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1 e sequelae 6 weeks and 12 months after acute carbon monoxide poisoning.
2 tcome was cognitive sequelae six weeks after carbon monoxide poisoning.
3 eserved hippocampal neurogenesis after acute carbon monoxide poisoning.
4 was conducted during the first 7 days after carbon monoxide poisoning.
5 n-carbon monoxide-treated control, (2) acute carbon monoxide poisoning, (3) acute carbon monoxide poi
6 This review focuses on the unique aspects of carbon monoxide poisoning and hyperbaric oxygen therapy
7 in critically ill patients, including acute carbon monoxide poisoning, arterial gas embolism, severe
8 eviate delayed memory impairment after acute carbon monoxide poisoning by preserving adult neurogenes
10 ) acute carbon monoxide poisoning, (3) acute carbon monoxide poisoning followed by 7-day hyperbaric o
11 r B protein (TrkB-Fc) chimera, and (5) acute carbon monoxide poisoning followed by intracerebroventri
12 mly assigned patients with symptomatic acute carbon monoxide poisoning in equal proportions to three
13 ficient information has been published about carbon monoxide poisoning in the medical literature to d
15 ose deaths were classified as "unintentional carbon monoxide poisoning, not fire related," 49 (36%) m
17 l air embolism, its role in the treatment of carbon monoxide poisoning remains somewhat controversial
23 his suppression of adult neurogenesis by the carbon monoxide poisoning was appreciably alleviated by
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