

21 Y, 32:68, wt/wt; DBM mixed with cortical and cancellous bone chips 1:4 (DBMC) (11 mg total, of which

22 ociated with enhanced bone resorption in the cancellous bone compartment and with suppressed endocort

23 and there was a trend towards a decrease in cancellous bone connectivity.

24 The MAT- WT --> Kit(W/W-v) mice lost cancellous bone following 2 weeks of HU.

25 HU MAT- mice had elevated cancellous bone formation and resorption compared to oth

26 ls in postnatal mice dramatically stimulated cancellous bone formation via marked expansion of the os

27 hymal progenitors" (MMPs), are essential for cancellous bone formation.

28 Cancellous bone histomorphometry revealed an increased n

29 Cancellous bone histomorphometry revealed that the incre

30 ne marrow stromal cells (BMSCs) form cortico-cancellous bone in rodent models.

31 Histomorphometric parameters characterizing cancellous bone in the distal radius can be derived from

32 Furthermore, increased cancellous bone is abolished by Wnt inhibition but furth

33 ponent of the cascade of events that lead to cancellous bone loss during estrogen deficiency.

34 ontribute to the increase in osteoclasts and cancellous bone loss that occurs after loss of estrogen.

35 ion in mediating estrogen deficiency-induced cancellous bone loss was investigated in ovariectomized

36 with Debio0719 prevented ovariectomy-induced cancellous bone loss.

37 s associated with exaggerated disuse-induced cancellous bone loss.

38 Cancellous bone marrow R2' measured in the proximal femu

39 e, but not in adult mice, whereas epiphyseal cancellous bone mass decreased with loading in both youn

40 likely to accumulate in strut centers making cancellous bone more tolerant of stress concentrations a

41 , bone formation rate, and wall width in the cancellous bone of conditional knock-out mice.

42 These differences appeared whether light, cancellous bone or heavier endosteal bone was removed.

43 creased prevalence of apoptosis in vertebral cancellous bone osteocytes and osteoblasts that follows

44 lysis revealed thin cortical bone and sparse cancellous bone patterns.

45 of osteoblast recruitment during adult human cancellous bone remodeling is lacking.

46 Advanced visualization of cancellous bone significantly increased the detection of

47 evised a method for obtaining information on cancellous bone structure from iliac bone histomorphomet

48 Cancellous bone structure was treated as a quasi-regular

49 ant decrease in bone mass and alterations in cancellous bone structure.

50 rfaces of the humerus and the periosteal and cancellous bone surfaces of the mandible.

51 tes is essential for osteoclast formation in cancellous bone under physiological conditions, and RANK

52 Cancellous bone volume and cortical thickness were decre

53 Notch in the skeleton causes an increase in cancellous bone volume and enhanced osteoblastic differe

54 reatment was associated with preservation of cancellous bone volume and inhibition of osteoclast form

55 Cancellous bone volume and osteoid markers correlated wi

56 microstructural abnormalities such as lower cancellous bone volume and reduced trabecular thickness.

57 mined by dual-energy densitometry; decreased cancellous bone volume and trabecular width and increase

58 Cancellous bone volume fraction was lower in flight anim

59 e- and bone compartment-specific deficits in cancellous bone volume fraction.

60 Our results demonstrate low cancellous bone volume in adult patients with CF with lo

61 e characterized by a significant decrease in cancellous bone volume in the tibial and femoral metaphy

62 As the mice matured, cancellous bone volume was restored partially in male bu

63 usly over the calvaria of mice and increased cancellous bone volume when orally administered to rats.

64 7(-/-)) exhibit higher bone mineral density, cancellous bone volume, and mechanical strength compared

65 iblings, demonstrated a striking decrease in cancellous bone volume, connectivity, and trabecular num

66 m, and normalization of bone markers such as cancellous bone volume, trabecular number, osteoblast su

67 Cancellous bone volume/tissue volume was below normal co

68 Cancellous bone volumes of ARKO male mice are reduced co

69 ature osteocytes in mineralized cortical and cancellous bone was positive for sclerostin with diffuse

70 However, cancellous bone was preferentially lost in the metaphysi

71 ate loading, especially in those areas where cancellous bone was present.

72 Metal distribution within the part of cancellous bone was revealed for silver as well as for t

73 We demonstrate that in cancellous bone, the foam-like component of whole bones,

74 fiber reinforcement reached the strength of cancellous bone, which was much stronger than previous i

75 tion of a threaded titanium cage packed with cancellous bone.

76 matory process that demineralizes trabecular cancellous bone.

77 m in cortical bone, but tempers bone gain in cancellous bone.

78 c stem and progenitor cells (HSPCs) in human cancellous bone.

79 localized throughout the marrow cavities of cancellous bone.

80 in mouse femur is also present within human cancellous bone.

81 t of estrogens against endocortical, but not cancellous, bone resorption.

82 n derivative (EMD) and particulate anorganic cancellous bovine-derived bone xenograft (BDX) have both

83 In summary, both the particulate anorganic cancellous bovine-derived bone xenograft used alone and

84 This protection occurred in cancellous, but not cortical, bone and was associated wi

85 or the protective effect of estrogens on the cancellous, but not the cortical, bone compartment that

86 sayed demineralized bone matrix and cortical cancellous chips uniformly dispersed in a thermoplastic

87 garding use of a combination 50%/50% cortico-cancellous FDBA compared with a 100% cortical or 100% ca

88 in the cortical FDBA group compared with the cancellous FDBA group (P = 0.019).

89 neralized CT/other material was found in the cancellous FDBA group compared with the cortical FDBA gr

90 new bone formation between the cortical and cancellous FDBA groups (P = 0.857).

91 s FDBA compared with a 100% cortical or 100% cancellous FDBA in ridge preservation.

92 ion in non-molar sites using 50%/50% cortico-cancellous FDBA versus 100% cortical and 100% cancellous

93 ancellous FDBA, 100% cortical FDBA, and 100% cancellous FDBA when used in ridge preservation of non-m

94 omly assigned to each group (cortical versus cancellous FDBA).

95 or dimensional changes among 50%/50% cortico-cancellous FDBA, 100% cortical FDBA, and 100% cancellous

96 100% cancellous FDBA; or 3) 50%/50% cortico-cancellous FDBA.

97 preservation in humans using cortical versus cancellous FDBA.

98 ancellous FDBA versus 100% cortical and 100% cancellous FDBA.

99 he following: 1) 100% cortical FDBA; 2) 100% cancellous FDBA; or 3) 50%/50% cortico-cancellous FDBA.

100 d ridge preservation with either cortical or cancellous freeze-dried bone allograft (FDBA) in non-mol

101 greater loss of lingual ridge height in the cancellous group.

102 ative concentration of HSPCs and BVFs within cancellous marrow was observed to diminish with increasi

103 ralized bone matrix (GDBM) by comparing with cancellous mineralized bone matrix (CMBM) and anorganic

104 Cortical and cancellous mineralized freeze-dried bone allografts (FDB

105 osteoblasts or osteocytes did not influence cancellous or cortical bone mass.

106 orticoid-induced osteoporosis, the number of cancellous osteoclasts increased, even though osteoclast

107 nous (type I) bone versus more trabeculated, cancellous (type III) bone.

108 e socket graft procedure plus buccal overlay cancellous xenograft (overlay group).