ライフサイエンスコーパス: cancer cell line

1 well with the in vitro values from the same cancer cell line.

2 sed CRISPR to KO PPIP5Ks in the HCT116 colon cancer cell line.

3 ability to inhibit proliferation of the HeLa cancer cell line.

4 n of a conventional HPV-16 positive cervical cancer cell line.

5 uld be modeled in an APC-mutated human colon cancer cell line.

6 NA12878 human genome and the HCC1954 breast cancer cell line.

7 537, in the estrogen-responsive MCF7 breast cancer cell line.

8 atic models using the SGC-7901/sFRP1 gastric cancer cell line.

9 levels in HCT116, HT29, and SW480 colorectal cancer cell lines.

10 HMECs) and estrogen receptor-positive breast cancer cell lines.

11 cit in colorectal, ovarian, renal and breast cancer cell lines.

12 fied their mRNA level in a panel of prostate cancer cell lines.

13 sponse of engineered T cells to their target cancer cell lines.

14 globally profile CARM1 substrates in breast cancer cell lines.

15 em cells with conditioned medium from breast cancer cell lines.

16 atients with breast cancer and in the NCI-60 cancer cell lines.

17 ransporters are known to induce apoptosis in cancer cell lines.

18 oxic toward normal cell lines as compared to cancer cell lines.

19 g in a large number of genomically annotated cancer cell lines.

20 sphorylation of EGFR protein in several lung cancer cell lines.

21 )-3-(1H-pyrrol-2-yl)acrylonitrile (ANI-7) in cancer cell lines.

22 ted genes modulate drug response in selected cancer cell lines.

23 ric proteins and we tested our prediction in cancer cell lines.

24 inhibitor, erlotinib, in Non-Small Cell Lung Cancer cell lines.

25 mation in vivo and induced apoptosis in lung cancer cell lines.

26 dels and clinically relevant patient-derived cancer cell lines.

27 50 of 3.3 nM) against a large number (93) of cancer cell lines.

28 tiproliferative activity against HT-29 colon cancer cell lines.

29 l four compounds are effective in five liver cancer cell lines.

30 -refractory papillary and follicular thyroid cancer cell lines.

31 Taxol combined with Kanglaite on colorectal cancer cell lines.

32 AMC mouse colonocytes and Caco-2 human colon cancer cell lines.

33 ed at a ratio 1:6 (named PRP) using 24 human cancer cell lines.

34 n of these exhibited synergy in human breast cancer cell lines.

35 gression and apoptosis in several pancreatic cancer cell lines.

36 man colonic adenocarcinomas tested and colon cancer cell lines.

37 s for blocking proliferation in a variety of cancer cell lines.

38 MDA-MB-231) and ovarian (SKOV3 and SKOV3ip) cancer cell lines.

39 translocated from the cytoplasm to Golgi in cancer cell lines.

40 sing an average of 20 shRNAs per gene in 398 cancer cell lines.

41 th inhibition in NCI-H460 and MDA-MB-231met2 cancer cell lines.

42 -function screens performed in diverse human cancer cell lines.

43 GRIP1 in MCF-7 human breast and HepG2 liver cancer cell lines.

44 oring the mesenchymal identity in pancreatic cancer cell lines.

45 bal single-telomere-resolved analyses of two cancer cell lines.

46 investigators in analysing appropriate renal cancer cell lines.

47 porter cells recognize a ligand expressed by cancer cell lines.

48 d apoptosis in a panel of ER-positive breast cancer cell lines.

49 xicity against ovarian, breast, and prostate cancer cell lines.

50 d invasiveness of the SW480 and Hs578T human cancer cell lines.

51 ctures showed cytotoxic effects on different cancer cell lines.

52 ly determined versican expression by several cancer cell lines.

53 wound closing in three types of immortalized cancer cell lines.

54 cer tissues as well as pancreatic and breast cancer cell lines.

55 designed conjugate was studied in normal and cancer cell lines.

56 We also measured expression by 15 human cancer cell lines.

57 e arrest, and cell death in sensitive breast cancer cell lines.

58 hly invasive (T47D) and less invasive (MCF7) cancer cell lines.

59 o MYC/MYCN/MYCL1 expression in a panel of 35 cancer cell lines.

60 d HT29) and nonmetastatic (HepG2 and 1321NI) cancer cell lines.

61 al liposomes and cytotoxic activity on human cancer cell lines.

62 ion of GGPP, in a variety of different human cancer cell lines.

63 against Cisplatin-resistant SKOV3 and SW480 cancer cell lines.

64 them by perturbation experiments in several cancer cell lines.

65 have shown strong cytotoxic effects against cancer cell lines.

66 m large-scale chemical screening using human cancer cell lines.

67 encing the context-specificity phenotypes in cancer cell lines.

68 not been available across a large number of cancer cell lines.

69 C phenotype more lysis-resistant than breast cancer cell lines, a capacity to report protein expressi

70 DAC inhibitory activity in the human ovarian cancer cell line A2780, the human squamous carcinoma cel

71 Interestingly, chemosensitive cancer cell lines (A2780 and PEO1) displayed a glycolyti

72 nDRIVE, when applied to a large panel of pan-cancer cell lines, accurately predicted drug efficacy us

73 d sensitivity to statins in a large panel of cancer cell lines across multiple cancer types.

74 ltiple genomic data sets for three different cancer cell lines, allowing the identification of distin

75 lular superoxide dismutase (SOD3) in thyroid cancer cell lines although according to recent data, the

76 tosolic Ca(2+) concentration of HCT116 colon cancer cell line and modified the cytosolic Ca(2+) oscil

77 These abstracts spanned over 1467 cancer cell lines and 4373 genes representing a total of

78 vailable datasets to survey a total of 1,056 cancer cell lines and 9,250 primary tumors.

79 Among the genes validated using different cancer cell lines and antigens, we identified multiple l

80 CRISPR-Cas9 essentiality screens across 342 cancer cell lines and applied CERES to this data set.

81 pression to drug response in large panels of cancer cell lines and applying these models to tumor gen

82 oma proliferation, a) in vitro against colon cancer cell lines and b) in vivo on tumor growth in mice

83 hat it was broadly upregulated in many human cancer cell lines and cancers, including most notably in

84 iNVICT on simulated data as well as prostate cancer cell lines and cfDNA obtained from castration-res

85 ion inversely correlates with Snail level in cancer cell lines and clinical specimens.

86 rebrin is also upregulated in human prostate cancer cell lines and co-localizes with actin filaments

87 was extensively characterized in a panel of cancer cell lines and combined MEK/mTOR inhibition displ

88 cond application focuses on the RPPA data of cancer cell lines and contains >650 independent cell lin

89 reased FOXO1 expression increases pERK1/2 in cancer cell lines and correlates with increased pERK1/2

90 , whereas higher levels were seen in several cancer cell lines and during replicative senescence.

91 y AKT1 and AKT2 from breast cancer and colon cancer cell lines and flash-frozen tumor lysates with a

92 , which were tested against aggressive human cancer cell lines and for protein synthesis inhibitory a

93 ein levels of eight atypical cyclins in lung cancer cell lines and formalin-fixed and paraffin-embedd

94 increases proliferation and invasiveness of cancer cell lines and has liver oncogenic activity in mi

95 i) normal colon cells, ii) colon and breast cancer cell lines and iii) cancer stem-like cell subpopu

96 target of rapamycin signaling in human liver cancer cell lines and in both an in vitro and in vivo mo

97 ls constitutively in a panel of basal breast cancer cell lines and in more than one third of basal tu

98 ly, MB was effectively applied to normal and cancer cell lines and in the analysis of enzymatic kinet

99 s caused loss of c-IAP1 and XIAP in multiple cancer cell lines and in tumor xenografts, but not in he

100 ectal cancer and head and neck squamous cell cancer cell lines and in tumors from colorectal cancer p

101 entosis occurs constitutively in some human cancer cell lines and mitotic index correlates with cell

102 with the invasive ability of several bladder cancer cell lines and modulation of fibulin-3 expression

103 Finally, our data generated from both human cancer cell lines and mouse xenograft model showed that

104 evaluated as a radiosensitizer in colorectal cancer cell lines and murine xenograft models.

105 ein expression was confirmed in vitro in all cancer cell lines and normal primary dermal fibroblasts

106 sistant high-mesenchymal cell state in human cancer cell lines and organoids and show that it depends

107 As a result, prostate cancer cell lines and organoids derived from individuals

108 d accumulation of these proteins in prostate cancer cell lines and patient specimens and causing resi

109 analysis of transcriptional dysregulation in cancer cell lines and patient tumor specimens offers a p

110 we overexpressed ts-46 and ts-47 in two lung cancer cell lines and performed a clonogenic assay to ex

111 her supported by consistent results in human cancer cell lines and primary samples of human haematolo

112 ysis' (IMAHP) method to a panel of 41 breast cancer cell lines and show that deviations of the observ

113 MICU1 is overexpressed in a panel of ovarian cancer cell lines and that MICU1 overexpression correlat

114 s were positively correlated in human breast cancer cell lines and tissue specimens of primary breast

115 at NFAT3 is highly expressed in various skin cancer cell lines and tumor tissues.

116 ered that a small percentage of nonprostatic cancer cell lines and tumors express PSMA.

117 d4 This signature drove clustering of breast cancer cell lines and tumors into the common subtypes an

118 mor activity toward three different prostate cancer cell lines and was able to induce 60% tumor growt

119 on downstream of silenced NOX1 in both colon cancer cell lines and xenografts.

120 erative activity in vitro against a panel of cancer cell lines and, for selected highly active compou

121 ivity against a Pgp overexpressing resistant cancer cell line, and an important subset of the vinblas

122 -apoptotic proteins, possess cytotoxicity to cancer cell lines, and induce hallmarks of apoptosis.

123 s of MELK block the proliferation of various cancer cell lines, and MELK knockdown has been described

124 ial-mesenchymal transition in human prostate cancer cell lines, and stable overexpression of miR-194

125 Cancer cell lines are major model systems for mechanisti

126 Here we demonstrate, using a variety of cancer cell lines as well as activated primary T cells,

127 h heterogeneity was observed across multiple cancer cell lines as well as isogenically, suggesting th

128 pression was confirmed in a panel of thyroid cancer cell lines at the mRNA and protein levels.

129 In a panel of cancer cell lines, ATRi selectively induced replication

130 knockdown of LINC00152 using siRNAs in lung cancer cell lines, both cell proliferation and colony fo

131 5 stimuli and 7 inhibitors) in 14 colorectal cancer cell lines, building cell line-specific dynamic l

132 ansgenic cancer model and in a DLC1-positive cancer cell line but not in an isogenic DLC1-negative ce

133 lavones can induce apoptosis in a variety of cancer cell lines but have no effect on normal cells.

134 noxide (CO) reduced GSH/GSSG in three breast cancer cell lines by inhibiting CBS.

135 ced cellular cholesterol content in prostate cancer cell lines by inhibiting the activation of sterol

136 anti-proliferative activities against human cancer cell lines by inhibiting tubulin polymerization a

137 ld and reduces the toxicity of Taxol towards cancer cell lines by over 200-fold.

138 the effects of IL28 on the human intestinal cancer cell line Caco-2 in a wound-healing assay, and in

139 investigations using the anaplastic thyroid cancer cell line CAL-62 found that siRNA against TrkA, p

140 itivity of the cisplatin-resistant head-neck cancer cell line Cal27CisR by almost 7-fold.

141 Cancer cell lines carrying genetic aberrations that impa

142 have analyzed a panel of 17 KRAS mutant lung cancer cell lines classified as K-Ras-dependent or -inde

143 vitro experiments were performed with colon cancer cell line Colo320 (high Nrp-2 expression) and hum

144 gene expression data newly generated for 448 cancer cell lines, combined with publicly available data

145 The bioenergetics phenotype of ovarian cancer cell lines correlated with functional phenotypes

146 ed translation stress, and analysis of human cancer cell line data from Project Achilles further reve

147 Results on published datasets and in-house cancer cell line datasets followed by experimental valid

148 n and loss-of-function studies in colorectal cancer cell lines demonstrated that circCCDC66 controlle

149 R-221 and miR-17, are tested in human breast cancer cell lines, demonstrating the 70 approximately 90

150 Many mammalian cancer cell lines depend on glutamine as a major tri-car

151 stauprimide suppresses MYC transcription in cancer cell lines derived from distinct tissues.

152 Many cancer cell lines derived from hematopoietic and lymphoi

153 Prostate cancer cell lines derived from HiMyc tumors (HMVP2 and d

154 In most cancer cell lines derived from solid tumors, ABBV-075 tr

155 tal RNA extracted from human lung and breast cancer cell lines, discriminating between the cancer-pos

156 therapy because they are essential in breast cancer cell lines, druggable, enriched in stem-like brea

157 re tested against STAT3, A2780 and A2780cisR cancer cell lines, E. coli JW2496, and NF-kappaB.

158 fferences in the mechanotype of human breast cancer cell lines (Ea = 2.1 +/- 0.1 and 0.80 +/- 0.19 kP

159 for their effects against the growth of five cancer cell lines (EC109, HepG2, MCF7, MGC803, SKNSH).

160 nit of HIF-1 (HIF-1alpha) in a murine breast cancer cell line, EMT6.

161 ics of Drug Sensitivity in Cancer (GDSC) and Cancer Cell Line Encyclopedia (CCLE) are two major studi

162 ecules, and RNAseq data of cell lines in the Cancer Cell Line Encyclopedia (CCLE) to find potential d

163 iled against two compound panels, namely the Cancer Cell Line Encyclopedia and the Cancer Therapeutic

164 a from 65 kidney-derived cell lines from the Cancer Cell Line Encyclopedia and the COSMIC Cell Lines

165 is across all major types of cancer from the Cancer Cell Line Encyclopedia, which provides genetic an

166 Reducing NAPRT levels in a BRCA2-deficient cancer cell line exacerbated DNA damage in response to c

167 as found to be over-expressed in all ovarian cancer cell lines examined and upregulated by mutated TP

168 y, we found that cisplatin-resistant ovarian cancer cell lines exhibit lower levels of MOAP-1 accumul

169 on, some of RB-intact basal-like type breast cancer cell lines exhibited a similar phenotype followin

170 o restore endogenous levels of E-cadherin in cancer cell lines exhibiting strong or intermediate mese

171 Many human tumor types and cancer cell lines express the MAN2A1-FER fusion, which i

172 BACKGROUND & AIMS: Human tumors and liver cancer cell lines express the product of a fusion betwee

173 vestigate this question, we developed breast cancer cell lines expressing an inducible, constitutivel

174 f peptide exhibits an anti-tumor activity in cancer cell lines expressing wild-type p53.

175 discriminate between normal and human breast cancer cell lines (fibrocystic and metastatic states) as

176 We selected lung and breast cancer cell lines for the ability to infiltrate and grow

177 We integrate gene expression profiles of cancer cell lines from two E2F1-driven highly aggressive

178 Although cancer cell lines generally recapitulated the expression

179 reen in an isogenic pair of human colorectal cancer cell lines harboring mutant or wild-type KRAS.

180 mal cell state observed in human tumours and cancer cell lines has been associated with resistance to

181 Finally, we show that cancer cell lines have an OxPhos capacity that is insuff

182 ished in control tests with a non-metastatic cancer cell line (HeLa).

183 Depletion of La in various types of cancer cell lines impairs cell proliferation; however, t

184 horylation of the E1alpha subunit in the PC3 cancer cell line in vitro.

185 increases or decreases invasion of prostate cancer cell lines in 3D in vitro assays, respectively.

186 filaments, reduced the invasion of prostate cancer cell lines in 3D in vitro assays.

187 drebrin, also inhibited invasion of prostate cancer cell lines in 3D in vitro assays.

188 tress status in four breast and glioblastoma cancer cell lines in relation with their glycolytic phen

189 suppressed the invasive capacity of prostate cancer cell lines in vitro and in vivo Mechanistic inves

190 selective anti-cancer capacity on dozens of cancer cell lines in vitro and on subcutaneous xenograft

191 g, XDM-CBP was used in screenings of several cancer cell lines in vitro to study its inhibitory poten

192 en tested against a panel of four pancreatic cancer cell lines in vitro.

193 ns in vivo and following induction of EMT in cancer cell lines in vitro.

194 y in primary cancer specimens in vivo and in cancer cell lines in vitro.

195 n protein phosphatase activity for two colon cancer cell lines in which NOX1 expression was knocked d

196 Treatment of cancer cell lines in which RAB25 is pro-oncogenic with a

197 ive CBP/p300 inhibitor that acts on specific cancer cell lines, in particular malignant melanoma, bre

198 0.34 +/- 0.21 to 3.54 +/- 0.54 microM in 10 cancer cell lines including KBV200 cells with P-gp overe

199 pounds (9b-9n, 10) were evaluated on various cancer cell lines including, MCF-7 breast, HL-60 leukemi

200 e against a panel of hard-to-kill epithelial cancer cell lines (including triple-negative breast) and

201 compounds that are effective against certain cancer cell lines, including a drug-resistant line.

202 y validated the hybrid E/M status of certain cancer cell lines, including DU145 and A549.

203 In breast cancer cell lines, increased levels of centrosome amplif

204 cancer and genome-wide studies in a prostate cancer cell line indicate that ETV4 and MED25 occupy enh

205 sis with data from the Cell Miner Tool in 60 cancer cell lines indicated that although rapamycin acti

206 The utility of cancer cell lines is affected by the similarity to endog

207 ogues via LAT1 mediated transport in several cancer cell lines is reported.

208 Chemotaxis assay revealed that bladder cancer cell line J82 induced MDSC migration via CXCL2/MI

209 The same relationship is observed in a human cancer cell line (K562), and we postulate that four-gene

210 Ectopic expression of Snail in cancer cell lines lacking Snail profoundly represses 4E-

211 troduction of PTEN into a PTEN null prostate cancer cell line leads to dephosphorylation of PY342 but

212 n the current study, we use a human prostate cancer cell line, LNCaP as a model to perform whole huma

213 get as well as proliferation in the prostate cancer cell line, LNCaP.

214 tivation in an RB-intact luminal-type breast cancer cell line MCF-7 promoted a positive feed forward

215 ent-dependent cytotoxicity to GM2-expressing cancer cell line MCF-7.

216 ell as detection from real samples of breast cancer cell line, MCF-7.

217 ld based 3D-QSAR model based on human breast cancer cell line MCF7 in vitro anticancer activity, whic

218 To do this, we used the breast cancer cell line MCF7, which expresses a functional estr

219 cell line MDA-MB-231 and the luminal breast cancer cell line MCF7: a) a 3D collagen embedded multice

220 re potent at inhibiting the growth of breast cancer cell lines (MCF7, MCF7/VP16, BT474, T47D, ZR-75-1

221 al cell line panel to now include the breast cancer cell lines (MCF7, MCF7/VP16, BT474, T47D, ZR-75-1

222 s employing the triple-negative basal breast cancer cell line MDA-MB-231 and the luminal breast cance

223 the migration and invasiveness of the breast cancer cell line MDA-MB-231.

224 For in vivo studies, a human breast cancer cell line (MDA-231) was implanted in five mice pe

225 reened out as the most potent one on gastric cancer cell line(MGC803) through the investigation for t

226 By continuously exposing the gastric cancer cell line MKN45 to 5-FU for >100 passages, we est

227 Utilizing mouse embryonic fibroblast and cancer cell line models, here we show via ChIP-seq and b

228 TFs associated to mutant PIK3CA in isogenic cancer cell line models.

229 IHSF115 is cytotoxic for a variety of human cancer cell lines, multiple myeloma lines consistently e

230 ited the growth of multiple RAS-mutant human cancer cell lines of diverse tissue origin by blockade o

231 from a primary tumour-derived human prostate cancer cell line (OPCT-1), and its use to explore relati

232 has no effect on the fitness of basal breast cancer cell lines or cell lines from six other cancer ty

233 ds compared to monolayer cultures of ovarian cancer cell lines or primary cells.

234 ns missed by other algorithms in the ovarian cancer cell line OVCAR3.

235 Following infection of a human ovarian cancer cell line (OVCAR3) with a recombinant low-pathoge

236 th and DNA damage was studied in two ovarian cancer cell lines (OVCAR3 and A2780), normal hamster ova

237 Experiments involving a human cancer cell line panel and mouse xenografts revealed tha

238 armacology and genomic studies is the NCI-60 cancer cell line panel, which provides data for the larg

239 differences between the aggressive prostate cancer cell line PC3 and the even more aggressive, metas

240 spurs synergistic Golgi dispersal in several cancer cell lines, pinpointing a possible link between d

241 ducted in a cohort of low-passage pancreatic cancer cell lines, primary patient-derived xenografts, a

242 attenuated nicotine-induced BEAS2B and lung cancer cell line proliferation.

243 nhibitory effects on both PACE4 and prostate cancer cell lines proliferation.

244 d with mice bearing LNCaP and PC-3 (prostate cancer cell line; PSMA-negative) tumors.

245 ar accumulation of the PRMT5/WDR77 in breast cancer cell lines relative to immortalized breast epithe

246 using ChIP-seq) of 11 different human breast cancer cell lines representing five major molecular subt

247 HSulf-1 in OV202 and TOV2223 cells (ovarian cancer cell lines) resulted in increased lipid droplets

248 3 established and 12 patient derived ovarian cancer cell lines revealed significant bioenergetics div

249 n human aldosterone-producing adrenocortical cancer cell lines, roxithromycin inhibited KCNJ5MUT-indu

250 A cancer cell-line screen revealed that B-cell lines are p

251 Evaluation of 13-d in a panel of cancer cell lines showed a selective inhibition of proli

252 Biological investigation of 2-5 on human cancer cell lines showed enhancement of antiproliferativ

253 Finally, biological assays on specific cancer cell lines showed that 19 can impair CXCL12-media

254 A validation study on 21 breast cancer cell lines showed that our prediction agrees with

255 Su demonstrated tumor uptake in all 3 breast cancer cell lines studied.

256 s to suppress let-7 target genes in multiple cancer cell lines such as HMGA2 and MYC.

257 neration of tumorspheres in Notch expressing cancer cell lines, suggesting its potential as a therape

258 d mitogenic signaling in non-small cell lung cancer cell lines, suggesting that targeting TACC3 has p

259 of 19 ovarian cancer (OC) and 8 of 14 breast cancer cell lines tested.

260 sting of four defined derivatives of a colon cancer cell line that resulted from consecutive epitheli

261 gnificantly by UbV.7.2 expression in a colon cancer cell line that was treated with the chemotherapy

262 her with recently reported evidence in human cancer cell lines that ETAA1 activates ATR kinase throug

263 ate (PC3) and breast (MDA-MB-231 and BT-549) cancer cell lines the mTOR inhibitor RAD001 (everolimus)

264 In Ras mutant pancreatic cancer cell lines, the phosphorylation and dimerization

265 contrast to all other HPV-positive cervical cancer cell lines, they harbored a gain-of-function muta

266 nalyzed the proteomes of 10 human pancreatic cancer cell lines to a depth of >8,700 quantified protei

267 of additional EGFR-dependent HNSCC and lung cancer cell lines to EGFR blockade, they are unable to c

268 s by direct and indirect assays in 12 breast cancer cell lines to estimate the spontaneous occurrence

269 individual amino acids from human colorectal cancer cell lines to investigate isotope discrimination

270 onse of a set of colon, breast, and leukemia cancer cell lines to small-molecule inhibitors against D

271 genome-scale shRNA viability screen in human cancer cell lines to systematically identify genes that

272 intracellular reactive oxygen species of two cancer cell lines to the H2O2-containing environments mi

273 tigated the sensitivity of a panel of breast cancer cell lines to treatment with various types of HER

274 nsive gene expression analysis in ER+ breast cancer cell lines, to reveal the targets of miR-500a-5p

275 enetic modulation of PPARD in human prostate cancer cell lines validated the tumor suppressive activi

276 ukemia Jurkat cells compared with a panel of cancer cell lines via inducing caspase-dependent apoptos

277 mutation-rewired signaling networks in 1,001 cancer cell lines via KNMPx.

278 athway in individual cells from a colorectal cancer cell line was determined by applying a Bayesian c

279 e folate receptor (FOLR1) overexpressed in a cancer cell line was measured by flow cytometry using a

280 5 and HTC116) and ovarian (OVCAR3 and SKOV3) cancer cell lines was profiled with non-targeted metabol

281 effect on FSTL1, normal cell BEAS2B and lung cancer cell lines was treated with nicotine and the resu

282 ffect on Caco-2, HeLa (cancer) and MDCK (non-cancer) cell lines was established.

283 By profiling the response of over 120 human cancer cell lines, we derived an expression-based algori

284 ng a series of experiments in human prostate cancer cell lines, we validate the highest ranking predi

285 ingle nuclei to 424 single cells in a breast cancer cell line, which shows a high concordance (93.34%

286 our findings in MCF-7 and MDA-MB-231 breast cancer cell lines, which harbor lower hsa-miR-125b level

287 7 expression in androgen-responsive prostate cancer cell lines, which was accompanied by enhanced epi

288 emcitabine for growth inhibition of multiple cancer cell lines, while demonstrating little cytotoxici

289 ukemia cell line as well as the MCF-7 breast cancer cell line with chromatin interactions.

290 ise gene targeting to produce isogenic colon cancer cell lines with a knockout/rescue system for GALN

291 ly measure glutamine-derived ammonia in lung cancer cell lines with differential expression of glutam

292 s in BXPC-3 and PANC-1 cells, two pancreatic cancer cell lines with high and low TF expression levels

293 Similarly, human colorectal cancer cell lines with increased KRAS mutant allele freq

294 vels including cell surface DR5 in different cancer cell lines with limited or no effects on the leve

295 use intestinal epithelia and adenomas, human cancer cell lines with or without drug treatments, and h

296 of HOTAIR-overexpressing ovarian and breast cancer cell lines with PNAs decreased invasion and incre

297 16790A reduced viability of two basal breast cancer cell lines with pronounced endogenous overexpress

298 cellular metabolic profiles of four prostate cancer cell lines with varying degrees of aggressiveness

299 t significantly inhibited growth of multiple cancer cell lines without affecting normal cell growth a

300 udy genome-wide chromatin structure in human cancer cell lines, yet numerous technical challenges lim