ライフサイエンスコーパス: carcinogenic

1 than PAC in two categories (eutrophication, carcinogenics).

2 matic hydrocarbons (PAHs), some of which are carcinogenic.

3 s DNA damage in vivo and is considered to be carcinogenic.

4 elminthes and major human pathogens, are not carcinogenic.

5 mation of bromate, which is considered to be carcinogenic.

6 f-heterozygosity (LOH), which is potentially carcinogenic.

7 demonstrated to be cytotoxic, mutagenic, and carcinogenic.

8 ssified radiofrequency radiation as possibly carcinogenic.

9 at ubiquitous APOBEC-mediated mutagenesis is carcinogenic.

10 he first studied aflatoxins were found to be carcinogenic.

11 s, including carbon nanotubes (CNTs), may be carcinogenic.

12 Aristolochic acids (AA) are nephrotoxic and carcinogenic.

13 s infection with only a few helminth species carcinogenic?

14 s infection with only a few helminth species carcinogenic?

15 ed nonharmful and mitigate the generation of carcinogenic acridine products known to form when advanc

16 ious forms of cancer have been linked to the carcinogenic activities of microorganisms(1-3).

17 eutic agent as it has exhibited several anti-carcinogenic activities.

18 c health, due to the potential endocrine and carcinogenic activity of residues, accumulating in edibl

19 n the treatment of ulcerative colitis, and a carcinogenic activity under host inflammatory conditions

20 Several PAHs are toxic, and a subset exhibit carcinogenic activity.

21 The mechanism by which carcinogenic adducts inhibit DNA synthesis and cause mut

22 Carcinogenic adducts, such as 2-aminofluorene (AF) and N

23 h of consumers due to its ability to produce carcinogenic aflatoxins.

24 e and exceptionally sensitive to detect this carcinogenic agent in commercial products with a LOD (3s

25 ocarbon (PAH); this ubiquitous environmental carcinogenic agent is found in tobacco smoke, charcoal-g

26 rafish, rainbow trout, hamsters, and mice by carcinogenic agents (methylcholanthrene, thioacetamide),

27 directly targeted, avoidance of exposure to carcinogenic agents is likely to prevent other non-commu

28 l density, with no requirement for exogenous carcinogenic agents.

29 imetric indicator compound for many types of carcinogenic alkylating agents.

30 incidence of common cancers, probably due to carcinogenic amines (HAAs) formed upon meat heating.

31 n cell culture models, but most mutagens are carcinogenic and are poorly tolerated.

32 Top1cc can be trapped by carcinogenic and endogenous DNA lesions and by camptothe

33 ears, because arsenic species are considered carcinogenic and found at high concentrations in the sam

34 ,7,8-tetrachlorodibenzo-p-dioxin, TCDD) is a carcinogenic and highly toxic industrial byproduct that

35 e a class of ON compounds known to be highly carcinogenic and include species formed from nicotine de

36 Formalin is carcinogenic and is detrimental to public health.

37 Acrylamide is a carcinogenic and neurotoxic process contaminant that is

38 the potential for human exposure and adverse carcinogenic and non-cancer health effects.

39 re to curcumin, a natural compound with anti-carcinogenic and telomerase activity-reducing properties

40 nd most used DNA intercalators, but presents carcinogenic and teratogenic properties.

41 These compounds present carcinogenic and/or mutagenic properties and offer a ris

42 Some OPFRs are considered carcinogenic and/or neurodevelopmental toxicants, and ch

43 Regarding teratogenic, carcinogenic, and immunotoxic nature of ochratoxin A (OT

44 uced in cooked meats, and 4-aminobiphenyl, a carcinogenic aromatic amine present in tobacco smoke, we

45 Melanin may thus be carcinogenic as well as protective against cancer.

46 a neglected, tropical disease caused by the carcinogenic Asian liver fluke, Opisthorchis viverrini.

47 variants to assist further investigation of carcinogenic association and the development of diagnost

48 Known toxic and carcinogenic azaarenes, such as benzo[c]acridine and dib

49 Although the carcinogenic basis of AFB1 has been ascribed to its muta

50 tive hydrogenation, a potential approach for carcinogenic benzene removal from gasoline, is probed us

51 Z is not thought to be particularly toxic or carcinogenic, but little is known about its effect on de

52 s tend to live longer, have more potentially carcinogenic cells, and undergo more cell divisions.

53 ong-term consequences, but these may include carcinogenic change or granulomatous inflammation.

54 drugs, understanding the mode of actions of carcinogenic chemicals, and monitoring the genotypic tox

55 r example, to control emissions of toxic and carcinogenic combustion by-products, which also greatly

56 ted that DNA adducts of aristolochic acid, a carcinogenic component of Aristolochia herbs used in tra

57 yclic aromatic hydrocarbons (PAHs) are known carcinogenic components of tobacco smoke found in settle

58 Aflatoxin M1 (AFM1), a potentially carcinogenic compound, is found in milk obtained from an

59 Acetaldehyde is a naturally-occurring carcinogenic compound, present in different food items,

60 Our methodology entails an exposure to carcinogenic compounds during the animal's regeneration

61 Trihalomethanes (THMs) are conditionally carcinogenic compounds formed during chlorine disinfecti

62 AAs are nephrotoxic and carcinogenic compounds found in Aristolochia herbaceous

63 nalysis for very low concentrations of these carcinogenic compounds was developed and compared by app

64 izing reagents and avoiding the use of toxic carcinogenic compounds.

65 m stem cell dynamics, to detect and classify carcinogenic compounds.

66 -2003) of 34,018 workers without exposure to carcinogenic confounders.

67 allowing the bacteria to persist and induce carcinogenic consequences in the gastric niche.

68 allowing the bacteria to persist and induce carcinogenic consequences in the gastric niche.

69 umans, animals and crops, a source of potent carcinogenic contaminants of food, and an important gene

70 s other Cr supplements, was also oxidized to carcinogenic Cr(VI) and Cr(V) in living cells.

71 simple act of cooking can convert all of the carcinogenic Cr(VI) to the safer Cr(III).

72 The carcinogenic cyclic ether compound 1,4-dioxane is employ

73 be a potential previously unknown source of carcinogenic dibenzopyrenes to the environment.

74 N-Nitrosodimethylamine (NDMA) is a carcinogenic disinfection byproduct from water chloramin

75 c mechanisms that explain the geographic and carcinogenic diversity of HPV58 variants are still poorl

76 N(2)-Furfuryl-deoxyguanosine (fdG) is carcinogenic DNA adduct that originates from furfuryl al

77 re interactions between a DNA polymerase and carcinogenic DNA adducts in real-time by single-molecule

78 ctivity with DNA templates modified with the carcinogenic DNA adducts, 2-aminofluoene (AF) or N-acety

79 BPDE), which then can react with DNA to form carcinogenic DNA adducts.

80 protection against potentially mutagenic and carcinogenic DNA damage.

81 methylbenzidine(TMB) as a new remarkable non-carcinogenic DNA indicator for genosensing purposes, whi

82 Endogenous and carcinogenic DNA lesions can also trap Top1cc.

83 Initial exposure of osteoblasts to carcinogenic doses of IR in vitro and in vivo induced RB

84 ive immune system; however, it is considered carcinogenic due to its strong association with lymphoid

85 be was made possible by using Sudan red III (carcinogenic dye), which was oxidized by PPD doped in P-

86 the first time, that arsenite may exert its carcinogenic effect by targeting cysteine residues in th

87 radiation oncology, has potentially greater carcinogenic effect compared with sparsely ionizing radi

88 Statins have reported anti-carcinogenic effects and may be chemoprotective.

89 a DNA repair enzyme that protects cells from carcinogenic effects of alkylating agents; however, MGMT

90 he exact molecular mechanisms underlying the carcinogenic effects of arsenic remain incompletely unde

91 dicate that a high-salt diet potentiates the carcinogenic effects of cagA(+) H. pylori strains.

92 hanisms involved including withdrawal of the carcinogenic effects of calcineurin inhibitors and/or th

93 These data suggest that carcinogenic effects of densely ionizing radiation are m

94 Carcinogenic effects of hexavalent chromium in waters ar

95 The enhanced carcinogenic effects of hormones on ATF3-deficient prost

96 n carcinogenesis and suggests that the known carcinogenic effects of inflammation may be at least par

97 d is one of the most sensitive organs to the carcinogenic effects of IR, and we have recently highlig

98 omain) family known to mediate the toxic and carcinogenic effects of xenobiotics.

99 We showed significant carcinogenic effects on female animals while revealing a

100 urrently exposed to molecules with potential carcinogenic effects such as 17beta-estradiol, the most

101 erintuitive conclusion that UVR has anti-BCC carcinogenic effects that can explain, at least in part,

102 omatic hydrocarbon with strong mutagenic and carcinogenic effects.

103 ted with supply chain production, except for carcinogenic effects.

104 ry metabolites of fungi that cause toxic and carcinogenic effects.

105 on (IR) is a public concern due to its known carcinogenic effects.

106 which early-life iAs exposure elicits latent carcinogenic effects.

107 Arsenic is a ubiquitous and carcinogenic environmental element that enters the biosp

108 sample preparation procedure for analysis of carcinogenic ethyl carbamate (EC) in wine by GC-MS-SIM i

109 l DNA damage and repair processes as well as carcinogenic exposures or cancer related aberrations of

110 tures can be readily assigned to known human carcinogenic exposures or endogenous mechanisms of mutag

111 nds and endogenous nitrosation are important carcinogenic factors, but human evidence of their role i

112 ur findings suggest that minimal exposure to carcinogenic fibers may significantly increase the risk

113 The latter is known for being carcinogenic for vertebrates, potentially causing enviro

114 ge of these polymers is the reduction of the carcinogenic formaldehyde.

115 icro RNA (miRNA), are able to knock down the carcinogenic genes by targeting the mRNA expression, whi

116 HPV16 was the most common carcinogenic genotype detectable in 25% of urine, 33.8%

117 door air pollution and particulate matter as carcinogenic (Group 1).

118 hyl-6-phenylimidazo[4,5-b]pyridine (PhIP), a carcinogenic HAA produced in cooked meats, and 4-aminobi

119 ), but higher reactivity in forming the more carcinogenic haloacetonitrile after chlorination (244%-o

120 uracy of tumor diagnoses for their impact on carcinogenic hazard characterization.

121 ecognised toxicity and especially due to the carcinogenic hazard which they present.

122 elevance of the in vitro positive results to carcinogenic hazard.

123 ydrocarbon (PAH)-contaminated media, wherein carcinogenic hazards are evaluated using a dose-addition

124 While potential carcinogenic health risks present for both adult and chi

125 le heavy metal exposure, no potential of non carcinogenic health risks was found, while carcinogenic

126 n carcinogenic health risks was found, while carcinogenic health risks were found only for As.

127 4,5-f]quinoline (IQ), a highly mutagenic and carcinogenic heterocyclic amine found in cooked meats, w

128 -Amino-9H-pyrido[2,3-b]indole (AalphaC) is a carcinogenic heterocyclic aromatic amine formed during t

129 l-6-phenylimidazo[4,5-b]pyridine (PhIP) is a carcinogenic heterocyclic aromatic amine formed in cooke

130 Carcinogenic hexavalent chromium [Cr(VI)] requires cellu

131 alignant lesions, to define the effects of a carcinogenic host environment on the virulence phenotype

132 Urine-based carcinogenic HPV detection had a clinical sensitivity of

133 Although urine-based HPV carcinogenic HPV detection was lower (58.3%) than cervic

134 samples were tested for antibodies against 8 carcinogenic HPV genotypes (16, 18, 31, 33, 35, 45, 52,

135 neoplasia (HGAIN) attributable to individual carcinogenic HPV genotypes and estimated the range of th

136 Prevalence of multiple (>/= 2) carcinogenic HPV genotypes increased from 30.9% in cases

137 on were associated with an increased risk of carcinogenic HPV infection (odds ratio [OR], 3.65; 95% c

138 real-time PCR assay for the detection of 13 carcinogenic HPV types (the H13 assay; Hybribio, Hong Ko

139 of naturally occurring antibodies against 8 carcinogenic HPV types with subsequent infections.

140 risk of HSIL in women infected with multiple carcinogenic HPV types.

141 ultivalent vaccines that include most of the carcinogenic HPV types; continued surveillance postvacci

142 an oral alpha mucosal HPV detected, 1.3% had carcinogenic HPV, and 0.4% had HPV-16; similar patterns

143 g increased the risk of anal precancer among carcinogenic HPV-positive men but had no association wit

144 The fractions of HGAIN cases attributable to carcinogenic HPV16/18 targeted by currently licensed biv

145 to 61.5%, and the fractions attributable to carcinogenic HPV16/18/31/33/45/52/58 targeted by an inve

146 Carcinogenic human papillomaviruses (HPVs) cause a large

147 This is the first study to utilize carcinogenic hypoxia as an environmentally relevant expe

148 The behavior of late passage hMSCs in a carcinogenic hypoxia environment follows a profile simil

149 We suggest a mechanism by which carcinogenic hypoxia modulates the activity of three cri

150 Carcinogenic impacts range over 4 orders of magnitude, f

151 chloromethane was classified as likely to be carcinogenic in humans based primarily on evidence of ca

152 Most HAAs are carcinogenic in long-term animal studies.

153 he keratin-14 promoter and showed that E7 is carcinogenic in mice.

154 llomaviruses (HPV) have been suspected to be carcinogenic in nonmelanoma skin cancers (NMSC), but the

155 Sudan I is a carcinogenic industrial azo-dye, forbidden for use in fo

156 Interleukin-6 (IL-6) is a pro-carcinogenic inflammatory cytokine that plays a pivotal

157 methylarsenite [MAs(III)] to less toxic and carcinogenic inorganic arsenite [As(III)] by C-As bond c

158 indings leading first to the hypothesis that carcinogenic insults leave characteristic imprints on th

159 The recognition that tobacco smoke is carcinogenic led to the most significant and successful

160 beta) inserts, albeit slowly, T opposite the carcinogenic lesion O6-methylguanine (O6MeG) approximate

161 The carcinogenic mechanism depends mainly on the expression

162 colorectal neoplasia and may help elucidate carcinogenic mechanisms for distinct pathways.

163 its overexpression has been associated with carcinogenic mechanisms in several human cancers.

164 wn to be involved in a number of potentially carcinogenic mechanisms such as altering gene transcript

165 r metabolite levels for TSNAs (including the carcinogenic metabolite 4-(methylnitrosamino)-1-(3-pyrid

166 or human serum albumin (HSA) adducts of the carcinogenic metabolite benzo[a]pyrene diol epoxide (BPD

167 iliary and fecal elimination of PhIP and its carcinogenic metabolites and may affect PhIP-induced car

168 ion of environmental procarcinogens to their carcinogenic metabolites in both animals and humans.

169 -), but not Bcrp1;Mdr1a/b;Mrp(-/-) mice, the carcinogenic metabolites N2-OH-PhIP (2-hydroxyamino-1-me

170 metabolites such as butyrate and potentially carcinogenic metabolites such as secondary bile acids.

171 Carcinogenic metal nickel is a widespread environmental

172 Here we report that exposure to arsenic, a carcinogenic metal, decreased cellular levels of SLBP by

173 organic arsenite (As(III)) to more toxic and carcinogenic methylarsenite (MAs(III)).

174 nner, we created a quantitative map for this carcinogenic mutation in this healthy population and ide

175 a >2 fold inhibition of biosynthesis of the carcinogenic mycotoxin and secondary metabolite, aflatox

176 monitoring of Ochratoxin A (OTA), a natural carcinogenic mycotoxin produced by Aspergillus and Penic

177 ght hapten, Ochratoxin A (OTA), is a natural carcinogenic mycotoxin produced by Aspergillus and Penic

178 A (OTA) is a widespread and abundant natural carcinogenic mycotoxin produced by several species of As

179 Aflatoxins are the most potent genotoxic and carcinogenic mycotoxins.

180 are highly toxic, mutagenic, teratogenic and carcinogenic mycotoxins.

181 e concerns over the formation of potentially carcinogenic N-nitrosamine byproducts from reactions bet

182 e been raised about the potential release of carcinogenic N-nitrosamines and N-nitramines formed by r

183 However, the formation of potentially carcinogenic N-nitrosamines and N-nitramines from reacti

184 however, has been linked to the formation of carcinogenic N-nitrosamines as byproducts during chloram

185 -based CO2 capture facilities by potentially carcinogenic N-nitrosamines formed from reactions betwee

186 The occurrence of eight carcinogenic N-nitrosamines in biosolids from 74 wastewa

187 the precursors and reactions leading to the carcinogenic N-nitrosodimethylamine (NDMA) during drinki

188 ammonium polymers leads to the formation of carcinogenic N-nitrosodimethylamine (NDMA).

189 s quantification is important because of its carcinogenic nature and its usually presence in alcoholi

190 sistent organic pollutants, whose documented carcinogenic, neurological, and respiratory toxicities a

191 e threshold value of 10(-6), suggesting that carcinogenic Ni still was a concern to the residents.

192 their use has been linked to the presence of carcinogenic nitrosamines in treated drinking water.

193 mines present in all amine solvents can form carcinogenic nitrosamines.

194 trosate in the desorber, forming potentially carcinogenic nitrosamines.

195 5-b]pyridine (PhIP) is considered as a human carcinogenic or mutagenic compound that is produced from

196 hymine dimer which is potentially mutagenic, carcinogenic, or lethal to the organism.

197 The efficient removal of trace carcinogenic organic pollutants, such as polycyclic arom

198 The formation of bromate, a potentially carcinogenic ozonation byproduct, could be significantly

199 ic approach that assumes dose additivity for carcinogenic PAHs and (ii) a bioassay-based approach tha

200 reviewed, were unable to successfully remove carcinogenic PAHs from contaminated soils to concentrati

201 cal methods for determining 11 mutagenic and carcinogenic PAHs, as well as two EPA indicator PAHs in

202 five-ring PAHs, including five of the seven carcinogenic PAHs, with removals up to 80%.

203 ency equivalency factors (PEFs) for targeted carcinogenic PAHs.

204 has a high predictability, particularly for carcinogenic PAHs.

205 er, it remains unclear at which stage in the carcinogenic pathway fiber may act or which food sources

206 the miR-183 family regulates zinc levels and carcinogenic pathways in prostate cells.

207 Accumulating evidence indicates that common carcinogenic pathways may underlie digestive system canc

208 the same regular chow throughout the entire carcinogenic period.

209 Considering that flutriafol is a toxic and carcinogenic pesticide, as well as the increase in the n

210 These results indicate that host carcinogenic phenotypes modify cagT4SS function via alte

211 mine dimers is one of the most important DNA carcinogenic photolesions induced by ultraviolet irradia

212 Bolstering the immune response to carcinogenic polyaromatic hydrocarbons may be an effecti

213 n the induction of cell-mediated immunity to carcinogenic polyaromatic hydrocarbons.

214 ionship between lung cancer mortality rates, carcinogenic polycyclic aromatic hydrocarbon (PAH) emiss

215 ceedances for PM and benzo[a]pyrene B[a]P, a carcinogenic polycyclic aromatic hydrocarbon (PAH) of MW

216 , bioaccessibility, and dermal absorption of carcinogenic polycyclic aromatic hydrocarbons (cPAHs) in

217 DNA adducts derived from carcinogenic polycyclic aromatic hydrocarbons like benzo

218 Risk-based analysis of several carcinogenic POPs indicated that the fish with the highe

219 are generated at concentrations below their carcinogenic potencies; it is unclear how exposure leads

220 (a)pyrene-equivalents (a combined measure of carcinogenic potency across 7 different PAHs), which wer

221 Levels of benzo[a]pyrene, phenanthrene, and carcinogenic potency of PAH mixtures were highest when s

222 dicated MPFs with orders of magnitude higher carcinogenic potency than predicted by established in vi

223 a serious public health risk because of its carcinogenic potential and as climate pollutant.

224 codons of HPV58 variants representing higher carcinogenic potential and/or that are under positive se

225 The carcinogenic potential of CagA is linked to its polymorp

226 Because of the carcinogenic potential of DMA, C. pulverulentus should n

227 Data on the relative carcinogenic potential of human papillomavirus (HPV) typ

228 Although the carcinogenic potential of iron has been shown, evidence

229 this study was to investigate the long-term carcinogenic potential of MTM particulate matter (PMMTM)

230 These results provide new evidence for the carcinogenic potential of PMMTM and support further risk

231 ection to studies aimed at defining the full carcinogenic potential of this organism.

232 r ecotoxicity potential, 0-370 kg benzene-eq carcinogenic potential, and 2800-71,000 MT toluene-eq no

233 CNTs), a widely used nanomaterial with known carcinogenic potential, can affect cancer-associated fib

234 omoting events are required to unleash their carcinogenic potential.

235 hydrocarbons (PAHs) which are known to have carcinogenic potential.

236 ctive mutational signatures that explain its carcinogenic potential.

237 One way to interrogate the complex carcinogenic process and interactions between aberrant e

238 by arresting, slowing down, or reversing the carcinogenic process before invasion into surrounding ti

239 d, in the last decade, as key players in the carcinogenic process, with many candidates identified as

240 rations occur from the very beginning of the carcinogenic process.

241 standing of racial disparities in cancer and carcinogenic process.

242 tation landscape of melanoma reveals diverse carcinogenic processes across its subtypes, some unrelat

243 the involvement of CLIP2 in the fundamental carcinogenic processes including apoptosis, mitogen-acti

244 enome-wide DNA methylation change during the carcinogenic progress has not yet been explored.

245 enetic changes are an important mechanism in carcinogenic progression, but how these changes differ b

246 Carcinogenic properties are linked to the ability of the

247 Ms) have provided valuable insights into the carcinogenic properties of various human tumor viruses,

248 ng but also exert anti-inflammatory and anti-carcinogenic properties.

249 are environmental pollutants with toxic and carcinogenic properties.

250 We then used the model to investigate the carcinogenic property of Bisphenol A (BPA), an environme

251 d SCID mice models also revalidated the anti-carcinogenic property of nifetepimine.

252 gs showed that levels of both non-cancer and carcinogenic risk for PAE congeners in wheat were higher

253 a result of the widely reported potential of carcinogenic risk from x-ray based examinations, many st

254 Importantly, elevated carcinogenic risk is found at doses that yield undetecta

255 dence of imaging-related low-dose (<100 mGy) carcinogenic risk is nonexistent; it is a hypothetical r

256 rd quotient, selenium health benefit values, carcinogenic risk of arsenic, maximum safe consumption a

257 a)pyrene, the marker used for evaluating the carcinogenic risk of PAHs in food, was not detected in t

258 tual risks far greater than the hypothetical carcinogenic risk purportedly avoided.

259 nt was conducted to assess the potential non-carcinogenic risk resulting from wine consumption.

260 Current paradigms of carcinogenic risk suggest that genetic, hormonal, and en

261 timates revealed that the excessive lifetime carcinogenic risk to individuals exposed to biomass burn

262 fic DNA hypomethylation of genes that confer carcinogenic risk, with greatest hypomethylation at the

263 ancer (IARC) Programme for the Evaluation of Carcinogenic Risks to Humans has been criticized for sev

264 Experimental and clinical studies support a carcinogenic role for PTGS2 (cyclooxygenase-2), which is

265 The pro-carcinogenic role of Epsins suggests that they are poten

266 H2 histone methyltransferase (EZH2), but the carcinogenic role of these mutations is unclear.

267 ase that develops via one of the two primary carcinogenic routes: chemical carcinogenesis through exp

268 Fumonisins are a family of carcinogenic secondary metabolites produced by members o

269 osteoporosis while minimizing or eliminating carcinogenic side effects.In this study, we sought to de

270 However, the molecular mechanisms of the carcinogenic signaling pathway remain largely unclear.

271 Condensate and stormwater contained numerous carcinogenic solvents used in resin synthesis, endocrine

272 uces the blank level and avoids the use of a carcinogenic species.

273 -nitrosodipropylamine, which is 10-fold less carcinogenic than NDMA, at 20-fold lower yields.

274 (60.7%; 37/61), which further supported the carcinogenic theory of field cancerization.

275 Although radiation exposure is potentially carcinogenic, there are limited data on cumulative expos

276 mesenchymal stem/stromal cells (MSCs) in non-carcinogenic thyroids and papillary thyroid cancer (PTC)

277 pollution and airborne particulate matter as carcinogenic to humans (Group 1) for lung cancer.

278 s of the > 100 agents classified as Group 1, carcinogenic to humans (IARC Monographs Volume 100, part

279 TCE is carcinogenic to humans by all routes of exposure and pos

280 ever, MeI is highly volatile and potentially carcinogenic to humans if inhaled.

281 clic aromatic hydrocarbon, is classified as "carcinogenic to humans" and is ubiquitous in the environ

282 sment classifying red and processed meat as "carcinogenic to humans" on the basis of the positive ass

283 iate classification of a number of agents as carcinogenic to humans.

284 ancer (IARC) recently declared air pollution carcinogenic to humans.

285 pollution and airborne particulate matter as carcinogenic to humans.

286 confirm or refute the hypothesis that TFE is carcinogenic to humans.

287 lkaloids have been shown to be precursors of carcinogenic tobacco specific N'-nitrosamines.

288 the correlational structures of 26 probable carcinogenic toxics, and estimated odds ratios by brain

289 We conclude that Ate1-KO results in carcinogenic transformation of cultured fibroblasts, sug

290 is likely that Plk1 plays an active role in carcinogenic transformation.

291 -molecule sequencing of somatically acquired carcinogenic translocations at extremely low levels (<10

292 d neoplasms in the absence of any additional carcinogenic treatment.

293 < 0.001), HPV18 (P(trend) = 0.07), and other carcinogenic types (P(trend) < 0.001).

294 as 83.3% overall [Kappa = 0.9] and 86.8% for carcinogenic types [Kappa = 0.92]).

295 cious in preventing infections with the most carcinogenic types of HPV (HPV 16 and HPV 18) at the cer

296 positive results for HPV16, HPV18, and other carcinogenic types.

297 e sole mechanism in humans for the repair of carcinogenic UV irradiation-induced photoproducts in the

298 portant, as the 4-hydroxylated metabolite is carcinogenic, whereas the 2-hydroxylated metabolite is n

299 gents that may be allergenic and potentially carcinogenic, which represents a potentially hazardous c

300 emoval and preconcentration of two important carcinogenic xanthine dyes named rhodamine-B (RB) and rh