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1 plays a critical role in cellular stress and cancer prevention.
2 2 has been suggested to offer a strategy for cancer prevention.
3 yanins in P40 may provide extra benefits for cancer prevention.
4 re and perhaps extraordinary opportunity for cancer prevention.
5 ion with direct therapeutic implications for cancer prevention.
6 to determine the optimal use of aspirin for cancer prevention.
7 benefit-to-harm ratio of the drug for breast cancer prevention.
8 iruses (gammaHVs) is an important target for cancer prevention.
9 hallenge and an enormous opportunity in skin cancer prevention.
10 or our understanding of cancer causation and cancer prevention.
11 al and human intervention studies on tea and cancer prevention.
12 d to development of anticancer therapies and cancer prevention.
13 to assess the possible role of vitamin D in cancer prevention.
14 atase inhibitors to antiestrogens for breast cancer prevention.
15 ave transformed the practice of oncology and cancer prevention.
16 ould be an important strategy for pancreatic cancer prevention.
17 edback loop may be an important strategy for cancer prevention.
18 therapy may offer some therapeutic value in cancer prevention.
19 aped curve for micronutrients in relation to cancer prevention.
20 reening tests that offer the opportunity for cancer prevention.
21 types have the potential to improve cervical cancer prevention.
22 rly, sustained ART is a crucial component of cancer prevention.
23 odel of breast cancer, with implications for cancer prevention.
24 entially can play an important role in colon cancer prevention.
25 ing such alterations, therefore, may lead to cancer prevention.
26 and provides novel insights into its role in cancer prevention.
27 signaling pathway may be important in breast cancer prevention.
28 is kinase is a relevant strategy for mammary cancer prevention.
29 ess costly, are keys to continued success in cancer prevention.
30 nable to pharmacologic manipulation for skin cancer prevention.
31 re to alcohol has important implications for cancer prevention.
32 Sun-protective behavior affects skin cancer prevention.
33 functional foods or nutraceuticals for colon cancer prevention.
34 itamin D supplementation is useful in breast cancer prevention.
35 umor initiation, which is highly relevant to cancer prevention.
36 sed on nfGNPs for HPV detection and cervical cancer prevention.
37 ategy, namely, reverse-engineering precision cancer prevention.
38 r guiding public health strategies of breast cancer prevention.
39 that arise, and novel strategies for ovarian cancer prevention.
40 role of dietary selenium (Se) in colorectal cancer prevention.
41 issues from various types of insults and for cancer prevention.
42 g both YAP and AKT in liver size control and cancer prevention.
43 potential to save millions of lives through cancer prevention.
44 garding all things cancer related, including cancer prevention.
45 rivate practice would have a career focus in cancer prevention.
46 crobiome in breast carcinogenesis and breast cancer prevention.
47 mplementation of policies to improve primary cancer prevention.
52 Despite the huge potential importance of cancer prevention and clinical success stories such as t
53 r risk, and evaluate the potential impact of cancer prevention and control strategies on cancer rates
56 ly important questions that are critical for cancer prevention and early detection in women carrying
62 dence-informed frameworks for cost-effective cancer prevention and management are essential for deliv
63 ouraging exercise fitness in the clinics for cancer prevention and may promote the development of new
64 a possible molecular basis for zinc-induced cancer prevention and Orai1-SOCE signaling pathway in ca
72 gnize the potential of obesity management in cancer prevention and that excess body weight in women i
75 ves and analogues and their potential use in cancer prevention and therapy, which are related to thei
88 ning frequency and behaviors related to skin cancer prevention and to investigate whether these assoc
89 ning frequency and behaviors related to skin cancer prevention and to investigate whether these assoc
90 nvestigating how ASCO might favorably affect cancer prevention and treatment in resource-poor countri
91 3 into wild-type p53 have been suggested for cancer prevention and treatment, but they face a variety
93 een metformin use and a beneficial effect on cancer prevention and treatment, which has led to increa
113 omized trials demonstrating effectiveness in cancer prevention and widely recommended by guidelines f
114 rtance of sun protection and facilitate skin cancer prevention and, therefore, decrease the skin canc
118 ffects of anti-oestrogen treatment in breast cancer prevention, and suggest that fibroblast growth fa
119 n to cancer risk, discusses implications for cancer prevention, and suggests future research directio
120 t few years, with improvements in screening, cancer prevention, and treatment, and is best defined fo
121 owever, data on the risk-benefit profile for cancer prevention are insufficient and no definitive rec
124 indicated they would likely be interested in cancer prevention as a career focus, although only 12% t
125 of men: the Alpha-Tocopherol, Beta-Carotene Cancer Prevention (ATBC) Study and the Prostate, Lung, C
129 have drawn a great deal of attention toward cancer prevention because of their wide safety margin.
131 netically engineered mouse models but weaker cancer prevention benefit in chemically induced rat mode
132 ty would have been an effective strategy for cancer prevention, but the reality is that worldwide obe
133 health information, including advice on skin cancer prevention, but their effectiveness may be affect
134 ajor role in regulating genome stability and cancer prevention by controlling the key proteins involv
135 ty reduction with early cancer detection and cancer prevention by detection and removal of cancer pre
136 ns unclear whether MSA exerts its effects on cancer prevention by influencing angiogenesis within Se
137 ogen detoxification-independent mechanism of cancer prevention by ITCs has been attributed at least i
138 se findings suggest opportunities for breast cancer prevention by modifying individual estrogen metab
141 gy will undoubtedly continue contributing to cancer prevention by using traditional epidemiologic stu
144 al trials of supplemental micronutrients for cancer prevention completed over the past 20 years, incl
146 burden of cancer, societal attitudes towards cancer prevention, effects of inequitable treatment and
147 ctal cancer, at least in men, and colorectal cancer prevention efforts should encourage the achieveme
149 herence to the American Cancer Society (ACS) cancer prevention guidelines is associated with a reduct
151 ons, the mechanisms linking COX blockade and cancer prevention have long been an area of active inves
152 ials that have led to licensure for cervical cancer prevention have used the disease endpoint of cerv
153 abstracted from the American Cancer Society Cancer Prevention II Study cohort, with baseline ascerta
154 immune systems are considered important for cancer prevention, immunosurveillance, and control of ca
155 bservation that NSAIDs are most effective in cancer prevention in APC(min/+) mice if the mice are tre
158 formin, a first-line diabetes drug linked to cancer prevention in retrospective clinical analyses, in
159 ld therefore be a strong candidate for liver cancer prevention in the context of aberrant Smad3 signa
164 tobacco smoke-constituent biomarkers in lung cancer prevention, including improved lung cancer risk a
165 es of nutrients often suggested for prostate cancer prevention, including lycopene, long-chain n-3 fa
166 d from 51 hospitals into the Ohio Colorectal Cancer Prevention Initiative from January 1, 2013, to Ju
173 vidence comparing ICR to CCR with respect to cancer prevention is controversial and inconclusive.
180 herence to the WCRF/AICR recommendations for cancer prevention may lower the risk of developing most
182 tion to tamoxifen and raloxifene as a breast cancer prevention medication, although exemestane is not
183 t cancer of 1.66% or greater, discuss breast cancer prevention medications with their primary care pr
184 d provide incentive for reducing weight as a cancer prevention method in overweight and obese individ
185 pants were recruited through the multicenter Cancer Prevention Network and randomly assigned to group
187 sta Family Foundation for Research in Breast Cancer Prevention of the California Pacific Medical Cent
188 physical activity, and weight management for cancer prevention on the basis of the most comprehensive
189 physical activity, and weight management for cancer prevention on the basis of the most comprehensive
190 atase inhibitors to antiestrogens for breast cancer prevention.Oncogene advance online publication, 2
192 sent a novel therapeutic approach for breast cancer prevention or treatment based on its pharmacologi
193 dulation of selective sirtuin members to aid cancer prevention or treatment under defined conditions.
196 cal Adjuvant Breast and Bowel Project Breast Cancer Prevention P-1), MA.17, and BIG 1-98 (Breast Inte
198 ic evaluation at a time when global cervical cancer prevention policy continues to evolve and evidenc
199 tudy uncover a potential target for cervical cancer prevention, provide insight into the risk assessm
200 tudy was to examine whether adherence to the cancer prevention recommendations of the World Cancer Re
208 with the potential to accelerate advances in cancer prevention, screening, and early detection across
210 ies, such as anti-oestrogen drugs for breast cancer prevention, should be targeted to high-risk group
211 explore known and potential various types of cancer prevention strategies and focus on nonvaccine-bas
212 dominally obese individuals may be important cancer prevention strategies as well as adjuvant therapi
218 y mortality from the American Cancer Society Cancer Prevention Study II (ACS CPS-II) cohort have been
219 residence of 668,629 American Cancer Society Cancer Prevention Study II (CPS-II) cohort participants
220 te cancer mortality among 363,726 men in the Cancer Prevention Study II (CPS-II) cohort, of whom 7,45
221 249 postmenopausal, cancer-free women in the Cancer Prevention Study II (CPS-II) Nutrition Cohort, en
222 cer among 43,512 men and 56,011 women in the Cancer Prevention Study II (CPS-II) Nutrition Cohort; 10
223 centrations and lung cancer mortality in the Cancer Prevention Study II (CPS-II), a large prospective
225 articipants from the American Cancer Society Cancer Prevention Study II cohort were linked to modeled
226 bestos-exposed blue collar male workers from Cancer Prevention Study II for whom occupational and smo
228 ofessionals Follow-up Study (1986-2004), the Cancer Prevention Study II Nutrition Cohort (1992-2004),
229 ort studies: (1) the American Cancer Society Cancer Prevention Study II Nutrition Cohort and (2) the
231 cipants (60,059 men and 73,196 women) in the Cancer Prevention Study II Nutrition Cohort during the p
232 ned among 55,983 men and 66,655 women in the Cancer Prevention Study II Nutrition Cohort from 1992 to
233 62,792 men and 69,520 women enrolled in the Cancer Prevention Study II Nutrition Cohort in 1992, we
235 ta-Carotene Cancer Prevention Study, and the Cancer Prevention Study II Nutrition Cohort to investiga
236 ake with risk of NHL and NHL subtypes in the Cancer Prevention Study II Nutrition Cohort, a prospecti
237 es of Health-AARP Diet and Health Study, the Cancer Prevention Study II Nutrition Cohort, the Health
238 n Retired Persons Diet and Health Study, the Cancer Prevention Study II Nutrition Cohort, the Multiet
243 study cohort of the American Cancer Society Cancer Prevention Study II were correlated with air-poll
244 ditive scale between these risk factors from Cancer Prevention Study II, a large prospective US cohor
245 ake with fatal oral/pharyngeal cancer in the Cancer Prevention Study II, a prospective US cohort stud
249 e within the Alpha-Tocopherol, Beta-Carotene Cancer Prevention Study, a cohort study of Finnish male
250 Within the Alpha-Tocopherol, Beta-Carotene Cancer Prevention Study, a randomized controlled trial c
251 g Trial, the Alpha-Tocopherol, Beta-Carotene Cancer Prevention Study, and the Cancer Prevention Study
252 and women from 23 states who enrolled in the Cancer Prevention Study-3 cohort during 2006-2009 and ha
253 (random sub-cohort N=1,685) from the Korean Cancer Prevention Study-II (KCPS-II) (N=145,842) was use
256 r rectal cancer between 1992 and 2007 in the Cancer Prevention Study-II Nutrition Cohort, a prospecti
259 s been recognized as an important target for cancer prevention, the mechanism by which energy restric
260 ce that prebiotics play a role in colorectal cancer prevention, their role of satiety and weight mana
261 vital role in normal homeostatic control and cancer prevention, they also strongly encourage the emer
263 regulation of the pathway might be useful in cancer prevention, treatment, and regenerative medicine
267 o participated in the Selenium and Vitamin E Cancer Prevention Trial (2001-2011) with present analyse
271 tive randomized trials, such as the Prostate Cancer Prevention Trial (PCPT), Reduction by Dutasteride
274 initial report of the Selenium and Vitamin E Cancer Prevention Trial (SELECT) found no reduction in r
275 from the large-scale Selenium and Vitamin E Cancer Prevention Trial (SELECT) showed that antioxidant
276 te cancer risk in the Selenium and Vitamin E Cancer Prevention Trial (SELECT) through unknown mechani
277 was ancillary to the Selenium and Vitamin E Cancer Prevention Trial (SELECT), a randomized clinical
278 s (REDUCE) trial, and Selenium and Vitamin E Cancer Prevention Trial (SELECT), have provided practiti
279 rostate cancer in the Selenium and Vitamin E Cancer Prevention Trial (SELECT), plays an integral role
280 cer among 9,559 participants in the Prostate Cancer Prevention Trial (United States and Canada, 1994-
281 prostate cancer prevention trials (Prostate Cancer Prevention Trial [PCPT] and Selenium and Vitamin
282 tion Trial [PCPT] and Selenium and Vitamin E Cancer Prevention Trial [SELECT]) were examined to defin
283 R (Study of Tamoxifen and Raloxifene) breast cancer prevention trial and in other clinical trials.
286 IENTS AND METHODS Raw data from the Prostate Cancer Prevention Trial were used to model chemopreventi
287 carriers leading to an international breast cancer prevention trial, and insights into the intricate
290 d, double-blind, placebo-controlled, primary cancer prevention trial; participants were Finnish male
291 from the placebo arms of two large prostate cancer prevention trials (Prostate Cancer Prevention Tri
292 large, population-based, Phase III prostate cancer prevention trials have shown a significant benefi
293 nly used to determine eligibility for breast cancer prevention trials was greatly increased relative
294 sion Among screened men enrolled in prostate cancer prevention trials, differences in risk factor est
295 ys essential roles in organ size control and cancer prevention via restricting its downstream effecto
298 ns and Relevance: Important barriers to skin cancer prevention were lack of knowledge, the belief tha
299 plant-based foods seem promising for stomach cancer prevention, while vitamin C lowers the risk of es
300 t to develop a consensus statement on breast cancer prevention, with a focus on medical and therapeut
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