ライフサイエンスコーパス: carbonyl cyanide m-chlorophenyl hydrazone

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1 en the TM proton gradient was abolished with Carbonyl cyanide m-chlorophenyl hydrazone.

2 hondrial oxidative phosphorylation uncoupler carbonyl cyanide m-chlorophenyl hydrazone (2 microM) als

3 It is eliminated by the protonophore carbonyl cyanide m-chlorophenyl hydrazone and is approxi

4 function induced by mitochondrial ionophore, carbonyl cyanide m-chlorophenyl hydrazone and other resp

5 The effects of carbonyl cyanide m-chlorophenyl hydrazone (CCCP), an inh

6 Herein, carbonyl cyanide m-chlorophenyl hydrazone (CCCP)-induced

7 After addition of carbonyl cyanide m-chlorophenyl hydrazone (CCCP, 1-2 mic

8 Carbonyl cyanide m-chlorophenyl hydrazone (CCCP; 5 micro

9 nol (CEP), calcimycin (a Ca2+ ionophore) and carbonyl cyanide m-chlorophenyl hydrazone (CCCP; a mitoc

10 a2+]c, were abolished by a proton ionophore, carbonyl cyanide m-chlorophenyl-hydrazone (CCCP), and by

11 other cells, mitochondrial depolarization by carbonyl cyanide m-chlorophenyl hydrazone did not induce

12 port, while the protonmotive-force-inhibitor carbonyl cyanide m-chlorophenyl hydrazone had no effect.

13 nsensitive to ATP or treatment with NH4Cl or carbonyl cyanide m-chlorophenyl hydrazone, indicating th

14 tochondria or parkin-mediated mitophagy upon carbonyl cyanide m-chlorophenyl hydrazone-induced mitoch

15 Although the uncoupler carbonyl cyanide m-chlorophenyl hydrazone inhibited AGT(

16 of Opa1 splice variants but does not affect carbonyl cyanide m-chlorophenyl hydrazone or apoptosis-i

17 n those treated with TNF or uncoupling agent carbonyl cyanide m-chlorophenyl hydrazone, suggesting an

18 polarizing mitochondria with antimycin A1 or carbonyl cyanide m-chlorophenyl-hydrazone, the stimulati

19 Using carbonyl cyanide m-chlorophenyl hydrazone treatment, we

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