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1 months for pancreatic NETs v 7.3 months for carcinoid tumors).
2 months for pancreatic NETs v 18.8 months for carcinoid tumors).
3 s, 33% among pheochromocytomas, and 7% among carcinoid tumors).
4 en ovale; 1 of these also had a primary lung carcinoid tumor.
5 trophic gastritis and multiple large gastric carcinoid tumors.
6 e stomach that were determined to be mucosal carcinoid tumors.
7 all loci tested in both typical and atypical carcinoid tumors.
8 otreotide to treat symptomatic patients with carcinoid tumors.
9 le development of various cancers, including carcinoid tumors.
10 ong patients with metastatic or unresectable carcinoid tumors.
11 ctive in pancreatic endocrine tumors than in carcinoid tumors.
12 eans for diagnosis of primary and metastatic carcinoid tumors.
13 ent of adverse effects caused by products of carcinoid tumors.
14 d to be an option for selected patients with carcinoid tumors.
15 , A2, and A3Rs were present in BON cells and carcinoid tumors.
16 ents (4 male, 4 female) with primary hepatic carcinoid tumors.
17 has been commonly observed in patients with carcinoid tumors.
18 ease stabilization in patients with advanced carcinoid tumors.
19 s of tumorlets from intramucosal spread from carcinoid tumors.
20 like cells and to the development of gastric carcinoid tumors.
21 ssion is useful in the assessment of gastric carcinoid tumors.
22 nts who had undergone resection of bronchial carcinoid tumors.
24 ction in paired normal and tumor DNA from 17 carcinoid tumors, 5 SCLCs, and 38 NSCLCs to determine th
26 ed metastatic neuroendocrine tumors (20 with carcinoid tumors and 11 with other tumors) treated with
27 including 15 of 16 (94%) cases of multifocal carcinoid tumors and 7 of 8 (88%) cases of multifocal pa
28 trointestinal carcinoids comprise 90% of all carcinoid tumors and all carcinoids have malignant poten
29 nts with small tumors, as well as those with carcinoid tumors and bronchioloalveolar cell carcinoma,
31 patients were studied: subjects with typical carcinoid tumors and coexisting tumorlets (n = 5), typic
32 ur cases, including 16 multifocal intestinal carcinoid tumors and eight multifocal pancreatic endocri
33 e investigated whether multifocal intestinal carcinoid tumors and multifocal pancreatic endocrine tum
36 ference in chromosome 3p alterations between carcinoid tumors and SCLCs favors a stochastic rather th
37 of neuroendocrine tumors (five patients with carcinoid tumors and three patients with islet cell tumo
41 ighly up-regulated in human gastrointestinal carcinoid tumors, and we sought to define its pathogenic
42 gnetic resonance imaging, metastatic orbital carcinoid tumors appear as nonspecific tumor masses.
47 described significant prognostic features of carcinoid tumors as site of origin, age, sex, stage at d
48 ctive analysis of 150 patients with GI tract carcinoid tumors at the Massachusetts General Hospital w
49 ell lines as models for small intestinal or 'carcinoid' tumor biology are considered appropriate.
50 l deaths (1 was not related to the patient's carcinoid tumor but was due to a second coexistent cance
51 RNA is low to undetectable in human SCLC and carcinoid tumors, but the HGFL/MSP tyrosine kinase recep
52 vasoactive substances into the circulation, carcinoid tumors can cause right-sided valvular heart di
54 etion, acute interstitial nephritis, gastric carcinoid tumor, cardiovascular risk with clopidogrel an
55 The radiographic responses in patients with carcinoid tumors comprised a minor response in 2 patient
56 matic responses in patients with functioning carcinoid tumors comprised complete resolution in 3 of t
57 neuroendocrine tumors; its activity against carcinoid tumors could not be definitively determined in
58 study indicates that methylation profile of carcinoid tumors differs from PETs, reflecting different
59 ed for more aggressive surgical treatment of carcinoid tumors, especially in the setting of hepatic m
61 imbalance was shown to be an early event in carcinoid tumor formation by virtue of the absence of al
67 dy demonstrates that (1) incidence rates for carcinoid tumors have changed, (2) the most common gastr
71 w evidence with respect to the prevalence of carcinoid tumors in the middle and right lower lobe, wit
72 that characteristically are up-regulated in carcinoid tumors, including neurotensin and connective t
73 he current biologic understanding of gastric carcinoid tumors, including the role of hypergastrinemia
75 derstanding of the biologic basis of gastric carcinoid tumors increases, the treatment will likely be
84 ted in patients with a patent foramen ovale, carcinoid tumor of the lung, and active carcinoid syndro
88 ears of follow-up, 60 adenocarcinomas and 80 carcinoid tumors of the small intestine were diagnosed.
89 trast, we noted a markedly elevated risk for carcinoid tumors of the small intestine with saturated f
97 thyroid tumors, one gastrinoma, and one lung carcinoid tumor) showed allelic loss that placed the MEN
98 enrolled in the Natural History of Familial Carcinoid Tumor study at the National Institutes of Heal
100 tment outcome was analyzed for patients with carcinoid tumors (the most common tumors in this study),
101 st to the relatively conserved karyotypes of carcinoid tumors, the karyotypes of SCLC tumors and cell
103 d chromogranin A messenger RNA abundance, in carcinoid tumor tissue and macroscopically normal corpus
109 The median overall survival in patients with carcinoid tumors was 47 mo (95% confidence interval, 32-
110 xtracted from archival tissue sections of 35 carcinoid tumors was assessed for LOH with eight polymor
111 ranscript and protein levels indicative of a carcinoid tumor were identified in one acute appendiciti
112 ad undergone complete resection of bronchial carcinoid tumors were associated with increased local-re
114 wo hundred forty-nine patients with advanced carcinoid tumors were randomized to either doxorubicin w
115 with advanced pancreatic endocrine tumors or carcinoid tumors were treated with rhEndostatin administ
116 increased considerably, primarily because of carcinoid tumors which are now the most common small bow
117 We report a case of a patient with a known carcinoid tumor who developed a left orbital mass that d
119 luramine, ergot drugs, or 5-HT released from carcinoid tumors (with or without accompanying 5-HT(2A)
120 similar to those of two samples of pulmonary carcinoid tumors, with an average correlation coefficien
121 agnosis, management, and treatment of rectal carcinoid tumors, with special emphasis on minimally inv
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