ライフサイエンスコーパス: cantharidin

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1 ses 1 and 2A (okadaic acid, calyculin A, and cantharidin).

2 adaic acid, microcystin-LR, calyculin A, and cantharidin.

3 phase of the dermal inflammatory response to cantharidin.

4 nventional therapies such as cryotherapy and cantharidin.

5 adaic acid, microcystin-LR, calyculin A, and cantharidin.

6 te preferentially with males that had fed on cantharidin.

7 (0.5, 10, or 30 nM), calyculin A (10 nM), or cantharidin (100 nM); and the selective PP1 inhibitor NI

8 ever, inhibition of protein phosphatase with cantharidin (30 microM) did not interfere with the expre

9 When treated with cantharidin, a chemical inhibitor of PP2A, the phot1-5 m

10 o a comparable extent, both okadaic acid and cantharidin also induce aberrant centrosome replication,

11 exon 7 inclusion, we synthesized analogs of cantharidin, an inhibitor of PP1, and PP2A.

12 We found that cantharidin, an inhibitor of types 1 and 2A protein phos

13 d by the bioluminescent glow rhythm, whereas cantharidin and calyculin A caused large phase delays bu

14 protein phosphatase inhibitors okadaic acid, cantharidin and calyculin A inhibited the stimulatory pr

15 gest that protein phosphatases, sensitive to cantharidin and okadaic acid, are likely involved in reg

16 earance of cortical arrays, but calyculin A, cantharidin, and staurosporine, which caused swelling, d

17 They are attracted to cantharidin baits in the field and feed on the compound

18 The terpenoid cantharidin (CNT), previously used to treat various mali

19 Staurosporine and the phosphatase inhibitor, cantharidin, delay the asymmetric expression of DR5revGF

20 se inhibitor, okadaic acid, calyculin A, and cantharidin did not block light-induced phase shifts.

21 At 24 hours, cantharidin formed blisters of similar volume in both se

22 Males that ingest cantharidin secrete cantharidin from a cephalic gland.

23 The phosphatase 2A inhibitor, cantharidin (> or = 10 micromol/L), also blocked the IFN

24 the phosphatase inhibitors okadaic acid and cantharidin in organ elongation assays.

25 to the protein phosphatase type 2A inhibitor cantharidin in the absence of a pathogen.

26 ase inhibitors okadaic acid, fostriecin, and cantharidin increased 17,20 lyase activity, suggesting i

27 Also, the PP2A inhibitor cantharidin induced Mad2 phosphorylation and inhibited t

28 xudate mediators and cellular recruitment in cantharidin-induced skin blisters at 24 and 72 hours.

29 ne this, we found two types of responders to cantharidin-induced skin blisters in male healthy volunt

30 s either by in vitro culture or in resolving cantharidin-induced skin blisters led to an equivalent i

31 les can be rendered acceptable to females if cantharidin is added to their cephalic gland.

32 A major fraction of the acquired cantharidin is stored by the male in the large accessory

33 ne/threonine protein phosphatases (fluoride, cantharidin, metal-chelating agents).

34 e inhibitors, okadaic acid, calyculin A, and cantharidin, prevented all of these apoptotic characteri

35 Eggs endowed with cantharidin proved relatively invulnerable to attack by

36 rescued by treatment with staurosporine and cantharidin, respectively.

37 Males that ingest cantharidin secrete cantharidin from a cephalic gland.

38 The amount of cantharidin (Spanish fly) that the Neopyrochroa flabella

39 chroa flabellata have a natural affinity for cantharidin (Spanish fly).

40 (calyculin A, microcystin, okadaic acid, and cantharidin) that inhibit PP 1 and 2A were found to stim

41 epresents only a small fraction of the total cantharidin the male accumulates systemically following

42 that reduced basipetal transport in rcn1 and cantharidin-treated wild-type plants also restored a nor

43 Furthermore, cantharidin treatment of wild-type seedlings mimics the

44 Cantharidin-unfed males can be rendered acceptable to fe

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