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1 er by the prototypical Na(+) channel blocker carbamazepine.
2 he antiepileptic drug topiramate, but not by carbamazepine.
3 accumulated to a greater degree in bile than carbamazepine.
4 alproate and for lower verbal abilities with carbamazepine.
5 s alleviated by the sodium channel inhibitor carbamazepine.
6 with epilepsy or seizure when compared with carbamazepine.
7 nts compared with topiramate and secondarily carbamazepine.
8 agents, and possibly the antiepileptic agent carbamazepine.
9 non-inferiority of lamotrigine compared with carbamazepine.
10 rotein-induced EAE treated with phenytoin or carbamazepine.
11 c treatments such as lithium, valproate, and carbamazepine.
12 n the third year, they received lithium plus carbamazepine.
13 taking placebo to 3015 minutes while taking carbamazepine.
14 taking placebo to 45.3 minutes while taking carbamazepine.
15 ally relevant thermal stimuli was reduced by carbamazepine.
16 s the best candidate enzyme for metabolizing carbamazepine.
17 intenance period to 136 minutes while taking carbamazepine.
18 enance period and 32 awakenings while taking carbamazepine.
19 ndetectable or significantly lower levels of carbamazepine.
20 d with pain during treatment with placebo or carbamazepine.
21 40s who developed DIHS after treatment with carbamazepine.
22 pharmacological rescue by glibenclamide and carbamazepine.
23 87, 0.85-0.90), and treatment (gabapentin vs carbamazepine, 0.71, 0.59-0.86; topiramate vs carbamazep
28 ere validated using metabolites (10,11-epoxy carbamazepine, 10,11-dihydroxy carbamazepine, and acridi
29 uding 10,11-dihydro-10-hydroxycarbamazepine, carbamazepine-10,11-epoxide, acridone-N-carbaldehyde, 4-
31 o valproate (mean 97, 95% CI 94-101) than to carbamazepine (105, 102-108; p=0.0015), lamotrigine (108
32 of patients were receiving monotherapy with carbamazepine (237 patients), phenobarbitone/primidone (
33 e paralleled by pharmacological coupling, as carbamazepine (30 muM) depolarizes S214T activation, as
34 thoxazole (2-33 days), naproxen (6-19 days), carbamazepine (355-1,624 days), and ciprofloxacin were n
35 e taking placebo to 9.1 minutes while taking carbamazepine (400 mg then 200 mg/day), and a reduction
36 taking placebo to 231.9 minutes while taking carbamazepine (400 mg/day), and a reduction in total TIP
37 European centres and randomly assigned them carbamazepine 600 mg daily (n=230) or vigabatrin 2 g dai
38 93%), indomethacin (98%), timolol (50%), and carbamazepine (70%) were assigned to the high %F region;
39 95% CI 5.5% to 8.3%) compared with 2.6% with carbamazepine (95% CI 1.9% to 3.5%) and 2.3% with lamotr
44 ates suggest a non-significant advantage for carbamazepine against lamotrigine (0.91 [0.77-1.09]), to
48 his study we have investigated the effect of carbamazepine, an anti-convulsant, as it is known to hav
51 mazepine was only transformed to 10,11-epoxy carbamazepine and 10,11-dihydroxy carbamazepine as a dea
52 ard and feedback inhibition is unaffected by carbamazepine and additional commonly used Na(+) channel
53 by the use-dependent sodium channel blocker carbamazepine and by a blocker of reverse Na-Ca exchange
56 ies showed complete elimination of (14)C for carbamazepine and fluoxetine treatments and partial elim
58 vidence that despite structural differences, carbamazepine and glibenclamide compete for binding to K
59 HLA) associations: abacavir and HLA-B*57:01, carbamazepine and HLA-B*15:02, allo-purinol and HLA-B*58
61 laimed wastewater-irrigated produce excreted carbamazepine and its metabolites in their urine, while
65 endent rapamycin as well as mTOR-independent carbamazepine and minoxidil, markedly attenuated cyst fo
66 uticals acetaminophen, sulfamethoxazole, and carbamazepine and on the pharmaceutical intermediate 5-m
70 s treatment with the sodium channel blockers carbamazepine and oxcarbazepine, which although effectiv
72 treatments with antiepileptic drugs, such as carbamazepine and phenytoin, which are sodium channel bl
73 f sodium channels, in contrast to drugs like carbamazepine and phenytoin, which bind tightly to fast-
75 Triazolam, oral midazolam, St. John's Wort, carbamazepine and pimozide, are contraindicated with DAA
78 mug L(-1)), and several compounds, including carbamazepine and sulfamethoxazole, were detected throug
79 One of the pharmacological treatments is Carbamazepine and the most prevalent surgical treatments
84 thium and certain anticonvulsants, including carbamazepine and valproic acid, are effective antimanic
87 xposed to phenytoin, 98 for those exposed to carbamazepine, and 92 for those exposed to valproate.
88 (10,11-epoxy carbamazepine, 10,11-dihydroxy carbamazepine, and acridine) as primary substrates and f
89 nt mood stabilizers, e.g., valproic acid and carbamazepine, and atypical antipsychotic drugs (APDs),
90 Lamotrigine, 10,11,-dihydro-10,11,-dihydroxy-carbamazepine, and carbamazepine were the most persisten
91 ine, olanzapine, ziprasidone, valproic acid, carbamazepine, and citalopram were associated with highe
92 ions of triclosan, triclocarban, miconazole, carbamazepine, and diphenhydramine in plant tissues that
94 he tricyclic anticonvulsant drugs phenytoin, carbamazepine, and lamotrigine block neuronal voltage-ga
95 orketamine, O-desmethyltramadol, diclofenac, carbamazepine, and methoxetamine were not substantially
97 to treatment with mood stabilizers (lithium, carbamazepine, and sodium valproate) and plasma levels o
98 Finally, rapamycin as well as spermidine, carbamazepine, and tamoxifen could also rescue the motor
99 hree other autophagy activators (spermidine, carbamazepine, and tamoxifen) in a FTLD-U mouse model wi
100 lsant mood stabilizers (AMS), valproic acid, carbamazepine, and zonisamide, but not lithium, also pre
101 ely prescribed anticonvulsants phenytoin and carbamazepine are potent inducers of cytochrome P450 enz
105 o significant difference for patients taking carbamazepine at randomization, because of a low rate of
106 sample volume were evaluated for five drugs: carbamazepine, atenolol, sulfamethazine, diazepam, and a
107 odynamic analysis, and functional profiling, carbamazepine attenuated pain in patients with IEM due t
108 onic or uncharged trimethoprim and uncharged carbamazepine, but did not affect sequestration of fast-
109 lizers, valproic acid (VPA), zonisamide, and carbamazepine, but not the typical APD haloperidol, incr
110 Advanced oxidation can efficiently degrade carbamazepine, but the toxicity and persistence of the o
112 gate transport of phenytoin, lamotrigine and carbamazepine by using seven in-vitro transport models.
114 hether the products of advanced oxidation of carbamazepine can be biotransformed and ultimately miner
117 95% CI and p values for no difference) were: carbamazepine (CBZ) 0.43 (-0.19 to 1.05) p=0.17; lamotri
120 re we show that the autophagy-enhancing drug carbamazepine (CBZ) decreased the hepatic load of ATZ an
123 al sunlight photolysis of the pharmaceutical carbamazepine (CBZ) in the presence of dissolved organic
127 ental emission rates of alprazolam (APZ) and carbamazepine (CBZ) through domestic WWTPs (both sludge
128 rated TPs of the recalcitrant pharmaceutical carbamazepine (CBZ) were used for a target screening for
130 hlighting this difficulty, it is unclear why carbamazepine (CBZ), a frontline AED with a known molecu
133 ed the HLA-B *1502 genetic predisposition to carbamazepine (CBZ)-induced Stevens-Johnson syndrome (SJ
135 sing preliminary evidence include memantine, carbamazepine, citalopram, and prazosin, but none of the
136 tine, paroxetine, sertraline, norfluoxetine, carbamazepine, clozapine, flurbiprofen, and sulfobromoph
137 ve metabolites of acetaminophen, diclofenac, carbamazepine, clozapine, p-cresol, 4-ethylphenol, and 3
138 esults show that the oxidation byproducts of carbamazepine containing a hydroxyl or carbonyl group ca
139 ically distinct compounds, glibenclamide and carbamazepine, correct biogenesis defects in ATP-sensiti
140 ths was 90% taking lacosamide and 91% taking carbamazepine-CR (absolute treatment-difference: -1.3%,
141 in the lacosamide group and 308 (70%) in the carbamazepine-CR group completed 6 months of treatment w
142 tients taking lacosamide and 43 (10%) taking carbamazepine-CR had serious treatment-emergent adverse
143 44 patients taking lacosamide and 442 taking carbamazepine-CR were included in the full analysis set
144 ay for lacosamide and 800 or 1200 mg/day for carbamazepine-CR) over 2 weeks with a 1-week stabilisati
146 doses of 100 mg/day lacosamide or 200 mg/day carbamazepine-CR, uptitration to the first target level
151 ), anti-anxiety diazepam (DZP), anti-seizure carbamazepine (CZP) drugs and their metabolites in groce
154 tigated the fate and uptake of (14)C-labeled carbamazepine, diclofenac, fluoxetine, and orlistat in s
155 In patients who achieved therapeutic levels, carbamazepine did not alter the clinical or electromyogr
156 r several other protein misfolding diseases, carbamazepine did not correct KATP channel trafficking d
161 transformation products of the (14)C-labeled carbamazepine during UV/hydrogen peroxide advanced oxida
162 ons of widely monitored HHCB, triclosan, and carbamazepine explained 71-82% of the variability in the
163 ile fish bioconcentrated both fluoxetine and carbamazepine from exposure water, fluoxetine accumulate
164 Patients were randomly assigned to receive carbamazepine, gabapentin, lamotrigine, oxcarbazepine, o
165 , and they were randomly assigned to receive carbamazepine, gabapentin, lamotrigine, oxcarbazepine, o
166 iprofloxacin, trimethoprim, propranolol, and carbamazepine (>80%) was achieved within 3 h of electrol
167 nnel inhibitors, including sulfonylureas and carbamazepine, have been shown to correct channel traffi
168 luding clopidogrel, pegylated-interferon and carbamazepine, have led to the identification of specifi
169 e, lamotrigine was significantly better than carbamazepine (hazard ratio [HR] 0.78 [95% CI 0.63-0.97]
172 interaction was observed for lamotrigine or carbamazepine in any of the seven validated in-vitro tra
173 harmacogenetic testing for HLA-B*1502 before carbamazepine in patients of certain Asian ethnicities a
174 e-Carlo based pharmacokinetic simulations of Carbamazepine in treatment of Trigeminal Neuralgia.
175 including human leukocyte antigen loci with carbamazepine-induced dermatologic outcome and MC4R with
176 ated the relationship between HLA-B*1502 and carbamazepine-induced SJS and TEN and that reported suff
177 lationship between the HLA-B*1502 allele and carbamazepine-induced SJS and TEN in Han-Chinese, Thai,
178 for the relationship between HLA-B*1502 and carbamazepine-induced SJS and TEN was 79.84 (95% CI, 28.
180 bacavir hypersensitivity and HLA-B*57:01 and carbamazepine-induced Stevens-Johnson syndrome and HLA-B
181 lysis, the degree and timing of lithium- and carbamazepine-induced thyroid changes and their subseque
185 he widely used anticonvulsant pharmaceutical carbamazepine is recalcitrant in many environmental nich
187 achieving a 12-month remission (lamotrigine-carbamazepine) is 0 (-8 to 7) and 5 (-3 to 12), suggesti
188 ebo were significantly better tolerated than carbamazepine (lamotrigine, RR 5.24, 1.07-26.32; placebo
190 ntiepileptics approved for bipolar disorder (carbamazepine, lamotrigine, and valproate) are associate
191 ed to maternal IQs among children exposed to carbamazepine, lamotrigine, or phenytoin but not among t
192 ified as either exposed to an antiepileptic (carbamazepine, lamotrigine, or valproate) or not exposed
193 fects exist across four commonly used drugs (carbamazepine, lamotrigine, phenytoin and valproate).
194 epilepsy on antiepileptic drug monotherapy (carbamazepine, lamotrigine, phenytoin, or valproate) bet
195 ho were taking a single antiepileptic agent (carbamazepine, lamotrigine, phenytoin, or valproate) in
196 men with epilepsy receiving monotherapy (ie, carbamazepine, lamotrigine, phenytoin, or valproate) wer
198 ion of the KATP channel opener diazoxide and carbamazepine led to enhanced mutant channel function wi
200 ption coefficients increased in the order of carbamazepine < diclofenac < fluoxetine < orlistat.
201 elimination of (14)C, increased in the order carbamazepine < diclofenac < fluoxetine and orlistat.
203 These findings suggest that phenytoin and carbamazepine may substantially increase the risk for ca
204 contraceptive steroids and the inhibition of carbamazepine metabolism by certain macrolide antibiotic
209 re exposed to valproate monotherapy, 1718 to carbamazepine monotherapy and 2198 to lamotrigine monoth
210 mal fields; Group III, 14 patients receiving carbamazepine monotherapy; Group IV, 20 normal individua
211 cated agents were lamotrigine, azithromycin, carbamazepine, moxifloxacin, cephalexin, diclofenac, and
212 polar patients using lithium, valproate, and carbamazepine (N = 718) and then examined the patient de
213 similar irrespective of the system, and with carbamazepine no significant degradation was obtained.
215 s further evident at a cellular level, where carbamazepine normalized the hyperexcitability of dorsal
216 is usually resistant to pharmacotherapy, but carbamazepine normalizes activation of Na(V)1.7-V400M mu
217 technology was used to assess the effect of carbamazepine on firing of DRG neurons carrying S241T mu
221 We recruited 34 epilepsy patients taking carbamazepine or phenytoin in monotherapy whose physicia
222 g epilepsy patients from the enzyme-inducers carbamazepine or phenytoin to the noninducing drugs leve
224 er calcium concentrations in subjects taking carbamazepine or valproate compared with those taking ot
225 notherapy, patients may have received either carbamazepine or valproate in combination with lithium f
226 h anticonvulsants (phenytoin, phenobarbital, carbamazepine, or a combination) at the same time as ant
227 erphenazine) and a mood stabilizer (lithium, carbamazepine, or valproate), 37 patients were randomly
228 gned to 1 year of lithium and then 1 year of carbamazepine, or vice versa; in the third year, they re
229 nic use of many antiepileptic drugs, such as carbamazepine, oxcarbazepine, and topiramate, enhances h
230 tments of BD such as valproate, lamotrigine, carbamazepine, oxcarbazepine, antipsychotics, electrocon
236 NTDs related to exposure to FAAs (including carbamazepine, phenobarbital, phenytoin, primidone, sulf
237 cytochrome P450 enzyme (cP450) inducing AED (carbamazepine, phenytoin, and phenobarbital), a cP450 in
238 ere significantly less in subjects receiving carbamazepine, phenytoin, and valproate than in those re
239 esults of the one-step CEDIA for three AEDs (carbamazepine, phenytoin, and valproic acid), in the pre
242 h common drugs as trimethoprim, triamterene, carbamazepine, phenytoin, phenobarbital, and primidone,
243 f bone mass in women, and both phenytoin and carbamazepine produce increases in serum lipids and C-re
244 sy patients, switch from either phenytoin or carbamazepine produced significant declines in total cho
246 sing measured photolysis rates for atenolol, carbamazepine, propranolol, and sulfamethoxazole in wetl
247 ed in 13 units with a serum concentration of carbamazepine ranging from 3.5 to 8.4 mg/l, which was wi
248 actory solid tumors and to determine whether carbamazepine reduces oxaliplatin-induced neurotoxicity.
249 this study, a mechanism is proposed by which carbamazepine resists biodegradation, and a previously u
251 1.7-V400M mutant channels from a family with carbamazepine-responsive inherited erythromelalgia.
253 .62, 95% credible interval [CrI] 0.38-1.03), carbamazepine (RR 0.68, 0.44-1.06), imipramine (RR 0.95,
256 n identity test for monoclonal antibodies to carbamazepine, sirolimus, tacrolimus, cyclosporine, cort
259 peroning mechanism wherein glibenclamide and carbamazepine stabilize the heteromeric subunit interfac
260 xaliplatin); regimen B (to determine whether carbamazepine starting 24 hours before and ending 48 hou
261 ment with 300 microM lidocaine or 300 microM carbamazepine, suggesting that lacosamide, lidocaine, an
262 nd that the wastewater borne MPs diclofenac, carbamazepine, sulfamethoxazole, acesulfame, sucralose,
264 r the HLA-B*1502 allele before initiation of carbamazepine therapy in patients of Asian ancestry, but
268 the first 6 weeks from randomisation, showed carbamazepine to be significantly more effective than vi
269 at the observed uptake in the fluoxetine and carbamazepine treatments was due to the parent compounds
271 users (36.5%), valproate users (42.4%), and carbamazepine users (42.2%) with bipolar disorder diagno
273 ) and toxic epidermal necrolysis (TEN) among carbamazepine users, especially in some racial/ethnic po
275 ineffective, while valproate, gabapentin and carbamazepine varied in their potencies, with only the l
277 on treatment with low doses of rapamycin and carbamazepine was able to attenuate cyst formation as ef
279 sformation of trimethoprim, propranolol, and carbamazepine was attributed to direct electron transfer
282 icking rescue by reversible sulfonylureas or carbamazepine was facilitated by the KATP channel opener
285 d-state-fermentation conditions, 10,11-epoxy carbamazepine was further metabolized via acridine and 1
290 and functional analysis to be responsive to carbamazepine, was assessed in a double-blind, placebo-c
293 hium, quetiapine, olanzapine, bupropion, and carbamazepine were associated with high morbidity indice
294 -dihydro-10,11,-dihydroxy-carbamazepine, and carbamazepine were the most persistent (t1/2 = 12 +/- 2.
295 ants such as sucralose, sulfamethoxazole and carbamazepine, which are typical wastewater markers in r
296 taking placebo to 274.1 minutes while taking carbamazepine, while patient 2 reported a reduction of t
297 dwater and revealed persistence for the drug carbamazepine, while the herbicide MCPA (2-methyl-4-chlo
299 attenuation of all compounds studied except carbamazepine within an area similar to that typical of
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