ライフサイエンスコーパス: cannabinoid receptor antagonist

コーパス検索結果 (１語後でソート)

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1 This burst firing was suppressed by a cannabinoid receptor antagonist.

2 sues of both animals to SR 141716A, a potent cannabinoid receptor antagonist.

3 monstrations of precipitated withdrawal with cannabinoid receptor antagonists.

4 is (CoMFA) approach on a set of arylpyrazole cannabinoid receptor antagonists.

5 s previously reported to function as a CB(1) cannabinoid receptor antagonist (1) .

6 The CB1 cannabinoid receptor antagonist AM 251 [N-(piperidin-1-y

7 st WIN 55,212-2, and is abolished by the CB1 cannabinoid receptor antagonist AM 251.

8 ation was reduced after co-infiltration with cannabinoid receptor antagonists (AM 281 and SR 144528).

9 n intracellular [Ca2+] and is blocked by the cannabinoid receptor antagonist AM251, indicating that i

10 this reduction in firing was blocked by the cannabinoid receptor antagonist AM251.

11 globin but was blocked completely by the CB1 cannabinoid receptor antagonists AM251 [N-(piperidin-1-y

12 t (LA) but not affected by administration of cannabinoid receptor antagonist (AM251).

13 eatment with the peripherally restricted CB1 cannabinoid receptor antagonists, AM6546 and URB447.

14 ed bone loss and that CB1- and CB2-selective cannabinoid receptor antagonists are a new class of oste

15 f the acetaminophen metabolite AM 404 and of cannabinoid receptor antagonists as well as data from ti

16 SR141716, a recently developed CB1 cannabinoid receptor antagonist, blocks acute effects of

17 n contrast to Hp, which functions as a CB(1) cannabinoid receptor antagonist, both RVD-Hpalpha and VD

18 tant implications for the therapeutic use of cannabinoid receptor antagonists in humans.

19 d with cannabidiol and THC was unaffected by cannabinoid receptor antagonist, indicating it to be can

20 nistered cannabinoid receptor agonists and a cannabinoid receptor antagonist influenced rats' perform

21 that in rat cerebellar granule neurons, CB1 cannabinoid receptor antagonists inhibit axonal growth r

22 ed by the specific central cannabinoid (CB1) cannabinoid receptor antagonist N-(piperidin-1-yl)-5-(4-

23 pparent K(d) of 74 and 35 nM, respectively); cannabinoid receptor antagonists reduced or eliminated s

24 Local infusion of the CB(1)-cannabinoid receptor antagonist, rimonabant, into the du

25 A cannabinoid receptor antagonist significantly increased

26 The CB1 cannabinoid receptor antagonist SR 141716A abolished the

27 ceptor was intrathecal administration of the cannabinoid receptor antagonist SR 141716A.

28 ld readily be prevented by the selective CB1 cannabinoid receptor antagonist SR-141716A.

29 Pretreatment with the cannabinoid receptor antagonist, SR 141716A, blocked the

30 roduced by Win55212-2 was blocked by the CB1 cannabinoid receptor antagonist, SR141716 (1 microM).

31 ffect was blocked by coadministration of the cannabinoid receptor antagonist SR141716A (5 microg).

32 to the NAc and was completely blocked by the cannabinoid receptor antagonist SR141716A [N-piperidino-

33 The cannabinoid effect was blocked by the cannabinoid receptor antagonist SR141716A and the Gi/Go

34 he above effects of WIN-2 was blocked by the cannabinoid receptor antagonist SR141716A, showing a dep

35 tion of the drug and was reversed by the CB1 cannabinoid receptor antagonist SR141716A.

36 was blocked by pertussis toxin and the CB1 (cannabinoid) receptor antagonist SR141716A.

37 The cannabinoid receptor antagonist, SR141716A, failed to bl

38 iting work under way on the use of selective cannabinoid receptor antagonists to help patients stop s

39 that this activity, which was not blocked by cannabinoid receptor antagonists, was mediated by periph

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