ライフサイエンスコーパス: carboxyl ester lipase

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1 se, pancreatic lipase-related protein 2, and carboxyl-ester lipase.

2 Inhibition of carboxyl ester lipase activity in HepG2 cells reduced hy

3 ic triglyceride lipase, but close to that of carboxyl ester lipase, after a long lag phase, which cou

4 Confocal microscopy of immunostained carboxyl ester lipase and scavenger receptor BI showed a

5 arginine residues in bile salt activation of carboxyl ester lipase (CEL) activity.

6 This study evaluated the contributions of carboxyl ester lipase (CEL) and pancreatic triglyceride

7 y deletion mutations in the last exon of the carboxyl ester lipase (CEL) gene, resulting in a CEL pro

8 Pancreatic carboxyl ester lipase (CEL) hydrolyzes cholesteryl ester

9 To study the role of carboxyl ester lipase (CEL) in hepatic retinoid (vitamin

10 ble number of tandem repeat (VNTR) domain of carboxyl ester lipase (CEL) presents an opportunity to s

11 G2 cells and primary hepatocytes showed that carboxyl ester lipase (CEL), a broad-spectrum lipase sec

12 nes favoring the production of R-9-PAHSA and carboxyl ester lipase (CEL), a PAHSA degradative enzyme,

13 Bile salt-stimulated carboxyl ester lipase (CEL), also called cholesterol est

14 Previously we showed that carboxyl ester lipase (CEL)-deficient (CELKO) mice have

15 Carboxyl ester lipase (CEL, also called cholesterol este

16 Carboxyl ester lipase (CEL; EC 3.1.1.13) hydrolyzes chol

17 Carboxyl-ester lipase (CEL) maturity-onset diabetes of t

18 When added to cultures of HepG2 cells, carboxyl ester lipase cofractionated with scavenger rece

19 Previous work showed that carboxyl ester lipase enhanced selective uptake of chole

20 selective uptake in primary hepatocytes from carboxyl ester lipase knockout mice.

21 ly reduced in lipid rafts from the livers of carboxyl ester lipase-null mice compared with control an

22 The data suggest that carboxyl ester lipase plays a physiological role in hepa

23 eriments were performed to determine whether carboxyl ester lipase plays a role in scavenger receptor

24 hydrolytic activity is likely to derive from carboxyl ester lipase rather than MGL.

25 of cell-surface proteoglycans also prevented carboxyl ester lipase stimulation of cholesteryl ester u

26 ock scavenger receptor BI function prevented carboxyl ester lipase stimulation of selective uptake in

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