ライフサイエンスコーパス: cancer tissue

1 es revealed that Myo9b is upregulated in the cancer tissue.

2 gion-specific lipid signatures in colorectal cancer tissue.

3 ys are frequently found to be coactivated in cancer tissue.

4 tagged biomarker in a thin section of breast cancer tissue.

5 e as surrogate markers of DNA methylation in cancer tissue.

6 omplexity to the aberrant epigenome found in cancer tissue.

7 racemase, which is overexpressed in prostate cancer tissue.

8 ncer tissue array, we confirmed elevation in cancer tissue.

9 roximates a mitotic clock in both normal and cancer tissue.

10 es with an expression-based mitotic index in cancer tissue.

11 d kinase 4 (PAK4) is amplified in pancreatic cancer tissue.

12 ptor EP2 was overexpressed in human prostate cancer tissue.

13 how Th17 cells are recruited to the cervical cancer tissue.

14 with beta-catenin and PKM2 levels in breast cancer tissues.

15 EG3 expression has been reported in multiple cancer tissues.

16 ular location annotations for bioimages from cancer tissues.

17 te for detection of MGA expression in breast cancer tissues.

18 levels of matriptase activation in prostate cancer tissues.

19 ession of Siah2 and AKR1C3 in human prostate cancer tissues.

20 nes were further validated in human prostate cancer tissues.

21 anscript in three of 122 primary endometrial cancer tissues.

22 lectively and significantly higher in breast cancer tissues.

23 up-regulated and co-localized in human renal cancer tissues.

24 nsistent dysregulated expression in multiple cancer tissues.

25 was then applied to MALDI-IMS of human lung cancer tissues.

26 llular carcinoma, breast cancer, and ovarian cancer tissues.

27 ventional LAMP and nested PCR in 50 cervical cancer tissues.

28 ive gastric cancer cell model and in gastric cancer tissues.

29 iated with the nucleus of malignant prostate cancer tissues.

30 n can distinguish between normal and bladder cancer tissues.

31 highly expressed in human epithelial ovarian cancer tissues.

32 reduced in breast cancer cells and clinical cancer tissues.

33 wth, even though it was expressed by ovarian cancer tissues.

34 st cancer cell lines as well as human breast cancer tissues.

35 nature" that is distinct from primary breast cancer tissues.

36 ection of DNA samples taken from oesophageal cancer tissues.

37 posed to ambient temperatures, as well as in cancer tissues.

38 n of pVHL and accumulation of KLF4 in breast cancer tissues.

39 ir protein and mRNA expression in normal and cancer tissues.

40 erated from cancer cell lines and from human cancer tissues.

41 d prostate cell lines as well as in clinical cancer tissues.

42 evated pVHL and reduced KLF4 levels in colon cancer tissues.

43 ely correlated with CD8(+) T cells in breast cancer tissues.

44 n the cytoplasm in prostate premalignant and cancer tissues.

45 pecifically over-expressed in human prostate cancer tissues.

46 as or loss of p53, and in primary pancreatic cancer tissues.

47 42-5p and DOHH in a cohort of human prostate cancer tissues.

48 LATS2 expression in microdissected prostate cancer tissues.

49 nostaining analysis in IM, SPEM, and gastric cancer tissues.

50 nd PTPH1 protein expression in primary colon cancer tissues.

51 RIL are found at elevated levels in prostate cancer tissues.

52 mprehensive view of the molecular changes in cancer tissues.

53 CDH17 and MUC13 was up-regulated in gastric cancer tissues.

54 r absolute quantitation of AKT peptides from cancer tissues.

55 to efficiently differentiate cancer from non-cancer tissues.

56 st prevalent bacterial species in colorectal cancer tissues.

57 elated with Sox2 expression in human ovarian cancer tissues.

58 res regarding the intrinsic heterogeneity of cancer tissues.

59 expression in stromal cells in human gastric cancer tissues.

60 man metastatic castration-resistant prostate cancer tissues.

61 significantly overexpressed in human breast cancer tissues.

62 ased levels of both KLF4 and PRMT5 in breast cancer tissues.

63 l landscape of BRAF and other drivers across cancer tissues.

64 In contrast, in matched cancer tissue, 5' tRH abundance was reduced, and relativ

65 plicates and expresses transgene products in cancer tissue after intravenous infusion, in a dose-rela

66 ted in large-scale human genetic studies and cancer tissues, all of which occur close to two regulato

67 sequential Ga(3+)-Fe(3+)-IMAC to human lung cancer tissue allowed the identification of 2560 unique

68 cally) was significantly decreased in breast cancer tissues (although the transcript levels were comp

69 at IQGAP1 is overexpressed in HER2(+) breast cancer tissue and binds directly to HER2.

70 f RORalpha was downregulated in human breast cancer tissue and cell lines, and that reduced mRNA leve

71 ession appears to be down-regulated in liver cancer tissue and cell lines.

72 virus, has been isolated from human prostate cancer tissue and from activated CD4(+) T cells and B ce

73 obtain chemical images of cross sections of cancer tissue and hair samples sandwiched between infrar

74 ant correlation with Met expression in colon cancer tissue and is highly prognostic for occurrence of

75 w elevated La protein expression in cervical cancer tissue and its correlation with aberrant CCND1 pr

76 have determined its distribution in prostate cancer tissue and its role in prostate tumorigenesis usi

77 al studies of BRCA1 protein in frozen breast cancer tissue and MCF7 and HeLa cell lines revealed BRCA

78 In total, 20 cases of primary breast cancer tissue and resected BrM (10 estrogen receptor [ER

79 hort harbored patient-matched primary breast cancer tissue and resected BrM.

80 This was clinically tested in prostate cancer tissue and shown that cytoplasmic PML and CRM1 co

81 nalase expression is increased in pancreatic cancer tissue and that it functions as a growth factor.

82 st highly suppressed gene in both colorectal cancer tissue and the L1-NF-kappaB pathway.

83 st perturbed gene expression levels in human cancer tissue and the most perturbed expression levels i

84 her characterize metabolic changes in breast cancer tissue and the tumor microenvironment.

85 XMRV) was first identified in human prostate cancer tissue and was later found in a high percentage o

86 st abundantly expressed galectin in prostate cancer tissue and was markedly upregulated during diseas

87 equence in the TGFBR2 gene of 32 MSI-H colon cancer tissues and 6 cell lines (HCT116, LS180, LS411N,

88 Utilizing resected human lung cancer tissues and a p21CIP1/WAF1-deficient, K-rasG12D-e

89 tein expression are down-regulated in breast cancer tissues and cell lines compared with adjacent nor

90 Studies of both human liver cancer tissues and cell lines uncovered that DDD cleavag

91 alyzed proteomic and pharmacogenomic data in cancer tissues and cell lines using a global statistical

92 expression was reduced in most human breast cancer tissues and cell lines.

93 dicted E-cadherin-targeting miRNAs in breast cancer tissues and cells showed that miR-221 was abundan

94 e cancer (CRPC) cell lines, primary prostate cancer tissues and circulating tumor cells (CTCs) to inv

95 hat PPARalpha is overexpressed in pancreatic cancer tissues and clofibrate-mediated PPARalpha activat

96 ymerase chain reaction in 915 primary breast cancer tissues and correlated with known clinicopatholog

97 rrelated with TRIB2 expression in both liver cancer tissues and established liver cancer cell lines.

98 oblastoma cell line U87MG and primary breast cancer tissues and found that 26-45% of all genes with s

99 of CagA on HER-2 expression in human gastric cancer tissues and gastric mucosal tissues of H. pylori

100 signal was increased significantly in breast cancer tissues and highly correlated with ex vivo expres

101 ncer, demonstrable with primary human breast cancer tissues and human breast cancer cell lines.

102 etection of KRAS G12V mutation in colorectal cancer tissues and is presented herein.

103 that PIPKIgamma is highly expressed in lung cancer tissues and its expression level is critical for

104 e tissue, was reduced in both human prostate cancer tissues and LNCaP-B cells.

105 C5 was significantly hypermethylated in lung cancer tissues and lung cancer cell lines compared with

106 tient survival in examinations of colorectal cancer tissues and paired normal colorectal mucosa tissu

107 , POSTN was highly enriched in the stroma of cancer tissues and produced mainly by CAFs.

108 IGF-IR is overexpressed in invasive bladder cancer tissues and promotes motility and invasion of uro

109 ha in breast cancer cell lines, human breast cancer tissues and Runx3(+/-) mouse mammary tumors.

110 tial markers for gastric cancer in patients' cancer tissues and sera based on: (i) genome-scale trans

111 In lung cancer tissues and single cells, p66(Shc) expression inv

112 NAc-T3, is overexpressed in human pancreatic cancer tissues and suppression of GalNAc-T3 significantl

113 We performed RNA-Seq on T1 and T2 bladder cancer tissues and used publicly available bladder cance

114 These cells were found in prostate cancer tissues and were related to tumor aggressiveness

115 fy epialleles that differ between normal and cancer tissue, and define a measure of global epigenetic

116 l for identifying clusters of cancer and non-cancer tissue, and identifying a cancer CpG island hyper

117 Extracellular adenosine is elevated in cancer tissue, and it negatively regulates local immune

118 rmal colorectal tissue is subtle compared to cancer tissue, and the simultaneous FP/HW Raman endoscop

119 ion was detected in 17 (56.7%) of 30 gastric cancer tissues, and eight (47%) of them showed HER-2 DNA

120 was decreased in the majority of human liver cancer tissues, and its reduced expression was significa

121 n both cancer cell lines and advanced breast cancer tissues, and the levels of TRIM28 and TWIST1 are

122 -B is markedly down-regulated in human renal cancer tissues; and the overexpression of CXCR3-B in ren

123 RT-qPCR of FPE colon cancer tissue applied to four large independent populati

124 ne expression differences between normal and cancer tissue are anticorrelated with local network entr

125 Increases in stem cell population in breast cancer tissues are associated with tumor recurrence and

126 wed that higher levels of sGCbeta1 in breast cancer tissues are correlated with greater survival prob

127 of cancer, but iron regulatory mechanisms in cancer tissues are largely unknown.

128 carcinoma (NSCLC), especially in those whose cancer tissues are unavailable or in those that have acq

129 immunohistochemical analysis of 162 prostate cancer tissue array samples representing a range of Glea

130 Using a human breast cancer tissue array, we confirmed elevation in cancer ti

131 In human prostate cancer tissue arrays, Gal-1 expression correlated with t

132 f amplified FRET to determine pAkt status in cancer tissues as candidate biomarker for the identifica

133 vered that Pyk2 was overexpressed in bladder cancer tissues as compared to normal tissue controls.

134 p54(nrb)/Nono is highly expressed in breast cancer tissues as compared with the adjacent normal tiss

135 TS but negatively with YAP/TAZ in pancreatic cancer tissues as well as pancreatic and breast cancer c

136 tantly, CDK20 is overexpressed in human lung cancer tissues, as determined by immunostaining.

137 Assessment of key breast cancer tissue biomarkers is often done using nonquantita

138 elastase was expressed by TAN within breast cancer tissues but not by breast cancer cells.

139 protein in skin, normal cervix, and cervical cancer tissues, but not in larynx.

140 ch regions are frequently hypermethylated in cancer tissues, but not methylated in normal tissues.

141 oxin (Prx) enzymes are overexpressed in most cancer tissues, but their specific signaling role in can

142 hrome P450 1B1 (CYP1B1) promoters of bladder cancer tissues by bisulfite sequence analysis and methyl

143 We assessed Merlin expression in breast cancer tissues by immunohistochemistry and by real-time

144 in human chronic pancreatitis and pancreatic cancer tissues, Cad-11 expression was significantly incr

145 how variations in the cellular properties of cancer tissue can influence cancer biomarker ion images.

146 are colocalized in plasma membrane of breast cancer tissue cells and breast cancer cell lines BT474 a

147 ture GPI-anchored proteins from human breast cancer tissues, cells, and serum for proteomic analysis.

148 have been charted between normal and breast cancer tissues, changes in higher-order chromatin organi

149 and CYSLTR2 gene expression was decreased in cancer tissue compared to control at 0.26-fold and 0.23-

150 iR-183, -96, and -182 are higher in prostate cancer tissue compared with normal prostate.

151 rotein levels are decreased in human gastric cancer tissue compared with surrounding normal gastric t

152 ere highly overexpressed in about 36% of 136 cancer tissues compared to adjacent tissues, in which FE

153 mRNA isoform, SIGIRR(DeltaE8), in colorectal cancer tissues compared to paired nontumor tissues.

154 ave significantly lower expression in breast cancer tissues compared to paired normal breast tissue.

155 is upregulated in tamoxifen-resistant breast cancer tissues compared to their primary counterparts.

156 els were significantly downregulated in lung cancer tissues compared with adjacent non-cancerous tiss

157 promoter is highly methylated in pancreatic cancer tissues compared with non-cancerous tissues.

158 cell carcinoma, this score was decreased in cancer tissues compared with normal mucosa, validating t

159 We show that human colon cancer tissues contain distinct cell populations whose t

160 Cancer tissue contained significantly increased levels o

161 macrophage-associated signatures from large cancer tissue datasets.

162 Immunoperoxidase staining of bladder cancer tissues demonstrated that (1) PGDH is highly expr

163 Remarkably, pDCs isolated from human colon cancer tissues displayed a strong surface expression of

164 Both benign and cancer tissues displayed characteristic histology and ex

165 e rapid intraoperative detection of residual cancer tissue during breast-conserving surgery.

166 in real time for accurate identification of cancer tissues, especially these without any obvious str

167 cally classify sample regions in human renal cancer tissue ex-vivo into tumor or benign tissue based

168 sue Bank (BCCTB), a vital resource of breast cancer tissue for researchers to support and promote cut

169 r and pSmad2 immunohistochemically in breast cancer tissue from 1,045 patients in the Shanghai Breast

170 Here, we show that in cancer tissue from primary GIST patients as well as in c

171 of CD44v6 messenger RNA (mRNA) in pancreatic cancer tissues from 136 patients.

172 was performed on adjacent normal and breast cancer tissues from 96 premenopausal women with known cl

173 and overexpression of BCL-2 were observed in cancer tissues from metabolic organs.

174 munohistochemical staining in primary breast cancer tissues from patients with different stages of ca

175 s in APC(min/+) mice and in human colorectal cancer tissues, functioned redundantly to promote the Wn

176 n isoforms are preferentially upregulated in cancer tissues, genome-wide.

177 Because up-regulation of Siglec ligands in cancer tissue has been observed, the characterization of

178 lf-renewal and tumor growth in heterogeneous cancer tissues, has stimulated interests in developing n

179 However, most DW-MRI studies of cancer tissues have relied on simplistic mathematical mo

180 A genomic analysis of colorectal cancer tissues identified a somatic mutation (P301S) in

181 , the detection and localization of prostate cancer tissue in patients with primary or recurrent dise

182 nstrated a high prediction power to diagnose cancer tissue in real time during routine surgical proce

183 e unrestricted multiple 'omics data for each cancer tissue in The Cancer Genome Atlas as ready-to-ana

184 R and PTEN transcript expression in prostate cancer tissues in contrast to the positive correlation i

185 um (F.) nucleatum was abundant in colorectal cancer tissues in patients with recurrence post chemothe

186 maging and therapeutic agents selectively to cancer tissues in vivo.

187 ine of copy number analysis of heterogeneous cancer tissues including relevant processing steps.

188 alysis of AR binding sites in human prostate cancer tissues, including castrate-resistant prostate ca

189 wn the absence of expression of POX in human cancer tissues, including renal cancer.

190 ly, immunohistochemical analysis of prostate cancer tissue indicated that nuclear localization of fat

191 mRNA compared with other genotypes in breast cancer tissues, indicating that rs17506395 may be a func

192 Modulation of immunologic interactions in cancer tissue is a promising therapeutic strategy.

193 IL-20 expression in breast cancer tissue is associated with a poor clinical outcome

194 Recently it has been observed that cancer tissue is characterised by an increased variabili

195 Expression of aromatase in breast cancer tissue is driven by different promoters than thos

196 Cancer tissue is stiffer than its normal counterpart.

197 release of potent anticancer products within cancer tissues is a promising approach in chemotherapy.

198 ing formalin-fixed, paraffin-embedded (FFPE) cancer tissues is becoming the standard for identifying

199 strate that the transcriptional diversity of cancer tissues is largely explained by in vivo multiline

200 Furthermore, PRMT5 expression in prostate cancer tissues is significantly higher than that in beni

201 Although the stroma of cancer tissues is the main source of POSTN, it is still

202 ich has been detected in almost all kinds of cancer tissues, is considered as an important tumor mark

203 One mechanism by which cancer tissues limit the host immune response is via upr

204 pression by immunohistochemistry in a breast cancer tissue microarray (n = 377) with approximately 15

205 Furthermore, analysis of a human lung cancer tissue microarray revealed a significant, positiv

206 Analysis of a comprehensive breast cancer tissue microarray revealed a tight correlation be

207 In an ovarian cancer tissue microarray, TR3 protein expression was ele

208 ncer progression, we surveyed human prostate cancer tissue microarrays and found that the RAS/MAPK pa

209 Here, using human lung cancer tissue microarrays and fresh frozen tissues, we f

210 In this study, invasive and in situ breast cancer tissue microarrays containing luminal A, luminal

211 Breast cancer tissue microarrays revealed an inverse correlatio

212 The analysis of prostate cancer tissue microarrays revealed that tumors with redu

213 Immunohistochemical analysis of prostate cancer tissue microarrays showed significant P-AR S213 e

214 sification of breast cancer cells and breast cancer tissue microarrays with this system correlated wi

215 Using data mining and human breast cancer tissue microarrays, we found that Ctr9, the key s

216 ts expression was elevated in human prostate cancer tissue microarrays.

217 pes, whereas organoids derived from prostate cancer tissue mimic the histology of the tumor.

218 8 were expressed by 17%-50% of human gastric cancer tissues (MUC13, OLFM4, CDH17, KRT20, MUC5AC, LGAL

219 The presence in cancer tissue of Ag-specific, activated tumor infiltrati

220 mmune checkpoint blockade, regardless of the cancers' tissue of origin.

221 e cancer metabolic phenotypes are defined by cancer tissue-of-origin and environment and that these f

222 oblastoma cells (U87), which formed 3D brain cancer tissues on the chip, and used the GBM chip to per

223 for additional surgeries to excise unremoved cancer tissue or alternatively in the removal of excess

224 ed to a number of effects off their intended cancer tissue or molecular targets.

225 In acidic environments, such as in cancer tissue or the subcellular endosome, BTZ dissociat

226 gical specimens confirmed that most cervical cancer tissues overexpressed STIM1 and Orai1, accompanie

227 By improving cancer tissue oxygenation and extending the normalizatio

228 that PER2 was significantly up-regulated in cancer tissues (p < 0.005).

229 g expression of altered genes in BRAF-driven cancer tissue, parallel RNA-seq was also undertaken of t

230 many protein mutations, especially in human cancer tissues, prediction of their likely functional im

231 Moreover, our findings for cancer tissues presenting concomitant cytidine deaminase

232 DNA methylation profiling performed on cancer tissues prior to chemo/radiotherapy identified on

233 s and differentiation, with implications for cancer, tissue regeneration, and inflammation.

234 s to be up-regulated in both lung and breast cancer tissues relative to normal adjacent tissue, we fo

235 19.3% and 21.8% in localized and metastatic cancer tissues, respectively (P-value < 2 x 10(-16)).

236 ession analysis in human metastatic prostate cancer tissue revealed that beta1 was markedly upregulat

237 urity', or the percentage of cancer cells in cancer tissue sample, will bias the clustering results i

238 tative histological data of human colorectal cancer tissue samples (n = 20) including tumor cells, ly

239 analysis of >27,000 CpG sites in endometrial cancer tissue samples (n = 64) and control samples (n =

240 is associated with caveolin-1 in pancreatic cancer tissue samples and cell lines, and predicts the m

241 unohistochemical staining of lung and breast cancer tissue samples demonstrated that increased S6K2 e

242 our previous findings that lung and thyroid cancer tissue samples exhibited increased Sec62 protein

243 assay with 1178 paraffin-embedded colorectal cancer tissue samples from 2 prospective cohort studies.

244 methylation are recapitulated in endometrial cancer tissue samples obtained from patients treated wit

245 our studies is our analysis of human breast cancer tissue samples that indicated association of lowe

246 itor the differences in PSA concentration in cancer tissue samples which holds great promise in clini

247 detected with the proposed immunosensor for cancer tissue samples which holds great promise in clini

248 or source of HB-EGF production in pancreatic cancer tissue samples, and that macrophages are present

249 issense mutations of Ack1 were identified in cancer tissue samples, but the effects on Ack1 activity,

250 d proteomics, which allow analysis of breast cancer tissue samples, leading to the first large-scale

251 d genes in a series of 4552 human normal and cancer tissue samples.

252 tween miR-218 levels and Slug/ZEB2 levels in cancer tissue samples.

253 8 cancer proteins was performed on a breast cancer tissue section to illustrate the potential of thi

254 Our recent data suggest that human breast cancer tissues show a redox imbalance (reducing) compare

255 A parallel analysis of colorectal cancer tissues showed differential expression of 83 miRN

256 Analysis of prostate cancer tissues showed that the presence of a TMPRSS2-ERG

257 Analyses of human breast cancer tissues showed that ZNF24 and VEGF levels were in

258 s frequently found in tumour cells and human cancer tissues showing high levels of phospho-FADD(Ser19

259 further confirmed from data in human breast cancer tissues showing that CNNM3 levels correlate posit

260 at elevated CDK11(p110) expression in breast cancer tissues significantly correlated with poor differ

261 ch by imaging Papanicolaou smears and breast cancer tissue slides over a large field-of-view of ~20 m

262 we developed a library of 76 healthy and 20 cancer tissue-specific reconstructions.

263 Examination of human prostate cancer tissue specimens shows a reduction of LZTS2 prote

264 In 95 pancreatic cancer tissue specimens, KLF10 expression was inversely

265 1 expression was evaluated in the pancreatic cancer tissue specimens.

266 e-containing peptides identified in prostate cancer tissue studies carried out in our group were dete

267 demonstrated that IL-35 is produced in human cancer tissues, such as large B cell lymphoma, nasophary

268 were significantly higher in invasive breast cancer tissues than in breast adenocarcinoma tissue.

269 are expressed, along with Chk1, in fetal and cancer tissues than in normal tissues.

270 higher expression of PPARalpha in pancreatic cancer tissues than in tumor-adjacent tissues and that t

271 lncRNA GAS5 had lower expression in stomach cancer tissues than the normal counterparts.

272 nsive metabolic phenotyping method in breast cancer tissue that uses desorption electrospray ionizati

273 interleukin-22 (IL-22) in patient colorectal cancer tissues that was produced predominantly by CD4(+)

274 activity of a dual AI/SERM, while in breast cancer tissue the antiestrogenic SERM activity of a dual

275 In human breast cancer tissues, the levels of p54(nrb) and SREBP-1a prot

276 Moreover, in colorectal cancer tissues, the Sin1 protein but not mRNA was signif

277 thod to human cancer cell lines and biopsied cancer tissue, thereby illustrating its efficiency, repr

278 thus, suppression of aromatase expression in cancer tissues through the down-regulation of breast tum

279 d specific ASE characteristics in normal and cancer tissues, thus indicating that cisASE has potentia

280 In cancer tissues, TTT-RUVBL complex mRNAs were elevated an

281 tative SPECT imaging of preneoplastic breast cancer tissue using (111)In-labeled cCPE.

282 ia lesion (CIN2/3) samples and five cervical cancer tissues using rolling-circle amplification of tot

283 DOT1L expression and H3K79me2 in colorectal cancer tissues was a predictor of poor patient survival.

284 Most SIGIRR detected in human colon cancer tissues was cytoplasmic, whereas in nontumor tiss

285 188 normal breast and 1247 malignant breast cancer tissues, we observed the loss of KLLN in multiple

286 ere highly expressed specifically in gastric cancer tissues were analyzed for their association with

287 esponse of breast cancer cell lines and lung cancer tissues were evaluated to compare the performance

288 tudy examines HER2 testing of primary breast cancer tissue when performed with immunohistochemistry (

289 RBP2 was overexpressed in human lung cancer tissues where its depletion impaired cell prolife

290 s commonly upregulated in primary human lung cancer tissues where its expression level correlates wit

291 rmal colon mucosa but downregulated in colon cancer tissues, where in both settings its expression co

292 expressed in primary and metastatic prostate cancer tissues, where it functions as an autocrine growt

293 showed that MUC16 is overexpressed in breast cancer tissues whereas not expressed in non-neoplastic d

294 R2+) but not in HER2-negative (HER2-) breast cancer tissues, whereas both HER2+ and HER2- tumors expr

295 ression of CXCR4 and SHH in human pancreatic cancer tissues, whereas their expression is not observed

296 ting PIP2, is positively expressed in breast cancer tissues, which correlates intimately with the pro

297 n of this pathway was detected in human lung cancer tissues with concomitant downregulation of BAMBI,

298 s in formalin-fixed paraffin-embedded breast cancer tissue, with the goal of assessing the prognostic

299 Using primary colorectal cancer tissues, xenograft models, and MAPK reporter cons

300 lon tumor cell lines and primary human colon cancer tissue xenografted into immunodeficient mice.