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1 ompetition between tumor-infiltrating lymphocytes (TIL) and cancer cells for metabolic resources often renders T cells dy
2 ttranslational modifications important to tumorigenesis and cancer cell growth, here we report a chemoproteomic analysis
3                                                     Bladder cancer ranges from unaggressive and usually noninvasive tumor
4                         Annual mortality rates among breast cancer patients were significantly greater in LMICs in South
5 cies including humans and proteomic data to classify breast cancer tumours.
6 11 confers distinct features to ER-negative DCIS.com breast cancer cells, leading to populations enriched with highly pla
7 i at which gene expression could potentially explain breast cancer risk phenotypes.
8  human serum or 0.024 wt % of the total protein from breast cancer cell lysates.
9  change for >35% of patients diagnosed with invasive breast cancer and refined OS estimates.
10 vity against various human cancer cells, killing SW48 colon cancer cells in particular with a submicromolar half maximum
11 gh Wnt signaling has been intensively studied in colorectal cancer, it remains unclear whether activity in the tumor-asso
12 ther insight into the complex genetic architecture of cross-cancer susceptibility.
13 ix of fragmented alleles, and the contribution of different cancer deposits to ctDNA is largely unknown.
14 vivo data, potentially providing an important biomarker for cancer diagnosis and treatment.
15 bunit complex that folds many of the proteins essential for cancer development.
16 r growth and progression, and also pose major obstacles for cancer therapy.
17                             Thus, ATR is a major target for cancer therapies in homologous recombination-deficient cancer
18  most protocols of adoptive cell transfer (ACT) therapy for cancer, but is limited by short exposure and high toxicities.
19  is dysregulated in several malignancies, including gastric cancer, and is an important biomarker in drug discovery.
20            A total gastrectomy and an esophagectomy for GEJ cancer show largely comparable results with regard to long-te
21 S-CECR2-1 exhibits cytotoxic activity against various human cancer cells, killing SW48 colon cancer cells in particular w
22 icating a conditional pro-survival function of caspase-8 in cancer.
23                         Information on molecular changes in cancer-specific gene expression facilitates efficient targete
24                       DynaFit revealed that cell fitness in cancer cell lines, primary cancer cells, and fibroblasts unde
25 e found that expression of the catalytic domain of PDE3A in cancer cells lacking PDE3A is sufficient to confer sensitivit
26 iated pathways for cell survival and apoptosis signaling in cancer remain to be elucidated.
27 R] 5.01; 95% Confidence Interval [CI] 3.50-7.61) and kidney cancer (OR 2.50; 95% CI 1.69-3.72).
28 tiology of hepatitis B, the stage of Barcelona Clinic Liver Cancer (BCLC) B and C, and the presence of cirrhosis, respect
29 rding to patients' receipt of initial assessments by a lung cancer nurse specialist and according to trust-level reported
30 jority of miR-21-5p isolated from human non-small cell lung cancer (NSCLC) tissue possesses 3'-terminal 2'Ome.
31 reased lung CYP2A expression may alter smoking-related lung cancer risk and tissue damage from other inhaled toxins.
32                  Interestingly, prognostic outcomes of many cancer types have been linked with the expression levels of s
33 now recognized as an important regulator of cell migration, cancer cell invasion, and vesicular content release, we sough
34             This gene-centric model has shaped the field of cancer biology and advanced understanding of cancer pathophys
35 d the field of cancer biology and advanced understanding of cancer pathophysiology.
36 oenvironment (TME) and ascites-derived spheroids in ovarian cancer (OC) facilitate tumor growth and progression, and also
37 it revealed that cell fitness in cancer cell lines, primary cancer cells, and fibroblasts under unhindered growth conditi
38           Tropomyosin receptor kinases (TRKs) are promising cancer therapeutic targets.
39 mprised of methylation patterns specific to either prostate cancer or prostate normal epithelium.
40                        The major cause of death in prostate cancer patients can be attributed to metastatic spread of dis
41                                  Lethal metastatic prostate cancer seems to arise from a single clone in the primary tumo
42  can result in overtreatment and undertreatment of prostate cancer.
43 ent achievements in the accuracy of image-based AI for skin cancer diagnosis to address the effects of varied representat
44 rkel cell carcinoma (MCC), an extremely lethal form of skin cancer.
45  an EC(50) of low sub-micromolar range among various tested cancer cell lines such as A2780 (0.23 muM), PC3 (0.48 muM), a
46 a from The Cancer Imaging Archive and genomic data from The Cancer Genome Atlas from 110 patients from five institutions
47                                       Imaging data from The Cancer Imaging Archive and genomic data from The Cancer Genom
48            Genetic testing for hereditary predisposition to cancer is warranted in UM patients with strong personal or fa
49 nslated to clinical trials and it will further translate to cancer management.
50 this success, the interest in integrating nanomedicine with cancer immunotherapy to further improve clinical response and